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1. Full Text Plasmodium vivax transcriptomes reveal stage-specific chloroquine response and differential regulation of male and female gametocytes
by Kim, Adam and Popovici, Jean and Menard, Didier and Serre, David
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 371 - 9
Studies of Plasmodium vivax gene expression are complicated by the lack of in vitro culture system and the difficulties associated with studying clinical... 
GENETIC DIVERSITY | FORMS | INFECTIONS | COMMITMENT | FALCIPARUM | PACKAGE | MULTIDISCIPLINARY SCIENCES | MALARIA | EXPRESSION | Cell Cycle - genetics | Multigene Family | Genes, Protozoan - genetics | Humans | Plasmodium vivax - genetics | Male | Plasmodium falciparum - drug effects | Trophozoites - genetics | RNA, Messenger - metabolism | Trophozoites - drug effects | Chloroquine - pharmacology | Genes, Protozoan - drug effects | Plasmodium vivax - pathogenicity | Plasmodium falciparum - genetics | Female | Malaria, Falciparum - drug therapy | Plasmodium vivax - drug effects | Transcriptome - drug effects | Antimalarials - pharmacology | Gene Expression Regulation - drug effects | Malaria, Vivax - drug therapy | Genome, Protozoan - drug effects | Genome, Protozoan - genetics | Malaria - drug therapy | Cell Cycle - drug effects | Vector-borne diseases | Trophozoites | Malaria | Genes | Chloroquine | Infections | Gametocytes | Parasites | Gene expression
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2. Full Text DNA damage regulation and its role in drug-related phenotypes in the malaria parasites
by Gupta, Devendra Kumar and Patra, Alok Tanala and Zhu, Lei and Gupta, Archana Patkar and Bozdech, Zbynek
Scientific Reports, ISSN 2045-2322, 04/2016, Volume 6, Issue 1, p. 23603
DNA of malaria parasites, Plasmodium falciparum, is subjected to extraordinary high levels of genotoxic insults during its complex life cycle within both the... 
OXIDATIVE STRESS | BASE EXCISION-REPAIR | ARTEMISININ RESISTANCE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | DOUBLE-STRAND BREAKS | TOXOPLASMA-GONDII | MISMATCH-REPAIR | HOMOLOGOUS RECOMBINATION | RESISTANT PLASMODIUM-FALCIPARUM | SACCHAROMYCES-CEREVISIAE | Transcription, Genetic - drug effects | Genes, Protozoan | Gene Expression Profiling | DNA Repair - genetics | Plasmodium falciparum - drug effects | Protozoan Proteins - genetics | DNA Breaks, Double-Stranded | Artemisinins - pharmacology | Histone Code - drug effects | Protein Processing, Post-Translational - drug effects | DNA, Protozoan - drug effects | Plasmodium falciparum - genetics | Acetylation | Chromatin - drug effects | Hydroxamic Acids - pharmacology | Chromatin - ultrastructure | Drug Resistance, Multiple - genetics | Protozoan Proteins - biosynthesis | Antimalarials - pharmacology | Gene Expression Regulation - drug effects | Phenotype | Animals | DNA, Protozoan - genetics | DNA Damage | Chromatin - genetics | Methyl Methanesulfonate - toxicity | Malaria | Transcription | DNA damage | Genotoxicity | Artemisinin | Genomes | Parasites | DNA repair | Antimalarial agents | Deoxyribonucleic acid--DNA | DNA sequencing | Life cycles
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3. Full Text Mitotic Evolution of Plasmodium falciparum Shows a Stable Core Genome but Recombination in Antigen Families
by Bopp, Selina E. R and Manary, Micah J and Bright, A. Taylor and Johnston, Geoffrey L and Dharia, Neekesh V and Luna, Fabio L and McCormack, Susan and Plouffe, David and McNamara, Case W and Walker, John R and Fidock, David A and Denchi, Eros Lazzerini and Winzeler, Elizabeth A
PLoS Genetics, ISSN 1553-7390, 02/2013, Volume 9, Issue 2, p. e1003293
Malaria parasites elude eradication attempts both within the human host and across nations. At the individual level, parasites evade the host immune responses... 
