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Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6691
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 937 - 959
Until recently, significant advances in our understanding of the mechanisms of blood pressure regulation arose from studies of monogenic forms of hypertension... 
blood pressure | uromodulin | exome | hypertension | natriuretic peptide, brain | genome-wide association study | COMMON VARIANTS | ALDOSTERONE-PRODUCING ADENOMAS | CARDIAC & CARDIOVASCULAR SYSTEMS | KELCH-LIKE 3 | SECONDARY HYPERTENSION | SOMATIC MUTATIONS | MINERALOCORTICOID RECEPTOR | NATRIURETIC-PEPTIDE | MISSING HERITABILITY | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | AMBULATORY BLOOD-PRESSURE | GENOME-WIDE ASSOCIATION | Kidney Diseases - physiopathology | Paraganglioma - physiopathology | Oligonucleotide Array Sequence Analysis | Sodium, Dietary - adverse effects | Blood Pressure - genetics | Humans | Hypertension - drug therapy | Male | Sympathetic Nervous System - physiopathology | Hypotension - genetics | Kidney Diseases - genetics | Pheochromocytoma - physiopathology | Hypertension, Pregnancy-Induced - genetics | Bartter Syndrome - genetics | Hyperaldosteronism - physiopathology | Glucocorticoids - physiology | Female | Blood Pressure - physiology | Neoplastic Syndromes, Hereditary - genetics | Hypertension - genetics | Mineralocorticoids - physiology | Genetic Predisposition to Disease | Genome-Wide Association Study | Models, Cardiovascular | Paraganglioma - genetics | Hyperaldosteronism - genetics | Hypertension, Pregnancy-Induced - physiopathology | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Hypertension - physiopathology | Pregnancy | Adrenal Gland Neoplasms - physiopathology | Sodium, Dietary - pharmacokinetics | Neoplastic Syndromes, Hereditary - physiopathology | Pheochromocytoma - genetics | Bartter Syndrome - physiopathology | Polymorphism, Single Nucleotide | Mutation | Adrenal Gland Neoplasms - genetics
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e108578
Background: Pre-term birth (PTB) remains the leading cause of infant mortality and morbidity. Its etiology is multifactorial, with a strong genetic component.... 
ETHNIC-DIFFERENCES | INTERLEUKIN-6 IL-6 | PREMATURE RUPTURE | TUMOR-NECROSIS-FACTOR | INCREASED RISK | MULTIDISCIPLINARY SCIENCES | RACIAL DISPARITY | FACTOR-ALPHA GENE | FAMILIAL PATTERNS | FETAL MEMBRANES | TNF-ALPHA | Genetic Predisposition to Disease | Premature Birth - genetics | Receptors, Estrogen - genetics | Infant Mortality | Humans | Autoimmunity - genetics | Infant | Fetal Membranes, Premature Rupture - epidemiology | Premature Birth - epidemiology | STAT1 Transcription Factor - genetics | Interferon Regulatory Factor-3 - genetics | Polymorphism, Genetic | Pregnancy | Fetal Membranes, Premature Rupture - genetics | NF-kappa B - genetics | Pregnancy Complications - genetics | Receptors, Glucocorticoid - genetics | Female | Infant, Newborn | Pregnancy Complications - epidemiology | PPAR gamma - genetics | Membranes | Regulators | Transcription factors | Racial differences | Glucocorticoids | Identification methods | Pathogenesis | Genes | Premature birth | Genomics | Estrogens | Childrens health | Inflammatory response | Genomes | Single-nucleotide polymorphism | Birth | Pathways | Etiology | Genetic factors | Stat1 protein | Cytokines | Infant mortality | Inflammation | Metabolism | Morbidity | Children & youth | Medicine | Studies | Interferon regulatory factor 3 | Womens health | Collagen | Interferon | Peroxisome proliferator-activated receptors
Journal Article
Journal Article
Comparative Biochemistry and Physiology, Part A, ISSN 1095-6433, 10/2014, Volume 176, pp. 59 - 64
Chronic endogenous glucocorticoid (GC) excess in mammals is associated with metabolic dysfunction and dyslipidemia that are characterized by increased plasma... 
Pectoralis major muscle | Broilers | Corticosterone | Cholesterol metabolism | Apolipoproteins B - metabolism | Gene Expression - genetics | Muscle, Skeletal - metabolism | Receptors, Glucocorticoid - metabolism | Cholesterol - genetics | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - genetics | Chickens - metabolism | Glucocorticoids - genetics | Glucocorticoids - metabolism | Chickens - genetics | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - genetics | Chickens - physiology | Stress, Physiological - physiology | Receptors, LDL - genetics | Corticosterone - genetics | Stress, Physiological - genetics | RNA, Messenger - genetics | Receptors, LDL - metabolism | Up-Regulation - genetics | Apolipoproteins B - genetics | Cholesterol - metabolism | Down-Regulation - genetics | Triglycerides - metabolism | Cholesterol 7-alpha-Hydroxylase - genetics | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - metabolism | Animals | Cholestanetriol 26-Monooxygenase - genetics | Cholestanetriol 26-Monooxygenase - metabolism | Cholesterol 7-alpha-Hydroxylase - metabolism | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - metabolism | Receptors, Glucocorticoid - genetics | Corticosterone - metabolism | Transcription, Genetic - genetics | Triglycerides - genetics | PHYSIOLOGY | ZOOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | Deregulation | RNA | Gene expression | Cholesterol | Cytochrome P-450
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e14739
Journal Article
Nature Neuroscience, ISSN 1097-6256, 01/2013, Volume 16, Issue 1, pp. 33 - 41
Although the fact that genetic predisposition and environmental exposures interact to shape development and function of the human brain and, ultimately, the... 
POSTTRAUMATIC-STRESS-DISORDER | METHYLATION | GLUCOCORTICOID-RECEPTOR | POLYMORPHISMS | ABUSE | INCREASES | RISK | MODERATION | EXPRESSION | NEUROSCIENCES | ADULT DEPRESSION | Neuroimaging | Organophosphates - metabolism | Stress Disorders, Post-Traumatic - etiology | Humans | Middle Aged | Male | Gene-Environment Interaction | Young Adult | Dexamethasone - pharmacology | Transfection | HEK293 Cells | Adult | Female | Neurons - metabolism | Peptide Elongation Factor 1 - genetics | Hydrocortisone - blood | Neurons - drug effects | Child | Cell Line | Genetic Predisposition to Disease | Introns - genetics | Gene Frequency | Gene Expression Regulation | Models, Molecular | Genotype | Logistic Models | DNA Methylation - genetics | Signal Transduction - genetics | Hippocampus - cytology | Tacrolimus Binding Proteins - genetics | Stress Disorders, Post-Traumatic - blood | Signal Transduction - drug effects | Child Abuse - psychology | Glucocorticoids - chemistry | Glucocorticoids - pharmacology | Polymorphism, Single Nucleotide - genetics | Stress Disorders, Post-Traumatic - genetics | DNA Methylation - drug effects | Peptide Elongation Factor 1 - metabolism | Cohort Studies | Care and treatment | Corticosteroids | DNA | Physiological aspects | Research | Methylation | