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PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e58496
Background: Porphyromonas gingivalis is a major pathogen of periodontal disease that affects a majority of adults worldwide. Increasing evidence shows that... 
INTERLEUKIN-8 SECRETION | GRAM-NEGATIVE BACTERIA | HETEROGENEOUS EXPRESSION | CELL ACTIVATION | LIPOPOLYSACCHARIDE-BINDING PROTEIN | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | INNATE HOST-DEFENSE | PERIODONTAL-DISEASE | Gingiva - immunology | Humans | NF-kappa B - immunology | NF-kappa B - metabolism | Porphyromonas gingivalis - chemistry | Toll-Like Receptor 4 - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | MAP Kinase Signaling System - immunology | Toll-Like Receptor 4 - metabolism | Gingiva - metabolism | Gingiva - pathology | Lipid A - immunology | MAP Kinase Signaling System - drug effects | Lipid A - pharmacology | Fibroblasts | Gingiva - microbiology | Lipid A - chemistry | Porphyromonas gingivalis - immunology | Cytokines - biosynthesis | Acylation | Cytokines - immunology | Pathogenesis | Genes | Inflammatory response | Lipids | Cell interactions | Kinases | Tissues | Lipid A | Lipopolysaccharides | Interleukin 6 | Signal transduction | Receptors | Pathways | Granulocyte colony-stimulating factor | E coli | Rodents | Toll-like receptors | Bacteria | Gram-negative bacteria | Interleukin 8 | NF-κB protein | Periodontics | Cytokines | Granulocyte-macrophage colony-stimulating factor | Diabetes mellitus | Inflammation | TLR4 protein | Dentistry | Gene expression | Medicine | Studies | Signaling | Periodontal disease | TLR2 protein | CXCL10 protein | Adults | Transduction | Gingiva | Cardiovascular diseases | Human behavior | Monocyte chemoattractant protein 1
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 04/2017, Volume 118, Issue 4, pp. 819 - 828
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2014, Volume 4, Issue 1, p. 4828
Periodontitis has been implicated as a risk factor for metabolic disorders such as type 2 diabetes, atherosclerotic vascular diseases, and non-alcoholic fatty... 
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | POLYMERASE-CHAIN-REACTION | PORPHYROMONAS-GINGIVALIS | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR-DISEASE | CORONARY-HEART-DISEASE | DIET-INDUCED OBESITY | PERIODONTAL-DISEASE | ADIPOSE-TISSUE | Glucose Intolerance - metabolism | Inflammation - pathology | Ileum - pathology | Metabolic Diseases - pathology | Diabetes Mellitus, Type 2 - microbiology | Periodontitis - microbiology | Glucose Intolerance - microbiology | Ileum - metabolism | Male | Glucose Intolerance - pathology | Diabetes Mellitus, Type 2 - metabolism | Endotoxemia - pathology | Periodontitis - metabolism | Dental Plaque - microbiology | Gingiva - metabolism | Inflammation - metabolism | Microbiota | Gingiva - pathology | Insulin Resistance - physiology | Tight Junction Proteins - metabolism | Disease Models, Animal | Endotoxemia - microbiology | Porphyromonas gingivalis | Inflammation - microbiology | Cytokines - metabolism | Mice, Inbred C57BL | Periodontitis - pathology | Insulin - metabolism | Animals | Metabolic Diseases - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Endotoxemia - metabolism | Gingiva - microbiology | Ileum - microbiology | Metabolic Diseases - microbiology | Liver diseases | Diabetes mellitus | Oral administration | Dental plaque | Inflammation | Gene expression | Insulin | Ileum | Glucose tolerance | Intolerance | Fatty liver | Vascular diseases | Periodontitis | Arteriosclerosis | Rodents | Gingiva | Endotoxemia | Metabolic disorders
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, p. e0201855
Delayed gingival wound healing is widely observed in periodontal patients with diabetes. However, the molecular mechanisms of the impaired function of gingival... 
DIABETES-MELLITUS | CELLS | ACTIVATION | INHIBITION | ANTIOXIDANTS | MULTIDISCIPLINARY SCIENCES | NONSURGICAL PERIODONTAL THERAPY | GROWTH | ACETYL-L-CYSTEINE | EXPRESSION | INSIGHTS | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Oxidative Stress - physiology | Male | RNA, Messenger - metabolism | Cell Movement - physiology | Gingiva - injuries | Gingiva - metabolism | Gingiva - pathology | Gingiva - drug effects | Superoxide Dismutase-1 - metabolism | Adult | Female | Wound Healing - drug effects | Cell Survival - physiology | Fibroblasts - metabolism | Cell Survival - drug effects | Glucose - adverse effects | Cell Proliferation - physiology | Cells, Cultured | Antioxidants - pharmacology | Fibroblasts - pathology | Cell Movement - drug effects | Fibroblasts - drug effects | Acetylcysteine - pharmacology | Glucose - metabolism | Aged | Cell Proliferation - drug effects | Wound Healing - physiology | Oxidative Stress - drug effects | Oxidative stress | Toxicity | Acetylcysteine | Cytotoxicity | Superoxide dismutase | Glucose | Kinases | Assaying | Antioxidants | Angiogenesis | Hyperglycemia | Heme | Fibroblasts | Tumor necrosis factor-TNF | Periodontics | Wound healing | Lactase | Diabetes mellitus | Heme oxygenase (decyclizing) | Superoxide | Inflammation | Dentistry | Metabolism | Gene expression | Patients | Tetrazolium salt | Oxygenase | Molecular modelling | Antibiotics | N-Acetyl-L-cysteine | Diabetes | Gingiva | Viability | Cell migration | Apoptosis
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 2996 - 3005
Gingiva fibroblasts express 12 chemokine receptors. Fourteen chemokines were used to study proliferation (top), migration (middle), and secretion of wound... 
cytokine | migration | chemokine | gingiva | proliferation | PERIODONTAL-DISEASES | PHYSIOLOGY | PHENOTYPIC DIFFERENCES | KERATINOCYTES | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | HUMAN GINGIVAL FIBROBLASTS | GROWTH-FACTOR | ACCUMULATION | EXPRESSION | SKIN INFLAMMATION | Fibroblasts - secretion | Hepatocyte Growth Factor - secretion | Humans | Dose-Response Relationship, Drug | Receptors, CCR4 - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Gingiva - metabolism | Gingiva - pathology | Gingiva - drug effects | Gingiva - secretion | Interleukin-6 - metabolism | Wound Healing - drug effects | Fibroblasts - metabolism | Chemokines, CC - pharmacology | Fibroblasts - pathology | Interleukin-6 - secretion | Receptors, CCR4 - agonists | Hepatocyte Growth Factor - metabolism | Cell Movement - drug effects | Receptors, CCR3 - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Ligands | Cell Proliferation - drug effects | Tissue Inhibitor of Metalloproteinase-1 - secretion | Receptors, CCR3 - agonists | Cell Line, Transformed | Wound healing | Flow cytometry | CXCL12 protein | Leukocyte migration | Mucosa | CXCL13 protein | CX3CR1 protein | Stimulation | Tissue inhibitor of metalloproteinase 1 | Interleukin 6 | CCL22 protein | Receptors | CXCR5 protein | Fibroblasts | CCL20 protein | Activation analysis | Secretion | CXCR2 protein | Chemokine receptors | CXCR4 protein | CCR6 protein | Cytometry | Healing | CXCL11 protein | Gingiva | CCR9 protein | Chemokines | Cell migration | Index Medicus | Original | Original s
Journal Article