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Cellular Signalling, ISSN 0898-6568, 02/2014, Volume 26, Issue 2, pp. 240 - 252
In late stages of cancer, TGF-β promotes the metastasis process by enhancing the invasiveness of cancer cells and inducing the epithelial-to-mesenchymal... 
Cell proliferation | Smad2 | TGF-β | CDC 25A | NFAT | TRANSCRIPTION FACTORS | PHOSPHATASE | C-MYC | CELL BIOLOGY | HUMAN COLORECTAL-CARCINOMA | TGF-beta | GENE | MESENCHYMAL TRANSITIONS | NUCLEAR FACTOR | EXPRESSION | T-CELLS | CYCLIN | Cyclin D1 - metabolism | Cadherins - metabolism | Cyclin-Dependent Kinase 4 - genetics | Humans | Epithelial-Mesenchymal Transition - drug effects | Smad2 Protein - antagonists & inhibitors | Cyclin E - genetics | Breast Neoplasms - metabolism | cdc25 Phosphatases - antagonists & inhibitors | Smad2 Protein - genetics | Female | cdc25 Phosphatases - metabolism | Smad2 Protein - pharmacology | Cyclin-Dependent Kinase 2 - metabolism | NFATC Transcription Factors - metabolism | Smad2 Protein - metabolism | Cyclin-Dependent Kinase 2 - genetics | cdc25 Phosphatases - genetics | Cyclin-Dependent Kinase 4 - metabolism | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Cyclin D1 - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Cell Proliferation - drug effects | Cyclin E - metabolism | NFATC Transcription Factors - antagonists & inhibitors | Transforming Growth Factor beta - metabolism | Apoptosis | NFATC Transcription Factors - genetics | RNA, Small Interfering - metabolism | Progressions | Regulators | Control | Breast | Activation | Switching | Tumors | Cancer
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 4/2003, Volume 161, Issue 2, pp. 281 - 294
The proper segregation of sister chromatids in mitosis depends on bipolar attachment of all chromosomes to the mitotic spindle. We have identified the small... 
Mitosis | Kinetochores | Chromatids | Microtubules | HeLa cells | Cellular immunity | Chromosomes | Anaphase | Cells | Mitotic spindle apparatus | Chemical biology | Spindle assembly checkpoint | Chromosome segregation | KINASE-ACTIVITY | MAD2 | BUDDING YEAST | spindle assembly checkpoint | TENSION | G/M TRANSITION | mitosis | HISTONE H3 PHOSPHORYLATION | MAMMALIAN-CELLS | CELL BIOLOGY | kinetochores | chromosome segregation | chemical biology | VERTEBRATE SOMATIC-CELLS | Cell Cycle Proteins - drug effects | Protein Kinases - metabolism | RNA, Small Interfering - genetics | Paclitaxel - pharmacology | Humans | Nocodazole - pharmacology | Chromosome Segregation - drug effects | Mitosis - genetics | Aurora Kinase B | Microtubules - drug effects | Spindle Apparatus - genetics | Cell Cycle Proteins - genetics | Genes, cdc - physiology | Indoles - pharmacology | Kinetochores - enzymology | Protein-Serine-Threonine Kinases - metabolism | Aneugens - pharmacology | Eukaryotic Cells - enzymology | Polyploidy | Protein Kinases - drug effects | Separase | Protein-Serine-Threonine Kinases - genetics | Genes, cdc - drug effects | Aurora Kinases | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Anaphase - drug effects | Phenotype | Anaphase - genetics | Animals | Eukaryotic Cells - drug effects | Mitosis - drug effects | Microtubules - genetics | Microtubules - enzymology | Protein-Serine-Threonine Kinases - drug effects | Eukaryotic Cells - cytology | Kinetochores - drug effects | Spindle Apparatus - drug effects | HeLa Cells | Thiones - pharmacology | Endopeptidases | Chromosome Segregation - genetics | Spindle Apparatus - enzymology | Research | mitosis; chromosome segregation; kinetochores; spindle assembly checkpoint; chemical biology
Journal Article
Immunity, ISSN 1074-7613, 02/2016, Volume 44, Issue 2, pp. 343 - 354
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 4/2003, Volume 161, Issue 2, pp. 267 - 280
The Aurora/Ipl1 family of protein kinases plays multiple roles in mitosis and cytokinesis. Here, we describe ZM447439, a novel selective Aurora kinase... 
