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PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e80883
.... This effect was associated with reduced expressions of MMP-2 and TIMP-2 mRNA and protein levels... 
MIGRATION | INACTIVATION | INVASION | COLORECTAL-CANCER | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | TISSUE INHIBITOR | FLAVONOIDS | PROGNOSTIC-SIGNIFICANCE | NF-KAPPA-B | CARCINOMA | Transcription, Genetic - drug effects | Protein Binding - genetics | Nitriles - pharmacology | DNA, Neoplasm - metabolism | Humans | Cell Survival - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Transcription Factor AP-1 - metabolism | Cell Movement - genetics | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Protein Binding - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Mouth Neoplasms - enzymology | Cell Survival - drug effects | Butadienes - pharmacology | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | RNA, Messenger - genetics | Signal Transduction - genetics | Down-Regulation - drug effects | Cell Movement - drug effects | Matrix Metalloproteinase 2 - genetics | Matrix Metalloproteinase Inhibitors - pharmacology | Signal Transduction - drug effects | Cell Line, Tumor | Mouth Neoplasms - pathology | Kaempferols - pharmacology | Prevention | Medical research | Care and treatment | Squamous cell carcinoma | RNA | Analysis | Medicine, Experimental | Metastasis | Cardiovascular diseases | Mouth cancer | Cancer | Biotechnology | Food plants | Transcription factors | Tongue | Phosphorylation | Transcription | Oral cancer | Laboratories | Gene regulation | Colorectal cancer | Otolaryngology | Biochemistry | Matrix metalloproteinase | Tissue inhibitor of metalloproteinase 2 | Cancer therapies | Metastases | Angiogenesis | Signal transduction | Cell cycle | Physiology | Metalloproteinase | Plant-based foods | Food | Kaempferol | Activator protein 1 | Melanoma | Extracellular signal-regulated kinase | c-Jun protein | Plant protection | Breast cancer | Gene expression | Gelatinase A | Signaling | Bone cancer | Chemotherapy | Flavonoids | Hospitals | Molecular modelling | Cell migration | Apoptosis
Journal Article
The Plant cell, ISSN 1532-298X, 2013, Volume 25, Issue 8, pp. 2907 - 2924
The INDUCER OF CBF EXPRESSION (ICE)—C-REPEAT BINDING FACTOR/DRE BINDING FACTOR1 (CBF/DREB1) transcriptional pathway plays a critical role in modulating cold stress responses in Arabidopsis thaliana... 
Proteins | Altitude tolerance | Transcription factors | RESEARCH ARTICLES | Genes | Acclimatization | Gene expression regulation | Plants | Freezing | Plant cells | Seedlings | DEFENSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | COLD-ACCLIMATION | LOW-TEMPERATURE | ANTHOCYANIN ACCUMULATION | PLANT SCIENCES | CELL BIOLOGY | REDUCES CHILLING INJURY | METHYL JASMONATE | GENE | MALE-STERILE | ABSCISIC-ACID | PROTEINS | Transcription, Genetic - drug effects | Arabidopsis - physiology | Adaptation, Physiological - drug effects | Structure-Activity Relationship | Arabidopsis Proteins - drug effects | Arabidopsis Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Adaptation, Physiological - genetics | Stress, Physiological - drug effects | Repressor Proteins - metabolism | Protein Structure, Tertiary | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Stress, Physiological - genetics | Signal Transduction - genetics | Cyclopentanes - pharmacology | Mutation - genetics | Arabidopsis - genetics | Transcription Factors - metabolism | Up-Regulation - drug effects | Oxylipins - metabolism | Two-Hybrid System Techniques | Gene Expression Regulation, Plant - drug effects | Phenotype | Signal Transduction - drug effects | Genes, Plant - genetics | Models, Biological | Oxylipins - pharmacology | Protein Binding | Cyclopentanes - metabolism | Trans-Activators - metabolism | Arabidopsis thaliana | Physiological aspects | Instrument industry | Chemical properties
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0116747
