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Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 454 - 464
Dense deposit disease and glomerulonephritis with isolated C3 deposits are glomerulopathies characterized by deposits of C3 within or along the glomerular... 
complement | glomerulonephritis | clinical immunology | membranoproliferative glomerulonephritis (MPGN) | LONG-TERM | HEMOLYTIC-UREMIC SYNDROME | MESANGIOCAPILLARY GLOMERULONEPHRITIS | GLOMERULONEPHRITIS TYPE-II | MACULAR DEGENERATION | FACTOR-H DEFICIENCY | GLOMERULAR-FILTRATION-RATE | UROLOGY & NEPHROLOGY | MUTATIONS | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | MEMBRANE COFACTOR PROTEIN | Humans | Child, Preschool | Male | Complement System Proteins - metabolism | Young Adult | Time Factors | DNA Mutational Analysis | Renal Insufficiency - immunology | Child | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Risk Assessment | Complement C3 - metabolism | Gene Frequency | Risk Factors | Glomerulonephritis - genetics | Glomerulonephritis - immunology | Glomerulonephritis - mortality | Biomarkers - blood | Kidney Glomerulus - pathology | Disease Progression | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Phenotype | Complement Factor H - metabolism | Glomerulonephritis, Membranoproliferative - mortality | Glomerulonephritis - pathology | Adolescent | Age of Onset | Membrane Cofactor Protein - genetics | Kidney Glomerulus - immunology | Mutation | Complement Factor I - metabolism | Haplotypes | Complement C3 Nephritic Factor - genetics | Glomerulonephritis - therapy | Infant | Case-Control Studies | Adult | Complement System Proteins - genetics | Female | Membrane Cofactor Protein - metabolism | Complement Pathway, Alternative - genetics | France | Renal Insufficiency - genetics | Kaplan-Meier Estimate | Complement Factor I - genetics | Treatment Outcome | Chi-Square Distribution | Glomerulonephritis, Membranoproliferative - therapy | Biopsy | Complement C3 Nephritic Factor - metabolism | Complement Factor H - genetics
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 465 - 473
C3 glomerulonephritis (C3GN) is a recently described disorder that typically results from abnormalities in the alternative pathway (AP) of complement. Here, we... 
C3GN | proteomics | C3 glomerulopathy | MPGN | alternative pathway of complement | C3 glomerulonephritis | MACULAR DEGENERATION | CLASSIFICATION | RISK | PROTEIN 5 | DENSE DEPOSIT DISEASE | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | Complement C3 - analysis | Predictive Value of Tests | Recurrence | Follow-Up Studies | Humans | Middle Aged | Complement System Proteins - immunology | Laser Capture Microdissection | Molecular Sequence Data | Male | Glomerulonephritis, Membranoproliferative - complications | Complement System Proteins - analysis | Young Adult | Time Factors | DNA Mutational Analysis | Mass Spectrometry | Adult | Autoantibodies - analysis | Complement System Proteins - genetics | Female | Complement Pathway, Alternative - genetics | Proteomics - methods | Child | Amino Acid Sequence | Glomerulonephritis, Membranoproliferative - pathology | Genetic Predisposition to Disease | Proteinuria - immunology | Treatment Outcome | Complement C3 Nephritic Factor - analysis | Hematuria - immunology | Kidney Transplantation | Minnesota | Kidney Glomerulus - pathology | Glomerulonephritis, Membranoproliferative - therapy | Glomerulonephritis, Membranoproliferative - genetics | Glomerulonephritis, Membranoproliferative - immunology | Biopsy | Adolescent | Aged | Kidney Glomerulus - immunology | Mutation | Complement Factor H - genetics
Journal Article
Journal Article
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 2015, Volume 10, Issue 4, pp. 592 - 600
Background and objectives Steroid-resistant nephrotic syndrome is a rare kidney disease involving either immune-mediated or genetic alterations of podocyte... 
UROLOGY & NEPHROLOGY | PATTERNS | THERAPY | MUTATIONS | CYCLOPHOSPHAMIDE | NPHS2 | Recurrence | Age Distribution | Prospective Studies | Glomerulosclerosis, Focal Segmental - genetics | Humans | Immunosuppressive Agents - therapeutic use | Nephrosis, Lipoid - epidemiology | Child, Preschool | Infant | Male | Nephrotic Syndrome - genetics | Nephrotic Syndrome - congenital | Young Adult | DNA Mutational Analysis | Female | Registries | Nephrotic Syndrome - therapy | Retrospective Studies | Child | Nephrotic Syndrome - diagnosis | Infant, Newborn | Latin America - epidemiology | Genetic Predisposition to Disease | Nephrosis, Lipoid - diagnosis | Glomerulosclerosis, Focal Segmental - epidemiology | Risk Factors | Europe - epidemiology | Treatment Outcome | Kidney Transplantation | Nephrosis, Lipoid - therapy | Genetic Markers | Nephrosis, Lipoid - genetics | Glomerulonephritis, Membranoproliferative - therapy | Remission Induction | Glomerulosclerosis, Focal Segmental - diagnosis | Glomerulonephritis, Membranoproliferative - genetics | Middle East - epidemiology | Phenotype | Biopsy | Adolescent | Age of Onset | Nephrotic Syndrome - epidemiology | Glomerulonephritis, Membranoproliferative - diagnosis | Glomerulosclerosis, Focal Segmental - therapy | Mutation | Glomerulonephritis, Membranoproliferative - epidemiology | podocytopathies | nephrin | children | WT1 | Original | podocin
Journal Article