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Nature Neuroscience, ISSN 1097-6256, 03/2008, Volume 11, Issue 3, pp. 251 - 253
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 52, pp. 22480 - 22485
Huntington disease (HD) is an inherited neurological disorder caused by a polyglutamine expansion in the protein huntingtin and is characterized by selective... 
Proteins | Brain | Phenotypes | Neuroscience | Astrocytes | Transgenic animals | Neurons | Neuroglia | Huntington disease | Mice | Polyglutamine | Glia | Glutamate | Neurodegeneration | Excitotoxicity | NEURODEGENERATIVE DISEASE | CONTRIBUTE | MULTIDISCIPLINARY SCIENCES | neurodegeneration | polyglutamine | GLIAL-CELLS | glia | GLUTAMATE TRANSPORT | PATHOGENESIS | NEURONS | INTRANUCLEAR INCLUSIONS | ACCUMULATION | excitotoxicity | glutamate | TRANSGENIC MICE | Glial Fibrillary Acidic Protein - genetics | Humans | Huntington Disease - pathology | Sp1 Transcription Factor - metabolism | DNA Primers - genetics | Gliosis - pathology | Aging - genetics | Base Sequence | Excitatory Amino Acid Transporter 2 - genetics | Nuclear Proteins - genetics | Huntington Disease - physiopathology | Recombinant Proteins - metabolism | Promoter Regions, Genetic | Gene Expression | Gliosis - genetics | Nerve Tissue Proteins - physiology | Brain - physiopathology | Mice, Transgenic | Recombinant Proteins - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Nerve Tissue Proteins - genetics | Aging - pathology | Huntingtin Protein | Astrocytes - physiology | Phenotype | Animals | Aging - physiology | Brain - pathology | Huntington Disease - genetics | Glutamic Acid - metabolism | Nuclear Proteins - physiology | Mutation | Huntington's chorea | Physiological aspects | Development and progression | Genetic aspects | Risk factors | Nerve proteins | Biological Sciences
Journal Article
Journal Article
Gene, ISSN 0378-1119, 10/2015, Volume 571, Issue 1, pp. 81 - 90
Globoid cell leukodystrophy (GLD) is an autosomal recessive, lysosomal storage disease caused by deficiency of the enzyme galactocerebrosidase (GALC). The... 
Neurotrophic factor | Adeno-associated virus | Lysosomal storage disease | Purkinje cells | Globoid cell leukodystrophy | TWITCHER MOUSE | PURKINJE-CELLS | BONE-MARROW-TRANSPLANTATION | SPINAL-CORD | PSYCHOSINE | MURINE MODEL | SPINOCEREBELLAR ATAXIA | GENETICS & HEREDITY | EXPRESSION | KRABBE-DISEASE | Immunohistochemistry | Brain-Derived Neurotrophic Factor - genetics | Dependovirus - genetics | Genetic Vectors - administration & dosage | Neuroglia - pathology | Purkinje Cells - ultrastructure | Insulin-Like Growth Factor I - genetics | Mice, Neurologic Mutants | Glial Cell Line-Derived Neurotrophic Factor - metabolism | Galactosylceramidase - genetics | Ciliary Neurotrophic Factor - genetics | Brain-Derived Neurotrophic Factor - metabolism | Leukodystrophy, Globoid Cell - therapy | Microscopy, Electron, Transmission | Gene Expression | Gliosis - genetics | Purkinje Cells - metabolism | Cerebellum - metabolism | Gliosis - metabolism | Mice, Inbred C57BL | Cerebellar Diseases - therapy | Glial Cell Line-Derived Neurotrophic Factor - genetics | Kaplan-Meier Estimate | Reverse Transcriptase Polymerase Chain Reaction | Genetic Vectors - genetics | Cerebellum - pathology | Ciliary Neurotrophic Factor - metabolism | Animals | Cerebellar Diseases - genetics | Neuroglia - metabolism | Leukodystrophy, Globoid Cell - genetics | Cerebellum - ultrastructure | Purkinje Cells - pathology | Insulin-Like Growth Factor I - metabolism | Galactosylceramidase - metabolism | Genetic Therapy - methods | Enzymes | Analysis | Genes | Genetic research | Mice | Ciliary neurotrophic factor | Gene therapy | Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 12/2011, Volume 19, Issue 12, pp. 2152 - 2162
Journal Article
Stroke, ISSN 0039-2499, 09/2011, Volume 42, Issue 9, pp. 2589 - 2594
Background and Purpose-Activation of Notch worsens ischemic brain damage as antisense knockdown or pharmacological inhibition of the Notch pathway reduces the... 
apoptosis | focal ischemia | brain ischemia | inflammation | neuroregeneration | neuroprotection | CELLS | BRAIN-DAMAGE | PROLIFERATION | DEATH | CLINICAL NEUROLOGY | STROKE | TUMOR-NECROSIS-FACTOR | PERIPHERAL VASCULAR DISEASE | NF-KAPPA-B | UP-REGULATION | Amyloid Precursor Protein Secretases - genetics | Inflammation - pathology | Microglia - metabolism | Coculture Techniques | Brain Ischemia - genetics | Immunity, Innate - genetics | NF-kappa B - metabolism | Brain Ischemia - immunology | Inflammation - metabolism | Cell Nucleus - metabolism | Microglia - pathology | Gliosis - immunology | Active Transport, Cell Nucleus - genetics | Cytokines - genetics | Gliosis - genetics | Mice, Transgenic | Inflammation - therapy | Amyloid Precursor Protein Secretases - metabolism | Cell Nucleus - genetics | Active Transport, Cell Nucleus - immunology | Mice | Receptor, Notch1 - antagonists & inhibitors | Oligopeptides - pharmacology | Receptor, Notch1 - genetics | Cell Nucleus - immunology | Amyloid Precursor Protein Secretases - immunology | Gliosis - therapy | NF-kappa B - immunology | Brain Ischemia - metabolism | Gliosis - pathology | Microglia - immunology | Inflammation Mediators - metabolism | Cytokines - immunology | Inflammation Mediators - immunology | Cell Line | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Brain Ischemia - therapy | Gliosis - metabolism | Receptor, Notch1 - immunology | Inflammation - immunology | Receptor, Notch1 - metabolism | Immunity, Innate - immunology | Animals | NF-kappa B - genetics | Brain Ischemia - pathology | Inflammation - genetics | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Cytokines - biosynthesis | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2011, Volume 6, Issue 8, p. e23782
Journal Article
Neuron, ISSN 0896-6273, 02/2012, Volume 73, Issue 4, pp. 685 - 697
Journal Article
PLoS Genetics, ISSN 1553-7390, 07/2017, Volume 13, Issue 7, p. e1006846
Journal Article