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1. Full Text The Ngal reporter mouse detects the response of the kidney to injury in real time
by Lin, Chyuan-Sheng and Forster, Catherine S and Paragas, Neal and Deng, Shi-Xian and Viltard, Melanie and Kulkarni, Ritwij and Samstein, Benjamin and Mori, Kiyoshi and Landry, Donald W and Barasch, Jonathan and Yu, Wenqiang and Devarajan, Prasad and D'Agati, Vivette and Qiu, Andong and Kalandadze, Avtandil and Gong, Gangli and Zhang, Qingyin and Ratner, Adam J and Schmidt-Ott, Kai M and Liu, Yidong
Nature Medicine, ISSN 1078-8956, 02/2011, Volume 17, Issue 2, pp. 216 - 222
Many proteins have been proposed to act as surrogate markers of organ damage, yet for many candidates the essential biomarker characteristics that link the... 
MEDICINE, RESEARCH & EXPERIMENTAL | RAT-KIDNEY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | INNATE IMMUNE-RESPONSE | GELATINASE-ASSOCIATED LIPOCALIN | CELL BIOLOGY | EARLY URINARY BIOMARKER | SERUM CREATININE | GLOMERULAR-FILTRATION-RATE | EPITHELIAL-CELLS | CARDIAC-SURGERY | RENAL INJURY | Oncogene Proteins - blood | Oncogene Proteins - genetics | Kidney - pathology | Male | Acute-Phase Proteins - genetics | Mice, Mutant Strains - physiology | Mice, Mutant Strains - genetics | Oncogene Proteins - physiology | Acute-Phase Proteins - physiology | Dose-Response Relationship, Drug | Kidney - metabolism | Gentamicins - pharmacology | Lipocalins - blood | Lipocalins - genetics | Lipocalins - physiology | Female | Kidney - drug effects | Gene Expression Regulation - genetics | Gene Expression Regulation - physiology | Biomarkers - blood | Cisplatin - pharmacology | Kidney - injuries | Genes, Reporter - physiology | Animals | Lipocalin-2 | Lipid A - pharmacology | Genes, Reporter - drug effects | Mice | Cell receptors | Kidneys | Luciferase | Genes | Physiological aspects | Genetic aspects | Research | Injuries | Proteins | Biomarkers | Kidney diseases | Cellular biology | Rodents
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2. Full Text In vitro endocrine disruption potential of organophosphate flame retardants via human nuclear receptors
by Kojima, Hiroyuki and Takeuchi, Shinji and Itoh, Toshihiro and Iida, Mitsuru and Kobayashi, Satoshi and Yoshida, Takahiko
Toxicology, ISSN 0300-483X, 2013, Volume 314, Issue 1, pp. 76 - 83
Highlights • Nuclear receptor activities of OPFRs were studied by in vitro reporter gene assays. • TPhP and TCP acted as ERα/β and PXR agonists as well as AR... 
Emergency | Human | Flame retardant | Reporter gene assay | Nuclear receptor | Organophosphate | XENOBIOTIC RECEPTOR | POLYBROMINATED DIPHENYL ETHERS | PREGNANE-X-RECEPTOR | PHOSPHATE | ANDROGEN | ESTROGEN | REPORTER GENE ASSAYS | 200 PESTICIDES | PHARMACOLOGY & PHARMACY | TOXICOLOGY | CONGENERS | PLASTICIZERS | Transcription, Genetic - drug effects | Cricetinae | Cricetulus | Receptors, Cytoplasmic and Nuclear - drug effects | Humans | Cercopithecus aethiops | Receptors, Cytoplasmic and Nuclear - agonists | Endocrine Disruptors | Dose-Response Relationship, Drug | Flame Retardants - toxicity | Animals | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Plasmids - genetics | beta-Galactosidase - metabolism | Genes, Reporter - drug effects | Organophosphorus Compounds - toxicity | COS Cells | CHO Cells | Endocrine disruptors | Fireproofing agents | Index Medicus | Phosphates | Receptors | Houses | TCP (protocol) | Flame retardants | Textiles | Organophosphates
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3. Full Text Binding Selectivity of Abaloparatide for PTH-Type-1-Receptor Conformations and Effects on Downstream Signaling
by Hattersley, Gary and Dean, Thomas and Corbin, Braden A and Bahar, Hila and Gardella, Thomas J
Endocrinology, ISSN 0013-7227, 01/2016, Volume 157, Issue 1, pp. 141 - 149
The PTH receptor type 1 (PTHR1) mediates the actions of two endogenous polypeptide ligands, PTH and PTHrP, and thereby plays key roles in bone biology. Based... 
