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PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, pp. e0159442 - e0159442
Millions of mild traumatic brain injuries (TBIs) occur every year in the United States, with many people subject to multiple head injuries that can lead to... 
MILD | ASTROCYTE ACTIVATION | OPTIC-NERVE | TRAUMATIC BRAIN-INJURY | BEHAVIORAL IMPAIRMENTS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | COGNITIVE IMPAIRMENT | SPINAL-CORD-INJURY | CONCUSSION | AXONAL INJURY | Optic Tract - physiopathology | Memory Disorders - physiopathology | Neurons - pathology | Optic Nerve - pathology | Corpus Callosum - metabolism | Brain Concussion - pathology | Brain Concussion - physiopathology | Corpus Callosum - physiopathology | Neuroglia - pathology | Optic Tract - pathology | Cerebral Cortex - pathology | Memory Disorders - metabolism | Head Injuries, Closed - metabolism | Male | Gliosis - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Head Injuries, Closed - physiopathology | Gliosis - pathology | Neurofilament Proteins - genetics | Pyramidal Tracts - physiopathology | Pyramidal Tracts - metabolism | Neurons - metabolism | Optic Nerve - physiopathology | Pyramidal Tracts - pathology | Memory Disorders - pathology | Disease Models, Animal | Psychomotor Performance | Gene Expression | Head Injuries, Closed - pathology | Gliosis - metabolism | Mice, Inbred C57BL | Hippocampus - pathology | Optic Tract - metabolism | Hippocampus - metabolism | Animals | Corpus Callosum - pathology | Neuroglia - metabolism | Mice | Neurofilament Proteins - metabolism | Hippocampus - physiopathology | Brain Concussion - metabolism | Optic Nerve - metabolism | Complications and side effects | Care and treatment | Head injuries | Research | Memory, Disorders of | Risk factors | Intervals | Brain | Traumatic brain injury | Gait | Neuronal-glial interactions | Visual system | Histopathology | Neurodegeneration | Neurofilament protein | Physiology | Acute effects | Stains | Injuries | Head | Optic nerve | Motor ability | Abnormalities | Cortex (visual) | Inflammation | Trauma | Corpus callosum | Optic tract | Medicine | Axons | Injury prevention | Brain research | Gliosis | Chronic traumatic encephalopathy | Cell death | Spinal cord injuries | Laboratory animals | Human behavior | Apoptosis | Animal cognition | Index Medicus
Journal Article
Pain, ISSN 0304-3959, 2008, Volume 140, Issue 3, pp. 411 - 419
Provoked vestibulodynia (PVD) is a common form of chronic vulvar pain with unknown aetiology. Central pain regulatory mechanisms have been suggested to be... 
Neuronal degeneration | Cerebral alterations | Structural magnetic resonance imaging | Clinical pain | Microglial activation | Voxel-based morphometry | POSTTRAUMATIC-STRESS-DISORDER | BASAL GANGLIA | FIBROMYALGIA PATIENTS | NEUROSCIENCES | CLINICAL NEUROLOGY | NEUROPATHIC PAIN | HIPPOCAMPAL VOLUME | MIGRAINE PATIENTS | CHRONIC-FATIGUE | ANESTHESIOLOGY | CHRONIC BACK-PAIN | VESTIBULITIS SYNDROME | Pain, Intractable - physiopathology | Substantia Nigra - physiopathology | Pain Threshold - physiology | Substantia Nigra - pathology | Humans | Gliosis - physiopathology | Hypertrophy - pathology | Hypertrophy - physiopathology | Gliosis - pathology | Young Adult | Encephalitis - physiopathology | Vulvar Vestibulitis - physiopathology | Microglia - pathology | Adult | Female | Pain, Intractable - pathology | Vulvar Vestibulitis - etiology | Gliosis - etiology | Physical Stimulation | Pain, Intractable - etiology | Hypertrophy - etiology | Vulvar Vestibulitis - pathology | Basal Ganglia - physiopathology | Brain - physiopathology | Encephalitis - pathology | Basal Ganglia - pathology | Hippocampus - pathology | Magnetic Resonance Imaging | Pain Measurement - methods | Brain - pathology | Brain Mapping | Chronic Disease - psychology | Hippocampus - physiopathology | Stress, Psychological - complications | Encephalitis - etiology | Brain | Specific gravity | Backache | Young women | Chronic pain | Index Medicus
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 12/2007, Volume 114, Issue 6, pp. 619 - 631
Neuroimaging studies indicate reduced volumes of certain gray matter regions in survivors of prematurity with periventricular leukomalacia (PVL). We... 
