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Journal of clinical oncology, ISSN 1527-7755, 12/2013, Volume 31, Issue 34, pp. 4333 - 4342
Journal Article
BMC medical genomics, ISSN 1755-8794, 2010, Volume 3, Issue 1, pp. 26 - 26
Background: Hyperactivation of the Ras signaling pathway is a driver of many cancers, and RAS pathway activation can predict response to targeted therapies... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Neoplasms - metabolism | Lung Neoplasms - drug therapy | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Databases, Genetic | ras Proteins - metabolism | Antibodies, Monoclonal - therapeutic use | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Antibodies, Monoclonal, Humanized | Mitogen-Activated Protein Kinase Kinases - metabolism | RNA Interference | Colorectal Neoplasms - drug therapy | Female | Cetuximab | Proto-Oncogene Proteins c-akt - metabolism | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | ras Proteins - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Breast Neoplasms - drug therapy | Breast Neoplasms - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Mutation | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | RNA, Small Interfering - metabolism | Ras genes | Physiological aspects | Genetic aspects | Research | Gene expression | Protein kinases | Risk factors | Cancer | Index Medicus
Journal Article
The New England journal of medicine, ISSN 1533-4406, 09/2013, Volume 369, Issue 11, pp. 1023 - 1034
Journal Article
PLoS genetics, ISSN 1553-7390, 2015, Volume 11, Issue 12, pp. e1005634 - e1005634
Journal Article
PloS one, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166626 - e0166626
Ectopic programmed cell death ligand 1 (PD-L1) expression in non-small cell lung cancers (NSCLCs) is related to immune evasion by cancer, and it is a molecular... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lung Neoplasms - drug therapy | RNA, Small Interfering - genetics | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Proto-Oncogene Proteins p21(ras) - genetics | Transcription Factor AP-1 - genetics | Humans | ErbB Receptors - genetics | Lung Neoplasms - pathology | Cell Movement - genetics | Nitriles - administration & dosage | Phorbols - administration & dosage | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | STAT3 Transcription Factor - genetics | Lung Neoplasms - genetics | Cell Adhesion - genetics | Mitogen-Activated Protein Kinases - biosynthesis | Adenocarcinoma - drug therapy | B7-H1 Antigen - genetics | STAT3 Transcription Factor - biosynthesis | Protein Kinase Inhibitors - administration & dosage | MAP Kinase Signaling System - drug effects | Butadienes - administration & dosage | Transcription Initiation Site | Mitogen-Activated Protein Kinases - genetics | Mutation | Genes | Luciferase | B cells | Genetic transcription | Comparative analysis | Lung cancer, Non-small cell | Mitogens | Protein kinases | Apoptosis | Adenocarcinoma | Cluster analysis | Biotechnology | Chromatin | Transcription factors | Immunoprecipitation | Transcription | Downstream effects | Lung cancer | Science | Oncology | Metastasis | Kinases | K-Ras protein | Lymphocytes | L1 gene | Antigens | Epidermal growth factor receptors | Activator protein 1 | Stat3 protein | Extracellular signal-regulated kinase | Non-small cell lung carcinoma | MAP kinase | Tumor cell lines | Gene expression | Lymphoma | 1-Phosphatidylinositol 3-kinase | Signaling | Inhibitors | Immune checkpoint | Lungs | Protein kinase | Cell death | PD-L1 protein | Dimethyl sulfoxide | Ligands | Acetic acid | Phorbol 12-myristate 13-acetate | Index Medicus | Lymphomas
Journal Article
PloS one, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e73616 - e73616
Recent studies have suggested that aberrant K-ras signaling is responsible for triggering immunological responses and inflammation-driven tumorigenesis... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Apoptosis - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Colorectal Neoplasms - genetics | Humans | Middle Aged | Cell Survival - genetics | Apoptosis - genetics | Male | Gene Expression Profiling | Interferon-gamma - metabolism | Interleukins - metabolism | Dose-Response Relationship, Drug | Interleukin-23 - genetics | Interleukins - genetics | Interleukin-23 - metabolism | Aged, 80 and over | Adult | Female | Interferon-gamma - genetics | Colorectal Neoplasms - metabolism | Caco-2 Cells | Cell Survival - drug effects | Polyenes - pharmacology | Interleukin-17 - genetics | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Disease Progression | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Interleukin-17 - metabolism | Polyunsaturated Alkamides - pharmacology | Aged | Mutation | Colorectal Neoplasms - pathology | Neoplasm Staging | Laboratories | Chemical engineering | Genes | Crosstalk | Lung cancer | Interleukin | Colorectal carcinoma | Colorectal cancer | Interleukin 22 | Interleukin 23 | Biology | Kinases | Medical schools | K-Ras protein | Cell growth | Immunology | Interleukins | Intestine | Tumor necrosis factor-TNF | Tumorigenesis | Inhibition | Cytokines | Granulocyte-macrophage colony-stimulating factor | Cell division | Inflammation | Patients | Interleukin 17 | Signaling | Pancreatic cancer | γ-Interferon | Interferon | Aberration | Cancer | Apoptosis | Index Medicus
Journal Article
The FEBS journal, ISSN 1742-464X, 02/2019, Volume 286, Issue 3, pp. 459 - 478
Matrix metalloproteinases (MMPs) are tissue‐remodeling enzymes involved in the processing of various biological molecules. MMPs also play important roles in... 
NOX4 | HIC‐5 | metastasis | MMP9 | ROS | HIC-5 | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | RNA, Small Interfering - genetics | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Extracellular Matrix - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Focal Adhesions - pathology | LIM Domain Proteins - metabolism | RNA, Messenger - metabolism | Breast Neoplasms - metabolism | Matrix Metalloproteinase 9 - metabolism | Heterografts | Lung Neoplasms - secondary | Cellular Senescence | Matrix Metalloproteinase 9 - genetics | Female | Intracellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins B-raf - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Lung Neoplasms - genetics | NADPH Oxidase 4 - genetics | Signal Transduction | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | LIM Domain Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Mitochondria - metabolism | Mitochondria - pathology | NADPH Oxidase 4 - metabolism | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins B-raf - genetics | Focal Adhesions - metabolism | Cell Line, Tumor | LIM Domain Proteins - genetics | Mice, Inbred NOD | Mice | Extracellular Matrix - pathology | RNA, Small Interfering - metabolism | Cancer cells | Metastasis | Cancer | Cell proliferation | Biotechnology | Hydrogen peroxide | Matrix metalloproteinases | Epithelial cells | Matrix metalloproteinase | NAD(P)H oxidase | Metastases | Cell adhesion & migration | Angiogenesis | Signal transduction | Mitochondria | NOX4 protein | Metalloproteinase | Mammary gland | Tumor cells | Invasiveness | Tumor cell lines | Gene expression | Gelatinase B | Signaling | Lungs | Mutation | Cell migration | mRNA stability | Tumors
Journal Article
Journal Article