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Nature (London), ISSN 1476-4687, 2014, Volume 511, Issue 7510, pp. 488 - 492
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0132977
.... Recent evidences have highlighted the correlation of BORIS/CTCFL expression with poor overall survival of different cancer patients... 
TESTIS ANTIGEN BORIS | HEPATOCELLULAR-CARCINOMA | SIDE POPULATION | IN-VITRO | INDUCED APOPTOSIS | TELOMERASE | MULTIDISCIPLINARY SCIENCES | IMPRINTED SITES | CTCF | IDENTIFICATION | TUMOR-INITIATING CELLS | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | RNA, Small Interfering - genetics | Cell Proliferation | Epithelial Cells - metabolism | Polycomb Repressive Complex 1 - metabolism | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | Polycomb Repressive Complex 1 - genetics | Telomerase - genetics | ATP-Binding Cassette Transporters - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | ATP-Binding Cassette Transporters - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Telomerase - metabolism | Neoplasm Proteins - genetics | DNA-Binding Proteins - antagonists & inhibitors | Nanog Homeobox Protein | Signal Transduction | Isoenzymes - genetics | Spheroids, Cellular - metabolism | Epithelial Cells - pathology | Retinal Dehydrogenase - genetics | DNA-Binding Proteins - genetics | Organ Specificity | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | Phenotype | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Biomarkers, Tumor - genetics | Cell Movement | RNA, Small Interfering - metabolism | RNA | Genes | Colorectal cancer | Stem cells | Genetic aspects | Gene expression | Cancer | Protein binding | Neurosciences | Germ cells | Telomerase reverse transcriptase | Populations | Dyes | Mesenchyme | Oct-4 protein | Gene regulation | Oncology | Spheres | Drug resistance | Tissues | Metastases | DNA-binding protein | CD44 antigen | Cell cycle | Life sciences | Colon | Telomerase | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Antigens | Cell survival | Tumor cells | Invasiveness | Cervix | Survival | Brain research | Molecular modelling | Medical prognosis | Epigenetics | Breast | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Molecular cancer therapeutics, ISSN 1538-8514, 2016, Volume 15, Issue 5, pp. 1082 - 1094
Journal Article
Molecular cell, ISSN 1097-2765, 2009, Volume 35, Issue 6, pp. 856 - 867
Previous studies have suggested that the HIF transcription factors can both activate and inhibit gene expression... 
RNA | CELLCYCLE | SIGNALING | BREAST-CANCER | EPHRIN-A3 | SPECIFICITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYDROXYLATION | MICRORNA TARGET PREDICTIONS | RNAS | HIF-1-ALPHA | HIF-ALPHA | SIGNATURE | RENAL-CELL CARCINOMA | CELL BIOLOGY | Receptor, Fibroblast Growth Factor, Type 5 - metabolism | Neoplasm Transplantation | Up-Regulation | Cell Proliferation | Immunoprecipitation | Pancreatic Neoplasms - metabolism | Oligonucleotide Array Sequence Analysis | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Male | MicroRNAs - metabolism | Receptor, Fibroblast Growth Factor, Type 5 - genetics | Head and Neck Neoplasms - metabolism | Cell Hypoxia | Time Factors | Cell Transformation, Neoplastic - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Base Sequence | Eukaryotic Initiation Factor-2 - metabolism | Transcription, Genetic | Reproducibility of Results | Transduction, Genetic | Response Elements | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Stress, Physiological - genetics | Pancreatic Neoplasms - pathology | Gene Silencing | Gene Expression Profiling - methods | Pancreatic Neoplasms - genetics | Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Homeodomain Proteins - genetics | Head and Neck Neoplasms - pathology | Transcription Factors - metabolism | Animals | Mice, Nude | Cell Line, Tumor | Protein Binding | Head and Neck Neoplasms - genetics | Mice | Cell Transformation, Neoplastic - pathology | Analysis | Pancreatic cancer | Radiation | Genetic research | DNA binding proteins | Gene expression | Hypoxia | microRNA | mir-210 | tumor initiation | gene expression
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2012, Volume 287, Issue 39, pp. 32800 - 32824
...) characterized by high levels of CD44v3 and aldehyde dehydrogenase-1 (ALDH1) expression. These tumor cells also express several stem cell markers... 
