X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (7490) 7490
Book Chapter (35) 35
Dissertation (19) 19
Conference Proceeding (5) 5
Publication (4) 4
Book / eBook (2) 2
Magazine Article (2) 2
Government Document (1) 1
Journal / eJournal (1) 1
Web Resource (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (4318) 4318
neurosciences (3649) 3649
gliosis (3511) 3511
male (3025) 3025
humans (2789) 2789
gliosis - pathology (2366) 2366
female (2040) 2040
mice (1940) 1940
rats (1850) 1850
brain (1644) 1644
astrocytes (1448) 1448
disease models, animal (1386) 1386
clinical neurology (1291) 1291
gliosis - metabolism (1199) 1199
immunohistochemistry (1178) 1178
brain - pathology (1127) 1127
inflammation (1089) 1089
gliosis - physiopathology (1005) 1005
astrocytes - metabolism (983) 983
microglia (957) 957
neurons (908) 908
astrocytes - pathology (861) 861
glial fibrillary acidic protein - metabolism (856) 856
pathology (852) 852
mice, inbred c57bl (820) 820
middle aged (807) 807
adult (802) 802
neurology (790) 790
expression (779) 779
rats, sprague-dawley (737) 737
neurodegeneration (731) 731
central-nervous-system (719) 719
neurons - pathology (689) 689
rodents (649) 649
cells, cultured (640) 640
glial fibrillary acidic protein (638) 638
analysis (628) 628
hippocampus (600) 600
gliosis - etiology (592) 592
aged (591) 591
brain - metabolism (581) 581
magnetic resonance imaging (548) 548
alzheimers-disease (546) 546
alzheimer's disease (535) 535
nervous system (529) 529
mice, transgenic (528) 528
astrocytes - drug effects (527) 527
reactive gliosis (518) 518
apoptosis (516) 516
microglia - metabolism (513) 513
rats, wistar (489) 489
proteins (485) 485
fibrillary acidic protein (477) 477
neurons - metabolism (477) 477
hippocampus - pathology (467) 467
neurodegenerative diseases (467) 467
biochemistry & molecular biology (461) 461
neuroprotection (447) 447
cell biology (444) 444
activation (442) 442
time factors (441) 441
gliosis - chemically induced (429) 429
research (419) 419
oxidative stress (418) 418
animals, newborn (416) 416
astrocyte (415) 415
cells (412) 412
epilepsy (402) 402
cerebral cortex - pathology (395) 395
mice, knockout (392) 392
gene expression (391) 391
gliosis - genetics (389) 389
central nervous system (377) 377
microglia - pathology (369) 369
disease (366) 366
animal models (361) 361
ischemia (360) 360
cytokines (359) 359
degeneration (358) 358
multidisciplinary sciences (358) 358
neuroinflammation (358) 358
gliosis - drug therapy (350) 350
reactive astrocytes (347) 347
microglia - drug effects (343) 343
medicine (335) 335
hippocampus - metabolism (333) 333
mouse model (330) 330
cortex (329) 329
spinal cord - pathology (328) 328
research article (325) 325
physiological aspects (321) 321
adolescent (317) 317
astrogliosis (313) 313
neuroscience (311) 311
transgenic mice (311) 311
injuries (307) 307
rat-brain (303) 303
atrophy (298) 298
neurons - drug effects (298) 298
neuroglia - metabolism (297) 297
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (7314) 7314
German (78) 78
French (53) 53
Japanese (51) 51
Spanish (30) 30
Russian (21) 21
Polish (12) 12
Portuguese (10) 10
Chinese (9) 9
Italian (4) 4
Korean (4) 4
Czech (3) 3
Turkish (2) 2
Danish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 4, p. e59586
Recent human trials of treatments for Alzheimer's disease (AD) have been largely unsuccessful, raising the idea that treatment may need to be started earlier... 
