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Nature genetics, ISSN 1546-1718, 2016, Volume 48, Issue 7, pp. 768 - 776
Glioblastoma (GBM) is the most common and aggressive primary brain tumor. To better understand how GBM evolves, we analyzed longitudinal genomic and... 
GENE FUSIONS | TGF-BETA | CANCER GENOMICS | LANDSCAPE | GLIOMA | GENETICS & HEREDITY | GENOMIC ALTERATIONS | MSH6 MUTATIONS | SEQUENCING DATA | BRAIN-TUMORS | TEMOZOLOMIDE | Cell Proliferation | Dacarbazine - therapeutic use | Genomics | Humans | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Gene Expression Regulation, Neoplastic | Transcriptome | Neoplasm Recurrence, Local - drug therapy | Clonal Evolution - genetics | Neoplasm Recurrence, Local - pathology | Neoplasm Grading | Glioblastoma - genetics | Tumor Suppressor Proteins - genetics | Dacarbazine - analogs & derivatives | Brain Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Survival Rate | Brain Neoplasms - drug therapy | Mutation - genetics | Antineoplastic Agents, Alkylating - therapeutic use | Latent TGF-beta Binding Proteins - genetics | DNA Modification Methylases - genetics | Transforming Growth Factor beta - genetics | Glioblastoma - pathology | Neoplasm Recurrence, Local - genetics | Biomarkers, Tumor - genetics | Temozolomide | Glioblastoma - drug therapy | Longitudinal Studies | Molecular targeted therapy | Care and treatment | Gene mutations | Development and progression | Genetic aspects | Genetic transcription | Glioblastoma multiforme | Health aspects | Methods | Brain cancer | Genomes | Gene expression | Cancer therapies | Studies | Archives & records | DNA methylation | Mathematical models | Mutation | Deoxyribonucleic acid--DNA | Tumors | Cancer
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 2, p. e8918
Background: Glioblastoma multiforme (GBM) is the most common and aggressive type of brain tumor in humans and the first cancer with comprehensive genomic... 
GLIOMAS | PROTEIN-INTERACTION NETWORKS | HUMAN-DISEASE | BIOLOGICAL NETWORKS | METABOLIC NETWORKS | MULTIDISCIPLINARY SCIENCES | FUNCTIONAL MODULES | GENES | CANCER GENOME | PIKE-A | INTEGRATED ANALYSIS | Genetic Predisposition to Disease | Humans | Brain Neoplasms - genetics | Signal Transduction - genetics | Gene Regulatory Networks | GTP-Binding Proteins - genetics | Tumor Suppressor Protein p53 - genetics | Phosphatidylinositol 3-Kinases - genetics | Algorithms | Glioblastoma - genetics | Retinoblastoma Protein - genetics | Models, Genetic | Software | GTPase-Activating Proteins - genetics | Mutation | Genes | Brain tumors | Genomics | Natural language interfaces | Genomes | Protein-protein interactions | Technology application | Mobile communication systems | Wireless communication systems | Language processing | Analysis | Genetic aspects | Computational linguistics | Tumor proteins | Health aspects | Protein kinases | Cancer | Brain | Copy number | Modules | p53 Protein | Brain cancer | Glioblastoma | Biology | Metastasis | Software development tools | Kinases | Proteins | Signal transduction | Alterations | Pathways | Cellular communication | Network analysis | Phylogenetics | Bioinformatics | Deoxyribonucleic acid--DNA | Cartography | Nucleotide sequence | Retinoblastoma protein | Metabolism | Gene expression | Glioblastoma multiforme | 1-Phosphatidylinositol 3-kinase | Endothelium | Signaling | Hypotheses | Passengers | Protein interaction | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Nature (London), ISSN 1476-4687, 2008, Volume 455, Issue 7216, pp. 1061 - 1068
Journal Article
European journal of human genetics : EJHG, ISSN 1018-4813, 01/2013, Volume 21, Issue 1, pp. 55 - 61
...European Journal of Human Genetics (2013) 21, 5561& 2013 Macmillan Publishers Limited All rights reserved 1018-4813/13 http://www.nature.com/EJHG Web End... 
