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PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0137923
Background Semaphorins (SEMAs) consist of a large family of secreted and membrane-anchored proteins that are important in neuronal pathfinding and axon... 
SUPPRESSOR | INHIBITION | INTEGRINS | MULTIDISCIPLINARY SCIENCES | PROGNOSTIC MARKER | INDUCTION | EXPRESSION | CARCINOMA | PROGRESSION | Matrix Metalloproteinases - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Cyclin E - genetics | Cell Movement - genetics | Proto-Oncogene Proteins c-akt - genetics | Neovascularization, Pathologic - pathology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | MAP Kinase Signaling System - genetics | Neoplasm Invasiveness - pathology | Cell Cycle Checkpoints - genetics | G1 Phase - genetics | Cyclin-Dependent Kinases - genetics | Mouth Neoplasms - genetics | Semaphorins - genetics | Cell Proliferation - genetics | Up-Regulation - genetics | Down-Regulation - genetics | Neoplasm Metastasis - genetics | Cyclin D1 - genetics | Neoplasm Metastasis - pathology | Cell Line, Tumor | Mouth Neoplasms - pathology | Neovascularization, Pathologic - genetics | Neoplasm Invasiveness - genetics | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Proteins | Immunohistochemistry | Squamous cell carcinoma | RNA | Growth | Proteases | Analysis | DNA polymerases | B cells | Metastasis | Oral cancer | Immunoblotting | Nervous system | AKT protein | Life assessment | Cyclin D1 | Inactivation | Cyclin-dependent kinase 4 | Maxillofacial surgery | Metastases | Cyclin-dependent kinase | Angiogenesis | Surgery | Penicillin | Cyclin-dependent kinase inhibitors | Medical research | Melanoma | Patients | Polymerase chain reaction | Hospitals | Correlation analysis | Axon guidance | Cell migration | Cell proliferation | Biotechnology | Laboratories | Science | Kinases | Neurogenesis | Cell cycle | Extracellular matrix | Cyclin E | G1 phase | Deactivation | Invasiveness | Secretion | Therapeutic applications | Breast cancer | Dentistry | Cyclins | Gelatinase B | Gelatinase A | Cyclin-dependent kinase 2 | Medicine | Cyclin-dependent kinase inhibitor p21 | Cell lines | Cyclin-dependent kinase inhibitor p27 | Diagnostic systems | Apoptosis
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 06/2013, Volume 210, Issue 6, pp. 1137 - 1151
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 5, pp. 2177 - 2182
The stromal compartment is increasingly recognized to play a role in cancer. However, its role in the transition from preinvasive to invasive disease is... 
Adenocarcinoma | Datasets | Metaplasia | Medical research | Genes | Fibroblasts | Inflammation | Digestive system diseases | Signatures | Cancer | gene set enrichment | CELLS | intraepithelial neoplasia | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | BARRETTS-ESOPHAGUS | FIBROBLASTS | BREAST-CANCER | microarray | GASTROESOPHAGEAL-REFLUX | inflammation | CHRONIC-PANCREATITIS | Barrett's esophagus | DIFFERENTIATION | EXPRESSION | PROGRESSION | Inflammation - pathology | Adenocarcinoma - pathology | Prognosis | Stromal Cells - pathology | Humans | Digestive System Neoplasms - immunology | Esophageal Neoplasms - pathology | Barrett Esophagus - pathology | Neoplasm Invasiveness - pathology | Digestive System Neoplasms - pathology | Serine Endopeptidases - genetics | Trans-Activators - genetics | Precancerous Conditions - pathology | Adenocarcinoma - genetics | Nuclear Proteins - genetics | Cytokines - genetics | Digestive System Neoplasms - genetics | Receptors, Cytokine - genetics | Barrett Esophagus - genetics | Oncogenes | Membrane Proteins - genetics | Adenocarcinoma - immunology | Stromal Cells - immunology | Inflammation - immunology | DNA-Binding Proteins - genetics | Esophageal Neoplasms - immunology | Precancerous Conditions - genetics | Precancerous Conditions - immunology | Esophageal Neoplasms - genetics | Gelatinases - genetics | Barrett Esophagus - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Inflammation - genetics | Neoplasm Invasiveness - genetics | Proto-Oncogene Proteins c-bcl-6 | Genetic aspects | Research | Biological Sciences | Barrett’s esophagus
Journal Article
Cancer Research, ISSN 0008-5472, 06/2018, Volume 78, Issue 11, pp. 2852 - 2863
Although prostate cancer is clinically manageable during several stages of progression, survival is severely compromised once cells invade and metastasize to... 