Pathology | Perinatal hypoxia–ischemia | Basal ganglia | Medicine & Public Health | Neurodevelopmental disability | Brainstem | Thalamus | Perinatal panencephalopathy | White matter gliosis | Perinatal hypoxia-ischemia | perinatal panencephalopathy | THALAMOCORTICAL CONNECTIONS | basal ganglia | LOW-BIRTH-WEIGHT | BRAIN-INJURY | ABNORMALITIES | COMPLICATION | PRETERM INFANTS | SUBPLATE NEURONS | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | MEMORY | neurodevelopmental disability | CEREBELLUM | brainstem | white matter gliosis | perinatal hypoxia-ischemia | thalamus | PLASTICITY | Neurons - pathology | Prevalence | Humans | Nerve Degeneration - physiopathology | Cerebellar Nuclei - growth & development | Infant | Male | Gliosis - physiopathology | Leukomalacia, Periventricular - pathology | Thalamus - pathology | Brain - growth & development | Gliosis - pathology | Cerebellar Nuclei - physiopathology | Leukomalacia, Periventricular - physiopathology | Gliosis - epidemiology | Premature Birth - physiopathology | Nerve Fibers, Myelinated - pathology | Female | Globus Pallidus - physiopathology | Brain Damage, Chronic - pathology | Globus Pallidus - pathology | Infant, Newborn | Leukomalacia, Periventricular - epidemiology | Globus Pallidus - growth & development | Comorbidity | Brain - physiopathology | Cerebellar Nuclei - pathology | Nerve Degeneration - epidemiology | Brain Damage, Chronic - physiopathology | Nerve Degeneration - pathology | Thalamus - growth & development | Thalamus - physiopathology | Brain - pathology | Brain Damage, Chronic - epidemiology | Index Medicus | Original Paper
Journal Article
Journal of NeuroVirology, ISSN 1355-0284, 10/2016, Volume 22, Issue 5, pp. 650 - 660
Methamphetamine (Meth) use is common among HIV-infected persons. It remains unclear whether Meth dependence is associated with long-lasting degenerative... 
Astrogliosis | Neurology | Neurosciences | Biomedicine | Immunology | Small vessel disease | Arteriolosclerosis | Methamphetamine | β-Amyloid | Infectious Diseases | Microgliosis | Virology | PROTRACTED ABSTINENCE | NEUROSCIENCES | ABUSERS | HIV-INFECTION | VIROLOGY | RESONANCE-SPECTROSCOPY | beta-Amyloid | CENTRAL-NERVOUS-SYSTEM | NEUROCOGNITIVE DISORDERS | WHITE-MATTER HYPERINTENSITIES | USERS | BRAIN | Glial Fibrillary Acidic Protein - genetics | Frontal Lobe - metabolism | White Matter - metabolism | Humans | Middle Aged | Male | Amphetamine-Related Disorders - pathology | Parietal Lobe - metabolism | Glial Fibrillary Acidic Protein - metabolism | Synaptophysin - genetics | Amyloid beta-Peptides - genetics | Autopsy | Amyloid beta-Peptides - metabolism | Amphetamine-Related Disorders - genetics | Alcoholism - complications | Prosencephalon - metabolism | Alcoholism - physiopathology | Prosencephalon - physiopathology | Synaptophysin - metabolism | Gene Expression | Gliosis - genetics | Parietal Lobe - pathology | White Matter - pathology | HIV Infections - complications | HIV Infections - physiopathology | Opioid-Related Disorders - complications | Opioid-Related Disorders - pathology | White