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Development (Cambridge), ISSN 0950-1991, 05/2016, Volume 143, Issue 10, pp. 1697 - 1709
Journal Article
Nature Structural & Molecular Biology, ISSN 1545-9993, 05/2009, Volume 16, Issue 5, pp. 492 - 498
Journal Article
Journal Article
Plant Physiology, ISSN 0032-0889, 10/2013, Volume 163, Issue 2, pp. 867 - 881
The plastid genome is highly conserved among plant species, suggesting that alterations of its structure would have dramatic impacts on plant fitness.... 
Oxidative stress | Leaves | Reactive oxygen species | Phenotypes | Chloroplasts | Genes | Genomes | SIGNALING AND RESPONSE | Plants | Plastids | Chlorophylls | VARIEGATION MUTANTS | RESPONSES | OXIDATIVE STRESS | GENE | CHLOROPLAST DEVELOPMENT | PHOTOSYSTEM-II | SINGLET OXYGEN | LIGHT STRESS | TOPOISOMERASE-IV | DNA-POLYMERASES | PLANT SCIENCES | Cell Death - radiation effects | Genome, Plastid - genetics | Inheritance Patterns - drug effects | Reactive Oxygen Species - metabolism | Cellular Reprogramming - radiation effects | Arabidopsis - growth & development | Adaptation, Physiological - drug effects | Stress, Physiological - radiation effects | Arabidopsis Proteins - metabolism | Arabidopsis - radiation effects | Cell Nucleus - metabolism | Photosynthesis - genetics | Adaptation, Physiological - genetics | Cell Nucleus - radiation effects | Light | Plastids - drug effects | Genomic Instability - drug effects | Inheritance Patterns - genetics | Plastids - genetics | Stress, Physiological - drug effects | Cell Death - drug effects | Signal Transduction - radiation effects | Cellular Reprogramming - genetics | DNA, Plant - genetics | Inheritance Patterns - radiation effects | Photosynthesis - radiation effects | Genomic Instability - radiation effects | Arabidopsis Proteins - genetics | Stress, Physiological - genetics | Arabidopsis - ultrastructure | Ciprofloxacin - pharmacology | Signal Transduction - genetics | Adaptation, Physiological - radiation effects | Mutation - genetics | Arabidopsis - genetics | Cellular Reprogramming - drug effects | Phenotype | Signal Transduction - drug effects | Photosynthesis - drug effects | Plastids - ultrastructure | Gene Rearrangement - genetics | Plastids - radiation effects | Cell Nucleus - drug effects | Arabidopsis thaliana | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Gene expression | Index Medicus
Journal Article
International Journal of Radiation Biology, ISSN 0955-3002, 10/2018, Volume 94, Issue 10, pp. 883 - 889
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2783 - 14
The importance of the tumor-associated stroma in cancer progression is clear. However, it remains uncertain whether early events in the stroma are capable of... 
BREAST-CANCER | NUCLEAR PTEN | CONTRALATERAL BREAST | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | FOLLOW-UP | DNA-REPAIR | TUMOR MICROENVIRONMENT | MOUSE MODELS | FIBROBLASTS | RADIATION-THERAPY | Epithelial Cells - metabolism | Gamma Rays - adverse effects | Epithelial Cells - drug effects | Humans | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | Mammary Glands, Human - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | Mammary Neoplasms, Experimental - pathology | Stromal Cells - drug effects | Genomic Instability - drug effects | Female | Antineoplastic Agents - pharmacology | Mammary Glands, Human - radiation effects | Mammary Glands, Human - drug effects | Genomic Instability - radiation effects | PTEN Phosphohydrolase - genetics | Epithelial Cells - radiation effects | PTEN Phosphohydrolase - deficiency | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Signal Transduction | Stromal Cells - metabolism | Radiation Tolerance - genetics | Mammary Neoplasms, Experimental - radiotherapy | Mammary Glands, Animal - drug effects | Mammary Glands, Animal - radiation effects | Breast Neoplasms - radiotherapy | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Breast Neoplasms - pathology | Cell Transformation, Neoplastic | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Cell Proliferation - radiation effects | Stromal Cells - radiation effects | Hyperplasia | Radiation | Paracrine signalling | Homology | Genomic instability | Epidermal growth factor | Clonal deletion | Deletion | Tumorigenesis | Inhibition | Mammary gland | Tensin | Radiation effects | Stability | Epidermal growth factor receptors | Stroma | Risk reduction | Mammary glands | Breast cancer | Radiation therapy | Epithelium | Patients | Radiation dosage | Breast | Diagnostic systems | PTEN protein | Cancer | Tumors | Index Medicus
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 08/2009, Volume 35, Issue 4, pp. 534 - 541
The molecular pathways leading from genomic instability to cellular senescence and/or cell death remain incompletely characterized. Using mouse embryonic... 
CELLCYCLE | DNA | HUMDISEASE | APOPTOSIS | ACTIVATION | TUMOR SUPPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | ONCOGENE-INDUCED SENESCENCE | CELLULAR SENESCENCE | TUMORIGENESIS | CANCER | MICE LACKING | P53 | CELL BIOLOGY | Gamma Rays | Cellular Senescence - drug effects | Apoptosis - genetics | Intracellular Signaling Peptides and Proteins - metabolism | DNA-Binding Proteins - metabolism | Aging - genetics | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - genetics | Cellular Senescence - radiation effects | Cell Cycle Proteins - genetics | Genomic Instability - drug effects | BRCA1 Protein - deficiency | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Hydrogen Peroxide - toxicity | Cellular Senescence - genetics | Genomic Instability - radiation effects | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Chromosomal Proteins, Non-Histone | Protein-Serine-Threonine Kinases - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Fibroblasts - pathology | Cell Transformation, Neoplastic - metabolism | Doxorubicin - toxicity | Mice, Knockout | BRCA1 Protein - genetics | Animals | Histones - genetics | Checkpoint Kinase 2 | Mice | Histones - metabolism | Tumor Suppressor p53-Binding Protein 1 | Aging - metabolism | Gene mutations | Cell death | Kidney diseases | Tumor proteins | Health aspects | Tumors | Protein binding | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 06/2014, Volume 4, Issue 1, pp. 5421 - 5421
Journal Article
Journal Article