Cellular mechanisms of multidrug resistance (MDR) are related to ABC transporters, apoptosis, antioxidation, drug metabolism, DNA repair and cell proliferation... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | STEM-CELLS | MDR1 | TRANSPORTERS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | MUTANT P53 | TUMOR-SUPPRESSOR PROTEIN | DRUG-RESISTANCE | OVARIAN-CANCER | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Drug Resistance, Multiple - drug effects | Genes, Neoplasm | Humans | Apoptosis - genetics | Epithelial-Mesenchymal Transition - drug effects | Gene Expression Profiling | DNA Repair - genetics | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | Dose-Response Relationship, Drug | MCF-7 Cells | Neoplastic Stem Cells - metabolism | Inhibitory Concentration 50 | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | DNA Repair - drug effects | Drug Resistance, Multiple - genetics | Tumor Suppressor Protein p53 - metabolism | Signal Transduction - genetics | Down-Regulation - drug effects | Cell Shape - drug effects | Up-Regulation - drug effects | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Signal Transduction - drug effects | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Drug resistance in microorganisms | Anthracyclines | Tumor proteins | Intermediate filament proteins | Genes | Cell proliferation | Transcription | Bcl-2 protein | Mesenchyme | Gene regulation | Cytology | AKT protein | Cytotoxicity | Drug resistance | Kinases | DNA repair | Cancer therapies | Doxorubicin | Cell surface | E-cadherin | Cell morphology | Proteins | MDR1 protein | Clonal deletion | CD44 antigen | Rodents | Cell cycle | Drug metabolism | Repair | Drug dosages | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | Glutathione | Enzymes | Ploidy | BRCA1 protein | Multidrug resistance | Breast cancer | Gene expression | Metabolism | 1-Phosphatidylinositol 3-kinase | Medicine | Hypotheses | Chemotherapy | Gene amplification | Pharmacy | Stem cells | Mutation | Codons | Surface markers | Apoptosis | Tumors | Deoxyribonucleic acid | DNA
Journal Article
BMC cancer, ISSN 1471-2407, 2015, Volume 15, Issue 1, p. 672
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 6, pp. 2188 - 2201
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2018, Volume 22, Issue 10, pp. 4935 - 4947
Journal Article
International journal of cancer, ISSN 0020-7136, 2018, Volume 143, Issue 11, pp. 2871 - 2883
.... SETD1A expression was upregulated in breast tumor tissue compared to that in normal breast tissue. Moreover, ER... 
breast cancer | tamoxifen resistance | estrogen receptor | miR‐1915‐3p | SETD1A | miR-1915-3p | ACTIVATION | PATTERNS | MECHANISMS | HSETD1A | THERAPY | ONCOLOGY | METHYLTRANSFERASE COMPLEX | CXXC FINGER PROTEIN-1 | HISTONE MODIFICATIONS | PROGRESSION | Cell Cycle - genetics | Apoptosis - drug effects | Humans | Cell Survival - genetics | Apoptosis - genetics | Cell Movement - genetics | MCF-7 Cells | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Cell Survival - drug effects | Cell Proliferation - genetics | Histone-Lysine N-Methyltransferase - genetics | Up-Regulation - genetics | Down-Regulation - drug effects | Breast Neoplasms - drug therapy | Down-Regulation - genetics | Up-Regulation - drug effects | Cell Movement - drug effects | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Tamoxifen - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | MicroRNAs - genetics | Cell Cycle - drug effects | Drug Resistance, Neoplasm - drug effects | Cell proliferation | Chromatin | Cell survival | Leukocyte migration | Genes | Estrogens | Methyltransferase | Estrogen receptors | Breast cancer | Tamoxifen | Survival | Metastases | Lysine | Transcription activation | Cell cycle | Methylation | Cell migration | Histone H3 | Cancer | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2018, Volume 115, Issue 43, pp. E10119 - E10126
Journal Article
by Dong, J and Tang, D and Gao, Z and Yu, R and Li, K and He, H and Terzaghi, W and Deng, X. W and Chen, H
The Plant cell, ISSN 1532-298X, 2014, Volume 26, Issue 9, pp. 3630 - 3645
.... Genetic analysis showed that each pif single mutant could enhance the det1-1 phenotype, and ectopie expression of each PIF in det1-1 partially suppressed the det1-1 phenotype, based on hypocotyl... 