VARIANT ANTIGEN | MUTATION-RATES | CYTOCHROME-B | ANTIMALARIAL-DRUGS | VAR GENE | GENETICS & HEREDITY | RESISTANCE | CHROMOSOMES | DNA-SEQUENCING DATA | MALARIA PARASITES | ECTOPIC RECOMBINATION | Malaria, Falciparum - genetics | Drug Resistance, Multiple - genetics | Antigens - drug effects | Drug Resistance, Multiple - drug effects | Humans | Host-Parasite Interactions - immunology | Plasmodium falciparum - drug effects | Plasmodium falciparum - immunology | Mitosis - genetics | Host-Parasite Interactions - genetics | Cytochromes b - genetics | Multidrug Resistance-Associated Proteins - immunology | Plasmodium falciparum - genetics | Malaria, Falciparum - immunology | Multidrug Resistance-Associated Proteins - genetics | Antigenic Variation - drug effects | Genome, Protozoan - drug effects | High-Throughput Nucleotide Sequencing | Atovaquone - administration & dosage | Antigenic Variation - genetics | Multidrug Resistance-Associated Proteins - metabolism | Antigens - genetics | Evolution, Molecular | Plasmodium falciparum | Microbial genetics | Antigens | Physiological aspects | Evolutionary genetics | Genetic aspects | Research | Health aspects | Mutation | Parasites | Malaria | Genes | Genomics
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4. Full Text Characterisation of 20S proteasome in Tritrichomonas foetus and its role during the cell cycle and transformation into endoflagellar form
by Pereira-Neves, Antonio and Gonzaga, Luiz and Menna-Barreto, Rubem F. S and Benchimol, Marlene
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0129165
Proteasomes are intracellular complexes that control selective protein degradation in organisms ranging from Archaea to higher eukaryotes. These structures... 
TRYPANOSOMA-BRUCEI | COLD-EXPOSURE | INTRACELLULAR-LOCALIZATION | PLASMODIUM-FALCIPARUM | MULTIDISCIPLINARY SCIENCES | GENE FAMILY | TOXOPLASMA-GONDII | ENTAMOEBA-HISTOLYTICA | S PROTEASOME | PATHOGEN TRICHOMONAS-VAGINALIS | PROTOZOAN PARASITE | Endoplasmic Reticulum - metabolism | Molecular Sequence Data | Tritrichomonas foetus - growth & development | Phylogeny | Tritrichomonas foetus - genetics | Protozoan Proteins - genetics | Endoplasmic Reticulum - ultrastructure | Protein Subunits - metabolism | Protozoan Proteins - metabolism | Acetylcysteine - analogs & derivatives | Endoplasmic Reticulum - drug effects | Life Cycle Stages - drug effects | Protein Subunits - genetics | Amino Acid Sequence | Microscopy, Electron, Transmission | Microscopy, Electron, Scanning | Flagella - ultrastructure | Blotting, Western | Proteasome Endopeptidase Complex - genetics | Sequence Homology, Amino Acid | Flagella - metabolism | Cell Cycle | Cysteine Proteinase Inhibitors - pharmacology | Acetylcysteine - pharmacology | Spores, Protozoan - metabolism | Protozoan Proteins - ultrastructure | Protein Subunits - antagonists & inhibitors | Proteasome Endopeptidase Complex - metabolism | Proteasome Endopeptidase Complex - classification | Microscopy, Fluorescence | Spores, Protozoan - drug effects | Spores, Protozoan - ultrastructure | Tritrichomonas foetus - metabolism | Ubiquitin | Proteolysis | Proteases | Analysis | Genomics | Cell cycle | Genomes | Transformation | Membranes | Serine | Differentiation (biology) | Homeostasis | Homology | Labelling | Parasites | Cytosol | Gene sequencing | Proteins | Eukaryotes | Lactacystin | Inhibition | Catalysis | Bioinformatics | Fractionation | Peptidase | Archaea | Fetuses | Gene expression | Electron microscopy | Cell differentiation | Proteasome inhibitors | Substrates | Cysts | Microscopy | Proteasomes | Immunofluorescence | Endoplasmic reticulum | Apoptosis
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5. Full Text Analysis of short RNAs in the malaria parasite and its red blood cell host
by Rathjen, Tina and Nicol, Clare and McConkey, Glenn and Dalmay, Tamas
FEBS Letters, ISSN 0014-5793, 2006, Volume 580, Issue 22, pp. 5185 - 5188
RNA interference (RNAi) is an RNA degradation process that involves short, double-stranded RNAs (dsRNA) as sequence specificity factors. The natural function... 
