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Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 1, pp. 153 - 162
Journal Article
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 07/2016, Volume 25, Issue 2, pp. 61 - 77
Aims: This preclinical study was aimed at determining whether pharmacological targeting of transcription factor NRF2, a master controller of many homeostatic... 
Original Research Communications | OXIDATIVE STRESS | IN-VITRO | TRANSCRIPTION FACTOR NRF2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | MPTP MOUSE MODEL | ALPHA-SYNUCLEIN | HEME OXYGENASE-1 | AUTOPHAGY | NF-KAPPA-B | EXPRESSION | NEUROPROTECTION | Immunohistochemistry | NF-E2-Related Factor 2 - agonists | Parkinson Disease - drug therapy | RNA, Messenger - metabolism | Autophagy | Brain - metabolism | Gliosis - pathology | Neuroprotective Agents - pharmacology | Dimethyl Fumarate - pharmacology | NF-E2-Related Factor 2 - genetics | Parkinson Disease - metabolism | Neuroprotective Agents - administration & dosage | Disease Models, Animal | Gene Expression | Gliosis - genetics | Parkinson Disease - pathology | Synucleins - genetics | Gliosis - metabolism | RNA, Messenger - genetics | Drug Repositioning | Synucleins - metabolism | Mice, Knockout | Phenotype | Animals | Mice | Dimethyl Fumarate - administration & dosage | Care and treatment | Parkinson's disease | Research | Gene expression | Dimethyl | Neuroprotection | Brain | Therapy | Oxidative stress | Basal ganglia | Multiple sclerosis | Mesencephalon | Toxicity | Substantia nigra | Parkinsons disease | Innovations | Synuclein | Proteins | Neurotoxicity | Autopsy | Movement disorders | Dopamine receptors | Recombinant | Phenotypes | Dopamine | Wound healing | Neurodegenerative diseases | Pharmacology | Inflammation | Disease control | Ganglia | Lewy bodies | Gliosis | Cell death | Phagocytosis | Index Medicus
Journal Article
Journal Article
Journal Article
Stroke, ISSN 0039-2499, 09/2011, Volume 42, Issue 9, pp. 2589 - 2594
Background and Purpose-Activation of Notch worsens ischemic brain damage as antisense knockdown or pharmacological inhibition of the Notch pathway reduces the... 
apoptosis | focal ischemia | brain ischemia | inflammation | neuroregeneration | neuroprotection | CELLS | BRAIN-DAMAGE | PROLIFERATION | DEATH | CLINICAL NEUROLOGY | STROKE | TUMOR-NECROSIS-FACTOR | PERIPHERAL VASCULAR DISEASE | NF-KAPPA-B | UP-REGULATION | Amyloid Precursor Protein Secretases - genetics | Inflammation - pathology | Microglia - metabolism | Coculture Techniques | Brain Ischemia - genetics | Immunity, Innate - genetics | NF-kappa B - metabolism | Brain Ischemia - immunology | Inflammation - metabolism | Cell Nucleus - metabolism | Microglia - pathology | Gliosis - immunology | Active Transport, Cell Nucleus - genetics | Cytokines - genetics | Gliosis - genetics | Mice, Transgenic | Inflammation - therapy | Amyloid Precursor Protein Secretases - metabolism | Cell Nucleus - genetics | Active Transport, Cell Nucleus - immunology | Mice | Receptor, Notch1 - antagonists & inhibitors | Oligopeptides - pharmacology | Receptor, Notch1 - genetics | Cell Nucleus - immunology | Amyloid Precursor Protein Secretases - immunology | Gliosis - therapy | NF-kappa B - immunology | Brain Ischemia - metabolism | Gliosis - pathology | Microglia - immunology | Inflammation Mediators - metabolism | Cytokines - immunology | Inflammation Mediators - immunology | Cell Line | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Brain Ischemia - therapy | Gliosis - metabolism | Receptor, Notch1 - immunology | Inflammation - immunology | Receptor, Notch1 - metabolism | Immunity, Innate - immunology | Animals | NF-kappa B - genetics | Brain Ischemia - pathology | Inflammation - genetics | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Cytokines - biosynthesis | Index Medicus
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 06/2014, Volume 39, Issue 12, pp. 2151 - 2162
Key questions remain regarding the processes governing gliogenesis following central nervous system injury that are critical to understanding both beneficial... 
