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The EMBO Journal, ISSN 0261-4189, 07/2004, Volume 23, Issue 13, pp. 2544 - 2553
Bach1 is a transcriptional repressor of heme oxygenase‐1 and β‐globin genes, both of which are known to be transcriptionally induced by heme. To test the... 
heme oxygenase | Maf | globin | heme | oxidative stress | Globin | Oxidative stress | Heme | Heme oxygenase | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Heme - metabolism | Transcription Factors - chemistry | Humans | Leucine Zippers | Molecular Sequence Data | Globins - metabolism | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | Chromatin Immunoprecipitation | Heme Oxygenase-1 | Transcription, Genetic | Active Transport, Cell Nucleus | Hemin - pharmacology | Heme Oxygenase (Decyclizing) - metabolism | Dimerization | Heme Oxygenase (Decyclizing) - genetics | Genes, Reporter | Repressor Proteins - metabolism | Fibroblasts - metabolism | Amino Acid Sequence | Cell Line | Green Fluorescent Proteins - metabolism | Zinc Fingers | Gene Expression Regulation | Glutathione Transferase - metabolism | Alanine - metabolism | Nuclear Proteins - metabolism | Recombinant Fusion Proteins - chemistry | Transcription Factors - genetics | Fanconi Anemia Complementation Group Proteins | Amino Acid Motifs | MafK Transcription Factor | Membrane Proteins | Transcription Factors - metabolism | Karyopherins - metabolism | Escherichia coli - genetics | Plasmids | Fibroblasts - drug effects | Erythroid-Specific DNA-Binding Factors | Spectrophotometry | Basic-Leucine Zipper Transcription Factors | Amino Acid Substitution | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Science (American Association for the Advancement of Science), ISSN 0036-8075, 6/2004, Volume 304, Issue 5678, pp. 1797 - 1800
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2008, Volume 295, Issue 1, pp. 39 - 47
In sickle cell disease, nitric oxide (NO) depletion by cell-free plasma hemoglobin and/or oxygen radicals is associated with arginine deficiency, impaired NO... 
Sickle cell disease | Microvascular regulation | Vasoreactivity | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Hemolysis - drug effects | Heme Oxygenase-1 - metabolism | Microcirculation - metabolism | Anemia, Sickle Cell - physiopathology | Humans | Anemia, Sickle Cell - drug therapy | Globins - metabolism | Nitroprusside - pharmacology | Microcirculation - drug effects | Tyrosine - analogs & derivatives | Nitric Oxide Synthase Type II - antagonists & inhibitors | Vasodilator Agents - metabolism | Membrane Proteins - metabolism | Nitric Oxide Donors - pharmacology | Citrulline - blood | Disease Models, Animal | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - blood | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase Type III | Hemoglobins - metabolism | Mice, Transgenic | Mice, Knockout | Dinoprostone - metabolism | Globins - genetics | Tyrosine - metabolism | Animals | Anemia, Sickle Cell - metabolism | Arginine - pharmacology | Cyclooxygenase 2 - metabolism | Hemodynamics - drug effects | Mice | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Anemia, Sickle Cell - genetics | Nitric Oxide Synthase Type II - metabolism | Index Medicus | vasoreactivity | microvascular regulation | sickle cell disease
Journal Article
10.