Red blood cell | MicroRNA | RNA interference | Malaria | TRYPANOSOMA-BRUCEI | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICRORNAS | malaria | red blood cell | CELL BIOLOGY | GENOMICS | BIOPHYSICS | PLASMODIUM-FALCIPARUM | GROWTH | microRNA | INTERFERENCE | CYCLE | RNA, Protozoan - genetics | Humans | MicroRNAs - metabolism | RNA Stability - drug effects | RNA, Ribosomal - genetics | Cell Differentiation - genetics | Plasmodium falciparum - genetics | Cloning, Molecular | RNA, Transfer - genetics | Plasmodium falciparum - metabolism | RNA, Double-Stranded - metabolism | Malaria - metabolism | Gene Expression Regulation - genetics | RNA, Transfer - metabolism | RNA, Ribosomal - metabolism | RNA Stability - genetics | Gene Expression Regulation - drug effects | RNA, Protozoan - metabolism | Animals | Cell Differentiation - drug effects | Erythrocytes - metabolism | RNA, Double-Stranded - genetics | RNA, Double-Stranded - pharmacology | Genome, Protozoan - drug effects | Genome, Protozoan - genetics | MicroRNAs - genetics | RNA, Small Interfering | Erythrocytes - parasitology | Malaria - genetics | Plasmodium falciparum | Gene expression | Analysis | Transfer RNA
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6. Full Text Genome-wide mutagenesis and multi-drug resistance in American trypanosomes induced by the front-line drug benznidazole
by Campos, Mônica C and Phelan, Jody and Francisco, Amanda F and Taylor, Martin C and Lewis, Michael D and Pain, Arnab and Clark, Taane G and Kelly, John M
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 14407 - 8
Chagas disease is caused by the protozoan parasite Trypanosoma cruzi and affects 5-8 million people in Latin America. Although the nitroheterocyclic compound... 
RECOMBINATION | IN-VITRO | CRUZI | POSACONAZOLE | TRANSPORTER | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | CHAGAS-DISEASE | RANDOMIZED-TRIAL | MECHANISMS | NIFURTIMOX | Trypanocidal Agents - metabolism | Mutagens - metabolism | Nitroimidazoles - pharmacology | Drug Resistance, Multiple | Mutagens - pharmacology | Trypanosoma cruzi - genetics | Mice, SCID | Trypanosoma cruzi - pathogenicity | Sequence Analysis, DNA | Mutagenesis - drug effects | Triazoles - pharmacology | Nitroimidazoles - metabolism | Animals | Trypanocidal Agents - pharmacology | Chagas Disease - drug therapy | Trypanosoma cruzi - drug effects | Female | Genome, Protozoan - drug effects | Mutation | Disease Models, Animal | Protozoa | Ergosterol | Vigilance | Benznidazole | Cross-resistance | Cloning | Multidrug resistance | Nitroreductase | Genomes | Parasites | Drug resistance | DNA repair | Mitochondria | Metabolites | Mutagenesis | Chagas' disease | Posaconazole | Deoxyribonucleic acid--DNA
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7. Full Text Genome-wide scans for the identification of Plasmodium vivax genes under positive selection
by Shen, Hai-Mo and Chen, Shen-Bo and Wang, Yue and Xu, Bin and Abe, Eniola Michael and Chen, Jun-Hu
Malaria Journal, ISSN 1475-2875, 06/2017, Volume 16, Issue 1, pp. 238 - 12
Background: The current trend of Plasmodium vivax cases imported from Southeast Asia into China has sharply increased recently, especially from the... 
