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1. Full Text Cross-ancestry genome-wide association analysis of corneal thickness strengthens link between complex and Mendelian eye diseases
by Iglesias, Aiana I and Mishra, Aniket and Vitart, Veronique and Bykhovskaya, Yelena and Höhn, René and Springelkamp, Henriët and Cuellar-Partida, Gabriel and Gharahkhani, Puya and Bailey, Jessica N. Cooke and Willoughby, Colin E and Li, Xiaohui and Yazar, Seyhan and Nag, Abhishek and Khawaja, Anthony P and Polašek, Ozren and Siscovick, David and Mitchell, Paul and Tham, Yih Chung and Haines, Jonathan L and Kearns, Lisa S and Hayward, Caroline and Shi, Yuan and van Leeuwen, Elisabeth M and Taylor, Kent D and Wang, Jie Jin and Rochtchina, Elena and Attia, John and Scott, Rodney and Holliday, Elizabeth G and Baird, Paul N and Xie, Jing and Inouye, Michael and Viswanathan, Ananth and Sim, Xueling and Bonnemaijer, Pieter and Rotter, Jerome I and Martin, Nicholas G and Zeller, Tanja and Mills, Richard A and Staffieri, Sana E and Jonas, Jost B and Schmidtmann, Irene and Boutin, Thibaud and Kang, Jae H and Lucas, Sionne E. M and Wong, Tien Yin and Beutel, Manfred E and Wilson, James F and Allingham, R. Rand and Brilliant, Murray H and Budenz, Donald L and Christen, William G and Fingert, John and Friedman, David S and Gaasterland, Douglas and Gaasterland, Terry and Hauser, Michael A and Kraft, Peter and Lee, Richard K and Lichter, Paul R and Liu, Yutao and Loomis, Stephanie J and Moroi, Sayoko E and Pericak-Vance, Margaret A and Realini, Anthony and Richards, Julia E and Schuman, Joel S and Scott, William K and Singh, Kuldev and Sit, Arthur J and Vollrath, Douglas and Weinreb, Robert N and Wollstein, Gadi and Zack, Donald J and Zhang, Kang and Donnelly, Peter and Barroso, Ines and Blackwell, Jenefer M and Bramon, Elvira and Brown, Matthew A and Casas, Juan P and Corvin, Aiden and Deloukas, Panos and Duncanson, Auey and Jankowski, Janusz and Markus, Hugh S and Mathew, Christopher G and Palmer, Colin N. A and Plomin, Robert and Rautanen, Anna and Sawcer, Stephen J and Trembath, Richard C and Wood, Nicholas W and Spencer, Chris C. A and Band, Gavin and Bellenguez, C. line and Freeman, Colin and Hellenthal, Garrett and Giannoulatou, Eleni and Pirinen, Matti and ... and Blue Mountains Eye Study-GWAS Grp and NEIGHBORHOOD Consortium and Wellcome Trust Case Control Consor and Blue Mountains Eye Study—GWAS group and Wellcome Trust Case Control Consortium 2 (WTCCC2)
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 1864 - 11
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lumican - metabolism | Glaucoma, Open-Angle - genetics | Humans | Corneal Diseases - metabolism | Corneal Dystrophies, Hereditary - ethnology | Corneal Diseases - genetics | Ehlers-Danlos Syndrome - genetics | Corneal Dystrophies, Hereditary - pathology | Loeys-Dietz Syndrome - metabolism | Mendelian Randomization Analysis | Fibrillin-1 - metabolism | Cornea - pathology | Keratoconus - pathology | Corneal Diseases - pathology | Decorin - genetics | Gene Expression | Eye Diseases, Hereditary - pathology | Lumican - genetics | ADAMTS Proteins - metabolism | Proteoglycans - metabolism | Ehlers-Danlos Syndrome - ethnology | European Continental Ancestry Group | Myopia - pathology | Cornea - metabolism | Myopia - ethnology | Loeys-Dietz Syndrome - pathology | Corneal Diseases - ethnology | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Cornea - abnormalities | Quantitative Trait Loci | Proteoglycans - genetics | ADAMTS Proteins - genetics | Transforming Growth Factor beta2 - metabolism | Corneal Dystrophies, Hereditary - genetics | Glaucoma, Open-Angle - pathology | Keratoconus - metabolism | Loeys-Dietz Syndrome - ethnology | Glaucoma, Open-Angle - ethnology | Myopia - metabolism | Ehlers-Danlos Syndrome - pathology | Marfan Syndrome - ethnology | Keratoconus - genetics | Glaucoma, Open-Angle - metabolism | Fibrillin-1 - genetics | Eye Diseases, Hereditary - ethnology | Genome-Wide Association Study | Quantitative Trait, Heritable | Corneal Dystrophies, Hereditary - metabolism | Marfan Syndrome - genetics | Eye Diseases, Hereditary - genetics | Myopia - genetics | Asian Continental Ancestry Group | Keratoconus - ethnology | Ehlers-Danlos Syndrome - metabolism | Loeys-Dietz Syndrome - genetics | Polymorphism, Single Nucleotide | Transforming Growth Factor beta2 - genetics | Genome, Human | Decorin - metabolism | Eye Diseases, Hereditary - metabolism | Glaucoma | Cornea | Genes | Myopia | Association analysis | Genomes | Gene expression | Tissues | Connective tissues | Keratoconus | Collagen | Eye diseases | Extracellular matrix | Index Medicus
