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STEM CELLS, ISSN 1066-5099, 08/2016, Volume 34, Issue 8, pp. 2026 - 2039
Shifting the balance away from tumor‐mediated immune suppression toward tumor immune rejection is the conceptual foundation for a variety of immunotherapy... 
Macrophage migration inhibitory factor | Tumor microenvironment | Immunotherapy | Glioblastoma | MIF | Cancer stem cells | Myeloid‐derived suppressor cells | Tumor immune suppression | Myeloid-derived suppressor cells | GLIOMA | SELF-RENEWAL | RECEPTOR | TUMOR-INITIATING CELLS | BRAIN-TUMORS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | IN-VITRO | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH | HEMATOLOGY | PROMOTES | PROGRESSION | Tumor Microenvironment - drug effects | Cell Survival - drug effects | Neoplastic Stem Cells - secretion | Neoplastic Stem Cells - drug effects | Humans | Mice, Inbred C57BL | Arginase - metabolism | Brain Neoplasms - pathology | Culture Media, Conditioned - pharmacology | Myeloid-Derived Suppressor Cells - drug effects | Macrophage Migration-Inhibitory Factors - secretion | Carcinogenesis - metabolism | Carcinogenesis - pathology | Animals | Brain Neoplasms - immunology | Glioblastoma - immunology | Mice, Nude | Glioblastoma - pathology | Immune Evasion - drug effects | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Female | Myeloid-Derived Suppressor Cells - metabolism | Analysis | Stem cells | T cells | Macrophages | Glioblastoma multiforme | Cancer | Cell proliferation | Populations | Leukocyte migration | Syngeneic grafts | Brain tumors | Stem cell transplantation | Cytotoxicity | Arginase | Lymphocytes T | Suppressor cells | Bearing | Rodents | Mathematical models | Immune system | Proximity | Cell survival | Immune response | Tumor cells | CXCR2 protein | Patients | Survival | Rejection | Depletion | Glioma | Migration inhibitory factor | Antitumor activity | Cell migration | Index Medicus
Journal Article
STEM CELLS, ISSN 1066-5099, 06/2013, Volume 31, Issue 6, pp. 1051 - 1063
Glioblastoma multiforme (GBM) is a life‐threatening brain tumor. Accumulating evidence suggests that eradication of glioma stem‐like cells (GSCs) in GBM is... 
Cancer stem cell | Glioblastoma stem cell | Neural stem cell | Glioblastoma | PLK1 | TARGET | SPECIFICITY | IDENTIFICATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | INHIBITION | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH | HEMATOLOGY | TRANSCRIPTION FACTOR | PROMOTES | EXPRESSION | PROGRESSION | LEUCINE-ZIPPER KINASE | Phosphorylation | Neoplastic Stem Cells - drug effects | Humans | Brain Neoplasms - pathology | Mitosis - genetics | Brain Neoplasms - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Dacarbazine - pharmacology | Forkhead Transcription Factors - metabolism | Dacarbazine - analogs & derivatives | HEK293 Cells | Cell Cycle Proteins - genetics | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Protein-Serine-Threonine Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Neural Stem Cells - drug effects | Protein-Serine-Threonine Kinases - genetics | Brain Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Brain Neoplasms - drug therapy | Forkhead Transcription Factors - genetics | Neural Stem Cells - pathology | Peptides - pharmacology | Up-Regulation - drug effects | Animals | Mitosis - drug effects | Mitosis - physiology | Glioblastoma - pathology | Cell Proliferation - drug effects | Mice | Glioblastoma - drug therapy | Neural Stem Cells - metabolism | Chemotherapy | Gliomas | Analysis | Stem cells | Development and progression | Genetic engineering | Health aspects | Cancer | Kinases | Index Medicus | Cancer Stem Cells
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2011, Volume 108, Issue 11, pp. 4274 - 4280
Glioblastoma (GBM) is the most malignant brain tumor and is highly resistant to intensive combination therapies and anti-VEGF therapies. To assess the... 
