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Journal of the National Cancer Institute, ISSN 0027-8874, 04/2011, Volume 103, Issue 8, pp. 645 - 661
Background Ionizing radiation (IR) is effectively used in cancer therapy. However, in subsets of patients, a few radioresistant cancer cells survive and cause... 
STEM-CELLS | IN-VITRO | ONCOLOGY | TYROSINE KINASE | HGF | CELL INVASION | RECEPTOR | EPITHELIAL-MESENCHYMAL TRANSITIONS | NF-KAPPA-B | SCATTER-FACTOR | PROTOONCOGENE | Humans | Neoplasm Invasiveness - prevention & control | Radiation Tolerance | Receptors, Growth Factor - antagonists & inhibitors | NF-kappa B - metabolism | RNA, Messenger - metabolism | NF-kappa B - radiation effects | Receptors, Growth Factor - genetics | Proto-Oncogene Proteins c-met - radiation effects | Signal Transduction - radiation effects | Protein-Serine-Threonine Kinases - metabolism | Radiation, Ionizing | Tumor Suppressor Proteins - metabolism | DNA-Binding Proteins - radiation effects | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Cell Cycle Proteins - metabolism | Cell Cycle Proteins - radiation effects | Receptors, Growth Factor - radiation effects | Proto-Oncogene Proteins c-met - drug effects | Ataxia Telangiectasia Mutated Proteins | Phosphorylation - radiation effects | Cell Line, Tumor | Mice | RNA, Small Interfering | Tumor Suppressor Proteins - radiation effects | Receptors, Growth Factor - metabolism | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Apoptosis - radiation effects | Up-Regulation - radiation effects | Transcription, Genetic - radiation effects | Transplantation, Heterologous | Sulfones - pharmacology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | Protein-Serine-Threonine Kinases - radiation effects | Tumor Suppressor Proteins - genetics | Polymerase Chain Reaction | Cell Cycle Proteins - genetics | Indoles - pharmacology | Neoplasms - radiotherapy | Gene Expression Regulation, Neoplastic - drug effects | In Situ Nick-End Labeling | Enzyme-Linked Immunosorbent Assay | Radiation-Sensitizing Agents - pharmacology | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | DNA-Binding Proteins - genetics | Cell Survival - radiation effects | Cell Movement - radiation effects | Proto-Oncogene Proteins c-met - genetics | Blotting, Northern | Animals | NF-kappa B - genetics | Protein Kinase Inhibitors - pharmacology | Neoplasms - pathology | Receptors, Growth Factor - drug effects | DNA Damage - radiation effects | Mitogen-Activated Protein Kinases - metabolism | Ionizing radiation | Physiological aspects | Genetic aspects | Cancer invasiveness | Research | Gene expression | Radiotherapy | Health aspects
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e83392
MicroRNA (miRNA)-mediated regulation of the cellular transcriptome is an important epigenetic mechanism for fine-tuning regulatory pathways. These include... 
INDUCED DNA-DAMAGE | TARGET GENES | HEPATOCELLULAR-CARCINOMA | NONMELANOMA SKIN-CANCER | THYROID-HORMONE | MULTIDISCIPLINARY SCIENCES | BASAL-CELL CARCINOMA | MALIGNANT-MELANOMA | MESENCHYMAL TRANSITION | IDENTIFICATION | RADIATION | Keratinocytes - radiation effects | Humans | Neoplasms, Radiation-Induced - etiology | MicroRNAs - metabolism | Carcinogenesis - metabolism | Carcinogenesis - radiation effects | Neoplasms, Radiation-Induced - genetics | Computer Simulation | Signal Transduction - radiation effects | Promoter Regions, Genetic | Membrane Proteins - genetics | Carcinogenesis - genetics | Cells, Cultured | MicroRNAs - radiation effects | Monomeric GTP-Binding Proteins - genetics | Gene Expression - radiation effects | Neoplasms, Radiation-Induced - metabolism | Ultraviolet Rays - adverse effects | Skin Neoplasms - metabolism | Keratinocytes - metabolism | Regulatory Elements, Transcriptional | Skin Neoplasms - genetics | Gene Regulatory Networks - radiation effects | MicroRNAs - genetics | Skin Neoplasms - etiology | Epigenetic inheritance | Ionizing radiation | MicroRNA | Analysis | Development and progression | Skin | Skin cancer | Phosphorylation | Homology | Biology | Regulatory sequences | Metastasis | Carcinogenesis | Metastases | Liver cancer | Carcinogens | Pathways | miRNA | Deoxyribonucleic acid--DNA | Medical research | Dermatology | U.V. radiation | Melanoma | Keratinocytes | Exposure | Gene expression | Ribonucleic acid--RNA | Signaling | Ultraviolet radiation | MicroRNAs | Irradiation | Cancer | Apoptosis | RNA | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0159998 - e0159998
Recent research found that Tiron was an effective antioxidant that could act as the intracellular reactive oxygen species (ROS) scavenger or alleviate the... 