Phenotypes | Transcription factors | Hypocotyls | Cocktails | RESEARCH ARTICLES | Genes | Gene expression regulation | Physiological regulation | Plants | Seedlings | Plant cells | PLANT | DET1 | SEED-GERMINATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEGATIVE REGULATOR | PLANT SCIENCES | CELL BIOLOGY | LIGHT-SIGNAL-TRANSDUCTION | COP9 SIGNALOSOME | GENE-EXPRESSION | DEGRADATION | TRANSCRIPTION FACTOR | E3 UBIQUITIN LIGASE | Darkness | Cell Wall - radiation effects | Arabidopsis - growth & development | Genes, Plant | Etiolation - drug effects | Arabidopsis Proteins - metabolism | Arabidopsis - radiation effects | Cell Wall - drug effects | Light | Protein Binding - drug effects | Protein Binding - radiation effects | Photosynthesis - radiation effects | Gene Expression Regulation, Plant - radiation effects | Arabidopsis - drug effects | Morphogenesis - radiation effects | Proteasome Inhibitors - pharmacology | Etiolation - radiation effects | Nuclear Proteins - metabolism | Transcriptome - genetics | Seedlings - drug effects | Mutation - genetics | Arabidopsis - metabolism | Gene Expression Regulation, Plant - drug effects | Phenotype | Indoleacetic Acids - pharmacology | Models, Biological | Photosynthesis - drug effects | Morphogenesis - drug effects | Seedlings - radiation effects | Seedlings - metabolism | Arabidopsis thaliana | Botanical research | Physiological aspects | Photoreceptors | Research | Photomorphogenesis
Journal Article
Neuropsychopharmacology (New York, N.Y.), ISSN 1740-634X, 2012, Volume 38, Issue 5, pp. 872 - 883
Stress and glucocorticoid hormones regulate hippocampal neurogenesis, but the molecular mechanisms underlying their effects are unknown... 
stem cells | Hedgehog signaling | prenatal stress | depression | PROGENITOR CELLS | INDUCED DOWN-REGULATION | ADULT NEUROGENESIS | PSYCHIATRY | MAJOR DEPRESSION | NEUROSCIENCES | MINERALOCORTICOID RECEPTOR | NEURAL STEM-CELLS | GENE | PHARMACOLOGY & PHARMACY | DENTATE GYRUS | EXPRESSION | Microtubule-Associated Proteins - metabolism | Humans | Male | Gene Expression Profiling | Receptors, Glucocorticoid - metabolism | Dose-Response Relationship, Drug | Dexamethasone - pharmacology | Transfection | Glucocorticoids - metabolism | Female | Fetus | Neurogenesis - drug effects | Neurons - metabolism | Hedgehogs - metabolism | Neurons - drug effects | Gene Expression Regulation - genetics | Neural Stem Cells - drug effects | Neural Stem Cells - physiology | Rats | Neuropeptides - metabolism | Hippocampus - cytology | Rats, Sprague-Dawley | Hormone Antagonists - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Neurogenesis - physiology | Receptors, Glucocorticoid - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Hydrocortisone - pharmacology | Mifepristone - pharmacology | Dexamethasone | Glucocorticoids | Astrocytes | Gliogenesis | Hormones | Gene expression | Hydrocortisone | Neurogenesis | Stress | Signal transduction | DNA microarrays | Hedgehog protein | Neuroblasts | Microtubule-associated protein 2 | Mineralocorticoid receptors | Neural stem cells | Notch protein | Differentiation | Hippocampus | stress | Animal models | hippocampus | Neuropharmacology | Receptor Pharmacology | glucocorticoids | Original | Biological Psychiatry | molecular signaling pathways
Journal Article