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Nature neuroscience, ISSN 1546-1726, 2013, Volume 16, Issue 12, pp. 1773 - 1782
...; however, the signals controlling C1q expression and function remain elusive. Previous work implicated an astrocyte-derived factor in regulating neuronal C1q expression... 
Journal Article
Plant physiology (Bethesda), ISSN 0032-0889, 6/2016, Volume 171, Issue 2, pp. 1128 - 1143
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 04/2015, Volume 284, Issue 2, pp. 142 - 151
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10431
... of PrCa models in 3D, to compare phenotypes, gene expression and metabolism between 2D and 3D cultures, and to evaluate... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GENE-EXPRESSION SIGNATURE | STEM-CELLS | MAMMARY EPITHELIA | METASTASIS | BIOLOGY | TUMOR-CELL INVASION | DIFFERENTIATION | CULTURE MODEL | LINES | Laminin - pharmacology | Epithelial Cells - drug effects | Humans | Mesoderm - drug effects | Spheroids, Cellular - pathology | Male | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Spheroids, Cellular - enzymology | Neoplasm Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA, Messenger - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Prostate - drug effects | Antineoplastic Agents - pharmacology | Collagen - pharmacology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Principal Component Analysis | Prostatic Neoplasms - drug therapy | Epithelium - drug effects | Prostatic Neoplasms - pathology | Epithelium - pathology | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Cell Shape - drug effects | Phenotype | Proteoglycans - pharmacology | Signal Transduction - drug effects | Models, Biological | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Drug Combinations | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Epigenetic inheritance | Growth | Oncology, Experimental | Genes | Research | Gene expression | Ionizing radiation | Stem cells | Physiological aspects | Models | Drug discovery | Drug therapy | Prostate cancer | Integrins | Cancer | Cell culture | Biotechnology | Transformation | Motility | Leukocyte migration | Mesenchyme | Epithelial cells | Homeostasis | AKT protein | Metastasis | Drug resistance | Tissues | Ovarian cancer | Cell adhesion & migration | Metastases | Rodents | Fibroblasts | Extracellular matrix | Basal lamina | Lipid metabolism | Medical research | Invasiveness | Phenotypic plasticity | Tumor cell lines | Metabolism | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Interferon | Three dimensional models | Prostate | Cell migration
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23522
Background: Aryl hydrocarbon receptor (AhR), a transcription factor of the bHLH/PAS family, is well characterized to regulate the biochemical and toxic effects of environmental chemicals... 
FAS LIGAND | INFLAMMATORY-BOWEL-DISEASE | IL-10(-/-) MICE | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ULCERATIVE-COLITIS | CHEMOKINE RECEPTORS | NF-KAPPA-B | T-CELLS | HELPER-CELL DIFFERENTIATION | Dextran Sulfate | T-Lymphocytes, Regulatory - metabolism | Epigenesis, Genetic | Colon - drug effects | Th17 Cells - drug effects | Flow Cytometry | Th17 Cells - metabolism | Polychlorinated Dibenzodioxins - pharmacology | Colitis - chemically induced | Receptors, Aryl Hydrocarbon - metabolism | Female | Receptors, CXCR3 - metabolism | Cytokines - metabolism | Colon - pathology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Gene Expression Regulation | Receptors, Aryl Hydrocarbon - genetics | Interleukin-17 - genetics | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Colon - metabolism | Mice, Knockout | Receptors, Aryl Hydrocarbon - agonists | T-Lymphocytes, Regulatory - drug effects | Animals | Cell Differentiation - drug effects | Colitis - metabolism | CpG Islands - genetics | Mice | Colitis - prevention & control | DNA Methylation - drug effects | CD4-Positive T-Lymphocytes - drug effects | Epigenetic inheritance | Herbicides | Analysis | Dioxin | Body weight | Weight loss | Colitis | Research | T cells | Methylation | Toxicity | Mucosa | Helper cells | TCDD | Hydrocarbons | Lymphocytes T | Body weight loss | Activation | Sodium sulfates | Lymph nodes | Demethylation | Cell activation | Aromatic compounds | Lymphocytes | Rodents | DNA methylation | Foxp3 protein | Colon | CpG islands | Cytokines | Lamina propria | T cell receptors | Inflammation | CXCR3 protein | Environmental effects | Interleukin 17 | Inflammatory bowel disease | Dextran | Sodium | Epigenetics | Differentiation
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e24099
...) and HDAC inhibitor Vorinostat (SAHA). Methodology/ Principal Findings: Re-expression of miR-34a in human pancreatic cancer stem cells (CSCs... 