IRRADIATION | OXIDATIVE STRESS | HUMAN SKIN | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MECHANISMS | SODIUM 4,5-DIHYDROXYBENZENE-1,3-DISULFONATE | INDUCTION | C-JUN | NF-KAPPA-B | PROTEIN-KINASES | Promoter Regions, Genetic - radiation effects | Binding Sites - radiation effects | Humans | Transcriptional Activation - drug effects | Skin Aging - radiation effects | 1,2-Dihydroxybenzene-3,5-Disulfonic Acid Disodium Salt - pharmacology | Promoter Regions, Genetic - drug effects | Transcription Factor AP-1 - metabolism | Dermis - drug effects | Cytoprotection - drug effects | Signal Transduction - radiation effects | Dermis - cytology | Matrix Metalloproteinase 1 - genetics | Fibroblasts - metabolism | MAP Kinase Signaling System - radiation effects | Dermis - metabolism | Skin Aging - drug effects | Transcriptional Activation - radiation effects | Cells, Cultured | Dermis - radiation effects | Matrix Metalloproteinase 3 - metabolism | Antioxidants - pharmacology | Ultraviolet Rays - adverse effects | MAP Kinase Signaling System - drug effects | Fibroblasts - radiation effects | Signal Transduction - drug effects | Fibroblasts - drug effects | Transcription Factor AP-1 - radiation effects | Cytoprotection - genetics | Matrix Metalloproteinase 1 - metabolism | Cytoprotection - radiation effects | Matrix Metalloproteinase 3 - genetics | Genetic aspects | Skin | Genetic transcription | RNA | Analysis | Enzyme-linked immunosorbent assay | Oxidative stress | Reactive oxygen species | Transcription factors | Senescence | Oncology | Activation | Matrix metalloproteinase | Kinases | Proteins | Antioxidants | Signal transduction | Consent | Transcription activation | Fibroblasts | Aging | Biocompatibility | Metalloproteinase | Interstitial collagenase | Stromelysin 1 | Wound healing | U.V. radiation | Activator protein 1 | MAP kinase | Gene expression | Medicine | Signaling | Collagen | In vivo methods and tests | Binding sites | Apoptosis | Index Medicus
Journal Article
STEM CELLS, ISSN 1066-5099, 02/2013, Volume 31, Issue 2, pp. 372 - 383
Hematopoietic stem and progenitor cells (HSPCs), which continuously maintain all mature blood cells, are regulated within the marrow microenvironment. We... 
Bone marrow | Irradiation | Prostaglandin E2 | Hematopoietic stem cells | Apoptosis | PROGENITOR CELLS | OSTEOBLASTIC MC3T3-E1 CELLS | WHOLE-BODY IRRADIATION | DUODENAL-ULCER | 16,16-DIMETHYL PROSTAGLANDIN-E2 | MACROPHAGES | BONE-MARROW | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CYCLOOXYGENASE-2 EXPRESSION | HEMATOLOGY | EXPOSURE | MEGAKARYOCYTE MATURATION | Whole-Body Irradiation | Gene Expression - drug effects | Apoptosis - drug effects | Apoptosis - radiation effects | Radiation Injuries, Experimental - immunology | Hematopoietic Stem Cells - pathology | Male | Actins - immunology | Actins - genetics | Cellular Microenvironment - radiation effects | Cyclooxygenase 2 - genetics | Radiation Injuries, Experimental - drug therapy | Radiation-Protective Agents - pharmacology | Bone Marrow Cells - drug effects | Macrophages - radiation effects | Cyclooxygenase 2 - immunology | Hematopoietic Stem Cells - radiation effects | Cell Differentiation - radiation effects | Hematopoietic Stem Cells - drug effects | Cell Survival - drug effects | Dinoprostone - pharmacology | Macrophages - pathology | Bone Marrow Cells - pathology | Mice, Transgenic | Bone Marrow Cells - radiation effects | Radiation Injuries, Experimental - pathology | Cell Survival - radiation effects | Gene Expression - radiation effects | Animals | Cell Differentiation - drug effects | Dinoprostone - analogs & derivatives | Macrophages - drug effects | Cell Proliferation - drug effects | Mice | Cellular Microenvironment - drug effects | Cell Proliferation - radiation effects | Medical colleges | Nuclear radiation | Analysis | Prostaglandins E | Information management | Macrophages
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e48674
Observations of enhanced growth of melanized fungi under low-dose ionizing radiation in the laboratory and in the damaged Chernobyl nuclear reactor suggest... 