INSULIN | STAT3 ACTIVATION | TUMOR INVASION | IGF-IR | ONCOLOGY | RECEPTOR | KINASE INHIBITOR | SYNTENIN | EXPRESSION | GROWTH-FACTOR-I | MELANOMA METASTASIS | Interleukin-8 - genetics | Prostatic Neoplasms - pathology | Interleukin-6 - genetics | Humans | Carcinogenesis - genetics | Male | Signal Transduction - genetics | Melanoma - pathology | Vascular Endothelial Growth Factor A - genetics | Cell Movement - genetics | Carcinogenesis - pathology | Syntenins - genetics | Cell Differentiation - genetics | Matrix Metalloproteinase 2 - genetics | Animals | Prostatic Neoplasms - genetics | Matrix Metalloproteinase 9 - genetics | Melanoma - genetics | Neoplasm Invasiveness - pathology | Cell Line, Tumor | Receptors, Somatomedin - genetics | Neoplasm Invasiveness - genetics | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | Biotechnology | Phosphorylation | Insulin-like growth factor I | Insulin-like growth factor-binding protein 2 | Insulin-like growth factors | Metastasis | Metastases | Interleukin 6 | Proteins | Dependence | Bioinformatics | Interleukin 8 | CRISPR | Cell survival | Melanoma | Organs | Stat3 protein | Data processing | Tumor cell lines | Insulin | Molecular chains | Gelatinase B | Gelatinase A | Cell lines | Prostate cancer | Prostate | Cell migration | Syndecan | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63165
Matrix metalloproteinases (MMPs) contribute to cancer through their involvement in cancer invasion and metastasis. We evaluated genetic variation in MMP1 (9... 
POPULATION | MATRIX-METALLOPROTEINASE-2 | WOMEN | MULTILOCUS GENOTYPE DATA | POLYMORPHISMS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | INFERENCE | MMP-9 | Genetic Predisposition to Disease - genetics | Matrix Metalloproteinases - genetics | Humans | Middle Aged | Risk Factors | Hispanic Americans | European Continental Ancestry Group | Matrix Metalloproteinase 2 - genetics | Breast Neoplasms - enzymology | Breast Neoplasms - genetics | Matrix Metalloproteinase 9 - genetics | Polymorphism, Single Nucleotide - genetics | Adult | Female | Matrix Metalloproteinase 3 - genetics | Matrix Metalloproteinase 1 - genetics | Women | Societies, clubs, etc | Oncology, Experimental | Genes | Development and progression | Breast cancer | Metastasis | Research | Risk factors | Women's organizations | Genetic research | Genetic aspects | Single nucleotide polymorphisms | Native Americans | Cancer | Health care | Matrix metalloproteinases | Single-nucleotide polymorphism | Matrix metalloproteinase | Medical diagnosis | Epidemiology | Population genetics | Metastases | Population | Genetics | Metalloproteinase | Age | Medical research | Stromelysin 1 | Health risks | Genetic diversity | Preventive medicine | Cultural differences | Survival | Minority & ethnic groups | Gelatinase B | Gelatinase A | Disease prevention | Studies | Ethnicity | Womens health | Biomarkers | Breast | Hormone replacement therapy | Health risk assessment | Tumors | Health disparities
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31251
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 02/2018, Volume 57, Issue 2, pp. 225 - 234
As one of the most frequently diagnosed cancers in women, the development and progression of epithelial ovarian carcinoma (EOC) remains an open area of... 