Contractile Proteins/genetics | MutL Protein Homolog 1 | Microsatellite Instability | Humans | Adenosine Triphosphatases/genetics | Child, Preschool | Parotid Neoplasms/complications | Male | Adaptor Proteins, Signal Transducing/genetics | Syndrome | Filamins | Pregnancy | Agenesis of Corpus Callosum/genetics | DNA-Binding Proteins/genetics | Glioblastoma/complications | Female | DNA Repair Enzymes/genetics | Mismatch Repair Endonuclease PMS2 | DNA Repair-Deficiency Disorders/etiology | Malformations of Cortical Development, Group II/genetics | Mutation | Nuclear Proteins/genetics | Child | Microfilament Proteins/genetics | gray matter heterotopia | childhood cancer | agenesis of corpus callosum | biallelic germline mutation | constitutional mismatch repair deficiency syndrome | LYNCH-SYNDROME | BIOCHEMISTRY & MOLECULAR BIOLOGY | GUIDELINES | BIALLELIC MUTATIONS | MSH6 | MALFORMATIONS | GENE | COLORECTAL-CANCER | GENETICS & HEREDITY | EARLY-ONSET | PMS2 | NEUROFIBROMATOSIS | Contractile Proteins - genetics | Malformations of Cortical Development, Group II - pathology | DNA Repair Enzymes - genetics | DNA Repair-Deficiency Disorders - etiology | Glioblastoma - genetics | Agenesis of Corpus Callosum - genetics | Parotid Neoplasms - therapy | Nuclear Proteins - genetics | Microfilament Proteins - genetics | Glioblastoma - diagnosis | DNA-Binding Proteins - genetics | Malformations of Cortical Development, Group II - genetics | Agenesis of Corpus Callosum - pathology | Glioblastoma - therapy | Parotid Neoplasms - complications | Parotid Neoplasms - diagnosis | Adaptor Proteins, Signal Transducing - genetics | Glioblastoma - complications | Adenosine Triphosphatases - genetics | DNA Repair-Deficiency Disorders - genetics | Parotid Neoplasms - genetics | Neuroimaging | Pediatrics | Yeast | Congenital defects | Brain tumors | Brain cancer | Genes | Oncology | Malignancy | Birth | Substantia grisea | MSH6 protein | Mismatch repair | Diagnosis | Children | Hematology | MLH1 protein | MSH2 protein | Patients | Corpus callosum | Hospitals | Genetic recombination | Cancer | Tumors
Journal Article
Acta neuropathologica, ISSN 1432-0533, 2018, Volume 135, Issue 4, pp. 601 - 615
textabstractThe optimal treatment for patients with low-grade glioma (LGG) WHO grade II remains controversial. Overall survival ranges from 2 to over 15 years... 
TMZ | DNA methylation | DDR genes | MGMT-STP27 | Randomized trial | Low-grade glioma | Pathology | Neurosciences | Medicine & Public Health | CELLS | METHYLATION | MGMT | SENSITIVITY | MISMATCH REPAIR | PATHOLOGY | TUMORS | NEUROSCIENCES | CLINICAL NEUROLOGY | TEMOZOLOMIDE | GLIOBLASTOMA | RADIOTHERAPY | EXPRESSION | Epigenesis, Genetic | Humans | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Male | Glioma - genetics | Temozolomide - therapeutic use | Neoplasm Grading | DNA Methylation - radiation effects | Glioma - pathology | Tumor Suppressor Proteins - genetics | Glioma - therapy | Adult | Female | Promoter Regions, Genetic | Discoidin Domain Receptors - genetics | Brain Neoplasms - genetics | Isocitrate Dehydrogenase - genetics | Treatment Outcome | Antineoplastic Agents, Alkylating - therapeutic use | DNA Modification Methylases - genetics | DNA | Progression-Free Survival | Brain Neoplasms - therapy | CpG Islands | DNA Methylation - drug effects | Medical colleges | Medical research | Care and treatment | DNA damage | Genes | Medicine, Experimental | Genetic aspects | Radiotherapy | Methylation | Cancer | Preservation | Brain tumors | Genotoxicity | Cognitive ability | Genomes | Radiation therapy | Survival | Quality of life | Glioma | Temozolomide | Isocitrate dehydrogenase | Deoxyribonucleic acid--DNA | CpG islands | Original Paper
Journal Article
PLoS genetics, ISSN 1553-7404, 2012, Volume 8, Issue 7, p. e1002772
Journal Article
Clinical cancer research, ISSN 1557-3265, 2016, Volume 22, Issue 9, pp. 2301 - 2310
Journal Article