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American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2013, Volume 49, Issue 2, pp. 167 - 179
Interstitial lung fibrosis can develop as a consequence of occupational or medical exposure, as a result of genetic defects, and after trauma or acute lung... 
Aging | Cytokines | Fibroblast | Collagen | Fibrosis | SURFACTANT PROTEIN-C | GROWTH-FACTOR-RECEPTOR | FLUORESCEIN ISOTHIOCYANATE | BIOCHEMISTRY & MOLECULAR BIOLOGY | collagen | NECROSIS-FACTOR-ALPHA | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | MURINE MODEL | fibroblast | PROVIDE CLUES | EPITHELIAL-CELLS | INDUCED PULMONARY-FIBROSIS | RESPIRATORY SYSTEM | TRANSFORMING GROWTH-FACTOR-BETA-1 | fibrosis | cytokines | aging | Respiratory Distress Syndrome, Adult - pathology | Lung Diseases, Interstitial - metabolism | Epithelial Cells - metabolism | Respiratory Distress Syndrome, Adult - physiopathology | Lung Diseases, Interstitial - pathology | Humans | Genetic Diseases, Inborn - genetics | Pulmonary Fibrosis - genetics | Genetic Diseases, Inborn - pathology | Respiratory Distress Syndrome, Adult - metabolism | Respiratory Mucosa - pathology | Respiratory Distress Syndrome, Adult - genetics | Pulmonary Alveoli - metabolism | Respiratory Mucosa - physiopathology | Genetic Diseases, Inborn - metabolism | Pulmonary Fibrosis - metabolism | Lung Diseases, Interstitial - physiopathology | Pulmonary Alveoli - physiopathology | Disease Models, Animal | Pulmonary Alveoli - pathology | Cell Communication | Epithelial Cells - pathology | Pulmonary Fibrosis - pathology | Animals | Lung Diseases, Interstitial - genetics | Pulmonary Fibrosis - physiopathology | Genetic Diseases, Inborn - physiopathology | Respiratory Mucosa - metabolism | Translational Review
Journal Article
Human Gene Therapy, ISSN 1043-0342, 10/2008, Volume 19, Issue 10, pp. 979 - 990
Journal Article
Journal of Medical Genetics, ISSN 0022-2593, 12/2011, Volume 48, Issue 12, pp. 856 - 859
ObjectiveTo investigate the potential contribution of germline sequence alterations in the BAP1 gene in uveal melanoma (UM) patients with possible... 
BREAST-CANCER | GENE | HIGH-RISK | GENETICS & HEREDITY | TUMOR-SUPPRESSOR | BRCA1 | UBIQUITIN HYDROLASE | BRCA1-ASSOCIATED PROTEIN-1 | Adenocarcinoma - pathology | Genetic Testing | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Genetic Diseases, Inborn - genetics | Lung Neoplasms - pathology | Male | Meningioma - genetics | Genetic Diseases, Inborn - pathology | Loss of Heterozygosity | Adenocarcinoma - metabolism | DNA Mutational Analysis | Melanoma - genetics | Tumor Suppressor Proteins - genetics | Germ-Line Mutation | Ubiquitin Thiolesterase - metabolism | Adult | Female | Adenocarcinoma - genetics | Genetic Diseases, Inborn - metabolism | Uveal Neoplasms - metabolism | Microsatellite Repeats | Lung Neoplasms - genetics | Melanoma - metabolism | Uveal Neoplasms - genetics | Genetic Predisposition to Disease - genetics | Tumor Suppressor Proteins - metabolism | Meningeal Neoplasms - genetics | Meningioma - metabolism | Uveal Neoplasms - pathology | Gene Silencing | Chromosomes, Human, Pair 3 - metabolism | Melanoma - pathology | Ubiquitin Thiolesterase - genetics | Meningioma - pathology | Pedigree | Meningeal Neoplasms - metabolism | Meningeal Neoplasms - pathology | Chromosomes, Human, Pair 3 - genetics | Genetic aspects | Research | BRCA mutations | Cancer
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 09/2014, Volume 111, Issue 38, pp. E4033 - E4042
Journal Article
Blood, ISSN 0006-4971, 01/2013, Volume 121, Issue 5, pp. 822 - 829
Journal Article