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PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 8, p. e105931
In order to test the hypothesis that in primary open angle glaucoma (POAG), an important cause of irreversible blindness, a spreading of neurodegeneration... 
OPEN-ANGLE GLAUCOMA | OPTIC-NERVE | RESTING-STATE NETWORKS | NERVE-FIBER LAYER | PATHWAY | MULTIDISCIPLINARY SCIENCES | SPATIAL STATISTICS | DIFFUSION | REGISTRATION | WHITE-MATTER ABNORMALITIES | VOXEL-BASED MORPHOMETRY | Cerebellar Cortex - physiopathology | White Matter - physiopathology | Visual Pathways - physiopathology | Humans | Middle Aged | Glaucoma, Open-Angle - pathology | Male | Case-Control Studies | Somatosensory Cortex - pathology | Atrophy | Middle Cerebellar Peduncle - pathology | Multimodal Imaging | Pyramidal Tracts - physiopathology | Visual Cortex - pathology | Adult | Female | Visual Pathways - pathology | Anterior Thalamic Nuclei - pathology | Pyramidal Tracts - pathology | Visual Cortex - physiopathology | Glaucoma, Open-Angle - physiopathology | Somatosensory Cortex - physiopathology | Anterior Thalamic Nuclei - physiopathology | Hippocampus - pathology | Middle Cerebellar Peduncle - physiopathology | White Matter - pathology | Magnetic Resonance Imaging | Cerebellar Cortex - pathology | Gray Matter - physiopathology | Gray Matter - pathology | Brain Mapping | Hippocampus - physiopathology | Glaucoma | Brain | Care and treatment | Anisotropy | Blindness | Cerebellum | Neurosciences | Visual cortex | Memory | Radiation | Substantia grisea | Visual system | Neurodegeneration | Surgery | Eye (anatomy) | Thalamus | Visual fields | Age | Optic nerve | Visual field | Abnormalities | Pyramidal tracts | Substantia alba | Patients | Medicine | Magnetic resonance imaging | Neural networks | Short term memory | Hippocampus | Structure-function relationships
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1303, pp. 131 - 143
Abstract Transsynaptic degeneration has been implicated in patients with primary open angle glaucoma (POAG) and animal models of chronic intraocular... 
Neurology | Visual system | Neural plasticity | Glial fibrillary acidic protein (GFAP) | Ischemia | Acute intraocular hypertension | Superior colliculus (SC) | Transsynaptic degeneration | Neuronal death | Degenerative disease | Glia and other non-neuronal cells | Lateral geniculate nucleus (LGN) | Glutamine synthetase (GS) | OPTIC-NERVE | EXPERIMENTAL GLAUCOMA | ASTROCYTES | NEUROSCIENCES | LATERAL GENICULATE-NUCLEUS | PARVOCELLULAR LAYERS | SUPERIOR COLLICULUS | RETINAL GANGLION-CELLS | FIBRILLARY ACIDIC PROTEIN | MULLER CELL | BRAIN | Rats, Wistar | Visual Pathways - physiopathology | Neuroglia - pathology | Nerve Degeneration - physiopathology | Retinal Degeneration - etiology | Gliosis - physiopathology | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | Geniculate Bodies - pathology | Geniculate Bodies - physiopathology | Retinal Ganglion Cells - pathology | Gliosis - pathology | Female | Visual Pathways - pathology | Glutamate-Ammonia Ligase - metabolism | Glaucoma - physiopathology | Gliosis - etiology | Disease Models, Animal | In Situ Nick-End Labeling | Acute Disease | Retina - physiopathology | Rats | Retinal Degeneration - physiopathology | Glaucoma - complications | Disease Progression | Nerve Degeneration - pathology | Superior Colliculi - physiopathology | Animals | Superior Colliculi - pathology | Axons - pathology | Glaucoma - pathology | Apoptosis - physiology | Nerve Degeneration - etiology | Retinal Degeneration - pathology | Early Diagnosis | Retina - pathology
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0131921
Damage to the eye from blast exposure can occur as a result of the overpressure air-wave (primary injury), flying debris (secondary injury), blunt force trauma... 
EYE | GLOBE | COMMOTIO-RETINAE | COMBAT OCULAR TRAUMA | MULTIDISCIPLINARY SCIENCES | INJURY | ETHANOL | MICE | MODEL | GLAUCOMA | Microglia - metabolism | Optic Nerve - pathology | Wounds, Nonpenetrating - complications | Blindness - physiopathology | Oxidative Stress - physiology | Nerve Degeneration - physiopathology | Blindness - pathology | Blast Injuries - complications | Eye Injuries - pathology | Microglia - pathology | Mice, Inbred DBA | Optic Nerve - physiopathology | Disease Models, Animal | Electroretinography | Eye Injuries - complications | Mice, Inbred C57BL | Retina - physiopathology | Olfactory Mucosa - physiopathology | Explosions | Nerve Degeneration - pathology | Blast Injuries - pathology | Eye Injuries - physiopathology | Animals | Olfactory Mucosa - pathology | Mice | Wounds, Nonpenetrating - pathology | Nerve Degeneration - etiology | Wounds, Nonpenetrating - physiopathology | Blindness - etiology | Retina - pathology | Blast Injuries - physiopathology | Glaucoma | Oxidative stress | Traumatic brain injury | Laboratories | Flight | Retina | Blast | Acuity | Eye | Wave propagation | Neurodegeneration | Eye injuries | Rodents | Nerves | Burns (injuries) | Optic nerve | Overpressure | Quaternary | Mortality | Visual acuity | Histology | Housing | Epithelium | Trauma | Studies | Brain research | Optical Coherence Tomography | Damage detection | Cell death | Photoreceptors | Olfactory epithelium | Apoptosis
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2009, Volume 5, Issue 4, pp. 348 - 360
Abstract Infection with several important pathogens could constitute risk factors for cognitive impairment, dementia, and Alzheimer's disease (AD) in... 
Neurology | Bacterial infection | Vascular risk factor | Vaccination | Inflammation | Alzheimer's disease | Viral infection | Dementia | DETECT CHLAMYDIA-PNEUMONIAE | HUMAN BRAIN-TISSUE | MILD COGNITIVE IMPAIRMENT | CLINICAL NEUROLOGY | APOLIPOPROTEIN-E | HERPES-SIMPLEX-VIRUS | OPEN-ANGLE GLAUCOMA | POLYMERASE-CHAIN-REACTION | HELICOBACTER-PYLORI INFECTION | VARICELLA-ZOSTER-VIRUS | EPSTEIN-BARR-VIRUS | Brain - microbiology | Humans | Central Nervous System Infections - physiopathology | Encephalitis, Viral - complications | Vaccines - therapeutic use | Virus Diseases - microbiology | Virus Diseases - complications | Encephalitis - physiopathology | Virus Diseases - physiopathology | Alzheimer Disease - prevention & control | Central Nervous System Infections - microbiology | Central Nervous System Bacterial Infections - complications | Central Nervous System Bacterial Infections - microbiology | Encephalitis - complications | Alzheimer Disease - microbiology | Alzheimer Disease - physiopathology | Brain - physiopathology | Encephalitis, Viral - physiopathology | Risk Factors | Central Nervous System Bacterial Infections - physiopathology | Central Nervous System Infections - complications | Animals | Encephalitis, Viral - microbiology | Brain - pathology | Encephalitis - microbiology | Development and progression | Drug therapy | Health aspects
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