Mitotic index | Mitosis | Kinetochores | Microtubules | DNA | Cell lines | HeLa cells | Cultured cells | Chromosomes | Cells | Chemical biology | Aneuploidy | Spindle checkpoint | ZM447439 | KINASE-ACTIVITY | LOCALIZATION | BUDDING YEAST | PHOSPHORYLATION | TENSION | mitosis | MAMMALIAN-CELLS | CELL BIOLOGY | chemical biology | CENTROSOME AMPLIFICATION | HISTONE H3 | aneuploidy | spindle checkpoint | Protein Kinases - metabolism | Protein Kinases - genetics | Paclitaxel - pharmacology | DNA Replication - drug effects | Humans | Chromosome Segregation - drug effects | Mitosis - genetics | Tumor Suppressor Protein p53 - genetics | Aurora Kinase B | Spindle Apparatus - genetics | Eukaryotic Cells - ultrastructure | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Benzamides - pharmacology | Mad2 Proteins | Phosphorylation - drug effects | Calcium-Binding Proteins - metabolism | Eukaryotic Cells - enzymology | Chromosomal Proteins, Non-Histone - metabolism | Kinetochores - metabolism | Enzyme Inhibitors - pharmacology | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Genes, cdc - drug effects | Aurora Kinases | Tumor Suppressor Protein p53 - drug effects | Anaphase - drug effects | Chromosomal Proteins, Non-Histone - genetics | Anaphase - genetics | Eukaryotic Cells - drug effects | Mitosis - drug effects | DNA Replication - genetics | Repressor Proteins - drug effects | Kinetochores - drug effects | Spindle Apparatus - drug effects | HeLa Cells | Quinazolines - pharmacology | Cell Cycle Proteins | Calcium-Binding Proteins - genetics | Chromosome Segregation - genetics | Research | Cytokinesis | Protein kinases | mitosis; spindle checkpoint; chemical biology; aneuploidy; ZM447439
Journal Article
Cell, ISSN 0092-8674, 2002, Volume 109, Issue 4, pp. 459 - 472
Fanconi anemia (FA) and ataxia telangiectasia (AT) are clinically distinct autosomal recessive disorders characterized by spontaneous chromosome breakage and... 
TARGETED DISRUPTION | CHECKPOINT PATHWAY | DNA-DAMAGE RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | S-PHASE | ATM-DEPENDENT PHOSPHORYLATION | NUCLEAR-COMPLEX | NIJMEGEN-BREAKAGE-SYNDROME | IONIZING-RADIATION | X-IRRADIATION | MAMMALIAN-CELLS | CELL BIOLOGY | Nucleic Acid Synthesis Inhibitors - pharmacology | Fanconi Anemia - metabolism | Humans | Ubiquitin - metabolism | Phosphoserine - antagonists & inhibitors | G1 Phase - drug effects | G2 Phase - drug effects | Genes, cdc - radiation effects | Cell Nucleus - metabolism | Cell Nucleus - radiation effects | Nuclear Proteins - deficiency | Fanconi Anemia - genetics | Signal Transduction - radiation effects | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins | Radiation, Ionizing | Ataxia Telangiectasia - physiopathology | Fanconi Anemia Complementation Group D2 Protein | G2 Phase - radiation effects | Mutation - radiation effects | Protein-Serine-Threonine Kinases - genetics | Genes, cdc - drug effects | Ataxia Telangiectasia - metabolism | Signal Transduction - genetics | Ubiquitin - genetics | Ataxia Telangiectasia Mutated Proteins | Phosphoserine - metabolism | S Phase - genetics | Mitomycin - pharmacology | Phosphorylation - radiation effects | S Phase - radiation effects | Mutation - drug effects | G1 Phase - radiation effects | Signal Transduction - drug effects | Fanconi Anemia - physiopathology | Ataxia Telangiectasia - genetics | S Phase - drug effects | HeLa Cells | Cell Nucleus - drug effects | Cell Cycle Proteins | Cell Line, Transformed | Tumor Suppressor Proteins | Cell research | Ionizing radiation | Mitomycin | Analysis | Causes of | Physiological aspects | Ataxia telangiectasia | Genetic aspects | Gene expression | Chromosomes | Cells | Fanconi's anemia
Journal Article
Glia, ISSN 0894-1491, 04/2006, Volume 53, Issue 6, pp. 649 - 659
The elevation of intracellular cAMP synergistically enhances the neuregulin‐dependent proliferation of cultured Schwann cells (SCs); however, the mechanism by... 