Matter - physiopathology | Microtubule-Associated Proteins - genetics | Putamen - pathology | Microtubule-Associated Proteins - metabolism | Gliosis - physiopathology | Parietal Lobe - physiopathology | DNA-Binding Proteins - metabolism | Gliosis - pathology | HIV Infections - pathology | Putamen - physiopathology | Opioid-Related Disorders - genetics | Adult | Female | Temporal Lobe - pathology | Prosencephalon - pathology | Alcoholism - pathology | Frontal Lobe - physiopathology | HIV Infections - genetics | Amphetamine-Related Disorders - physiopathology | DNA-Binding Proteins - genetics | Temporal Lobe - metabolism | Gliosis - complications | Temporal Lobe - physiopathology | Alcoholism - genetics | Aged | Opioid-Related Disorders - physiopathology | Amphetamine-Related Disorders - complications | Frontal Lobe - pathology | Putamen - metabolism | Index Medicus
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 08/2009, Volume 6, Issue 1, pp. 23 - 23
Background: Lyme neuroborreliosis (LNB) may present as meningitis, cranial neuropathy, acute radiculoneuropathy or, rarely, as encephalomyelitis. We... 
BORRELIA-BURGDORFERI | TUMOR-NECROSIS-FACTOR | NONHUMAN PRIMATE MODEL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RHESUS-MONKEY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CENTRAL-NERVOUS-SYSTEM | MENINGEAL INFLAMMATION | IMMUNOLOGY | CEREBROSPINAL-FLUID | NEUROSCIENCES | FACTOR-ALPHA | Lyme Neuroborreliosis - pathology | Neurons - pathology | Spinal Cord Diseases - physiopathology | Nerve Degeneration - physiopathology | Ganglia, Spinal - physiopathology | Gliosis - physiopathology | Macaca mulatta | Encephalitis - physiopathology | Lyme Neuroborreliosis - immunology | Spinal Cord - pathology | Leukocytosis - physiopathology | Lyme Neuroborreliosis - physiopathology | Neuroglia - immunology | Radiculopathy - physiopathology | Leukocytosis - microbiology | Gliosis - immunology | Ganglia, Spinal - immunology | Spinal Cord Diseases - immunology | Neurons - microbiology | Brain - physiopathology | Leukocytosis - immunology | Nerve Degeneration - microbiology | Neurons - immunology | Radiculopathy - microbiology | Encephalitis - immunology | Nerve Degeneration - immunology | Neuroglia - microbiology | Gliosis - microbiology | Spinal Cord - immunology | Meningitis - immunology | Animals | Apoptosis - immunology | Spinal Cord Diseases - microbiology | Ganglia, Spinal - pathology | Brain - pathology | Antibodies - blood | Chemokines - metabolism | Spinal Cord - physiopathology | Meningitis - microbiology | Meningitis - physiopathology | Brain - immunology | Encephalitis - microbiology | Radiculopathy - immunology | Prevention | Lyme disease | Usage | Care and treatment | Fluorescent antibody technique | Research | Immunofluorescence | T cells | Health aspects | Apoptosis | Index Medicus
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 04/2010, Volume 27, Issue 4, pp. 753 - 762
In this study we tested the hypothesis that long-term neuropathological outcome is worsened by hyperoxic compared to normoxic reperfusion in a rat global... 