tumorigenesis | lncRNA TDRG1 | epithelial ovarian carcinoma | progression miR‐93 | progression miR-93 | TARGET | TUMOR INVASION | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | MICRORNAS | RHOC | BREAST-CANCER | ONCOLOGY | CANCER STATISTICS | RECURRENCE | EXPRESSION | Neoplasms, Glandular and Epithelial - pathology | Cell Proliferation - genetics | Humans | Carcinogenesis - genetics | Ovarian Neoplasms - pathology | rhoC GTP-Binding Protein - genetics | Up-Regulation - genetics | Signal Transduction - genetics | Cell Movement - genetics | Disease Progression | Down-Regulation - genetics | Carcinogenesis - pathology | Ovarian Neoplasms - genetics | Proteins - genetics | Cell Differentiation - genetics | Matrix Metalloproteinase 2 - genetics | Neoplasms, Glandular and Epithelial - genetics | Carcinoma, Ovarian Epithelial | Cell Line, Tumor | Carcinoma - genetics | Female | MicroRNAs - genetics | Carcinoma - pathology | Gene Expression Regulation, Neoplastic - genetics | Genetic research | Development and progression | Carcinoma | RNA | Ovarian cancer | Cancer | Cell proliferation | Ovarian carcinoma | MiRNA | Homology | Matrix metalloproteinase | Gene expression | Tissues | Kinases | Ribonucleic acid--RNA | Gelatinase A | Ribosomal protein S6 kinase | Bcl-x protein | Metalloproteinase | Tumorigenesis | Diagnostic systems | Non-coding RNA | Binding sites | Cell migration
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e99955
Background: Cancer-associated fibroblasts (CAF) are activated fibroblasts in the cancer stroma and play an important role in cancer progression. Some reports... 
STROMAL MYOFIBROBLASTS | ANGIOGENESIS | EXPRESSION | MULTIDISCIPLINARY SCIENCES | ACTIVATION PROTEIN | INFILTRATION | Prognosis | Tissue Array Analysis | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Actins - metabolism | Male | Epithelial-Mesenchymal Transition - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Actins - genetics | Esophageal Neoplasms - pathology | Carcinoma, Squamous Cell - mortality | Tumor Microenvironment - genetics | Esophageal Neoplasms - metabolism | Gelatinases - metabolism | Serine Endopeptidases - genetics | Biomarkers, Tumor - metabolism | Esophageal Neoplasms - mortality | Female | Membrane Proteins - metabolism | Esophageal Squamous Cell Carcinoma | Fibroblasts - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism | Calcium-Binding Proteins - metabolism | Gene Expression | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Macrophages - pathology | Membrane Proteins - genetics | Fibroblasts - pathology | Macrophages - metabolism | Phenotype | Esophageal Neoplasms - genetics | Gelatinases - genetics | Receptor, Platelet-Derived Growth Factor alpha - genetics | Survival Analysis | Aged | Biomarkers, Tumor - genetics | Serine Endopeptidases - metabolism | Calcium-Binding Proteins - genetics | Health sciences | Correlation | Platelet-derived growth factor | Mesenchyme | Colorectal cancer | Smooth muscle | Metastasis | Macrophages | Proteins | Genotype & phenotype | Angiogenesis | Actin | Classification | Fibroblasts | Growth factors | Squamous cell carcinoma | Tumor cells | Markers | Stroma | Muscles | Histology | Breast cancer | Radiation therapy | Patients | Fibroblast activation protein | Esophagus | Pathology | Chemotherapy | Medical prognosis | Morphology | Diagnostic systems | Prostate | Cancer | Tumors
Journal Article
Journal of Molecular Endocrinology, ISSN 0952-5041, 2013, Volume 50, Issue 2, pp. R21 - R37
Polycystic ovary syndrome (PCOS) is the most common endocrinopathy associated with infertility and metabolic disorder in women of reproductive age. Dysfunction... 
