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1. Full Text AJKD Atlas of Renal Pathology: Focal Segmental Glomerulosclerosis
by Fogo, Agnes B., MD and Lusco, Mark A., MD and Najafian, Behzad, MD and Alpers, Charles E., MD
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 66, Issue 2, pp. e1 - e2
Nephrology | UROLOGY & NEPHROLOGY | Kidney Glomerulus - ultrastructure | Kidney - pathology | Atlases as Topic | Humans | Kidney Glomerulus - pathology | Glomerulosclerosis, Focal Segmental - pathology | Kidney - ultrastructure | Index Medicus
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2. Full Text MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis
by Ahuja, Tejinder S and Berns, Jeffrey S and Nelson, George W and Kimmel, Paul L and Winkler, Cheryl A and Johnson, Randall C and Freedman, Barry I and Kajiyama, Hiroshi and Vlahov, David and Trachtman, Howard and Smith, Michael W and McKenzie, Louise M and Cho, Monique E and Dart, Richard A and Briggs, William and Kopp, Jeffrey B and Korbet, Stephen M and Bowden, Donald W and Michel, Donna M and Mokrzycki, Michele H and Simon, Eric and Oleksyk, Taras and Schelling, Jeffrey R
Nature Genetics, ISSN 1061-4036, 10/2008, Volume 40, Issue 10, pp. 1175 - 1184
The increased burden of chronic kidney and end-stage kidney diseases (ESKD) in populations of African ancestry has been largely unexplained. To identify... 
UNITED-STATES | HAPLOTYPE MAP | GENETICS & HEREDITY | AFRICAN-AMERICANS | CHRONIC KIDNEY-DISEASE | HYPERTENSIVE NEPHROSCLEROSIS | ADMIXTURE | GENOME-SCAN | LOCUS | NEPHROPATHY | STAGE RENAL-DISEASE | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease - genetics | Glomerulosclerosis, Focal Segmental - genetics | Humans | Middle Aged | Risk Factors | African Americans - genetics | Myosin Heavy Chains - genetics | Male | Chromosome Mapping | DNA Primers - chemistry | Molecular Motor Proteins - genetics | Case-Control Studies | Lod Score | AIDS-Associated Nephropathy - genetics | Chromosomes, Human, Pair 22 - genetics | Haplotypes - genetics | Polymorphism, Single Nucleotide - genetics | Adult | Female | Hypertension - genetics | Genome, Human | Glomerulosclerosis, Focal Segmental - pathology | Genetic Linkage | Glomerulonephritis | Chromosome mapping | Demographic aspects | Genetic variation | African Americans | Genetic aspects | Research | Health aspects | Methods | Risk factors | Medical research | Genetics | Genes | Cancer | Index Medicus
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3. Full Text Focal segmental glomerulosclerosis is induced by microRNA-193a and its downregulation of WT1
by Gebeshuber, Christoph A and Kornauth, Christoph and Dong, Lihua and Sierig, Ralph and Seibler, Jost and Reiss, Martina and Tauber, Stefanie and Bilban, Martin and Wang, Shijun and Kain, Renate and Böhmig, Georg A and Moeller, Marcus J and Gröne, Hermann-Josef and Englert, Christoph and Martinez, Javier and Kerjaschki, Dontscho
Nature Medicine, ISSN 1078-8956, 04/2013, Volume 19, Issue 4, pp. 481 - 487
Focal segmental glomerulosclerosis (FSGS) is a frequent and severe glomerular disease characterized by destabilization of podocyte foot processes. We report... 