(PTH)/PTH-RELATED PROTEIN-RECEPTOR | PATHWAYS | STATES | LIGAND-BINDING | BIASED AGONISM | ENDOCRINOLOGY & METABOLISM | ERK1/2 ACTIVATION | PARATHYROID-HORMONE PTH | MECHANISMS | OSTEOPOROSIS | CAMP | Receptor, Parathyroid Hormone, Type 1 - chemistry | Receptor, Parathyroid Hormone, Type 1 - metabolism | Humans | Peptide Fragments - pharmacology | Guanosine 5'-O-(3-Thiotriphosphate) - chemistry | HEK293 Cells | Cell Membrane - metabolism | Bone Density Conservation Agents - metabolism | Cyclic AMP - metabolism | Binding Sites - drug effects | Cell Membrane - drug effects | Bone Density Conservation Agents - chemistry | Recombinant Proteins - metabolism | Peptide Fragments - metabolism | Second Messenger Systems - drug effects | Models, Molecular | Recombinant Proteins - chemistry | Parathyroid Hormone-Related Protein - chemistry | Parathyroid Hormone-Related Protein - metabolism | Bone Density Conservation Agents - pharmacology | Parathyroid Hormone-Related Protein - pharmacology | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Peptide Fragments - chemistry | MAP Kinase Signaling System - drug effects | Receptor, Parathyroid Hormone, Type 1 - genetics | Genes, Reporter - drug effects | Ligands | Protein Conformation | Kinetics | GTP-Binding Proteins - metabolism | Original Research
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4. Full Text Glyphosate-based herbicides are toxic and endocrine disruptors in human cell lines
by Gasnier, Céline and Dumont, Coralie and Benachour, Nora and Clair, Emilie and Chagnon, Marie-Christine and Séralini, Gilles-Eric
Toxicology, ISSN 0300-483X, 2009, Volume 262, Issue 3, pp. 184 - 191
Abstract Glyphosate-based herbicides are the most widely used across the world; they are commercialized in different formulations. Their residues are frequent... 
Emergency | HepG2 | Endocrine disruptor | Sexual steroids | Roundup | Glyphosate | ANDROGEN RECEPTOR | PESTICIDE EXPOSURE | HUMAN PLACENTAL MICROSOMES | COMET ASSAY | DNA-DAMAGE | ENVIRONMENTAL CHEMICALS | IN-VITRO | GENE-EXPRESSION | PREGNANT RATS | PHARMACOLOGY & PHARMACY | BISPHENOL-A | TOXICOLOGY | Glycine - analogs & derivatives | Transcription, Genetic - drug effects | Humans | Receptors, Androgen - metabolism | Aromatase - metabolism | Male | RNA, Messenger - metabolism | Estrogen Receptor beta - drug effects | Dose-Response Relationship, Drug | Estrogen Receptor beta - metabolism | Herbicides - administration & dosage | Aromatase - genetics | Glycine - toxicity | Receptors, Androgen - drug effects | Estrogen Receptor alpha - metabolism | Toxicity Tests - methods | Glycine - administration & dosage | DNA Damage - drug effects | Herbicides - toxicity | Comet Assay | Endocrine Disruptors - administration & dosage | Endocrine Disruptors - toxicity | Estrogen Receptor alpha - drug effects | Aromatase - drug effects | Adolescent | Cell Line, Tumor | Genes, Reporter - drug effects | Pesticide Residues - toxicity | Endocrine disruptors | Herbicides | Messenger RNA | Estrogen | Index Medicus
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5. Full Text Discovery of Novel Cell Wall-Active Compounds Using P-ywaC, a Sensitive Reporter of Cell Wall Stress, in the Model Gram-Positive Bacterium Bacillus subtilis
by Czarny, TL and Perri, AL and French, S and Brown, ED
ANTIMICROBIAL AGENTS AND CHEMOTHERAPY, ISSN 0066-4804, 06/2014, Volume 58, Issue 6, pp. 3261 - 3269
The emergence of antibiotic resistance in recent years has radically reduced the clinical efficacy of many antibacterial treatments and now poses a significant... 