Microglia - metabolism | Neurons - pathology | Age Factors | Memory Disorders - genetics | Cell Count | Humans | Memory Disorders - metabolism | Male | Memory Disorders - diagnosis | Alzheimer Disease - pathology | Alzheimer Disease - diagnosis | Gliosis - pathology | CA1 Region, Hippocampal - pathology | Amyloid beta-Protein Precursor - metabolism | CA3 Region, Hippocampal - metabolism | Microglia - pathology | CA1 Region, Hippocampal - metabolism | Neurons - metabolism | Memory Disorders - pathology | Disease Models, Animal | Biomarkers - metabolism | Gene Expression | Gliosis - genetics | Plaque, Amyloid - pathology | CA3 Region, Hippocampal - cytology | Gliosis - metabolism | Mice, Transgenic | Inflammation | Plaque, Amyloid - genetics | Stereotaxic Techniques | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Plaque, Amyloid - diagnosis | Plaque, Amyloid - metabolism | Mice | Gliosis - diagnosis | Alzheimer Disease - genetics | Early Diagnosis | Neurosciences | Disease | Hyperactivity | Neurobiology | Cognitive ability | Memory tasks | Neurodegeneration | Transgenic animals | Rodents | Aging | Tumor necrosis factor-TNF | Amyloid | Alzheimer's disease | Age | Plaques | Medical research | Cytokines | Neurodegenerative diseases | Medical treatment | Spatial analysis | Microglia | Medicine | Pathology | Brain research | Gliosis | Mutation | Hippocampus | Spatial memory | Dementia
Journal Article
Antioxidants & redox signaling, ISSN 1557-7716, 2016, Volume 25, Issue 2, pp. 61 - 77
Aims: This preclinical study was aimed at determining whether pharmacological targeting of transcription factor NRF2, a master controller of many homeostatic... 
Original Research Communications | OXIDATIVE STRESS | IN-VITRO | TRANSCRIPTION FACTOR NRF2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | MPTP MOUSE MODEL | ALPHA-SYNUCLEIN | HEME OXYGENASE-1 | AUTOPHAGY | NF-KAPPA-B | EXPRESSION | NEUROPROTECTION | Immunohistochemistry | NF-E2-Related Factor 2 - agonists | Parkinson Disease - drug therapy | RNA, Messenger - metabolism | Autophagy | Brain - metabolism | Gliosis - pathology | Neuroprotective Agents - pharmacology | Dimethyl Fumarate - pharmacology | NF-E2-Related Factor 2 - genetics | Parkinson Disease - metabolism | Neuroprotective Agents - administration & dosage | Disease Models, Animal | Gene Expression | Gliosis - genetics | Parkinson Disease - pathology | Synucleins - genetics | Gliosis - metabolism | RNA, Messenger - genetics | Drug Repositioning | Synucleins - metabolism | Mice, Knockout | Phenotype | Animals | Mice | Dimethyl Fumarate - administration & dosage | Care and treatment | Parkinson's disease | Research | Gene expression | Neuroprotection | Brain | Therapy | Oxidative stress | Basal ganglia | Multiple sclerosis | Mesencephalon | Toxicity | Substantia nigra | Parkinsons disease | Innovations | Synuclein | Proteins | Neurotoxicity | Autopsy | Movement disorders | Dopamine receptors | Phenotypes | Dopamine | Wound healing | Neurodegenerative diseases | Pharmacology | Inflammation | Disease control | Ganglia | Lewy bodies | Gliosis | Cell death | Phagocytosis | Index Medicus
Journal Article
Journal Article
Glia, ISSN 0894-1491, 2018, Volume 66, Issue 1, pp. 126 - 144
Stimulation of Na+/H+ exchanger isoform 1 (NHE1) in astrocytes causes ionic dysregulation under ischemic conditions. In this study, we created a Nhe1flox/flox... 