PI3‐K | forskolin | ErbB2/3 | proliferation | MAPK | heregulin | PI3-K | Heregulin | Proliferation | Forskolin | AXONS | NERVE | SIGNALING PATHWAYS | PROTEIN-KINASE-B | 3-KINASE | RECEPTOR | NEUROSCIENCES | GROWTH-FACTOR | CAMP | Extracellular Signal-Regulated MAP Kinases - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Adenylyl Cyclases - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | G1 Phase - drug effects | Neuregulin-1 | Intracellular Fluid - drug effects | Phosphatidylinositol 3-Kinases - drug effects | Up-Regulation - physiology | Genes, cdc - physiology | Phosphorylation - drug effects | Oncogene Protein v-akt - metabolism | Cyclic AMP - metabolism | Schwann Cells - drug effects | MAP Kinase Kinase 1 - antagonists & inhibitors | Colforsin - pharmacology | Oncogene Protein v-akt - drug effects | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Genes, cdc - drug effects | MAP Kinase Kinase 1 - metabolism | Adenylyl Cyclases - metabolism | Enzyme Activation - drug effects | G1 Phase - physiology | Nerve Tissue Proteins - drug effects | Cyclic AMP - pharmacology | Nerve Tissue Proteins - metabolism | Up-Regulation - drug effects | Intracellular Fluid - enzymology | Animals | Cell Cycle - physiology | S Phase - physiology | Cell Proliferation - drug effects | S Phase - drug effects | Cell Cycle - drug effects | Enzyme Activation - physiology | Schwann Cells - enzymology
Journal Article
Journal of Cell Science, ISSN 0021-9533, 04/2004, Volume 117, Issue 18, pp. 4277 - 4287
Journal Article
Blood, ISSN 0006-4971, 03/2016, Volume 127, Issue 13, pp. 1687 - 1700
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2008, Volume 107, Issue 1, pp. 265 - 278
Oxidative stress is one of the earliest events in Alzheimer’s disease (AD). A chemical genetic screen revealed that deregulated cyclin‐dependent kinase 5... 
cyclin‐dependent kinase 5 | peroxiredoxin II | peroxiredoxin I | Alzheimer’s disease | oxidative stress | reactive oxygen species | Oxidative stress | Reactive oxygen species | Alzheimer's disease | Cyclin-dependent kinase 5 | Peroxiredoxin II | Peroxiredoxin I | ALZHEIMERS-DISEASE | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAMMALIAN-CELLS | NEUROSCIENCES | CYTOCHROME-C-OXIDASE | ACTIVATOR P35 | BOVINE BRAIN | INDUCED NEURONAL DEATH | DOWN-SYNDROME | cyclin-dependent kinase 5 | CHEMICAL GENETIC APPROACH | CYCLIN-DEPENDENT KINASE-5 | Gene Expression Regulation, Enzymologic - drug effects | Mitochondria - enzymology | Reactive Oxygen Species - metabolism | Calcium Signaling - physiology | Oxidative Stress - physiology | Peroxiredoxins - metabolism | Nerve Degeneration - physiopathology | Nerve Degeneration - genetics | PC12 Cells | Up-Regulation - physiology | Genes, cdc - physiology | Neurons - metabolism | Cell Death - drug effects | Energy Metabolism - physiology | Alzheimer Disease - physiopathology | Nerve Degeneration - enzymology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Genes, cdc - drug effects | Alzheimer Disease - enzymology | Mitochondria - drug effects | Enzyme Activation - drug effects | Cyclin-Dependent Kinase 5 - metabolism | Neurotoxins - pharmacology | Up-Regulation - drug effects | Cyclin-Dependent Kinase 5 - antagonists & inhibitors | Animals | Cell Death - physiology | Calcium Signaling - drug effects | Gene Expression Regulation, Enzymologic - genetics | Feedback, Physiological - drug effects | Mice | Feedback, Physiological - physiology | Oxidative Stress - drug effects | Alzheimer Disease - genetics | Enzyme Activation - physiology | Energy Metabolism - drug effects | Neurology | Genetics | Biochemistry | Oxidation | Cellular biology | Alzheimers disease | Index Medicus
Journal Article