Plasmodium vivax | Haplotype-based detecting | Immune evasion | Drug resistance | Positive selection | Invasion | INFECTIOUS DISEASES | SIGNATURES | FALCIPARUM | DRUG-RESISTANCE | ISOLATE | TROPICAL MEDICINE | POPULATION-GENETICS | COMMUNITY | CHINA-MYANMAR BORDER | DIVERSITY | INFECTION | EVOLUTIONARY HISTORY | PARASITOLOGY | Genetic Variation | Genome, Protozoan | Plasmodium vivax - drug effects | Genes, Protozoan - drug effects | China | Plasmodium vivax - genetics | Malaria, Vivax - prevention & control | Drug Resistance | Antimalarials - pharmacology | Genomes | Research | Biological diversity
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8. Full Text Genomics illuminates parasite biology
by Foth, Bernardo J and Otto, Thomas D
Nature reviews. Microbiology, ISSN 1740-1526, 11/2014, Volume 12, Issue 11, pp. 727 - 727
MALARIA | MICROBIOLOGY | INFECTIONS | Anti-Infective Agents - pharmacology | Genomics | Humans | Plasmodium - genetics | Anti-Infective Agents - therapeutic use | Plasmodium - drug effects | Protozoan Proteins - genetics | Drug Resistance - genetics | Artemisinins - pharmacology | Animals | Artemisinins - therapeutic use | Malaria - parasitology | Genome, Protozoan - drug effects | Genome, Protozoan - genetics | Malaria - drug therapy | Mutation | Genetic research | Genetic aspects | Research | Parasites
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9. Full Text Growth of the protozoan parasite Entamoeba histolytica in 5-azacytidine has limited effects on parasite gene expression
by Ali, Ibne Kairim M and Ehrenkaufer, Gretchen M and Hackney, Jason A and Singh, Upinder
BMC Genomics, ISSN 1471-2164, 2007, Volume 8, Issue 1, pp. 7 - 7
Background: In higher eukaryotes DNA methylation regulates important biological functions including silencing of gene expression and protection from adverse... 
ANTISENSE INHIBITION | DEOXYRIBONUCLEIC ACIDS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CYSTEINE PROTEINASES | GENETICS & HEREDITY | DNA METHYLTRANSFERASE EHMETH | CPG METHYLATION | VIRULENCE | MAMMALIAN-CELLS | HISTONE DEACETYLASE | CYTOSINE METHYLATION | MYZUS-PERSICAE | Virulence - drug effects | Cricetinae | Protozoan Proteins - antagonists & inhibitors | Gene Silencing | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Protozoan Proteins - biosynthesis | Entamoeba histolytica - drug effects | Entamoeba histolytica - pathogenicity | Gene Expression Regulation - drug effects | Azacitidine - pharmacology | Genome, Protozoan - physiology | DNA Methylation | Animals | Genes, Protozoan - drug effects | Microarray Analysis | Genes, Protozoan - physiology | Mesocricetus | Entamoeba histolytica - growth & development | CHO Cells
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10. Full Text Biochemical and inhibition studies of glutamine synthetase from Leishmania donovani
by Kumar, Vinay and Kumar, Rohit and Yadav, Shailendra and Soumya, Neelagiri and Babu, Neerupudi Kishore and Singh, Sushma
Microbial Pathogenesis, ISSN 0882-4010, 06/2017, Volume 107, pp. 164 - 174
Leishmaniasis is a group of tropical diseases caused by protozoan parasites of the genus . is a protozoan parasite that causes visceral leishmaniasis, a fatal... 