MMP | blood–brain barrier | gliosis | cerebral edema | astrocyte end‐feet | neurovascular unit | astrocyte end-feet | blood-brain barrier | NEUROSCIENCES | Glial Fibrillary Acidic Protein - genetics | Cerebrovascular Circulation - physiology | Male | Cerebrovascular Circulation - genetics | Glial Fibrillary Acidic Protein - metabolism | Blood-Brain Barrier - ultrastructure | Gliosis - pathology | Matrix Metalloproteinase 9 - metabolism | Reperfusion | Infarction, Middle Cerebral Artery - complications | Gliosis - etiology | Disease Models, Animal | Microscopy, Electron, Transmission | Gliosis - genetics | Brain Infarction - diagnosis | Gene Expression Regulation - genetics | Gliosis - metabolism | Mice, Inbred C57BL | Sodium-Hydrogen Exchanger 1 - genetics | Mice, Transgenic | Infarction, Middle Cerebral Artery - pathology | Astrocytes - ultrastructure | Blood-Brain Barrier - pathology | Brain Infarction - etiology | Motor Activity - genetics | Sodium-Hydrogen Exchanger 1 - deficiency | Animals | Neurologic Examination | Mice | Brain Infarction - genetics | Astrocytes - metabolism | Stroke (Disease) | Brain damage | Ischemia | Cytochemistry | Proteases | Analysis | Occlusion | Matrix metalloproteinases | Hydrogen | Recombinase | Gene deletion | Corn oil | Clonal deletion | Blood-brain barrier | Nhe1 gene | Cerebral blood flow | Rodents | Cerebral infarction | Edema | Stroke | Tight junctions | Astrocytes | Na+/H+-exchanging ATPase | Inflammation | Tamoxifen | Ablation | Endothelial cells | Flox | Gelatinase B | Blood flow | Neurological diseases | Injury prevention | Gliosis | Infarction | Brain injury | Hypertrophy
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2012, Volume 73, Issue 4, pp. 685 - 697
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 2008, Volume 28, Issue 46, pp. 12039 - 12051
Reactive oxygen species (ROS) modulate intracellular signaling but are also responsible for neuronal damage in pathological states. Microglia, the resident CNS... 
Interleukin-1 | NADPH oxidase | Nitric oxide | Nox | Neuroinflammation | LPS | Microglia | nitric oxide | OXIDATIVE STRESS | interleukin-1 | CHRONIC GRANULOMATOUS-DISEASE | ALZHEIMERS-DISEASE | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | NEURONAL DAMAGE | neuroinflammation | SUPEROXIDE GENERATION | MOUSE MODEL | LIPOPOLYSACCHARIDE | HYDROGEN-PEROXIDE | microglia | PARKINSONS-DISEASE | Reactive Oxygen Species - metabolism | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nitrites - metabolism | rac GTP-Binding Proteins - metabolism | Male | Gliosis - enzymology | Gliosis - physiopathology | Cytochrome b Group - metabolism | Zymosan - metabolism | Encephalitis - physiopathology | Inflammation Mediators - pharmacology | rac GTP-Binding Proteins - genetics | NADPH Oxidases - genetics | Female | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | Corpus Striatum - enzymology | NADH, NADPH Oxidoreductases - genetics | Encephalitis - enzymology | Corpus Striatum - physiopathology | Microglia - drug effects | Mice, Inbred C57BL | Microglia - enzymology | Neuropeptides - metabolism | Cytochrome b Group - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Neurotoxins - pharmacology | Proteins - genetics | Animals | Proteins - metabolism | Lipopolysaccharides - pharmacology | Corpus Striatum - drug effects | Mice | Oxidative Stress - drug effects | rac1 GTP-Binding Protein | Index Medicus | Reactive Oxygen Species | Oxidative Stress | Nitrites | Neuropeptides | Lipopolysaccharides | Proteins | Life Sciences | Immunology | NADH, NADPH Oxidoreductases | Membrane Glycoproteins | Zymosan | rac GTP-Binding Proteins | Encephalitis | Neurotoxins | Inflammation Mediators | Cytochrome b Group | Gliosis | Corpus Striatum | NADPH Oxidase
Journal Article