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The Journal of cell biology, ISSN 0021-9525, 11/2003, Volume 163, Issue 4, pp. 847 - 857
.... At metastases, migratory fibroblasts sometimes revert to an epithelial phenotype, by a process involving regulation of the E-cadherin-β-catenin complex... 
Cell culture techniques | HCT116 cells | B lymphocytes | Epithelial cells | Cell adhesion | Colorectal cancer | Cadherins | Cultured cells | Slugs | Cells | β-catenin | Tumorigenesis | Slug | ERK | beta-catenin | TUMOR PROGRESSION | CYCLIN D1 | TRANSCRIPTION FACTOR SNAIL | REPRESSES E-CADHERIN | EPITHELIAL-MESENCHYMAL TRANSITIONS | COLON-CARCINOMA CELLS | CELL BIOLOGY | cell adhesion | COLORECTAL-CANCER | ADHESION SYSTEM | RECEPTOR ACTIVATION | tumorigenesis | UP-REGULATION | Cell Line | Epithelial Cells - metabolism | Cadherins - metabolism | beta Catenin | Humans | Cell Transformation, Neoplastic - metabolism | Adherens Junctions - metabolism | Down-Regulation - physiology | Neoplasm Invasiveness - physiopathology | Transcription Factors - metabolism | Animals | Cell Adhesion - physiology | Homeostasis - physiology | Cell Line, Tumor | Cytoskeletal Proteins - metabolism | Signal Transduction - physiology | Trans-Activators - metabolism | Cell Differentiation - physiology | Gene Expression Regulation, Neoplastic - physiology | Snail Family Transcription Factors | Repressor Proteins - metabolism | Mitogen-Activated Protein Kinases - metabolism | Human | Cytoskeletal Proteins | Down-Regulation | Biochemistry, Molecular Biology | Gene Expression Regulation, Neoplastic | Homeostasis | Epithelial Cells | Cellular Biology | Cell Adhesion | Life Sciences | Cell Transformation, Neoplastic | Adherens Junctions | Cell Differentiation | Cancer | cell adhesion; β-catenin; slug; ERK; tumorigenesis
Journal Article
Oncogene, ISSN 1476-5594, 2008, Volume 28, Issue 2, pp. 209 - 218
... ORIGINAL ARTICLE Epigenetic regulation of CD133 and tumorigenicity of CD133 ovarian cancer cells T Baba1,2, PA Convery1, N Matsumura1,2, RS Whitaker1, E Kondoh1,2... 
DNA methylation | Xenograft | Cancer-initiating cell | CD133 | Ovarian cancer | INITIATING CELLS | STEM-CELLS | ovarian cancer | cancer-initiating cell | BIOCHEMISTRY & MOLECULAR BIOLOGY | OVARIAN-CANCER | IDENTIFICATION | TUMORS | CELL BIOLOGY | GLIOBLASTOMA | xenograft | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | AC133 | CARCINOMA | HEMATOPOIETIC STEM | Neoplasm Transplantation | Humans | Neoplasm Proteins - physiology | Ascitic Fluid - pathology | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Peptides - genetics | Gene Expression Profiling | Promoter Regions, Genetic - drug effects | Antigens, CD - genetics | Ovarian Neoplasms - genetics | DNA Methylation | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Cell Division | Neoplastic Stem Cells - pathology | Female | Ovarian Neoplasms - metabolism | Carcinoma - pathology | Neoplasm Proteins - genetics | Glycoproteins - genetics | Peptides - physiology | Cisplatin - pharmacology | AC133 Antigen | Glycoproteins - physiology | Drug Resistance, Neoplasm - genetics | Animals | Epigenesis, Genetic - genetics | Mice, Nude | Cell Line, Tumor | Antigens, CD - physiology | Carcinoma - genetics | Mice | Mice, Inbred BALB C | Protein Processing, Post-Translational | Carcinoma - metabolism | Histones - metabolism | Chemotherapy | Genetic aspects | Research | Genetic regulation | Drug therapy | Health aspects | Cancer | Oncology | Cellular biology | Gene expression | Stem cells
Journal Article
Journal Article
Nature genetics, ISSN 1546-1718, 2013, Volume 45, Issue 7, pp. 739 - 746
Journal Article
Oncogene, ISSN 0950-9232, 08/2010, Volume 29, Issue 31, pp. 4436 - 4448
..., resulting, in turn, in the down-regulation of epithelial markers. Re-expression... 
