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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
The Plant cell, ISSN 1532-298X, 2013, Volume 25, Issue 8, pp. 2907 - 2924
The INDUCER OF CBF EXPRESSION (ICE)—C-REPEAT BINDING FACTOR/DRE BINDING FACTOR1 (CBF/DREB1) transcriptional pathway plays a critical role in modulating cold stress responses in Arabidopsis thaliana... 
Proteins | Altitude tolerance | Transcription factors | RESEARCH ARTICLES | Genes | Acclimatization | Gene expression regulation | Plants | Freezing | Plant cells | Seedlings | DEFENSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | COLD-ACCLIMATION | LOW-TEMPERATURE | ANTHOCYANIN ACCUMULATION | PLANT SCIENCES | CELL BIOLOGY | REDUCES CHILLING INJURY | METHYL JASMONATE | GENE | MALE-STERILE | ABSCISIC-ACID | PROTEINS | Transcription, Genetic - drug effects | Arabidopsis - physiology | Adaptation, Physiological - drug effects | Structure-Activity Relationship | Arabidopsis Proteins - drug effects | Arabidopsis Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Adaptation, Physiological - genetics | Stress, Physiological - drug effects | Repressor Proteins - metabolism | Protein Structure, Tertiary | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Stress, Physiological - genetics | Signal Transduction - genetics | Cyclopentanes - pharmacology | Mutation - genetics | Arabidopsis - genetics | Transcription Factors - metabolism | Up-Regulation - drug effects | Oxylipins - metabolism | Two-Hybrid System Techniques | Gene Expression Regulation, Plant - drug effects | Phenotype | Signal Transduction - drug effects | Genes, Plant - genetics | Models, Biological | Oxylipins - pharmacology | Protein Binding | Cyclopentanes - metabolism | Trans-Activators - metabolism | Arabidopsis thaliana | Physiological aspects | Instrument industry | Chemical properties
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0116747
Cellular mechanisms of multidrug resistance (MDR) are related to ABC transporters, apoptosis, antioxidation, drug metabolism, DNA repair and cell proliferation... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | STEM-CELLS | MDR1 | TRANSPORTERS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | MUTANT P53 | TUMOR-SUPPRESSOR PROTEIN | DRUG-RESISTANCE | OVARIAN-CANCER | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Drug Resistance, Multiple - drug effects | Genes, Neoplasm | Humans | Apoptosis - genetics | Epithelial-Mesenchymal Transition - drug effects | Gene Expression Profiling | DNA Repair - genetics | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | Dose-Response Relationship, Drug | MCF-7 Cells | Neoplastic Stem Cells - metabolism | Inhibitory Concentration 50 | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | DNA Repair - drug effects | Drug Resistance, Multiple - genetics | Tumor Suppressor Protein p53 - metabolism | Signal Transduction - genetics | Down-Regulation - drug effects | Cell Shape - drug effects | Up-Regulation - drug effects | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Signal Transduction - drug effects | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Drug resistance in microorganisms | Anthracyclines | Tumor proteins | Intermediate filament proteins | Genes | Cell proliferation | Transcription | Bcl-2 protein | Mesenchyme | Gene regulation | Cytology | AKT protein | Cytotoxicity | Drug resistance | Kinases | DNA repair | Cancer therapies | Doxorubicin | Cell surface | E-cadherin | Cell morphology | Proteins | MDR1 protein | Clonal deletion | CD44 antigen | Rodents | Cell cycle | Drug metabolism | Repair | Drug dosages | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | Glutathione | Enzymes | Ploidy | BRCA1 protein | Multidrug resistance | Breast cancer | Gene expression | Metabolism | 1-Phosphatidylinositol 3-kinase | Medicine | Hypotheses | Chemotherapy | Gene amplification | Pharmacy | Stem cells | Mutation | Codons | Surface markers | Apoptosis | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Cancer cell, ISSN 1535-6108, 2016, Volume 29, Issue 4, pp. 464 - 476
.... In this perspective we discuss how understanding the cancer epigenome is providing insights into disease pathogenesis and informing drug development... 