GAMMA-RADIATION | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | PHOTOLYASE/CRYPTOCHROME FAMILY | ESCHERICHIA-COLI | GENE-EXPRESSION | DOWN-REGULATION | SACCHAROMYCES-CEREVISIAE | EXOPHIALA DERMATITIDIS | BLUE-LIGHT | TRANSCRIPTIONAL RESPONSE | Cell Cycle - genetics | Reactive Oxygen Species - metabolism | Exophiala - physiology | Membrane Fluidity - genetics | Antioxidants - metabolism | Up-Regulation - radiation effects | DNA Repair - radiation effects | Gene Expression Profiling | Transcriptome - radiation effects | DNA Repair - genetics | RNA, Messenger - metabolism | Melanins - metabolism | Adaptation, Physiological - genetics | Biological Transport - radiation effects | Membrane Fluidity - radiation effects | Biological Transport - genetics | Exophiala - cytology | Microbial Viability - radiation effects | Genes, Fungal - genetics | Radiation, Ionizing | Reproducibility of Results | Exophiala - radiation effects | Cell Cycle - radiation effects | RNA, Messenger - genetics | Water - metabolism | Exophiala - genetics | Up-Regulation - genetics | Microbial Viability - genetics | Transcriptome - genetics | Adaptation, Physiological - radiation effects | Reverse Transcriptase Polymerase Chain Reaction | Carotenoids - biosynthesis | Gene Expression Regulation, Fungal - radiation effects | Ribosomes - genetics | Dose-Response Relationship, Radiation | Ribosomes - radiation effects | Fungi | Nuclear energy | Ionizing radiation | RNA | Analysis | Carbohydrate metabolism | DNA synthesis | Free radicals (Chemistry) | Nuclear facilities | Reactive oxygen species | Baking yeast | Yeast | Laboratories | Genomics | Genes | Science | Irradiated | Amino acids | Biosynthesis | Genomes | Antioxidants | Signal transduction | Membrane fluidity | Fluidity | Cell cycle | Deoxyribonucleic acid--DNA | Carbohydrates | Cell division | Exposure | Functional analysis | Gene expression | Metabolism | Ribonucleic acid--RNA | Biological activity | Radiation dosage | Studies | DNA biosynthesis | Irradiation | Genetic engineering | Nuclear reactors | Cell size | Mutation | Survivability | Radiation damage | Melanin | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
Plant, Cell & Environment, ISSN 0140-7791, 04/2014, Volume 37, Issue 4, pp. 961 - 977
The blue light photoreceptors cryptochromes are ubiquitous in higher plants and are vital for regulating plant growth and development. In spite of being... 
microarray | photomorphogenesis | anthocyanin | hypocotyl | abiotic stress | Anthocyanin | Hypocotyl | Microarray | Abiotic stress | Photomorphogenesis | SEEDLING DEVELOPMENT | ARABIDOPSIS HYPOCOTYL ELONGATION | F-BOX PROTEINS | VACUOLAR H+-ATPASE | BLUE-LIGHT | PLANT SCIENCES | EMBRYOGENESIS ABUNDANT PROTEINS | GENOME-WIDE ANALYSIS | NITRIC-OXIDE | DE-ETIOLATION | TRANSCRIPTION-FACTOR | Cell Wall - radiation effects | Brassica napus - drug effects | Cotyledon - drug effects | Cryptochromes - genetics | Molecular Sequence Data | Gene Expression Profiling | Plant Development - drug effects | Plant Development - genetics | Stress, Physiological - radiation effects | Plant Development - radiation effects | Brassica napus - radiation effects | Cotyledon - radiation effects | Light | Plants, Genetically Modified | Plant Epidermis - cytology | Plant Leaves - drug effects | Plant Proteins - metabolism | Stress, Physiological - drug effects | Germination - radiation effects | Signal Transduction - radiation effects | Plant Epidermis - radiation effects | Real-Time Polymerase Chain Reaction | Brassica napus - growth & development | Cryptochromes - metabolism | Germination - drug effects | Gene Expression Regulation, Plant - radiation effects | Brassica napus - genetics | Stress, Physiological - genetics | Cell Wall - genetics | Hypocotyl - growth & development | Plant Leaves - radiation effects | Signal Transduction - genetics | Hypocotyl - radiation effects | Seedlings - drug effects | Anthocyanins - metabolism | Abscisic Acid - pharmacology | Plant Proteins - genetics | Gene Expression Regulation, Plant - drug effects | Phenotype | Signal Transduction - drug effects | Plant Leaves - growth & development | Cotyledon - growth & development | Hypocotyl - drug effects | Plant Growth Regulators - metabolism | Seedlings - radiation effects | Seedlings - metabolism | Genetic research | Abscisic acid | Genetic engineering | Growth | Analysis | Genes
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 04/2017, Volume 26, Issue 11, pp. 563 - 582
Aims: Radiation-induced normal tissue toxicity is closely linked to endothelial cell (EC) damage and dysfunction (acute effects). However, the underlying... 