MEDICINE, RESEARCH & EXPERIMENTAL | AMINONUCLEOSIDE NEPHROSIS | NEPHRIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | KIDNEY DEVELOPMENT | MODEL | CELL BIOLOGY | GENE-EXPRESSION | MICE | GLOMERULI | CELL | PROTEIN PODOCALYXIN | PODOCYTE FUNCTION | Podocytes - metabolism | Kidney - pathology | Glomerulosclerosis, Focal Segmental - genetics | Humans | Male | Mice, Transgenic | Mice, Knockout | Down-Regulation - physiology | WT1 Proteins - physiology | Animals | Glomerulosclerosis, Focal Segmental - etiology | Doxycycline - pharmacology | Mice | MicroRNAs - physiology | Glomerulosclerosis, Focal Segmental - pathology | Glomerulonephritis | Development and progression | MicroRNA | Research | Health aspects | Proteins | Pathology | Kidney diseases | Gene expression | Ribonucleic acid--RNA | Tumors | Index Medicus
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4. Full Text Parietal epithelial cells participate in the formation of sclerotic lesions in focal segmental glomerulosclerosis
by Smeets, B and Kuppe, C and Sicking, E.M and Fuss, A and Jirak, P and Kuppevelt, A.H.M.S.M. van and Endlich, K and Wetzels, J.F.M and Grone, H.J and Floege, J and Moeller, M.J
Journal of the American Society of Nephrology, ISSN 1046-6673, 2011, Volume 22, Issue 7, pp. 1262 - 1274
The pathogenesis of the development of sclerotic lesions in focal segmental glomerulosclerosis (FSGS) remains unknown. Here, we selectively tagged podocytes or... 
THY-1.1 TRANSGENIC MICE | MURINE MODEL | ORIGIN | CRESCENTS | ALBUMINURIA | MOUSE | RATS | UROLOGY & NEPHROLOGY | COLLAPSING GLOMERULOPATHY | PODOCYTES | EXPRESSION | Epithelial Cells - metabolism | Humans | Desoxycorticosterone | Extracellular Matrix - metabolism | Rats | Epithelial Cells - pathology | Mice, Transgenic | Podocytes - pathology | Sodium Chloride, Dietary | Cell Lineage | Animals | Nephrectomy | Glomerulosclerosis, Focal Segmental - etiology | Thy-1 Antigens - genetics | Hyaluronan Receptors - metabolism | Mice | Glomerulosclerosis, Focal Segmental - pathology | Index Medicus | Basic Research
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5. Full Text MYO1E Mutations and Childhood Familial Focal Segmental Glomerulosclerosis
by Mele, Caterina and Iatropoulos, Paraskevas and Donadelli, Roberta and Calabria, Andrea and Maranta, Ramona and Cassis, Paola and Buelli, Simona and Tomasoni, Susanna and Piras, Rossella and Krendel, Mira and Bettoni, Serena and Morigi, Marina and Delledonne, Massimo and Pecoraro, Carmine and Abbate, Isabella and Capobianchi, Maria Rosaria and Hildebrandt, Friedhelm and Otto, Edgar and Schaefer, Franz and Macciardi, Fabio and Ozaltin, Fatih and Emre, Sevinc and Ibsirlioglu, Tulin and Benigni, Ariela and Remuzzi, Giuseppe and Noris, Marina and PodoNet Consortium
The New England Journal of Medicine, ISSN 0028-4793, 07/2011, Volume 365, Issue 4, pp. 295 - 306
Focal segmental glomerulosclerosis (FSGS) affects the podocytes. The molecular cause of over half the cases is unknown, and treatment is elusive. This study... 
ORGANIZATION | MEDICINE, GENERAL & INTERNAL | DISEASES | PROTEIN | GENE | RESISTANT NEPHROTIC SYNDROME | ACTIN CYTOSKELETON | GLOMERULAR PODOCYTES | IDENTIFICATION | ENDOCYTOSIS | CYCLOSPORINE | Kidney Glomerulus - blood supply | Glucocorticoids - therapeutic use | Glomerulosclerosis, Focal Segmental - genetics | Humans | Child, Preschool | Infant | Male | Mutation, Missense | Kidney Glomerulus - ultrastructure | Female | Myosin Type I - chemistry | Child | Drug Resistance | Genetic Linkage | Genome-Wide Association Study | Podocytes - metabolism | Myosin Type I - genetics | Genes, Recessive | Myosin Type I - metabolism | Podocytes - ultrastructure | Sequence Alignment | Animals | Pedigree | Age of Onset | Mice | Mutation | Glomerulosclerosis, Focal Segmental - pathology | Microscopy, Fluorescence | Care and treatment | Usage | Linkage (Genetics) | Gene mutations | Genetic aspects | Research | Kidney diseases | Diagnosis | End-stage renal disease | Motility | Transplants & implants | Glucocorticoids | Families & family life | Genomes | Proteins | Transfection | Next-generation sequencing | Rodents | Calcium-binding protein | Myosin | Children | Localization | Age | Calmodulin | Siblings | Linkage analysis | Urine | Filtration | Kidneys | Nephrotic syndrome | Electron microscopy | Patients | Microscopy | Biopsy | Index Medicus | Abridged Index Medicus
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6. Full Text A phase 1, single-dose study of fresolimumab, an anti-TGF-β antibody, in treatment-resistant primary focal segmental glomerulosclerosis
by Trachtman, Howard and Fervenza, Fernando C and Gipson, Debbie S and Heering, Peter and Jayne, David R.W and Peters, Harm and Rota, Stefano and Remuzzi, Giuseppe and Rump, L. Christian and Sellin, Lorenz K and Heaton, Jeremy P.W and Streisand, James B and Hard, Marjie L and Ledbetter, Steven R and Vincenti, Flavio
Kidney International, ISSN 0085-2538, 06/2011, Volume 79, Issue 11, pp. 1236 - 1243
Primary focal segmental glomerulosclerosis (FSGS) is a disease with poor prognosis and high unmet therapeutic need. Here, we evaluated the safety and... 