SNAI1 | StarD10 | breast cancer cell invasion | Nectin-1 | EMT | miR-661 | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR SNAIL | PHENOTYPE | E-CADHERIN | MIR-200 FAMILY | REPRESSORS ZEB1 | SIGNATURE | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | RECEPTORS | MICRORNA | Cell Adhesion Molecules - genetics | Epithelial Cells - metabolism | Oligonucleotide Array Sequence Analysis | Humans | MicroRNAs - metabolism | Gene Expression Profiling | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Nectins | Breast Neoplasms - metabolism | Epithelial Cells - physiology | Gene Expression - physiology | Female | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Gene Expression Regulation, Neoplastic - physiology | Snail Family Transcription Factors | Mesenchymal Stromal Cells - physiology | Transcription Factors - physiology | Cell Dedifferentiation - physiology | Cell Dedifferentiation - drug effects | Neoplasm Invasiveness | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Mesenchymal Stromal Cells - metabolism | Phosphoproteins - genetics | Transcription Factors - genetics | Cell Adhesion Molecules - metabolism | Transcription Factors - metabolism | Breast Neoplasms - genetics | Validation Studies as Topic | Breast Neoplasms - pathology | MicroRNAs - genetics | MicroRNAs - physiology | Cell Dedifferentiation - genetics | Messenger RNA | Physiological aspects | Development and progression | Genetic aspects | Breast cancer | Metastasis | Research | Risk factors | Proteins | Gene expression | Cell adhesion & migration | Life Sciences | Biochemistry, Molecular Biology | pathology | Cell Adhesion Molecules | Gene Expression Regulation, Neoplastic | Cell Dedifferentiation | Breast Neoplasms | Mesenchymal Stem Cells | genetics | Phosphoproteins | pharmacology | Gene Expression | physiology | drug effects | Epithelial Cells | MicroRNAs | metabolism | RNA, Small Interfering | Transcription Factors | RNA, Messenger
Journal Article
Oncogene, ISSN 1476-5594, 2012, Volume 32, Issue 13, pp. 1616 - 1625
...) and overexpression is correlated with poor survival for breast, colon and liver cancer patients. In this study, we show that HOTAIR expression is increased in pancreatic tumors compared with non-tumor tissue and is associated with more aggressive tumors... 
HOTAIR | Cell cycle progression | Pro-oncogenic | Invasion | Prognostic | ACTIVATION | pro-oncogenic | PROTEIN | CHROMATIN | DUCTAL ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | CELL BIOLOGY | prognostic | INTERFERON | invasion | LONG NONCODING RNA | REPRESSION | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | cell cycle progression | MIRNAS | Prognosis | Pancreatic Neoplasms - diagnosis | Humans | Transplantation, Heterologous | RNA, Long Noncoding - physiology | Carcinoma, Pancreatic Ductal - genetics | Gene Knockdown Techniques | Cell Transformation, Neoplastic - genetics | Biomarkers, Tumor - metabolism | Carcinoma, Pancreatic Ductal - diagnosis | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - physiology | Pancreatic Neoplasms - pathology | RNA, Small Interfering - pharmacology | Pancreatic Neoplasms - genetics | Biomarkers, Tumor - physiology | RNA, Long Noncoding - genetics | Carcinoma, Pancreatic Ductal - pathology | Animals | Cell Line, Tumor | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Mice | Cell Transformation, Neoplastic - drug effects | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | RNA | Pancreatic cancer | Physiological aspects | Genetic aspects | Research | Gene expression | Oncology | Cell cycle | Tumors
Journal Article
Cancer research (Chicago, Ill.), ISSN 1538-7445, 2018, Volume 78, Issue 15, pp. 4191 - 4202
...)-1-dependent regulation of mitogen-activated protein kinase (MAPK) signaling pathways contributes to chemotherapy-induced BCSC enrichment... 
