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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2012, Volume 109, Issue 13, pp. 5040 - 5045
Respiratory syncytial virus (RSV) is the major viral cause of severe pulmonary disease in young infants worldwide. However, the mechanisms by which RSV causes... 
Human respiratory syncytial virus | Respiratory syncytial virus infections | Intestines | Epithelial cells | Secretion | Cell lines | Cultured cells | Viruses | Infections | Chemokines | Primary airway epithelial cells | Virus-host interaction | Lung epithelium model | BRONCHIOLITIS | HUMAN AIRWAY EPITHELIUM | MULTIDISCIPLINARY SCIENCES | PARAINFLUENZA VIRUS | primary airway epithelial cells | CHILDREN | virus-host interaction | AGE 13 | INTERFERON | INFANTS | lung epithelium model | INFLUENZA-VIRUS | PROTEINS | CILIATED CELLS | Chemokines - secretion | Cilia - pathology | Respiratory Syncytial Viruses - physiology | Humans | Hyperplasia | Cytopathogenic Effect, Viral | Epithelial Cells - pathology | Mucus - secretion | Respiratory Mucosa - virology | Giant Cells - virology | Respiratory Mucosa - pathology | Epithelial Cells - secretion | Models, Biological | Epithelial Cells - virology | Cell Differentiation | Goblet Cells - pathology | Respiratory Syncytial Virus Infections - pathology | Respiratory Syncytial Virus Infections - virology | Bronchi - pathology | Child | Giant Cells - pathology | Respiratory Syncytial Viruses - pathogenicity | Apoptosis | Respiratory syncytial virus infection | Disease transmission | Physiological aspects | Development and progression | Research | Epithelium | Health aspects | Babies | Chronic obstructive pulmonary disease | Cells | Viral infections | Index Medicus | Biological Sciences | virus–host interaction
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2012, Volume 109, Issue 8, pp. 2772 - 2777
Journal Article
Oncogene, ISSN 0950-9232, 08/2017, Volume 36, Issue 34, pp. 4887 - 4900
Our recent perplexing findings that polyploid giant cancer cells (PGCCs) acquired embryonic-like stemness and were capable of tumor initiation raised two... 
OVARIAN TUMORIGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-CELLS | CELLULAR SENESCENCE | FAILURE | CLEAVAGE-STAGE EMBRYOS | ESCAPE | CELL BIOLOGY | IN-VITRO | CYTOKINESIS | THERAPY | ONCOLOGY | GENETICS & HEREDITY | DIFFERENTIATION | Embryonic Stem Cells - metabolism | Paclitaxel - pharmacology | Neoplastic Stem Cells - drug effects | Giant Cells - drug effects | Humans | Ovarian Neoplasms - pathology | Carcinogenesis - metabolism | Blastomeres - drug effects | Blastomeres - metabolism | Neoplastic Stem Cells - metabolism | Female | Ovarian Neoplasms - metabolism | Cell Differentiation - physiology | Ovarian Neoplasms - drug therapy | Neoplastic Stem Cells - physiology | Polyploidy | Giant Cells - metabolism | Blastomeres - pathology | Carcinogenesis - drug effects | Carcinogenesis - pathology | Drug Resistance, Neoplasm - genetics | Embryonic Stem Cells - drug effects | Cell Differentiation - drug effects | Embryonic Stem Cells - pathology | Cell Line, Tumor | Signal Transduction - physiology | Giant Cells - pathology | Cellular signal transduction | Research | Cell differentiation | Cancer cells | Blastomeres | Senescence | Carcinoma | Mesenchyme | Mitosis | Oct-4 protein | Embryo cells | Benign | Compaction | Cytokinesis | Embryos | Spheroids | Ovarian cancer | Yes-associated protein | Chemotherapy | Developmental stages | Somatic cells | Paclitaxel | Stem cells | Giant cells | Tumorigenesis | Budding | Cancer | Tumors | Index Medicus | Original
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, pp. e0125316 - e0125316
Missense mutations of the GJA1 gene encoding the gap junction channel protein connexin43 (Cx43) cause bone malformations resulting in oculodentodigital... 