Original Research Communications | radioprotection | mesenchymal stem cell therapy | vascular dysfunction | Radiotherapy | TOTAL-BODY IRRADIATION | PRO-INFLAMMATORY CYTOKINES | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | TISSUE | INJURY | MECHANISMS | ENDOCRINOLOGY & METABOLISM | radiotherapy | IONIZING-RADIATION | UP-REGULATION | PULMONARY-FIBROSIS | Salicylates - pharmacology | Endothelial Cells - ultrastructure | Mesenchymal Stromal Cells - cytology | Radiation Injuries - genetics | Superoxide Dismutase-1 - metabolism | Lung - radiation effects | Lung - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Radiation Injuries - therapy | Gene Expression | Lung - pathology | Endothelial Cells - metabolism | Endothelial Cells - radiation effects | Phenotype | Animals | Fibroblasts - radiation effects | Fibrosis | Superoxide Dismutase-1 - genetics | Radiation Injuries - pathology | Mice | Endothelial Cells - pathology | Radiation Injuries - metabolism | Radiation Injuries, Experimental | Mesenchymal Stem Cell Transplantation | Organometallic Compounds - pharmacology | Superoxide dismutase | Research | Lungs | Gene expression | Health aspects | Cellular therapy | Cell culture | Therapy | Mesenchyme | Toxicity | Radiation | Innovations | Stem cell transplantation | Paracrine signalling | Adoptive transfer | Antioxidants | Reduction | Bone marrow | Radiation injuries | Aorta | Acute effects | Radiation effects | Thorax | Radioprotection | Endothelial cells | Stem cells | Irradiation | Viability | Radiation damage | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e32035
Background: The sestrin family of stress-responsive genes (SESN1-3) are suggested to be involved in regulation of metabolism and aging through modulation of... 
TARGET | LOCALIZATION | APOPTOSIS | ACTIVATED PROTEIN-KINASE | GENE | PHOSPHORYLATION | BIOLOGY | P53 TUMOR-SUPPRESSOR | STRESS | IDENTIFICATION | FAMILY | AMP-Activated Protein Kinases - metabolism | Humans | Nuclear Proteins - metabolism | Cell Survival - radiation effects | Enzyme Activation - radiation effects | Subcellular Fractions - metabolism | Protein Subunits - metabolism | Breast Neoplasms - enzymology | Breast Neoplasms - radiotherapy | Phosphorylation - radiation effects | Breast Neoplasms - pathology | Models, Biological | Subcellular Fractions - radiation effects | Protein Binding - radiation effects | Cell Line, Tumor | Female | Signal Transduction - radiation effects | Cell Proliferation - radiation effects | Radiation, Ionizing | Ionizing radiation | Care and treatment | RNA | Cancer cells | Genetic research | Breast cancer | Health aspects | Protein kinases | Cancer | TOR protein | Phosphorylation | Immunoprecipitation | Lung cancer | Homeostasis | mRNA | Kinases | AMP-activated protein kinase | Cancer therapies | Proteins | LKB1 protein | Signal transduction | Cell growth | Rodents | Cell cycle | Aging | Polarity | Tumorigenesis | Oxidation | Localization | Background radiation | Deoxyribonucleic acid--DNA | Immunoglobulins | AMP | Aging (artificial) | I.R. radiation | siRNA | Metabolism | Mammals | Gene expression | Fatty acids | Energy balance | Pathology | Signaling | Mutagenesis | Insects | Cytoplasm | Tumors | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article