clinical study | FSGS | fresolimumab | TGF-β antagonism | ADULT PATIENTS | MYCOPHENOLATE-MOFETIL TREATMENT | PARTIAL REMISSION | STEROID-RESISTANT | TGF-beta antagonism | RANDOMIZED-TRIAL | RENAL-DISEASE | CONVERTING ENZYME-INHIBITORS | CYCLOSPORINE-A | UROLOGY & NEPHROLOGY | NEPHROTIC SYNDROME | GLOMERULAR-DISEASE | Biomarkers - urine | Kidney - pathology | United States | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Kidney - immunology | Antibodies, Monoclonal - therapeutic use | Male | Dose-Response Relationship, Drug | Young Adult | Glomerular Filtration Rate - drug effects | Glomerulosclerosis, Focal Segmental - drug therapy | Transforming Growth Factor beta - antagonists & inhibitors | Adult | Female | Glomerulosclerosis, Focal Segmental - physiopathology | Kidney - physiopathology | Transforming Growth Factor beta - immunology | Kidney - drug effects | Proteinuria - immunology | Europe | Antibodies, Monoclonal - pharmacokinetics | Infusions, Parenteral | Treatment Outcome | Creatinine - urine | Proteinuria - drug therapy | Antibodies, Monoclonal - administration & dosage | Biopsy | Glomerulosclerosis, Focal Segmental - immunology | Glomerulosclerosis, Focal Segmental - pathology | Index Medicus | Original
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7. Full Text Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis
by Akilesh, Shreeram and Suleiman, Hani and Yu, Haiyang and Stander, M. Christine and Lavin, Peter and Gbadegesin, Rasheed and Antignac, Corinne and Pollak, Martin and Kopp, Jeffrey B and Winn, Michelle P and Shaw, Andrey S
Journal of Clinical Investigation, ISSN 0021-9738, 10/2011, Volume 121, Issue 10, pp. 4127 - 4137
The specialized epithelial cell of the kidney, the podocyte, has a complex actin-based cytoskeleton. Dynamic regulation of this cytoskeleton is required for... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELL MOTILITY | DISEASE | RHO | DYNAMICS | PROTEINURIA | KIDNEY PODOCYTES | ACTIN CYTOSKELETON | REARRANGEMENTS | FILAMIN-A | NEF | Glomerulosclerosis, Focal Segmental - genetics | Humans | Molecular Sequence Data | Male | Neuropeptides - physiology | Gene Knockdown Techniques | rac GTP-Binding Proteins - antagonists & inhibitors | Cell Shape | Cell Membrane - pathology | Female | Cell Differentiation | Glomerulosclerosis, Focal Segmental - physiopathology | rac GTP-Binding Proteins - physiology | GTPase-Activating Proteins - physiology | Amino Acid Sequence | GTPase-Activating Proteins - antagonists & inhibitors | Mutant Proteins - genetics | Mutant Proteins - physiology | Podocytes - pathology | Sequence Homology, Amino Acid | Animals | Podocytes - physiology | Pedigree | Mice | GTPase-Activating Proteins - genetics | rac1 GTP-Binding Protein | Glomerulosclerosis, Focal Segmental - pathology | Neuropeptides - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
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8. Full Text Rare hereditary COL4A3/COL4A4 variants may be mistaken for familial focal segmental glomerulosclerosis
by Malone, Andrew F and Phelan, Paul J and Hall, Gentzon and Cetincelik, Umran and Homstad, Alison and Alonso, Andrea S and Jiang, Ruiji and Lindsey, Thomas B and Wu, Guanghong and Sparks, Matthew A and Smith, Stephen R and Webb, Nicholas J.A and Kalra, Philip A and Adeyemo, Adebowale A and Shaw, Andrey S and Conlon, Peter J and Charles Jennette, J and Howell, David N and Winn, Michelle P and Gbadegesin, Rasheed A
Kidney International, ISSN 0085-2538, 12/2014, Volume 86, Issue 6, pp. 1253 - 1259
Focal segmental glomerulosclerosis (FSGS) is a histological lesion with many causes, including inherited genetic defects, with significant proteinuria being... 
