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PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100777 - e100777
Inhibition of sodium glucose cotransporter 2 (SGLT2) has been reported as a new therapeutic strategy for treating diabetes. However, the effect of SGLT2... 
OXIDATIVE STRESS | ACTIVATION | HYPERGLYCEMIA | SGLT2 INHIBITOR | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | RATS | COMPLICATIONS | MELLITUS | PANCREATIC-FUNCTION | RENAL INJURY | Inflammation - pathology | Oxidative Stress | Kidney Cortex - metabolism | Body Weight - drug effects | Diabetic Nephropathies - drug therapy | Male | Kidney Function Tests | Inflammation - metabolism | Kidney - metabolism | Glomerular Mesangium - metabolism | Glomerular Mesangium - drug effects | Homeostasis - drug effects | Kidney - physiopathology | Disease Models, Animal | Diabetic Nephropathies - pathology | Gene Expression | Glucosides - pharmacology | Kidney Tubules, Proximal - pathology | Kidney - drug effects | Macrophages - pathology | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetic Nephropathies - genetics | Kidney Cortex - pathology | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Blood Glucose - drug effects | Glomerular Mesangium - pathology | Macrophages - metabolism | Animals | Insulin-Secreting Cells - drug effects | Kidney Tubules, Proximal - metabolism | Glucose - metabolism | Inflammation - genetics | Benzhydryl Compounds - pharmacology | Mice | Apoptosis | Oxidases | Type 2 diabetes | Blood sugar | Analysis | Body weight | Diabetic nephropathies | Hemoglobin | DNA polymerases | Gene expression | Health aspects | Oxidative stress | RNA-directed DNA polymerase | Syngeneic grafts | Homeostasis | Glucose | Macrophages | Blood | NAD(P)H oxidase | Biomedical materials | Hyperglycemia | Rodents | Biocompatibility | Blood pressure | Inhibition | Pancreas | Pharmaceutical sciences | Age | University graduates | Neurochemistry | Creatinine | Stresses | Kidneys | Cytokines | Diabetes mellitus | Osteopontin | Inflammation | Dentistry | Metabolism | Stress | Medicine | Studies | Inhibitors | Nephropathy | Sodium | Monocyte chemoattractant protein | Fibrosis | Infiltration | Diabetes | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 10/2014, Volume 86, Issue 4, pp. 828 - 836
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2181 - 2196
Diabetic nephropathy (DN) is among the most lethal complications that occur in type 1 and type 2 diabetics. Podocyte dysfunction is postulated to be a critical... 
EPITHELIAL-MESENCHYMAL TRANSITION | KIDNEY-DISEASE | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | TUBEROUS SCLEROSIS COMPLEX | SLIT DIAPHRAGM | GLOMERULAR PROTEIN | ANGIOTENSIN-II | NEPHRIN EXPRESSION | ENDOPLASMIC-RETICULUM STRESS | NEPHROTIC SYNDROME | Phosphorylation | Ribosomal Protein S6 Kinases - metabolism | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Multiprotein Complexes | Podocytes - enzymology | Diabetic Nephropathies - drug therapy | Endoplasmic Reticulum - metabolism | Male | Mechanistic Target of Rapamycin Complex 1 | Mice, Mutant Strains | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Glomerular Basement Membrane - pathology | Regulatory-Associated Protein of mTOR | Cell Differentiation | Membrane Proteins - metabolism | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Carrier Proteins - physiology | Proteins - physiology | Sirolimus - therapeutic use | Mice, Inbred C57BL | Podocytes - pathology | Mice, Knockout | Carrier Proteins - genetics | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing | Glomerular Mesangium - pathology | Animals | Podocytes - drug effects | Podocytes - physiology | Mice | Protein Processing, Post-Translational | TOR Serine-Threonine Kinases | Enzyme Activation | Proteins - antagonists & inhibitors | Diabetic nephropathies | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2013, Volume 183, Issue 4, pp. 1269 - 1280
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 02/2008, Volume 19, Issue 2, pp. 298 - 309
Several recent reports support the hypothesis that aldosterone contributes to the progression of renal injury. Mineralocorticoids increase the expression of... 
TISSUE GROWTH-FACTOR | INTERCELLULAR-ADHESION MOLECULE-1 | ACTIVATED PROTEIN-KINASE | CELL-VOLUME | RENIN-ANGIOTENSIN SYSTEM | GENE-EXPRESSION | UROLOGY & NEPHROLOGY | DIABETIC-NEPHROPATHY | HYPERTENSIVE-RATS | UP-REGULATION | RENAL INJURY | Antibody Specificity | Transcription, Genetic - drug effects | Phosphorylation | Male | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Glomerular Mesangium - enzymology | Mesangial Cells - drug effects | Immediate-Early Proteins - metabolism | Aldosterone - metabolism | I-kappa B Kinase - metabolism | Mesangial Cells - enzymology | Protein-Serine-Threonine Kinases - metabolism | Immediate-Early Proteins - immunology | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | Rats | Aldosterone - pharmacology | MAP Kinase Kinase 1 - metabolism | Rats, Sprague-Dawley | Antibodies - pharmacology | Glomerular Mesangium - pathology | Transcription, Genetic - physiology | Animals | Immediate-Early Proteins - genetics | Signal Transduction - drug effects | Intercellular Adhesion Molecule-1 - genetics | Fibrosis | Mesangial Cells - pathology | Protein-Serine-Threonine Kinases - immunology | Signal Transduction - physiology | RNA, Small Interfering | Connective Tissue Growth Factor | Index Medicus | Basic Research
Journal Article
Kidney International, ISSN 0085-2538, 11/2010, Volume 78, Issue 9, pp. 905 - 919
We recently found a markedly lower prevalence of vascular complications, including kidney disease, in diabetic patients with Gilbert syndrome, a congenital... 
albuminuria | NADPH oxidase | diabetic nephropathy | oxidative stress | ACTIVATION | RATS | BETA | VASCULAR COMPLICATIONS | HEME OXYGENASE | UROLOGY & NEPHROLOGY | KIDNEY | EXPRESSION | SERUM BILIRUBIN | Diabetes Mellitus, Experimental - drug therapy | Diabetic Nephropathies - etiology | Humans | Transforming Growth Factor beta1 - metabolism | NADPH Oxidases - metabolism | Male | Biliverdine - pharmacology | Time Factors | Glomerular Mesangium - metabolism | Superoxides - metabolism | Glomerular Mesangium - drug effects | Hyperbilirubinemia, Hereditary - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Disease Models, Animal | Diabetic Nephropathies - pathology | Angiotensin II - metabolism | Endothelial Cells - metabolism | Diabetic Nephropathies - metabolism | Down-Regulation | Mice, Inbred C57BL | Cells, Cultured | Rats | Antioxidants - pharmacology | Hyperbilirubinemia, Hereditary - complications | NADPH Oxidase 4 | Albuminuria - prevention & control | Bilirubin - blood | Glomerular Mesangium - pathology | Animals | Rats, Gunn | Glucose - metabolism | Cell Proliferation - drug effects | Mice | Oxidative Stress - drug effects | Blood Glucose - metabolism | Endothelial Cells - drug effects | Index Medicus
Journal Article
Journal Article
Journal Article