POSTTRANSCRIPTIONAL REGULATION | MEK | THERAPY | ONCOLOGY | DUSP6/MKP-3 | DOXORUBICIN | TRANSCRIPTIONAL REPRESSION | RESISTANCE | PHOSPHATASE-1 | PHASE-I | HYPOXIA-INDUCIBLE FACTORS | Phosphorylation - physiology | Transcription, Genetic - drug effects | MAP Kinase Signaling System - physiology | Dual-Specificity Phosphatases - metabolism | Neoplastic Stem Cells - drug effects | Humans | RNA, Messenger - metabolism | Triple Negative Breast Neoplasms - drug therapy | Hypoxia-Inducible Factor 1 - metabolism | Forkhead Box Protein O3 - metabolism | Transcription, Genetic - physiology | MAP Kinase Signaling System - drug effects | Neoplastic Stem Cells - metabolism | Signal Transduction - drug effects | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Female | Signal Transduction - physiology | Antineoplastic Agents - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Gene Expression Regulation, Neoplastic - physiology | Mitogen-Activated Protein Kinase Phosphatases - metabolism | Enrichment | Phosphorylation | Hypoxia-inducible factor 1 | Switches | Cytotoxicity | Activation | Kinases | Metastases | Proteins | Signal transduction | Pathways | KLF4 protein | Inhibition | FOXO3 protein | Hypoxia-inducible factors | Phenotypes | Extracellular signal-regulated kinase | MAP kinase | Breast cancer | Ribonucleic acid--RNA | Survival | Signaling | Chemotherapy | RNA-binding protein | Inhibitors | Protein kinase | Medical prognosis | Stem cells | Hypoxia | Pluripotency | Cancer | Tumors
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2007, Volume 128, Issue 2, pp. 257 - 267
Assembly of the eIF4E/eIF4G complex has a central role in the regulation of gene expression at the level of translation initiation... 
FACTOR EIF-4E | STIMULATION | MALIGNANT-TRANSFORMATION | PHOSPHORYLATION | CAP-DEPENDENT TRANSLATION | MESSENGER-RNA CAP | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING PROTEIN EIF4E | REGULATORS | MTOR INHIBITORS | CANCER | CELL BIOLOGY | Protein Binding - genetics | Humans | Nitro Compounds - chemistry | Eukaryotic Initiation Factor-4E - metabolism | Peptide Fragments - pharmacology | Eukaryotic Initiation Factor-4G - drug effects | Eukaryotic Initiation Factor-4G - metabolism | Fluorescence Polarization Immunoassay - methods | Thiazoles - isolation & purification | Antineoplastic Agents - isolation & purification | Cell Transformation, Neoplastic - genetics | Protein Binding - drug effects | Nitro Compounds - pharmacology | Antineoplastic Agents - pharmacology | Protein Biosynthesis - genetics | Gene Expression Regulation, Neoplastic - drug effects | Eukaryotic Initiation Factor-4E - genetics | Peptide Fragments - genetics | Gene Expression Regulation, Neoplastic - genetics | RNA, Messenger - drug effects | Drug Evaluation, Preclinical - methods | Oncogenes - genetics | Peptide Fragments - metabolism | Jurkat Cells | RNA, Messenger - genetics | Hydrazones | Models, Molecular | Antineoplastic Agents - chemistry | Cell Transformation, Neoplastic - metabolism | Eukaryotic Initiation Factor-4E - drug effects | Nitro Compounds - isolation & purification | Animals | Cell Line, Tumor | Oncogenes - drug effects | Feedback, Physiological - drug effects | Protein Biosynthesis - drug effects | Thiazoles - chemistry | Eukaryotic Initiation Factor-4G - genetics | Mice | Thiazoles - pharmacology | Cell Transformation, Neoplastic - drug effects | Feedback, Physiological - physiology | Cell Line, Transformed | Peptide Fragments | Protein Biosynthesis | Gene Expression Regulation, Neoplastic | Eukaryotic Initiation Factor-4E | Chemical Sciences | Fluorescence Polarization Immunoassay | Life Sciences | Thiazoles | Drug Evaluation, Preclinical | Feedback, Biochemical | Oncogenes | Analytical chemistry | Biochemistry, Molecular Biology | Antineoplastic Agents | Organic chemistry | Eukaryotic Initiation Factor-4G | Cell Transformation, Neoplastic | Nitro Compounds | Protein Binding | RNA, Messenger
Journal Article