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2. Full Text Mechanistic basis of Rho GTPase-induced extracellular matrix synthesis in trabecular meshwork cells
by Padmanabhan P. Pattabiraman and Ponugoti Vasantha Rao
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 03/2010, Volume 298, Issue 3, pp. 749 - 763
Serum response factor | Actomyosin | Extracellular signal-regulated kinases | Fibronectin | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Serum Response Factor - genetics | Lysophospholipids - metabolism | Fibronectins - biosynthesis | Phosphorylation | Extracellular Matrix Proteins - biosynthesis | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Extracellular Matrix - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Recombinant Fusion Proteins - metabolism | Aqueous Humor - metabolism | RNA Interference | Adenoviridae - genetics | Laminin - biosynthesis | Trabecular Meshwork - cytology | Transduction, Genetic | Serum Response Factor - metabolism | Cells, Cultured | Actomyosin - metabolism | Trabecular Meshwork - enzymology | Tenascin - biosynthesis | Mechanotransduction, Cellular | Stress Fibers - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Cytoskeleton - metabolism | Myosin Light Chains - metabolism | Enzyme Activation | Genetic Vectors | Mitogen-Activated Protein Kinase 1 - metabolism | Fibronectins | Physiological aspects | Extracellular matrix | Research | Guanosine triphosphatase | Protein kinases | Glaucoma | Cytoskeleton | Kinases | Ribonucleic acid--RNA | Risk factors | Cells | Index Medicus | Extracellular Matrix, Cell Interactions | extracellular signal-regulated kinases | serum response factor | fibronectin | actomyosin
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3. Full Text Glaucomatous MYOC mutations activate the IL-1/NF-kappa B inflammatory stress response and the glaucoma marker SELE in trabecular meshwork cells
by Itakura, Tatsuo and Peters, Donna M and Fini, M. Elizabeth
Molecular vision, ISSN 1090-0535, 09/2015, Volume 21, pp. 1071 - 1084
Biochemistry & Molecular Biology | Ophthalmology | Life Sciences & Biomedicine | Science & Technology | Cytoskeletal Proteins - genetics | Glaucoma, Open-Angle - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Glycoproteins - metabolism | Glaucoma, Open-Angle - pathology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Actins - genetics | E-Selectin - genetics | Forkhead Transcription Factors - metabolism | Interleukin-1beta - metabolism | Cytoskeletal Proteins - metabolism | Glaucoma, Open-Angle - metabolism | Lentivirus - genetics | Eye Proteins - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Trabecular Meshwork - pathology | Mutagenesis, Site-Directed | Genetic Vectors - chemistry | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | E-Selectin - metabolism | Inflammation | Fibroblasts - pathology | Forkhead Transcription Factors - genetics | Eye Proteins - metabolism | NF-kappa B - genetics | Models, Biological | Trabecular Meshwork - metabolism | Forkhead Box Protein O1 | Primary Cell Culture | Transforming Growth Factor beta2 - genetics | Endothelial Cells - pathology | Cell Line, Transformed | Amino Acid Substitution | Index Medicus
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4. Full Text Neurodegenerative and inflammatory pathway components linked to TNF-α/TNFR1 signaling in the glaucomatous human retina
by Yang, Xiangjun and Luo, Cheng and Cai, Jian and Powell, David W and Yu, Dahai and Kuehn, Markus H and Tezel, Gülgün
Investigative ophthalmology & visual science, ISSN 0146-0404, 10/2011, Volume 52, Issue 11, pp. 8442 - 8454
Life Sciences & Biomedicine | Ophthalmology | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Retina - metabolism | Humans | STAT Transcription Factors - metabolism | NF-kappa B - metabolism | Janus Kinases - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Nerve Degeneration - metabolism | Receptors, Death Domain - metabolism | Tandem Mass Spectrometry | DNA Methylation | Caspases - metabolism | Optic Nerve Diseases - metabolism | Aged, 80 and over | Chromatography, Liquid | Glaucoma - metabolism | Fluorescent Antibody Technique, Indirect | Computational Biology | DNA Primers | Blotting, Western | Eye Proteins - metabolism | Proteomics | Signal Transduction - physiology | Aged | Polymorphism, Single Nucleotide | Index Medicus
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5. Full Text 661W is a retinal ganglion precursor-like cell line in which glaucoma-associated optineurin mutants induce cell death selectively