Angiogenesis | Myeloid cells | Cell lines | Blood vessels | Antibodies | Cultured cells | Mice | Endothelial cells | Tumors | Cancer | Hypoxia-inducible factor 1 | Mouse model | Glioma | GLIOMAS | VASCULOGENIC MIMICRY | mouse model | BEVACIZUMAB | angiogenesis | MULTIDISCIPLINARY SCIENCES | MOUSE | HYDROXYLATION | TUMORS | STEM-LIKE CELLS | THERAPY | hypoxia-inducible factor 1 | glioma | DIFFERENTIATION | HIF-ALPHA | Endothelial Cells - metabolism | Humans | Spheroids, Cellular - metabolism | Spheroids, Cellular - pathology | Vascular Endothelial Growth Factor A - metabolism | Cell Hypoxia | Xenograft Model Antitumor Assays | Cell Fusion | Receptor, Epidermal Growth Factor - metabolism | Animals | Receptors, Vascular Endothelial Growth Factor - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glioblastoma - pathology | Biomarkers, Tumor - metabolism | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Cell Transdifferentiation | Glioblastoma - metabolism | Neovascularization, Pathologic - metabolism | Tumor Cells, Cultured | Endothelial Cells - pathology | Disease Models, Animal | Transdifferentiation | Cancer cells | Diagnosis | Neovascularization | Research | Properties | Glioblastoma multiforme | Brain | Signal transduction | Transplants & implants | Rodents | Hypoxia | Vascular endothelial growth factor | Cancer therapies | Cells | Animal models | Tumor cells | Brain tumors | Glioblastoma | Cell fusion | Xenografts | Differentiation | glioblastoma cells | recombinase | Hippocampus | Oncogenes | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e91519 - e91519
Glioblastomas (GBMs) are highly aggressive, invasive brain tumors with bad prognosis and unmet medical need. These tumors are heterogeneous being constituted... 
PROGENITOR CELLS | BREAST-CANCER | GROWTH-FACTOR RECEPTOR | MULTIDISCIPLINARY SCIENCES | DRUG DISCOVERY | COMMERCIALLY AVAILABLE ANTIBODIES | CENTRAL-NERVOUS-SYSTEM | GABA(B) RECEPTORS | HUMAN GLIOMA-CELLS | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | Astrocytes - cytology | Receptors, G-Protein-Coupled - metabolism | Humans | Astrocytes - pathology | Fetus - metabolism | Fetus - pathology | Gene Expression Profiling | Molecular Targeted Therapy | Fetus - cytology | Glioblastoma - genetics | Glioblastoma - pathology | Ploidies | Proteomics | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Cell Differentiation | Receptors, G-Protein-Coupled - genetics | Glioblastoma - drug therapy | Chemotherapy | Prognosis | Analysis | Brain tumors | Stem cells | G proteins | Health aspects | Glioblastoma multiforme | Cancer | Brain | Neurosciences | G protein-coupled receptors | Brain cancer | Crosstalk | Glioblastoma | Multiple myeloma | Clinical trials | Cell interactions | Kinases | Cancer therapies | Proteins | Angiogenesis | Receptors | Epidermal growth factor | Physiology | Immunoglobulins | Astrocytes | Invasiveness | Fetuses | Melanoma | Mass spectroscopy | Gene expression | Signaling | Properties (attributes) | Comparative studies | Neural stem cells | Mass spectrometry | Tumors | Index Medicus | Life Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0169932 - e0169932
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, pp. e96239 - e96239
Glioblastoma is the most common and lethal primary brain tumor. Tumor initiation and recurrence are likely caused by a sub-population of glioblastoma stem... 
GLIOMA-CELLS | CANCER-CELLS | GROWTH-FACTOR RECEPTOR | NERVOUS-SYSTEM | IN-VITRO | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | AKT PATHWAY | SPHEROID CULTURES | PRECURSOR CELLS | BRAIN-TUMORS | Neoplastic Stem Cells - cytology | Caspase 9 - genetics | Caspase 9 - metabolism | Humans | Caspase 3 - metabolism | Gene Expression Regulation, Neoplastic | Glycoproteins - metabolism | Peptides - genetics | 3' Untranslated Regions - genetics | Cell Survival - genetics | MicroRNAs - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Flow Cytometry | Peptides - metabolism | Bcl-2-Like Protein 11 | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Caspase 3 - genetics | Apoptosis Regulatory Proteins - genetics | Glioblastoma - metabolism | Membrane Proteins - metabolism | Cell Survival - physiology | Glycoproteins - genetics | Proto-Oncogene Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | Proto-Oncogene Proteins - genetics | AC133 Antigen | Apoptosis Regulatory Proteins - metabolism | Cell Line, Tumor | MicroRNAs - genetics | DNA microarrays | MicroRNA | Analysis | Luciferase | Stem cells | Glioblastoma multiforme | Apoptosis | Brain | Flow cytometry | Brain tumors | Brain cancer | Glioblastoma | Fluorescence | Oncology | Biochemistry | Neurosurgery | Caspase-3 | Cancer therapies | Genetics | Cell survival | MiRNA | Caspase | siRNA | Survival | Neurology | Brain research | Mutagenesis | Ribonucleic acids | Cell number | MicroRNAs | Cell lines | Caspase-9 | Neural stem cells | Annexin V | BIM protein | Cancer | Tumors | Index Medicus
Journal Article