SYSTEM | VANCOMYCIN | ENTEROCOCCUS | BETA-LACTAM ANTIBIOTICS | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | INDUCTION | INHIBITORS | LIQUID-MEDIUM | L-FORMS | EXPRESSION | SIGMA(W) | Transcription, Genetic - drug effects | Permeability - drug effects | Genes, Bacterial - genetics | Ligases - genetics | Cell Wall - genetics | Genes, Reporter - genetics | Bacillus subtilis - genetics | Dose-Response Relationship, Drug | Microbial Sensitivity Tests | Cell Wall - drug effects | High-Throughput Screening Assays | Small Molecule Libraries | Genes, Reporter - drug effects | Anti-Bacterial Agents - pharmacology | Genes, Bacterial - drug effects | Cell Membrane - drug effects | Bacillus subtilis - drug effects
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6. Full Text Fingerprinting of neurotoxic compounds using a mouse embryonic stem cell dual luminescence reporter assay
by Colaianna, Marilena and Colaianna, Marilena and Ilmjärv, Sten and Ilmjärv, Sten and Peterson, Hedi and Peterson, Hedi and Kern, Ilse and Kern, Ilse and Julien, Stephanie and Julien, Stephanie and Baquié, Mathurin and Baquié, Mathurin and Pallocca, Giorgia and Pallocca, Giorgia and Bosgra, Sieto and Bosgra, Sieto and Sachinidis, Agapios and Sachinidis, Agapios and Hengstler, Jan G and Hengstler, Jan G and Leist, Marcel and Leist, Marcel and Krause, Karl-Heinz and Krause, Karl-Heinz
Archives of Toxicology, ISSN 0340-5761, 1/2017, Volume 91, Issue 1, pp. 365 - 391
Identification of neurotoxic drugs and environmental chemicals is an important challenge. However, only few tools to address this topic are available. The aim... 
Biomedicine, general | Neurotoxicity | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | Neuronal differentiation | Pharmacology/Toxicology | In vitro screening | Mouse embryonic stem cells | Neuroactivity | HISTONE DEACETYLASE INHIBITOR | DEVELOPMENTAL NEUROTOXICITY | COLONY-STIMULATING FACTOR | FETAL VALPROATE SYNDROME | METHYL MERCURY | LUNG-CANCER | IN-VITRO | MULTIPLE-SCLEROSIS | TOXICOLOGY | PHASE-I | Drugs, Investigational - adverse effects | Coculture Techniques | Mouse Embryonic Stem Cells - cytology | Neurons - cytology | Tubulin - genetics | Promoter Regions, Genetic - drug effects | Mouse Embryonic Stem Cells - drug effects | Mouse Embryonic Stem Cells - metabolism | Tubulin - metabolism | Stromal Cells - drug effects | Cell Differentiation | Neurons - metabolism | Environmental Pollutants - toxicity | Peptide Elongation Factor 1 - genetics | Drug Evaluation, Preclinical | Neurons - drug effects | Biomarkers - metabolism | Cell Line | Cell Survival - drug effects | Environmental Monitoring | Pluripotent Stem Cells - cytology | Stromal Cells - metabolism | Neurotoxins - toxicity | Pluripotent Stem Cells - metabolism | Animals | Pluripotent Stem Cells - drug effects | Cell Communication - drug effects | Genes, Reporter - drug effects | Luminescent Proteins - genetics | Mice | Peptide Elongation Factor 1 - metabolism | Stromal Cells - cytology | Luminescent Proteins - metabolism | Embryonic stem cells | Analysis | Journalists | Neurophysiology | Rodents | Stem cells | Luminescence | Drug therapy | Embryos | Chemicals | Index Medicus | In vitro Systems
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7. Full Text Glyphosate induces human breast cancer cells growth via estrogen receptors
by Thongprakaisang, Siriporn and Thiantanawat, Apinya and Rangkadilok, Nuchanart and Suriyo, Tawit and Satayavivad, Jutamaad
Food and Chemical Toxicology, ISSN 0278-6915, 09/2013, Volume 59, pp. 129 - 136
Glyphosate is an active ingredient of the most widely used herbicide and it is believed to be less toxic than other pesticides. However, several recent studies... 