Morris water maze | Astrocyte | Hippocampus | Resuscitation | Cardiac arrest | Microglia | hippocampus | BRAIN-INJURY | 100-PERCENT OXYGEN | CARDIOPULMONARY | NORMOBARIC OXYGEN | RATS | FOREBRAIN ISCHEMIA | cardiac arrest | NEUROSCIENCES | CLINICAL NEUROLOGY | STROKE | CARDIAC-ARREST | resuscitation | astrocyte | microglia | CRITICAL CARE MEDICINE | Neurons - pathology | Hyperoxia - physiopathology | Rats, Inbred F344 | Nerve Degeneration - physiopathology | Learning Disorders - physiopathology | Male | Gliosis - physiopathology | Time | Learning Disorders - etiology | Oxygen Consumption - physiology | Oxygen - metabolism | Gliosis - pathology | Learning Disorders - pathology | Encephalitis - physiopathology | Cognition Disorders - etiology | Hyperoxia - complications | Neurons - physiology | Reperfusion Injury - complications | Cell Survival - physiology | Gliosis - etiology | Resuscitation - adverse effects | Disease Models, Animal | Brain Ischemia - complications | Cognition Disorders - physiopathology | Oxygen - toxicity | Reperfusion Injury - pathology | Cognition Disorders - pathology | Encephalitis - pathology | Rats | Brain Ischemia - physiopathology | Hippocampus - pathology | Nerve Degeneration - pathology | Hyperoxia - pathology | Animals | Brain Ischemia - pathology | Reperfusion Injury - physiopathology | Nerve Degeneration - etiology | Hippocampus - physiopathology | Encephalitis - etiology | Cerebral ischemia | Care and treatment | Neurons | Patient outcomes | Physiological aspects | Reperfusion (Physiology) | Research | Hippocampus (Brain) | Stroke | Ischemia | Hyperoxia | Rodents | Apoptosis | Index Medicus | Original
Journal Article
Stroke, ISSN 0039-2499, 10/2009, Volume 40, Issue 10, pp. 3369 - 3377
Background and Purpose-Intraventricular hemorrhage (IVH) is a common complication of prematurity that results in neurological sequelae, including cerebral... 
Neurobehavioral testing | Gliosis | Myelin | Ventriculomegaly | White matter | Germinal matrix hemorrhageintraventricular hemorrhage | myelin | ventriculomegaly | germinal matrix hemorrhage-intraventricular hemorrhage | GERMINAL MATRIX HEMORRHAGE | WHITE-MATTER | PRETERM INFANTS | NEONATAL POSTHEMORRHAGIC HYDROCEPHALUS | BLOOD-BRAIN-BARRIER | CLINICAL NEUROLOGY | white matter | BIRTH-WEIGHT INFANTS | PERIPHERAL VASCULAR DISEASE | gliosis | neurobehavioral testing | SURVIVORS | AGE | Lateral Ventricles - pathology | Age Factors | Brain - anatomy & histology | Diffuse Axonal Injury - physiopathology | Gliosis - physiopathology | Lateral Ventricles - blood supply | Cerebral Hemorrhage - physiopathology | Hypertrophy - pathology | Hypertrophy - physiopathology | Gliosis - pathology | Premature Birth - physiopathology | Nerve Fibers, Myelinated - pathology | Ultrasonography | Lateral Ventricles - physiopathology | Gliosis - etiology | Disease Models, Animal | Animals, Newborn | Microscopy, Electron, Transmission | Rabbits | Hypertrophy - etiology | Movement Disorders - pathology | Brain - physiopathology | Glycerol - toxicity | Diffuse Axonal Injury - etiology | Premature Birth - pathology | Animals | Movement Disorders - physiopathology | Aging - physiology | Diffusion Magnetic Resonance Imaging | Brain - pathology | Cerebral Hemorrhage - complications | Cerebral Hemorrhage - pathology | Diffuse Axonal Injury - pathology | Index Medicus | ventriculomegaly neurobehavioral testing | Germinal matrix hemorrhage-intraventricular hemorrhage
Journal Article
Synapse, ISSN 0887-4476, 12/2006, Volume 60, Issue 7, pp. 521 - 532
Histological examination of brain after a single high (40 mg/kg) dose of D ‐methamphetamine (METH) was used to determine the relationships between blood‐brain... 
