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Molecules (Basel, Switzerland), ISSN 1420-3049, 01/2011, Volume 16, Issue 2, pp. 1021 - 1043
..., Konkuk University, Chungju 380-701, Korea 2 Department of Pharmacology, Brain Science and Engineering Institute, CMRI, Kyungpook National University School... 
Neuroprotection | Neuroinflammation | Natural products | Neurodegeneration | Microglia | Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Encephalitis - prevention & control | Neuroprotective Agents - therapeutic use | Plant Extracts - chemistry | Stilbenes - therapeutic use | Biological Products - pharmacology | Fatty Alcohols - therapeutic use | Ginsenosides - pharmacology | Stilbenes - pharmacology | Benzyl Alcohols - therapeutic use | Biphenyl Compounds - therapeutic use | Ginsenosides - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Fatty Alcohols - pharmacology | Microglia - physiology | Neuroprotective Agents - pharmacology | Biphenyl Compounds - pharmacology | Encephalitis - drug therapy | Catechols - therapeutic use | Nerve Degeneration - prevention & control | Benzyl Alcohols - pharmacology | Resveratrol | Catechols - pharmacology | Catechin - therapeutic use | Glucosides - therapeutic use | Molecular Structure | Catechin - pharmacology | Biological Products - therapeutic use | Glucosides - pharmacology | Curcumin - therapeutic use | Microglia - drug effects | Curcumin - pharmacology | Phenyl Ethers - pharmacology | Nerve Degeneration - pathology | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Phenyl Ethers - therapeutic use | Catechin - analogs & derivatives | Index Medicus | neurodegeneration | natural products | neuroinflammation | microglia | neuroprotection
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2009, Volume 29, Issue 41, pp. 12982 - 12993
Estrogen, in addition to its genomic effects in brain, causes rapid and reversible changes to synaptic operations. We report here that these acute actions are... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Estradiol - analogs & derivatives | Synaptic Transmission - physiology | Age Factors | Rats, Long-Evans | Actins - metabolism | Neuronal Plasticity - drug effects | Estrogen Antagonists - pharmacology | Male | Neurons - cytology | Ginsenosides - pharmacology | Excitatory Postsynaptic Potentials - drug effects | Neural Pathways - physiology | Patch-Clamp Techniques - methods | Dose-Response Relationship, Drug | Excitatory Postsynaptic Potentials - physiology | Sapogenins - pharmacology | Neuronal Plasticity - physiology | Long-Term Potentiation - drug effects | Dendritic Spines - drug effects | Neurons - physiology | Female | Synaptic Transmission - drug effects | Estrogens - metabolism | Neurons - drug effects | Thiazolidines - pharmacology | Electric Stimulation - methods | Estradiol - pharmacology | Phenols - pharmacology | Animals, Newborn | Dendritic Spines - physiology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Ovariectomy - methods | Biophysics | Hippocampus - cytology | Rats, Sprague-Dawley | GABA Antagonists - pharmacology | Microscopy, Confocal | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Animals | Analysis of Variance | Signal Transduction - drug effects | Cytoskeleton - metabolism | Picrotoxin - pharmacology | Oxazoles - pharmacology | In Vitro Techniques | Index Medicus
Journal Article
Clinical and experimental pharmacology & physiology, ISSN 1440-1681, 01/2006, Volume 33, Issue 1-2, pp. 27 - 32
Journal Article
Journal Article
International journal of molecular sciences, ISSN 1661-6596, 09/2014, Volume 15, Issue 9, pp. 15426 - 15442
Lethal autophagy is a pathway leading to neuronal death caused by transient global ischemia. In this study, we examined the effect of Ginsenoside Rb1 (GRb1) on... 
Autophagy | Ginsenoside Rb1 | OGD | Transient global ischemia | PI3K/Akt | Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Neuroprotective Agents - therapeutic use | Oxygen - pharmacology | Reperfusion Injury - etiology | Microtubule-Associated Proteins - genetics | Rats, Wistar | Humans | Neuroprotective Agents - antagonists & inhibitors | Reperfusion Injury - drug therapy | Male | Ginsenosides - pharmacology | Autophagy - drug effects | Ginsenosides - therapeutic use | CA1 Region, Hippocampal - pathology | Neurons - ultrastructure | Neuroprotective Agents - pharmacology | Nerve Tissue Proteins - biosynthesis | Apoptosis Regulatory Proteins - genetics | Neurons - metabolism | Phytotherapy | Chromones - pharmacology | Neurons - drug effects | Neuroblastoma - pathology | Apoptosis Regulatory Proteins - biosynthesis | Beclin-1 | Ginsenosides - antagonists & inhibitors | Morpholines - pharmacology | Rats | Glucose - pharmacology | Proto-Oncogene Proteins c-akt - physiology | Random Allocation | Enzyme Activation - drug effects | Microtubule-Associated Proteins - biosynthesis | Nerve Tissue Proteins - genetics | CA1 Region, Hippocampal - blood supply | Up-Regulation - drug effects | Animals | Reperfusion Injury - prevention & control | Signal Transduction - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Cell Line, Tumor | Phosphatidylinositol 3-Kinases - physiology | Proteins | Neurons | Cells | Index Medicus | autophagy | transient global ischemia
Journal Article
Food and chemical toxicology, ISSN 0278-6915, 09/2017, Volume 107, Issue Pt A, pp. 362 - 372
The use of Panax ginseng and Panax quinquefolius in traditional Chinese medicine dates back to about 5000 years ago thanks to its several beneficial and... 
Medicinal plants | Herbal products | Compound K | Free radicals | Heme oxygenase | Ginsenosides | Food Science & Technology | Toxicology | Life Sciences & Biomedicine | Science & Technology | Plant Extracts - chemistry | Animals | Panax - toxicity | Plant Extracts - toxicity | Panax - chemistry | Panax - classification | Humans | Plant Roots - chemistry | Plant Roots - toxicity | Ginsenosides - chemistry | Ginsenosides - toxicity | Medical research | Metabolites | Nitric oxide | Medicine, Botanic | Heme | Medicine, Experimental | Pharmacology | Medicine, Herbal | Diabetes | Free radicals (Chemistry) | Dementia | Index Medicus | AUC, area under the curve | PI3K, phosphoinositide 3-kinase | Cmax, peak plasma concentration | GLUT, glucose transporter | ChAT, choline acetyl transferase | LD, lethal dose | PPD, protopanaxadiol | NO, nitric oxide | MMSE, mini-mental status examination | HO, heme oxygenase | R, ischemia | 2, half-life | reperfusion | Tmax, time to reach the Cmax | DA, dopamine | COX-2, cyclooxygenase-2 | HUVEC, human umbilical vein endothelial cells | Ser, serine | eNOS, endothelial nitric oxide synthase | ADAS-Cog, Alzheimer's disease assessment scale-cognitive subscale | PPT, protopanaxatriol | h, hour(s) | CBS, cystathionine-β-synthase | CGL, cystathionine-γ-lyase | Nrf2, nuclear factor-erythroid 2-related factor | ARI, acute respiratory illness | AD, Alzheimer's disease | IL, interleukin | iNOS, inducible nitric oxide synthase | Bid, BH3 interacting-domain death agonist
Journal Article