SOX2 | IN-VITRO | ISOFORMS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | MARKER NANOG | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | CD44 | BREAST | MICRORNA | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Drug Resistance, Neoplasm | Mouth Neoplasms - metabolism | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Neoplasm Proteins - genetics | Mouth Neoplasms - genetics | Cell Survival - drug effects | Nanog Homeobox Protein | Isoenzymes - genetics | Hyaluronic Acid - genetics | MicroRNAs - biosynthesis | Signal Transduction - genetics | Cisplatin - pharmacology | Retinal Dehydrogenase - genetics | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | RNA, Neoplasm - biosynthesis | Signal Transduction - drug effects | Epigenesis, Genetic - genetics | Hyaluronic Acid - metabolism | Octamer Transcription Factor-3 - metabolism | Mouth Neoplasms - pathology | RNA, Neoplasm - genetics | MicroRNAs - genetics | Hyaluronan | Self-renewal | MicroRNA | Signal Transduction | Extracellular Matrix | Head and Neck Cancer | Differentiation | Cancer Stem Cells
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2008, Volume 283, Issue 48, pp. 33394 - 33405
Micro-RNAs are similar to 21-25-nucleotide-long noncoding RNAs that regulate gene expression primarily at the post-transcriptional level in animals... 
SURVIVAL | C/EBP-ALPHA | MICRORNA FUNCTION | THERAPY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | TUMOR-SUPPRESSOR | TARGETS | DEGRADATION | HUMAN HEPATOCELLULAR CARCINOMAS | EXPRESSION | Lung Neoplasms - drug therapy | Histone Deacetylases - biosynthesis | Antibiotics, Antineoplastic - pharmacology | Caspase 7 - biosynthesis | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Apoptosis - genetics | MicroRNAs - metabolism | Proto-Oncogene Proteins - biosynthesis | RNA, Neoplasm - metabolism | Receptors, Growth Factor - genetics | Caspase 3 - genetics | Caspase 3 - biosynthesis | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Lung Neoplasms - genetics | Forkhead Transcription Factors - biosynthesis | Histone Deacetylases - genetics | Caspase 7 - genetics | Cell Survival | Proto-Oncogene Proteins c-met | Repressor Proteins - genetics | Proto-Oncogene Proteins - genetics | Poly(ADP-ribose) Polymerases - biosynthesis | Down-Regulation - drug effects | Forkhead Transcription Factors - genetics | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | Down-Regulation - genetics | Animals | Poly(ADP-ribose) Polymerases - genetics | Repressor Proteins - biosynthesis | Mice, Nude | Myeloid Cell Leukemia Sequence 1 Protein | Receptors, Growth Factor - biosynthesis | Cell Line, Tumor | Proto-Oncogene Proteins c-pim-1 - genetics | RNA, Neoplasm - genetics | Mice | MicroRNAs - genetics | Poly (ADP-Ribose) Polymerase-1 | Proto-Oncogene Proteins c-bcl-2 - genetics | Proto-Oncogene Proteins c-pim-1 - biosynthesis | Doxorubicin - pharmacology | Cell Movement | RNA-Mediated Regulation and Noncoding Rnas
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 529, Issue 7584, pp. 110 - 114
...). Genomic alterations that remove CTCF-associated boundaries allow aberrant enhancer-gene interactions and alter gene expression (17). Since CTCF binding is methylation... 
MAINTENANCE | METHYLATION | LANDSCAPE | DEMETHYLATION | MULTIDISCIPLINARY SCIENCES | INTEGRATED GENOMIC ANALYSIS | ARCHITECTURE | PHENOTYPE | EXPRESSION | 2-HYDROXYGLUTARATE | PRINCIPLES | Chromatin - metabolism | Up-Regulation | Humans | Glioma - genetics | Base Sequence | Glioma - pathology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Binding Sites | Chromatin - drug effects | Repressor Proteins - metabolism | Oncogenes - genetics | Insulator Elements - genetics | Glioma - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Isocitrate Dehydrogenase - genetics | DNA Methylation - genetics | Down-Regulation - drug effects | Mutation - genetics | CRISPR-Cas Systems - genetics | Insulator Elements - drug effects | Phenotype | Isocitrate Dehydrogenase - chemistry | Receptor, Platelet-Derived Growth Factor alpha - genetics | CCCTC-Binding Factor | CpG Islands - genetics | Protein Binding | Isocitrate Dehydrogenase - metabolism | Cell Proliferation - drug effects | Enhancer Elements, Genetic - genetics | Glutarates - metabolism | Cell Transformation, Neoplastic - drug effects | Chromatin - genetics | DNA Methylation - drug effects | Glioma - drug therapy | Complications and side effects | Care and treatment | Platelet-derived growth factor | Gliomas | Analysis | Influence | Genetic aspects | Research | Methylation | Oncogenes | DNA methylation | Epigenetics | Genomes | Mutation | Gene expression | Binding sites | Deoxyribonucleic acid--DNA | Tumors
Journal Article