focal segmental glomerulosclerosis | proteinuria | Alport syndrome | podocyte | Podocyte | Focal segmental glomerulosclerosis | Proteinuria | COL4A4 GENE | ADULTS | NATURAL-HISTORY | LAMININ BETA-2 | FEATURES | BASEMENT-MEMBRANE NEPHROPATHY | DISEASE | UROLOGY & NEPHROLOGY | DOMINANT ALPORT-SYNDROME | MUTATIONS | NEPHROTIC SYNDROME | Genetic Testing | Glomerulosclerosis, Focal Segmental - genetics | Humans | Middle Aged | Genotype | Male | Glomerulosclerosis, Focal Segmental - complications | Mutation, Missense | Autoantigens - genetics | Exome | Hearing Loss - genetics | Young Adult | Phenotype | Podocytes - ultrastructure | DNA Mutational Analysis | Adolescent | Collagen Type IV - genetics | Adult | Female | Glomerular Basement Membrane - ultrastructure | Child | Glomerulosclerosis, Focal Segmental - pathology | Hematuria - etiology | Proteinuria - etiology | Index Medicus
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9. Full Text Pathologic classification of focal segmental glomerulosclerosis: a working proposal
by D'Agati, Vivette D and Fogo, Agnes B and Bruijn, Jan A and Jennette, J.Charles
American Journal of Kidney Diseases, ISSN 0272-6386, 02/2004, Volume 43, Issue 2, pp. 368 - 382
GLOMERULAR TIP LESION | MUTATION | SCLEROSIS | ADULTS | UROLOGY & NEPHROLOGY | PROTEINURIA | NEPHROTIC SYNDROME | COLLAPSING GLOMERULOPATHY | NEPHROPATHY | PROGRESSION | CHILDREN | Glomerulosclerosis, Focal Segmental - classification | Microscopy, Electron | Biopsy | Humans | Glomerulosclerosis, Focal Segmental - pathology | Microscopy, Fluorescence | Index Medicus
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10. Full Text Glomerular parietal epithelial cells contribute to adult podocyte regeneration in experimental focal segmental glomerulosclerosis
by Eng, Diana G and Sunseri, Maria W and Kaverina, Natalya V and Roeder, Sebastian S and Pippin, Jeffrey W and Shankland, Stuart J
Kidney International, ISSN 0085-2538, 11/2015, Volume 88, Issue 5, pp. 999 - 1012
As adult podocytes cannot adequately proliferate following depletion in disease states, there has been interest in the potential role of progenitors in... 