Obesity | Polycystic ovary syndrome | Adipose tissue | Adipokines | ACTIVATED PROTEIN-KINASE | SUBCUTANEOUS ADIPOSE-TISSUE | SERUM LEPTIN LEVELS | adipose tissue | GELATINASE-ASSOCIATED LIPOCALIN | polycystic ovary syndrome | DIABETES-SUSCEPTIBILITY GENES | PLASMA VISFATIN CONCENTRATIONS | MESSENGER-RIBONUCLEIC-ACID | ENDOCRINOLOGY & METABOLISM | COLONY-ENHANCING FACTOR | BODY-MASS INDEX | adipokines | VASCULAR ENDOTHELIAL-CELLS | obesity | Resistin - genetics | Humans | Leptin - metabolism | Acute-Phase Proteins - genetics | Obesity - genetics | Lectins - metabolism | Polycystic Ovary Syndrome - complications | Nicotinamide Phosphoribosyltransferase - metabolism | Lipocalins - genetics | Adipokines - genetics | Female | Retinol-Binding Proteins, Plasma - metabolism | Cytokines - genetics | Lipocalins - metabolism | Proto-Oncogene Proteins - metabolism | Resistin - metabolism | Polycystic Ovary Syndrome - metabolism | Cytokines - metabolism | Acute-Phase Proteins - metabolism | Obesity - complications | Intercellular Signaling Peptides and Proteins | Proto-Oncogene Proteins - genetics | Adipokines - metabolism | Leptin - genetics | Chemokines - genetics | GPI-Linked Proteins - metabolism | Obesity - metabolism | Nicotinamide Phosphoribosyltransferase - genetics | Lipocalin-2 | Polycystic Ovary Syndrome - genetics | Lectins - genetics | Chemokines - metabolism | Retinol-Binding Proteins, Plasma - genetics | GPI-Linked Proteins - genetics
Journal Article
Oncogene, ISSN 0950-9232, 01/2013, Volume 32, Issue 4, pp. 491 - 501
Invasion and metastasis are the major features of malignant tumors that are responsible for 90% of cancer-related deaths. Recently, microRNAs have been... 
gastric carcinoma | metastasis | N-cadherin | miR-145 | SIGNATURES | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL INVASION | METALLOPROTEINASES | CELL BIOLOGY | BREAST-CANCER | COLON-CANCER | ONCOLOGY | TUMOR-METASTASIS | GROWTH | GENETICS & HEREDITY | EXPRESSION | PROGRESSION | Cadherins - metabolism | Humans | Stomach Neoplasms - metabolism | Cell Growth Processes - physiology | Gastric Mucosa - pathology | MicroRNAs - metabolism | Stomach Neoplasms - pathology | Gastric Mucosa - metabolism | Antigens, CD - genetics | Cell Movement - genetics | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Neoplasm Metastasis | Matrix Metalloproteinase 9 - genetics | Cadherins - genetics | Carcinoma - pathology | Stomach Neoplasms - genetics | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Down-Regulation | Matrix Metalloproteinase 2 - genetics | Cell Line, Tumor | Carcinoma - genetics | MicroRNAs - genetics | Carcinoma - metabolism | MicroRNA | Molecular genetics | Physiological aspects | Development and progression | Genetic aspects | Research | Metastasis | Health aspects | Stomach cancer | Cancer | Oncology | Tumors | Translation | Gastric mucosa | Gelatinase B | Western blotting | Gelatinase A | Metastases | Molecular modelling | N-Cadherin | miRNA | Metalloproteinase | Gastric cancer | Cell migration
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1423 - 12
Beta-transducin repeat containing E3 ubiquitin protein ligase (BTRC) is crucial for the degradation of I kappa B alpha. Our previous transcriptome sequencing... 