targeted therapy | epigenome | histone modification | chromatin | Cancer | Histone modification | Chromatin | Epigenome | Targeted therapy | SELECTIVE-INHIBITION | THERAPEUTIC STRATEGY | ONCOLOGY | LYSINE 27 METHYLATION | HISTONE H3 | DROSOPHILA-TRITHORAX | 11Q23 TRANSLOCATIONS | CHROMATIN MODIFICATIONS | EMERGING MECHANISMS | TRANSCRIPTIONAL REGULATION | SOMATIC MUTATIONS | CELL BIOLOGY | Transcription, Genetic - drug effects | Humans | Gene Expression Regulation, Neoplastic | Neoplasms - prevention & control | Molecular Targeted Therapy | Neoplasm Proteins - metabolism | Neoplasms - therapy | Histone Code - drug effects | Neoplasms - genetics | Protein Processing, Post-Translational - drug effects | Cell Transformation, Neoplastic - genetics | Antineoplastic Agents - pharmacokinetics | Epigenesis, Genetic - drug effects | Chromatin - drug effects | Epigenomics | Oncogene Proteins - metabolism | Therapies, Investigational | Clinical Trials as Topic | DNA, Neoplasm - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Epigenesis, Genetic - genetics | Chromosome Aberrations | Histone Deacetylase Inhibitors - therapeutic use | Mice | Models, Genetic | DNA, Neoplasm - genetics | Histones - metabolism | Mutation | Chromatin - genetics | DNA Methylation - drug effects | Drug Resistance, Neoplasm - drug effects | Epigenetic inheritance | Development and progression | Genetic aspects
Journal Article
Molecular and cellular biology, ISSN 0270-7306, 2007, Volume 27, Issue 13, pp. 4953 - 4967
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CELLS | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | GENE-EXPRESSION | TRANSCRIPTIONAL COACTIVATOR | DIFFERENTIATION | MICROARRAY DATA | TRANSGENIC MICE | CELL BIOLOGY | Gene Expression Regulation, Enzymologic - drug effects | Acetyltransferases - metabolism | Multienzyme Complexes - metabolism | Adipocytes - drug effects | Glucose Intolerance | AMP-Activated Protein Kinases | Oxidative Phosphorylation - drug effects | Adipose Tissue, White - cytology | Body Composition - drug effects | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Food Deprivation | p38 Mitogen-Activated Protein Kinases - metabolism | Homeostasis - drug effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Isoenzymes - genetics | Hydrogen Peroxide - pharmacology | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Multienzyme Complexes - genetics | Macrophages - cytology | Mitochondria - drug effects | Feeding Behavior - drug effects | Polyamines - metabolism | Macrophages - metabolism | Organ Size - drug effects | Animals | Adipocytes - metabolism | Fibroblasts - drug effects | Adipose Tissue, White - enzymology | Adipose Tissue, White - growth & development | Glucose - metabolism | Macrophages - drug effects | Trans-Activators - metabolism | Mice | Transcription Factors | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | White/cytology/drug effects/enzymology/growth | Hydrogen Peroxide/pharmacology | Macrophages/cytology/drug effects/metabolism | Mitochondria/drug effects/metabolism | Fibroblasts/drug effects/metabolism | p38 Mitogen-Activated Protein Kinases/metabolism | MEDICIN OCH HÄLSOVETENSKAP | Homeostasis/drug effects | Multienzyme Complexes/genetics/metabolism | Protein-Serine-Threonine Kinases/genetics/metabolism | Trans-Activators/genetics/metabolism | Enzymologic/drug effects | Glucose/metabolism | Adipose Tissue | Feeding Behavior/drug effects | Organ Size/drug effects | Oxidative Phosphorylation/drug effects | MEDICAL AND HEALTH SCIENCES | development | Acetyltransferases/metabolism | Adipocytes/drug effects/metabolism | Gene Expression Regulation | Adenosine Triphosphate/metabolism | Body Composition/drug effects | Polyamines/metabolism | Energy Metabolism/drug effects | Isoenzymes/genetics/metabolism | Phosphorylation/drug effects
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2014, Volume 40, Issue 3, pp. 387 - 392
.... We aimed to evaluate the effect of these biomaterials on odontogenic differentiation of human dental pulp stem cells (DPSCs... 
Endocrinology & Metabolism | Dentistry | differentiation | calcium-enriched mixture | stem cell | pulp regeneration | mineral trioxide aggregate | CEM cement | human | dental pulp capping | Biomaterials | ENRICHED MIXTURE CEMENT | TREATMENT OUTCOMES | TISSUE | PULPOTOMY | PERMANENT MOLARS | IN-VITRO | MATRIX PROTEIN-1 | DENTISTRY, ORAL SURGERY & MEDICINE | HUMAN ODONTOBLASTS | IRREVERSIBLE PULPITIS | Bone Morphogenetic Protein 2 - drug effects | Phosphoproteins - drug effects | Humans | Sialoglycoproteins - drug effects | Transforming Growth Factor beta1 - drug effects | Cell Culture Techniques | Calcium Compounds - pharmacology | Fibroblast Growth Factor 4 - drug effects | Silicates - pharmacology | Microscopy, Electron, Scanning | Bone Morphogenetic Protein 4 - drug effects | Calcification, Physiologic - drug effects | Odontogenesis - drug effects | Dentinogenesis - drug effects | Cells, Cultured | Dental Pulp - cytology | Cementogenesis - drug effects | Biocompatible Materials - pharmacology | Cell Adhesion - drug effects | Extracellular Matrix Proteins - drug effects | Cell Shape - drug effects | Oxides - pharmacology | Cytokines - drug effects | Cell Differentiation - drug effects | Stem Cells - drug effects | Root Canal Filling Materials - pharmacology | Silicate Cement - pharmacology | Aluminum Compounds - pharmacology | Cell Proliferation - drug effects | Drug Combinations | Biological products | Cytokines | Gene expression | Stem cells
Journal Article
Cell metabolism, ISSN 1550-4131, 2016, Volume 24, Issue 6, pp. 795 - 806
.... Without any obvious toxicity or deleterious effects, NMN suppressed age-associated body weight gain, enhanced energy metabolism, promoted physical activity, improved insulin sensitivity and plasma... 