progenitor | glomerulosclerosis | podocin | CD44 | glomerulus | ERK | Glomerulosclerosis | Glomerulus | Progenitor | Podocin | ACTIVATION | RENIN LINEAGE | INJURY | RENAL PROGENITORS | NEPHROPATHY | IN-VIVO | DISEASE | UROLOGY & NEPHROLOGY | MICE | PROTEINS | EXPRESSION | Cell Proliferation | Albuminuria - etiology | Epithelial Cells - chemistry | Intracellular Signaling Peptides and Proteins - analysis | Cyclin-Dependent Kinase Inhibitor p57 - analysis | Microfilament Proteins - analysis | Bowman Capsule - pathology | Glomerulosclerosis, Focal Segmental - complications | Podocytes - pathology | Membrane Proteins - analysis | Glomerulosclerosis, Focal Segmental - metabolism | MAP Kinase Signaling System | Hyaluronan Receptors - analysis | Epithelial Cells - physiology | Regeneration | Animals | Podocytes - physiology | beta-Galactosidase - metabolism | Stem Cells - physiology | Mice | Glomerulosclerosis, Focal Segmental - pathology | Podocytes - chemistry | Cell Movement | Disease Models, Animal | Index Medicus | synaptopodin | nephrin
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11. Full Text Glomerular aging and focal global glomerulosclerosis: A podometric perspective
by Hodgin, Jeffrey B and Bitzer, Markus and Wickman, Larysa and Afshinnia, Farsad and Wang, Su Q and O'Connor, Christopher and Yang, Yan and Meadowbrooke, Chrysta and Chowdhury, Mahboob and Kikuchi, Masao and Wiggins, Jocelyn E and Wiggins, Roger C
Journal of the American Society of Nephrology, ISSN 1046-6673, 12/2015, Volume 26, Issue 12, pp. 3162 - 3178
Kidney aging is associated with an increasing proportion of globally scarred glomeruli, decreasing renal function, and exponentially increasing ESRD... 
SEGMENTAL GLOMERULOSCLEROSIS | NUMBER | TYPE-1 DIABETIC-PATIENTS | DISEASE | URINE | RENAL-FUNCTION | INJURY | UROLOGY & NEPHROLOGY | KIDNEY | PODOCYTE LOSS | PROGRESSION | Age Factors | Cell Count | Humans | Middle Aged | Child, Preschool | Organ Size | Podocytes - pathology | Hypertrophy - pathology | Aging - pathology | Young Adult | Cell Nucleus - pathology | Adolescent | Aged, 80 and over | Adult | Aged | Child | Glomerulosclerosis, Focal Segmental - pathology | Index Medicus | aging | Clinical Research | glomerulosclerosis | podocyte
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12. Full Text Endothelial mitochondrial oxidative stress determines podocyte depletion in segmental glomerulosclerosis
by Daehn, Ilse and Casalena, Gabriella and Zhang, Taoran and Shi, Shaolin and Fenninger, Franz and Barasch, Nicholas and Yu, Liping and D'Agati, Vivette and Schlondorff, Detlef and Kriz, Wilhelm and Haraldsson, Borje and Bottinger, Erwin P and Sahlgrenska akademin and Institute of Medicine, Department of Molecular and Clinical Medicine and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1608 - 1621
Focal segmental glomerular sclerosis (FSGS) is a primary kidney disease that is commonly associated with proteinuria and progressive loss of glomerular... 
MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-FACTOR-BETA | NADPH-OXIDASE | TGF-BETA | MESANGIAL CELL | NITRIC-OXIDE SYNTHASE | INJURY | GLOMERULAR-FILTRATION BARRIER | DIABETIC-NEPHROPATHY | CHRONIC KIDNEY-DISEASE | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Oxidative Stress | Glomerulosclerosis, Focal Segmental - genetics | Humans | Male | RNA, Messenger - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Cell Line | Podocytes - metabolism | Signal Transduction | Endothelin-1 - genetics | Endothelin-1 - metabolism | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Mitochondria - metabolism | Podocytes - pathology | Receptor, Endothelin A - genetics | Glomerulosclerosis, Focal Segmental - metabolism | Animals | Receptors, Transforming Growth Factor beta - metabolism | Endothelium, Vascular - metabolism | Models, Biological | Endothelium, Vascular - pathology | Receptor, Endothelin A - metabolism | Mice | Mice, Inbred BALB C | Transforming Growth Factor beta - metabolism | Glomerulosclerosis, Focal Segmental - pathology | Oxidative stress | Mitochondria | Research | Kidney diseases | Transforming growth factors | Endothelium | Cell culture | Cell growth | Microscopy | Transgenic animals | Biopsy | Rodents | Mortality | Ligands | Software | Diabetes | Apoptosis | Index Medicus | Abridged Index Medicus | KIDNEY-DISEASE | CHRONIC | Endokrinologi och diabetes | Endocrinology and Diabetes
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