by Sayyad, Zuberwasim and Sirohi, Kapil and Radha, Vegesna and Swarup, Ghanshyam
Scientific reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 16855 - 13
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Biomarkers - metabolism | Cell Line | Mutagenesis, Site-Directed | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Thiophenes - pharmacology | Nuclear Proteins - metabolism | Transcription Factor Brn-3C - metabolism | Pyrimidines - pharmacology | Neural Stem Cells - cytology | Retinal Ganglion Cells - metabolism | Nerve Tissue Proteins - metabolism | Thy-1 Antigens - metabolism | Retinal Ganglion Cells - cytology | Tubulin - metabolism | Animals | Eye Proteins - metabolism | Cell Differentiation - drug effects | Glaucoma - pathology | Mice | Eye Proteins - genetics | Glaucoma - metabolism | Neural Stem Cells - metabolism | Staurosporine - pharmacology | Glaucoma | Retinal ganglion cells | Nestin | Tubulin | Cell death | Retina | Amyotrophic lateral sclerosis | Photoreceptors | Brn-3 protein | Synuclein | Apoptosis | Index Medicus
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6. Melanopsin-expressing retinal ganglion cells in aging and disease
by Esquiva, Gema and Hannibal, Jens
Histology and histopathology, ISSN 0213-3911, 12/2019, Volume 34, Issue 12, pp. 1299 - 1311
Pathology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Humans | Retinitis Pigmentosa - metabolism | Retinal Diseases - metabolism | Seasonal Affective Disorder - metabolism | Circadian Rhythm | Retinal Ganglion Cells - metabolism | Diabetic Retinopathy - metabolism | Migraine Disorders - metabolism | Retina - physiology | Macular Degeneration - metabolism | Animals | Photophobia - metabolism | Retina - cytology | Light | Rod Opsins - metabolism | Alzheimer Disease - metabolism | Aging | Aged | Mice | Parkinson Disease - metabolism | Glaucoma - metabolism
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7. Full Text Rapamycin and fasting sustain autophagy response activated by ischemia/reperfusion injury and promote retinal ganglion cell survival
by Russo, Rossella and Varano, Giuseppe Pasquale and Adornetto, Annagrazia and Nazio, Francesca and Tettamanti, Gianluca and Girardello, Rossana and Cianfanelli, Valentina and Cavaliere, Federica and Morrone, Luigi Antonio and Corasaniti, Maria Tiziana and Cecconi, Francesco and Bagetta, Giacinto and Nucci, Carlo
Cell death & disease, ISSN 2041-4889, 10/2018, Volume 9, Issue 10, pp. 981 - 18
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Survival - drug effects | Retina - metabolism | TOR Serine-Threonine Kinases - metabolism | Mice, Inbred C57BL | Male | Autophagy - physiology | Autophagosomes - metabolism | Retinal Ganglion Cells - metabolism | Autophagy-Related Protein 12 - metabolism | Sirolimus - pharmacology | Autophagy - drug effects | Sirolimus - administration & dosage | Animals | Adenylate Kinase - metabolism | Autophagy-Related Protein 5 - metabolism | Female | Mice | Fasting - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Beclin-1 - metabolism | Reperfusion Injury - metabolism | Sequestosome-1 Protein - metabolism | Glaucoma | Occlusion | Diabetic retinopathy | Retinopathy | Fasting | Cell survival | Diabetes mellitus | Homeostasis | Retina | Rapamycin | Autophagy | Retinal ganglion cells | Reperfusion | Ischemia | Neurodegeneration | Hypoxia | Phagocytosis | Index Medicus
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8. Full Text Role of the autotaxin-lysophosphatidic acid axis in glaucoma, aqueous humor drainage and fibrogenic activity
by Ho, Leona T.Y and Osterwald, Anja and Ruf, Iris and Hunziker, Daniel and Mattei, Patrizio and Challa, Pratap and Vann, Robin and Ullmer, Christoph and Rao, Ponugoti Vasanth
Biochimica et biophysica acta. Molecular basis of disease, ISSN 0925-4439, 01/2020, Volume 1866, Issue 1, pp. 165560 - 165560
Glaucoma | Trabecular meshwork | Aqueous humor | Autotaxin | Intraocular pressure | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Lysophospholipids - metabolism | Collagen Type I - metabolism | Phosphoric Diester Hydrolases - metabolism | Humans | Mice, Inbred C57BL | Intraocular Pressure - physiology | Male | NF-kappa B - metabolism | Ocular Hypertension - metabolism | Smad3 Protein - metabolism | Fibronectins - metabolism | Aqueous Humor - metabolism | Animals | Trabecular Meshwork - metabolism | Drainage - methods | Female | Mice | Glaucoma - metabolism | Connective Tissue Growth Factor - metabolism | Index Medicus | intraocular pressure | aqueous humor | glaucoma | trabecular meshwork
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