UBIQUITINATION | INVASION | GENE | MIR-339-5P | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | KINASE | ALPHA | ADAM10 | CANCER | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | RNA, Small Interfering - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | NF-KappaB Inhibitor alpha - genetics | NF-kappa B - metabolism | Esophageal Neoplasms - pathology | Matrix Metalloproteinase 9 - metabolism | beta-Transducin Repeat-Containing Proteins - metabolism | Neoplasm Grading | Matrix Metalloproteinase 9 - genetics | Esophageal Neoplasms - metabolism | Female | Vascular Cell Adhesion Molecule-1 - genetics | Chemokine CCL2 - metabolism | Tetraspanins - genetics | Tetraspanins - metabolism | beta-Transducin Repeat-Containing Proteins - genetics | Esophageal Squamous Cell Carcinoma | Signal Transduction | Chemokine CCL2 - genetics | Lymphatic Metastasis | Urokinase-Type Plasminogen Activator - genetics | Tetraspanins - antagonists & inhibitors | Intercellular Adhesion Molecule-1 - metabolism | Animals | Esophageal Neoplasms - genetics | NF-kappa B - genetics | Urokinase-Type Plasminogen Activator - metabolism | Mice, Nude | Intercellular Adhesion Molecule-1 - genetics | NF-KappaB Inhibitor alpha - metabolism | Cell Line, Tumor | Aged | Mice | MicroRNAs - genetics | Neoplasm Staging | Vascular Cell Adhesion Molecule-1 - metabolism | RNA, Small Interfering - metabolism | Ubiquitin | Translocation | NF-κB protein | Squamous cell carcinoma | Intercellular adhesion molecule 1 | Therapeutic applications | Transducin | Metastasis | Tumor necrosis factor-α | Tissues | Gene expression | Nuclear transport | Lymph nodes | Gelatinase B | Metastases | Esophagus | Degradation | Signaling | Ubiquitination | Transcription activation | Proteasomes | Biomarkers | Monocyte chemoattractant protein 1 | Ubiquitin-protein ligase
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2009, Volume 4, Issue 4, pp. e5168 - e5168
Background: Inflammatory cytokine genes have been proposed as good candidate genes for conferring susceptibility to diabetic nephropathy. In the present study,... 
MULTIDISCIPLINARY SCIENCES | DISEASE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | PROMOTER POLYMORPHISM | JAPANESE | CANDIDATE GENE | CCR5 | EPIDEMIOLOGY | PROGRESSION | RENAL-INSUFFICIENCY | TGF-BETA-1 | Interleukin-8 - genetics | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Receptors, CCR5 - genetics | Male | C-Reactive Protein - metabolism | Ethnic Groups - genetics | Matrix Metalloproteinase 9 - genetics | Adult | Female | Odds Ratio | Cytokines - genetics | Diabetes Mellitus, Type 2 - complications | Cytokines - immunology | Genetic Predisposition to Disease | Gene Frequency | Risk Factors | Chemokine CCL2 - genetics | Diabetic Nephropathies - genetics | Genotype | Transforming Growth Factor beta1 - genetics | India | Regression Analysis | Aged | Polymorphism, Single Nucleotide | Type 2 diabetes | Diabetics | Cytokines | Genes | Diabetic nephropathies | Disease susceptibility | Genetic aspects | Single nucleotide polymorphisms | Biotechnology | Nephrology | Risk | Genomes | Single-nucleotide polymorphism | Epidemiology | Genotype & phenotype | Rodents | Interleukin 8 | Enzyme-linked immunosorbent assay | Genotypes | CCR5 protein | Kidneys | Transforming growth factor-b1 | Diabetes mellitus | Health risks | Breast cancer | Inflammation | Regression analysis | Metabolism | Health education | Patients | Gelatinase B | Polymerase chain reaction | Studies | Genetic variance | Restriction fragment length polymorphism | Nephropathy | Genotyping | Alleles | Diabetes | Monocyte chemoattractant protein 1 | Polymorphism
Journal Article