neurodegeneration | blood‐brain barrier | seizures | Fluoro‐Jade C | hippocampus | methamphetamine | Blood-brain barrier | Neurodegeneration | Hippocampus | Methamphetamine | Fluoro-Jade C | Seizures | blood-brain barrier | FORMER AMPHETAMINE | NEUROTOXICITY | NEUROSCIENCES | DOMOIC ACID | IMPAIRMENT | INDIVIDUALS | INDUCED NEURODEGENERATION | MICROGLIA | SOMATOSENSORY CORTEX | RAT FOREBRAIN | EXPOSURE | Amygdala - physiopathology | Neurons - pathology | Amygdala - drug effects | Methamphetamine - adverse effects | Body Temperature - drug effects | Organic Chemicals | Immunoglobulin G | Blood-Brain Barrier - physiopathology | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Male | Amphetamine-Related Disorders - pathology | Gliosis - physiopathology | Hippocampus - drug effects | Body Temperature - physiology | Fever - chemically induced | Fever - physiopathology | Nerve Degeneration - chemically induced | Seizures - chemically induced | Cerebral Arteries - drug effects | Gliosis - pathology | Plant Lectins | Seizures - physiopathology | Seizures - pathology | Central Nervous System Stimulants - adverse effects | Neurons - drug effects | Amphetamine-Related Disorders - metabolism | Fever - pathology | Disease Models, Animal | Phagocytosis - drug effects | Microglia - drug effects | Mice, Inbred C57BL | Phagocytosis - physiology | Cerebral Arteries - physiopathology | Fluoresceins | Hippocampus - pathology | Amphetamine-Related Disorders - physiopathology | Blood-Brain Barrier - drug effects | Amygdala - pathology | Animals | Mice | Hippocampus - physiopathology | Index Medicus
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1303, pp. 131 - 143
Abstract Transsynaptic degeneration has been implicated in patients with primary open angle glaucoma (POAG) and animal models of chronic intraocular... 
Neurology | Visual system | Neural plasticity | Glial fibrillary acidic protein (GFAP) | Ischemia | Acute intraocular hypertension | Superior colliculus (SC) | Transsynaptic degeneration | Neuronal death | Degenerative disease | Glia and other non-neuronal cells | Lateral geniculate nucleus (LGN) | Glutamine synthetase (GS) | OPTIC-NERVE | EXPERIMENTAL GLAUCOMA | ASTROCYTES | NEUROSCIENCES | LATERAL GENICULATE-NUCLEUS | PARVOCELLULAR LAYERS | SUPERIOR COLLICULUS | RETINAL GANGLION-CELLS | FIBRILLARY ACIDIC PROTEIN | MULLER CELL | BRAIN | Rats, Wistar | Visual Pathways - physiopathology | Neuroglia - pathology | Nerve Degeneration - physiopathology | Retinal Degeneration - etiology | Gliosis - physiopathology | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | Geniculate Bodies - pathology | Geniculate Bodies - physiopathology | Retinal Ganglion Cells - pathology | Gliosis - pathology | Female | Visual Pathways - pathology | Glutamate-Ammonia Ligase - metabolism | Glaucoma - physiopathology | Gliosis - etiology | Disease Models, Animal | In Situ Nick-End Labeling | Acute Disease | Retina - physiopathology | Rats | Retinal Degeneration - physiopathology | Glaucoma - complications | Disease Progression | Nerve Degeneration - pathology | Superior Colliculi - physiopathology | Animals | Superior Colliculi - pathology | Axons - pathology | Glaucoma - pathology | Apoptosis - physiology | Nerve Degeneration - etiology | Retinal Degeneration - pathology | Early Diagnosis | Retina - pathology | Index Medicus
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 25, Issue 2, pp. 367 - 377
Abstract Disruption of the blood–brain barrier (BBB) is a characteristic finding in common neurological disorders. Human data suggest BBB disruption may... 