epilepsy | nestin‐expressing cells | gliosis | cortical injury | Cortical injury | Nestin-expressing cells | Gliosis | Epilepsy | STEM-CELLS | nestin-expressing cells | PAX6 | GLIAL-CELLS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | NESTIN EXPRESSION | NEURAL STEM/PROGENITOR CELLS | ASTROCYTE DYSFUNCTION | SPINAL-CORD-INJURY | REACTIVE GLIOSIS | INTERMEDIATE-FILAMENT | Cicatrix - etiology | Homeodomain Proteins - metabolism | Humans | Middle Aged | Epilepsy - surgery | Male | Gliosis - physiopathology | Epilepsy - physiopathology | Glial Fibrillary Acidic Protein - metabolism | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Gliosis - pathology | Young Adult | Neurophysiological Monitoring - adverse effects | Microglia - physiology | Time Factors | Cicatrix - physiopathology | Microglia - pathology | Adult | Female | Gliosis - etiology | Repressor Proteins - metabolism | Electrodes, Implanted - adverse effects | Macrophages - physiology | Macrophages - pathology | Brain - physiopathology | Neurophysiological Monitoring - instrumentation | PAX6 Transcription Factor | Minichromosome Maintenance Complex Component 2 - metabolism | Nestin - metabolism | Eye Proteins - metabolism | Adolescent | Brain - pathology | Brain - surgery | Cicatrix - pathology | Epilepsy - pathology | Paired Box Transcription Factors - metabolism | Drug resistance | Drug therapy | Intermediate filament proteins | Index Medicus | Disorders of the Nervous System
Journal Article
Brain Pathology, ISSN 1015-6305, 09/2018, Volume 28, Issue 5, pp. 644 - 655
Mesial temporal lobe epilepsy with hippocampal sclerosis (MTLE‐HS) is a heterogeneous syndrome. Surgery results in seizure freedom for most pharmacoresistant... 
epilepsy | ILAE classification | hypertrophy | CD34 | hippocampal sclerosis | DIAGNOSTIC METHODS | TEMPORAL-LOBE EPILEPSY | ADULT FEMALE RAT | GRANULE CELL DISPERSION | PATHOLOGY | INTERNATIONAL CONSENSUS CLASSIFICATION | NEUROSCIENCES | CLINICAL NEUROLOGY | TASK-FORCE | VACUOLATING NEURONAL TUMORS | SEIZURE | DENTATE GYRUS | Sclerosis - surgery | Antigens, CD34 - metabolism | Prognosis | Humans | Male | Electroencephalography | Epilepsy, Temporal Lobe - surgery | Sclerosis - diagnostic imaging | Hippocampus - diagnostic imaging | Sclerosis - metabolism | Epilepsy, Temporal Lobe - diagnostic imaging | Gliosis - pathology | Sclerosis - pathology | Adult | Female | Epilepsy, Temporal Lobe - pathology | Gliosis - surgery | Proto-Oncogene Proteins B-raf - metabolism | Biomarkers - metabolism | Hippocampus - surgery | Gliosis - metabolism | Epilepsy, Temporal Lobe - metabolism | Hippocampus - pathology | Gliosis - diagnostic imaging | Hippocampus - metabolism | Proto-Oncogene Proteins B-raf - genetics | Intermediate Filament Proteins - metabolism | Cohort Studies | Nestin | Epilepsy | Cognitive ability | Sclerosis | Eutrophication | Heterogeneity | Convulsions & seizures | Reproduction (copying) | Surgery | Seizures | Seizing | CD34 antigen | Temporal lobe | Stellate cells | Neurons | Glial fibrillary acidic protein | EEG | Pharmacology | Patients | Nodules | Gliosis | Magnetic resonance imaging | Granular materials | Biomarkers | Hippocampus | Tumors | Index Medicus | Life Sciences | Human health and pathology
Journal Article
Glia, ISSN 0894-1491, 01/2018, Volume 66, Issue 1, pp. 126 - 144
Stimulation of Na + /H + exchanger isoform 1 (NHE1) in astrocytes causes ionic dysregulation under ischemic conditions. In this study, we created a Nhe1... 
MMP | blood–brain barrier | gliosis | cerebral edema | astrocyte end‐feet | neurovascular unit | astrocyte end-feet | blood-brain barrier | NEUROSCIENCES | Glial Fibrillary Acidic Protein - genetics | Cerebrovascular Circulation - physiology | Male | Cerebrovascular Circulation - genetics | Glial Fibrillary Acidic Protein - metabolism | Blood-Brain Barrier - ultrastructure | Gliosis - pathology | Matrix Metalloproteinase 9 - metabolism | Reperfusion | Infarction, Middle Cerebral Artery - complications | Gliosis - etiology | Disease Models, Animal | Microscopy, Electron, Transmission | Gliosis - genetics | Brain Infarction - diagnosis | Gene Expression Regulation - genetics | Gliosis - metabolism | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Infarction, Middle Cerebral Artery - pathology | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Brain Infarction - etiology | Motor Activity - genetics | Sodium-Hydrogen Exchanger 1 - deficiency | Animals | Neurologic Examination | Mice | Brain Infarction - genetics | Astrocytes - metabolism | Stroke (Disease) | Brain damage | Ischemia | Cytochemistry | Proteases | Analysis | Occlusion | Matrix metalloproteinases | Hydrogen | Recombinase | Gene deletion | Corn oil | Clonal deletion | Blood-brain barrier | Nhe1 gene | Cerebral blood flow | Rodents | Deletion | Cerebral infarction | Edema | Stroke | Tight junctions | Astrocytes | Na+/H+-exchanging ATPase | Inflammation | Tamoxifen | Ablation | Endothelial cells | Flox | Gelatinase B | Blood flow | Neurological diseases | Injury prevention | Gliosis | Infarction | Brain injury | Hypertrophy | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2016, Volume 6, Issue 1, pp. 18980 - 18980
Journal Article