CORNEAL FIBROBLASTS | MARROW | MULTIDISCIPLINARY SCIENCES | IN-VIVO | TISSUE | GENE-EXPRESSION | MYOFIBROBLASTS | GROWTH-FACTOR | CX43 | DIFFERENTIATION | COMMUNICATION | Bone and Bones - pathology | Gap Junctions - metabolism | Humans | Middle Aged | Actins - metabolism | Gene Expression Regulation, Neoplastic | Child, Preschool | Hematopoietic Stem Cells - pathology | Monocytes - metabolism | Antigens, CD - genetics | Bone Neoplasms - pathology | Actins - genetics | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Giant Cell Tumor of Bone - pathology | Neoplastic Stem Cells - metabolism | Bone and Bones - metabolism | Monocytes - pathology | Neoplastic Stem Cells - pathology | Adult | Bone Neoplasms - genetics | Giant Cell Tumor of Bone - diagnosis | Child | Giant Cell Tumor of Bone - genetics | Connexin 43 - genetics | Gap Junctions - pathology | Alkaline Phosphatase - genetics | Bone Neoplasms - diagnosis | Connexin 43 - metabolism | Macrophages - pathology | Signal Transduction | Giant Cells - metabolism | Bone Marrow Cells - pathology | Receptors, Cell Surface - metabolism | Hematopoietic Stem Cells - metabolism | Antigens, Differentiation, Myelomonocytic - genetics | Macrophages - metabolism | Giant Cell Tumor of Bone - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Adolescent | Survival Analysis | Aged | Primary Cell Culture | Alkaline Phosphatase - deficiency | Bone Marrow Cells - metabolism | Giant Cells - pathology | Receptors, Cell Surface - genetics | Immunohistochemistry | Dysplasia | Muscle proteins | Tumors | Nests | Smooth muscle | Osteolysis | Proteins | Animal tissues | Bone marrow | Fibroblasts | Bone dysplasia | Biocompatibility | Growth factors | Medical research | Gap junctions | Cultures | Cell membranes (biology) | Survival | Golgi apparatus | CD163 antigen | Pathology | DNA microarrays | Stem cells | Mutation | Binding sites | Alkaline phosphatase | Phosphorylation | Membranes | Oculodentodigital dysplasia | Laboratories | Oncology | Connexin 43 | Macrophages | Western blotting | Defects | Biomedical materials | Missense mutation | Actin | Rodents | Giant cells | Coupling | Localization | Calcein | Communication | Cell survival | Muscles | Cell membranes | Monocytes | Bone | Cancer | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e59418 - e59418
Cell-in-cell structures refer to a unique phenomenon that one living cell enters into another living cell intactly, occurring between homotypic tumor cells or... 
NATURAL-KILLER-CELLS | MELANOMA | EMPERIPOLESIS | ENTOSIS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISEASE | TUMOR-CELLS | DEATH | CANCER | LYMPHOCYTES | Cell Communication - immunology | Stem Cells - immunology | Mitosis | Humans | Aneuploidy | Hepatocytes - pathology | Killer Cells, Lymphokine-Activated - pathology | Killer Cells, Natural - pathology | Killer Cells, Natural - immunology | Cell Differentiation | B-Lymphocytes - pathology | T-Lymphocytes - pathology | Tumor Cells, Cultured | Carcinoma - pathology | Carcinoma - immunology | Hepatocytes - immunology | Cell Transformation, Neoplastic - immunology | Killer Cells, Lymphokine-Activated - immunology | Animals | B-Lymphocytes - immunology | Giant Cells - immunology | Neoplasms - immunology | Stem Cells - pathology | T-Lymphocytes - immunology | Mice | Cell Transformation, Neoplastic - pathology | Neoplasms - pathology | Giant Cells - pathology | Surveys | Killer cells | B cells | Stem cells | Cell culture | Biotechnology | Animal models | Carcinoma | Laboratories | Cytology | Stem cell transplantation | Cytotoxicity | Cannibalism | Tissues | Nuclei | Hepatitis | Immunology | Lymphocytes | Etiology | Physiology | Life sciences | Trends | Antigens | Lymphokine-activated killer cells | Tumor cells | Melanoma | Inflammation | Shielding | Tumor cell lines | Immune systems | Monocytes | Hospitals | Surveillance | Lymphocytes B | Cells (biology) | Nuclei (cytology) | Tumors | Cancer | Apoptosis | Index Medicus
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2014, Volume 443, Issue 3, pp. 944 - 949
Journal Article