Neurology | Neurodegeneration | Sensory-motor cortex | Epilepsy | Cortical spreading depression | Blood–brain barrier | Blood-brain barrier | blood-brain barrier | epilepsy | TEMPORAL-LOBE EPILEPSY | neurodegeneration | EPILEPTIFORM DISCHARGES | MECHANISMS | NEUROSCIENCES | SPREADING DEPRESSION | CELL-DEATH | sensory-motor cortex | PILOCARPINE MODEL | TRAUMATIC BRAIN | SODIUM DEHYDROCHOLATE | ELECTROENCEPHALOGRAPHIC CONSEQUENCES | cortical spreading depression | PROPAGATION | Neocortex - pathology | Cerebrovascular Circulation - physiology | Dyskinesia, Drug-Induced - physiopathology | Rats, Wistar | Astrocytes - pathology | Atrophy - pathology | Blood-Brain Barrier - physiopathology | Nerve Degeneration - physiopathology | Gliosis - chemically induced | Male | Gliosis - physiopathology | Cortical Spreading Depression - physiology | Epilepsy - physiopathology | Nerve Degeneration - chemically induced | Serum Albumin - adverse effects | Cerebral Arteries - drug effects | Gliosis - pathology | Deoxycholic Acid - adverse effects | Time Factors | Neocortex - physiopathology | Neural Conduction - drug effects | Organ Culture Techniques | Astrocytes - drug effects | Atrophy - physiopathology | Cerebrovascular Circulation - drug effects | Rats | Cerebral Arteries - physiopathology | Neural Conduction - physiology | Dyskinesia, Drug-Induced - pathology | Neocortex - blood supply | Blood-Brain Barrier - drug effects | Nerve Degeneration - pathology | Cortical Spreading Depression - drug effects | Blood-Brain Barrier - pathology | Magnetic Resonance Imaging | Animals | Epilepsy - chemically induced | Cerebral Arteries - pathology | Atrophy - chemically induced | Epilepsy - pathology | Index Medicus
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2008, Volume 31, Issue 3, pp. 395 - 405
Abstract Amyotrophic lateral sclerosis (ALS) is a lethal disease affecting motoneurons. In familial ALS, patients bear mutations in the superoxide dismutase... 
Neurology | Neuroprotection | Motoneuron | Cell therapy | Astrocyte | SOD1 | Microglia | SPINAL-CORD | NEUROSCIENCES | RAT MODEL | DISEASE ONSET | NEURAL CELLS | cell therapy | neuroprotection | ADULT BONE-MARROW | motoneuron | ANIMAL-MODELS | NITRIC-OXIDE | ALS MICE | IN-VITRO DIFFERENTIATION | microglia | astrocyte | MARROW STROMAL CELLS | Microglia - metabolism | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Nerve Degeneration - physiopathology | Male | Gliosis - physiopathology | Movement Disorders - surgery | Motor Neurons - pathology | Mesenchymal Stromal Cells - cytology | Nerve Degeneration - prevention & control | Spinal Cord - pathology | Recovery of Function - physiology | Female | Gliosis - surgery | Cell Survival - physiology | Mesenchymal Stromal Cells - physiology | Disease Models, Animal | Astrocytes - cytology | Microglia - cytology | Nerve Degeneration - surgery | Amyotrophic Lateral Sclerosis - therapy | Myelitis - physiopathology | Gliosis - metabolism | Survival Rate | Treatment Outcome | Sex Characteristics | Mutation - genetics | Spinal Cord - surgery | Mesenchymal Stem Cell Transplantation - methods | Animals | Movement Disorders - physiopathology | Movement Disorders - etiology | Mice | Spinal Cord - physiopathology | Superoxide Dismutase-1 | Myelitis - therapy | Astrocytes - metabolism | Superoxide dismutase | Amyotrophic lateral sclerosis | Transplantation | Analysis | Stem cells | Index Medicus
Journal Article