T47D-KBluc | Human breast cancer | T47D | Glyphosate | Genistein | Estrogenic effect | ER-BETA | FOOD SCIENCE & TECHNOLOGY | HUMAN HEALTH | ROUNDUP | LINES | ISOFLAVONES | HERBICIDE GLYPHOSATE | PHYTO-ESTROGENS | TOXICOLOGY | FARM FAMILY EXPOSURE | PLACENTAL CELLS | Neoplasms, Hormone-Dependent - metabolism | Glycine - analogs & derivatives | Herbicides - antagonists & inhibitors | Humans | Transcriptional Activation - drug effects | Estrogen Antagonists - pharmacology | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Breast Neoplasms - metabolism | Estrogen Receptor beta - metabolism | Neoplasms, Hormone-Dependent - chemically induced | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Estrogen Receptor beta - genetics | Estrogens - chemistry | Glycine - toxicity | Estrogen Receptor alpha - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Estrogen Receptor beta - antagonists & inhibitors | Cell Survival - drug effects | Endocrine Disruptors - chemistry | Estrogen Receptor alpha - antagonists & inhibitors | Estrogens - toxicity | Herbicides - toxicity | Endocrine Disruptors - toxicity | Drug Synergism | Response Elements - drug effects | Estrogen Receptor alpha - genetics | Glycine - antagonists & inhibitors | Neoplasm Proteins - agonists | Genistein - pharmacology | Cell Line, Tumor | Genes, Reporter - drug effects | Cell Proliferation - drug effects | Breast Neoplasms - chemically induced | Care and treatment | Breast cancer | Estrogen | Cancer | Index Medicus
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8. Full Text Profiling of the Tox21 10K compound library for agonists and antagonists of the estrogen receptor alpha signaling pathway
by Huang, Ruili and Sakamuru, Srilatha and Martin, Matt T and Reif, David M and Judson, Richard S and Houck, Keith A and Casey, Warren and Hsieh, Jui-Hua and Shockley, Keith R and Ceger, Patricia and Fostel, Jennifer and Witt, Kristine L and Tong, Weida and Rotroff, Daniel M and Zhao, Tongan and Shinn, Paul and Simeonov, Anton and Dix, David J and Austin, Christopher P and Kavlock, Robert J and Tice, Raymond R and Xia, Menghang
Scientific Reports, ISSN 2045-2322, 07/2014, Volume 4, Issue 1, p. 5664
The U.S. Tox21 program has screened a library of approximately 10,000 (10K) environmental chemicals and drugs in three independent runs for estrogen receptor... 
BREAST-CANCER CELLS | ANTIESTROGENIC ACTIVITY | CHEMICALS | MULTIDISCIPLINARY SCIENCES | HUMAN HEALTH | IN-VITRO ASSAYS | PYRETHROID INSECTICIDES | MECHANISMS | IONIC LIQUIDS | PHYTOESTROGENS | ANDROGENIC ACTIVITY | Cell Line | Small Molecule Libraries - pharmacology | Estrogen Receptor alpha - agonists | Signal Transduction - drug effects | Humans | Protein Binding - drug effects | HEK293 Cells | Estrogen Receptor alpha - antagonists & inhibitors | Genes, Reporter - drug effects | Ligands | Structure-Activity Relationship | High-Throughput Screening Assays - methods | Signal transduction | Reporter gene | Estrogen | Cell lines | Estrogen receptors | High-throughput screening | Structure-activity relationships
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9. Full Text The progeny of Arabidopsis thaliana plants exposed to salt exhibit changes in DNA methylation, histone modifications and gene expression
by Bilichak, Andriy and Ilnystkyy, Yaroslav and Hollunder, Jens and Kovalchuk, Igor
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e30515
Plants are able to acclimate to new growth conditions on a relatively short time-scale. Recently, we showed that the progeny of plants exposed to various... 