BIOMARKERS | NERVOUS-SYSTEM | APOPTOSIS | TRAIL | SIGNALING PATHWAY | P53 PROTEIN | MULTIDISCIPLINARY SCIENCES | NOTCH | TUMOR-SUPPRESSOR | MICRORNAS | EXPRESSION | Cell Cycle - genetics | Chromatin - metabolism | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Humans | Spheroids, Cellular - pathology | Apoptosis - genetics | Epithelial-Mesenchymal Transition - drug effects | MicroRNAs - metabolism | Epithelial-Mesenchymal Transition - genetics | Pancreatic Neoplasms - drug therapy | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Epigenesis, Genetic - drug effects | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Hydroxamic Acids - pharmacology | Tumor Stem Cell Assay | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Spheroids, Cellular - metabolism | Pancreatic Neoplasms - genetics | Up-Regulation - genetics | Up-Regulation - drug effects | Azacitidine - pharmacology | Cell Movement - drug effects | Cell Line, Tumor | Hydroxamic Acids - therapeutic use | Cell Proliferation - drug effects | MicroRNAs - genetics | Cell Cycle - drug effects | Azacitidine - therapeutic use | Prevention | Epigenetic inheritance | Care and treatment | Chemotherapy | Chromatin | Pancreatic cancer | Stem cells | Development and progression | Tumor proteins | Cancer | Apoptosis | Bcl-2 protein | p53 Protein | Metastasis | Caspase-3 | Cancer therapies | Toxicology | Scholarships & fellowships | Cell growth | N-Cadherin | Restoration | Physiology | Tumorigenesis | Inhibition | Gene expression | SIRT1 protein | Pathology | Biomarkers | Notch protein | Mutation | Cell proliferation | Azacytidine | Histone deacetylase | Transcription | Mesenchyme | Laboratories | Leukemia | Gene regulation | Multiple myeloma | E-cadherin | Modulators | Cell cycle | miRNA | Inducers | Departments | Caspase | Pharmacology | Breast cancer | Tumor cell lines | Ribonucleic acid--RNA | Medicine | Cyclin-dependent kinase inhibitor p21 | Medical prognosis | Reagents | Epigenetics | Cyclin-dependent kinase inhibitor p27 | Prostate cancer | RNA | Ribonucleic acid
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2008, Volume 233, Issue 2, pp. 286 - 296
.... To investigate whether transient developmental exposure to MXC could cause adult ovarian dysfunction, we exposed Fischer rats to 20 μg/kg/day (low dose... 
Endocrine disruptors | Ovary | Folliculogenesis | Estrogen receptor-β | Development | Luteinizing hormone receptor | Steroidogenesis | Anti-Mullerian hormone | GRANULOSA-CELL DIFFERENTIATION | ALPHA | ESTROGEN-RECEPTOR-BETA | IN-VITRO | FOLLICULAR DEVELOPMENT | PHARMACOLOGY & PHARMACY | Estrogen receptor-beta | BISPHENOL-A | TOXICOLOGY | LH RECEPTORS | MOUSE OVARY | Immunohistochemistry | Cholesterol Side-Chain Cleavage Enzyme - metabolism | Rats, Inbred F344 | Luteinizing Hormone - metabolism | Sexual Maturation - drug effects | Estrogen Receptor beta - drug effects | Dose-Response Relationship, Drug | Estrogen Receptor beta - metabolism | Insecticides - administration & dosage | Ovarian Follicle - drug effects | Fertility - drug effects | Female | Ovulation - drug effects | Luteinizing Hormone - drug effects | Cholesterol Side-Chain Cleavage Enzyme - drug effects | Rats | Estrous Cycle - drug effects | Gene Expression Regulation - drug effects | Pregnancy | Methoxychlor - toxicity | Animals | Methoxychlor - administration & dosage | Progesterone - metabolism | Litter Size - drug effects | Anti-Mullerian Hormone - metabolism | Ovarian Follicle - pathology | Insecticides - toxicity | Index Medicus | ESTROGENS | LUTEINIZING HORMONE | ORGANIC CHLORINE COMPOUNDS | PROGESTERONE | LITTER SIZE | RATS | AGING | 60 APPLIED LIFE SCIENCES | FERTILITY | PESTICIDES | DOSES | GENES | OVARIES | RECEPTORS | MORPHOLOGY | folliculogenesis | ovary | development | anti-Mullerian hormone | endocrine disruptors | steroidogenesis | luteinizing hormone receptor |