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Neuroscience, ISSN 0306-4522, 2002, Volume 115, Issue 1, pp. 307 - 320
Journal Article
Neuroscience, ISSN 0306-4522, 2017, Volume 346, pp. 409 - 422
Journal Article
Brain, ISSN 0006-8950, 2013, Volume 136, Issue 1, pp. 147 - 167
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2008, Volume 28, Issue 27, pp. 6904 - 6913
There are critical postnatal periods during which even subtle interventions can have long-lasting effects on adult physiology. We asked whether an immune... 
Development | Lipopolysaccharide | Seizure | Interleukin-1β | Tumor necrosis factor α | Cytokine | cytokine | development | ACID-INDUCED SEIZURES | seizure | TEMPORAL-LOBE EPILEPSY | NECROSIS-FACTOR-ALPHA | KAINIC ACID | FEBRILE SEIZURES | LIFE IMMUNE CHALLENGE | NEUROSCIENCES | interleukin-1 beta | tumor necrosis factor alpha | STEREOLOGICAL ESTIMATION | EPILEPTIFORM ACTIVITY | RECEPTOR TRAFFICKING | TNF-ALPHA | lipopolysaccharide | Electric Stimulation | Age Factors | Injections, Intraventricular | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Male | Gliosis - physiopathology | Time | Epilepsy - physiopathology | Nerve Degeneration - chemically induced | Lipopolysaccharides | Afferent Pathways - immunology | Disease Susceptibility - immunology | Epilepsy - immunology | Tumor Necrosis Factor-alpha - immunology | Afferent Pathways - drug effects | Pyramidal Cells - drug effects | Gliosis - immunology | Hippocampus - growth & development | Action Potentials - drug effects | Organ Culture Techniques | Potassium Channel Blockers - pharmacology | Animals, Newborn | Hippocampus - immunology | Rats | Encephalitis - immunology | Nerve Degeneration - immunology | Rats, Sprague-Dawley | Action Potentials - physiology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Pyramidal Cells - immunology | Convulsants - pharmacology | Encephalitis - chemically induced | Hippocampus - physiopathology | Index Medicus | tumor necrosis factor α | interleukin-1β
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 9/2012, Volume 124, Issue 3, pp. 411 - 424
Journal Article
Glia, ISSN 0894-1491, 12/2016, Volume 64, Issue 12, pp. 2166 - 2180
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 25, Issue 2, pp. 392 - 400
Abstract Dopaminergic cells in the substantia nigra are highly vulnerable to the neurodegenerative process of Parkinson’s disease. Therefore, mechanisms that... 
Neurology | Pesticide | Substantia nigra | NADPH oxidase | Minocycline | Paraquat | Inflammation | Parkinson’s disease | Microglia | Parkinson's disease | substantia nigra | OXIDATIVE STRESS | paraquat | pesticide | RISK | NEURODEGENERATION | HERBICIDE PARAQUAT | ALPHA-SYNUCLEIN | MODEL | NEUROTOXICITY | NEUROSCIENCES | inflammation | minocycline | LIPOPOLYSACCHARIDE | microglia | PARKINSONS-DISEASE | Substantia Nigra - physiopathology | Substantia Nigra - pathology | Minocycline - pharmacology | Oxidative Stress - physiology | NADPH Oxidases - metabolism | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Male | Gliosis - physiopathology | Nerve Degeneration - chemically induced | Gliosis - pathology | Encephalitis - physiopathology | Microglia - physiology | Inflammation Mediators - pharmacology | NADPH Oxidases - drug effects | Dopamine - metabolism | Biomarkers - metabolism | Parkinsonian Disorders - physiopathology | Microglia - drug effects | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Biomarkers - analysis | Encephalitis - pathology | Neurotoxins - toxicity | Herbicides - toxicity | Nerve Degeneration - pathology | Substantia Nigra - drug effects | Animals | Parkinsonian Disorders - pathology | Lipopolysaccharides - pharmacology | Mice | Encephalitis - chemically induced | Oxidative Stress - drug effects | Paraquat - toxicity | Physiological aspects | NADPH-oxidase
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1283, pp. 127 - 138
Journal Article