Arabidopsis - physiology | Salt-Tolerant Plants - drug effects | Sodium Chloride - pharmacology | Protein Processing, Post-Translational - genetics | Promoter Regions, Genetic - drug effects | Reproduction, Asexual - physiology | Salt-Tolerance - drug effects | Dose-Response Relationship, Drug | Protein Processing, Post-Translational - drug effects | Plants, Genetically Modified | Salt-Tolerance - genetics | DNA Methylation - physiology | Stress, Physiological - physiology | Salt-Tolerant Plants - metabolism | Arabidopsis - drug effects | Breeding | Stress, Physiological - genetics | Gene Expression Regulation, Plant - genetics | DNA Methylation - genetics | Arabidopsis - metabolism | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Genes, Reporter - drug effects | Histones - metabolism | DNA Methylation - drug effects | Cluster Analysis | Salt-Tolerant Plants - genetics | Arabidopsis thaliana | Chromatin | DNA | Analysis | Genomics | Histones | Genetic research | Genomes | DNA binding proteins | Gene expression | Methylation | DNA structure | Growth conditions | Regulators | Salts | Introns | Exons | Exposure | Sodium chloride | Gene sequencing | Progeny | Sodium | DNA methylation | Flowers & plants | Mutation | Deoxyribonucleic acid--DNA | Deoxyribonucleic acid
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10. Full Text Effects of interferon alpha on vascular endothelial growth factor gene transcription and tumor angiogenesis
by von Marschall, Zofia and Scholz, Arne and Cramer, Thorsten and Schäfer, Georgia and Schirner, Michael and Öberg, Kjell and Wiedenmann, Bertram and Höcker, Michael and Rosewicz, Stefan and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Institutionen för medicinska vetenskaper
Journal of the National Cancer Institute, ISSN 0027-8874, 03/2003, Volume 95, Issue 6, pp. 437 - 448
Background: Interferon alpha (IFN-alpha) has antiangiogenic activity, although the underlying mechanism of action is unclear. Because human neuroendocrine (NE)... 
CELLS | INHIBITION | THERAPY | ONCOLOGY | SP1 | CLINICAL-APPLICATIONS | PROTEIN-KINASE | DOWN-REGULATION | EXPRESSION | CANCER | BINDING | Immunohistochemistry | Interferon-alpha - pharmacology | Transcription, Genetic - drug effects | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Luciferases - metabolism | Vascular Endothelial Growth Factors | Receptors, Vascular Endothelial Growth Factor - drug effects | Transplantation, Heterologous | Pregnancy Proteins - metabolism | Promoter Regions, Genetic - drug effects | RNA, Messenger - metabolism | Biomarkers, Tumor | Intercellular Signaling Peptides and Proteins - metabolism | Lymphokines - genetics | Receptors, Vascular Endothelial Growth Factor - metabolism | Time Factors | Lymphokines - metabolism | Antineoplastic Agents - pharmacology | Electrophoretic Mobility Shift Assay | Gene Expression Regulation, Neoplastic - drug effects | Receptors, Vascular Endothelial Growth Factor - genetics | Tumor Cells, Cultured | Lymphokines - drug effects | Enzyme-Linked Immunosorbent Assay | Neuroendocrine Tumors - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Angiogenesis Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Animals | Mice, Nude | Neovascularization, Pathologic - drug therapy | Endothelial Growth Factors - genetics | Neuroendocrine Tumors - drug therapy | Genes, Reporter - drug effects | Mice | Neovascularization, Pathologic - metabolism | Neuroendocrine Tumors - blood supply | Receptors, Immunologic - metabolism | Measurement | Physiological aspects | Vascular endothelial growth factor | Interferon alpha | Interferon-alpha/pharmacology | Endothelial Growth Factors/genetics/metabolism | Tumor Cells; Cultured | Tumor Markers; Biological | Neuroendocrine Tumors/blood supply/drug therapy/metabolism | Antineoplastic Agents/pharmacology | Pregnancy Proteins/metabolism | Luciferases/metabolism | Promoter Regions (Genetics)/drug effects | Research Support; Non-U.S. Gov't | Genes; Reporter/drug effects | Mice; Nude | Gene Expression Regulation; Neoplastic/drug effects | RNA; Messenger/metabolism | Receptors; Vascular Endothelial Growth Factor/drug effects/genetics/metabolism | Receptors; Immunologic/metabolism | Intercellular Signaling Peptides and Proteins/genetics/metabolism | Neovascularization; Pathologic/drug therapy/metabolism | Angiogenesis Inhibitors/pharmacology | Lymphokines/drug effects/genetics/metabolism | Transcription; Genetic/drug effects | Transplantation; Heterologous
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