Glutamine synthetase | Kinetics | Localization | Leishmania | MYCOBACTERIUM-TUBERCULOSIS | PROTEIN | MICROBIOLOGY | IMMUNOLOGY | IDENTIFICATION | DRUG TARGETS | STREPTOCOCCUS-SUIS | PREFRONTAL CORTEX | METABOLISM | LIVER | EXPRESSION | GENE EVOLUTION | Temperature | Molecular Weight | Genes, Protozoan - genetics | Genome, Protozoan | Metals | Phosphorylcholine - analogs & derivatives | Protozoan Proteins - drug effects | Protozoan Proteins - genetics | Base Sequence | Phosphorylcholine - antagonists & inhibitors | Protozoan Proteins - chemistry | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Leishmania donovani - enzymology | Recombinant Proteins - chemistry | Antibodies, Protozoan | Recombinant Proteins - genetics | Enzyme Activation - drug effects | Leishmania donovani - growth & development | Glutamate-Ammonia Ligase - immunology | Sequence Homology, Amino Acid | Glutamate-Ammonia Ligase - drug effects | Recombinant Proteins - immunology | Escherichia coli - genetics | Recombinant Proteins - drug effects | Glutamate-Ammonia Ligase - genetics | Glutamate-Ammonia Ligase - chemistry | DNA, Protozoan - genetics | Leishmaniasis - parasitology | Sequence Analysis | Protozoan Proteins - immunology | Hydrogen-Ion Concentration | Chemotherapy | Ligases | Analysis | Escherichia coli | Genomics | Leishmaniasis | Drug resistance | Glutamate | Glutamine | Cancer | Index Medicus
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11. Full Text Protein kinases as drug targets in trypanosomes and Leishmania
by Naula, Christina and Parsons, Marilyn and Mottram, Jeremy C
BBA - Proteins and Proteomics, ISSN 1570-9639, 2005, Volume 1754, Issue 1, pp. 151 - 159
Protein kinases represent promising drug targets for a number of human and animal diseases. The recent completion of the sequenced genomes of three... 
Chemotherapy | Trypanosoma | Protein kinase | Leishmania | Cell cycle | PROCYCLIC FORM | CRUZI | cell cycle | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHAGAS-DISEASE | BRUCEI | CYCLIN-DEPENDENT KINASE | chemotherapy | MEXICANA | protein kinase | HISTONE H1 KINASE | MITOTIC CYCLIN | FLAGELLAR-LENGTH | BIOPHYSICS | CELL-CYCLE | Protein Kinases - metabolism | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Trypanosomatina - growth & development | Trypanosoma cruzi - metabolism | Humans | Leishmania major - drug effects | Leishmania major - genetics | Trypanosoma cruzi - genetics | Life Cycle Stages - genetics | Animals | Trypanosomatina - metabolism | Models, Biological | Leishmania major - metabolism | Trypanosoma cruzi - drug effects | Mitogen-Activated Protein Kinases - genetics | Genome, Protozoan - drug effects | Protein Kinase Inhibitors - pharmacology | Cyclin-Dependent Kinases - genetics | Mitogen-Activated Protein Kinases - metabolism | Tyrosine | Casein | Protein kinases | Analysis | Cancer
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12. Full Text Cryptic Parasite Revealed. Improved Prospects for Treatment and Control of Human Cryptosporidiosis Through Advanced Technologies {star operator}
by Jex, Aaron R and Smith, Huw V and Nolan, Matthew J and Campbell, Bronwyn E and Young, Neil D and Cantacessi, Cinzia and Gasser, Robin B
Advances in Parasitology, ISSN 0065-308X, 2011, Volume 77, pp. 141 - 173
Cryptosporidium is an important genus of parasitic protozoa of humans and other vertebrates and is a major cause of intestinal disease globally. Unlike many... 
Cryptosporidium | Drug targets | Transcriptomics | Essentiality | Genomics | CELL-FREE CULTURE | IN-VITRO | INFLAMMATORY-BOWEL-DISEASE | CHRONIC DIARRHEA | PLASMODIUM-FALCIPARUM | PARVUM INFECTION | WHOLE GENOME AMPLIFICATION | RATIONAL DRUG DESIGN | HUMAN-IMMUNODEFICIENCY-VIRUS | OF-THE-LITERATURE | PARASITOLOGY | Cryptosporidium parvum - growth & development | Spores, Protozoan - growth & development | Cryptosporidiosis - diagnosis | Humans | Transcriptome | Biomedical Research - trends | Cryptosporidiosis - drug therapy | Cryptosporidiosis - parasitology | Cryptosporidium parvum - genetics | Protozoan Proteins - genetics | Antiprotozoal Agents - pharmacology | Cryptosporidium - growth & development | Animals | Cryptosporidium parvum - drug effects | Protozoan Proteins - metabolism | Cryptosporidium - genetics | Cryptosporidiosis - prevention & control | Cryptosporidium - drug effects |