NAD | NAD+ precursor | insulin sensitivity | eye function | anti-aging | mitochondria | aging | nicotinamide mononucleotide | glucose metabolism | NMN | energy metabolism | precursor | LIFE-SPAN | STEM-CELLS | ACTIVATION | MITOCHONDRIAL | NAD BIOSYNTHESIS | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | RIBOSIDE | SIRT1 | ADIPOSE-TISSUE | CELL BIOLOGY | Darkness | Aging - drug effects | Male | Muscle, Skeletal - metabolism | Nicotinamide Mononucleotide - administration & dosage | Aging - genetics | Time Factors | Lipids - blood | Muscle, Skeletal - drug effects | Cell Respiration - drug effects | Myeloid Cells - drug effects | Weight Gain - drug effects | NAD - metabolism | Physical Conditioning, Animal | Food | Lymphocytes - metabolism | Insulin - pharmacology | Administration, Oral | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Eating - drug effects | Bone Density - drug effects | Animals | Aging - physiology | Lymphocytes - drug effects | Myeloid Cells - metabolism | Nicotinamide Mononucleotide - pharmacology | Drinking - drug effects | Nicotinamide Mononucleotide - blood | Energy Metabolism - drug effects | Niacinamide | Medical colleges | Exercise | Pharmacy | Genes | Body weight | Physiological aspects | Muscles | Mice | Ophthalmology | Gene expression
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2010, Volume 30, Issue 21, pp. 7152 - 7167
... (Set domain, bifurcated 1)/Eset/Kmt1e are associated with repressive chromatin remodeling and expressed in adult brain, but potential effects on neuronal function and behavior remain unexplored... 
DEPRESSION | CHROMATIN REGULATION | DEACETYLASE INHIBITOR | DNA METHYLATION | GENE | SODIUM-BUTYRATE | MOUSE MODEL | HIPPOCAMPAL | METABOTROPIC GLUTAMATE RECEPTORS | NEUROSCIENCES | METHYL-D-ASPARTATE | Chromatin - metabolism | Immobility Response, Tonic - physiology | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Fear - drug effects | Green Fluorescent Proteins - genetics | Protein Methyltransferases - metabolism | Neurons - ultrastructure | Excitatory Amino Acid Agents - pharmacology | Exploratory Behavior - drug effects | Behavior, Animal - drug effects | Excitatory Postsynaptic Potentials - genetics | Animals, Newborn | Membrane Potentials - drug effects | Adaptation, Ocular - genetics | Maze Learning - physiology | Food Preferences - drug effects | Histone-Lysine N-Methyltransferase | Affect - physiology | Behavior, Animal - physiology | Mice, Transgenic | Avoidance Learning - physiology | Avoidance Learning - drug effects | Mice | Membrane Potentials - genetics | Electroshock - adverse effects | Age Factors | Food Preferences - physiology | Protein Methyltransferases - genetics | Adaptation, Ocular - drug effects | Motor Activity - drug effects | Fear - physiology | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Receptors, N-Methyl-D-Aspartate - genetics | Exploratory Behavior - physiology | Transfection - methods | Neurons - physiology | Chromatin Immunoprecipitation - methods | Immobility Response, Tonic - drug effects | Neurons - drug effects | Sucrose - administration & dosage | Affect - drug effects | Conditioning (Psychology) - drug effects | Conditioning (Psychology) - physiology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Gene Expression Regulation - physiology | Hippocampus - cytology | Maze Learning - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Gene Expression Regulation - drug effects | Motor Activity - genetics | Animals | Sweetening Agents - administration & dosage
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 03/2018, Volume 10, Issue 3, p. n/a
.... These effects coincided with decreased brown adipocyte lipid content and increased nutrient uptake by BAT, confirming increased BAT activity... 
GPR120 | brown adipose tissue | Ca2 | mitochondria | lipid metabolism | Ca | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | PROTEIN | INSULIN-SECRETION | THERMOGENESIS | POTENT | GLUCOSE-METABOLISM | GENE-EXPRESSION | ACID RECEPTOR GPR40 | ADIPOSE-TISSUE | Adipocytes, White - cytology | Adipocytes, Brown - metabolism | Receptors, G-Protein-Coupled - metabolism | Adipose Tissue, White - metabolism | Body Weight - drug effects | Lipids | Male | Receptors, G-Protein-Coupled - agonists | Adipocytes, White - drug effects | Biphenyl Compounds - pharmacology | Cell Respiration - drug effects | Adiposity - drug effects | Oxidation-Reduction | Adipocytes, Brown - drug effects | Uncoupling Protein 1 - metabolism | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Gene Expression Regulation - drug effects | Phenylpropionates - pharmacology | Animals | Receptors, G-Protein-Coupled - deficiency | Cell Differentiation - drug effects | Models, Biological | Oxygen Consumption - drug effects | Glucose - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Adipocytes, Brown - cytology | Adipocytes, White - metabolism | Adipose Tissue, White - drug effects | Physiological aspects | Obesity | G proteins | Body weight | Membrane proteins | Pharmacology & Drug Discovery | Metabolism
Journal Article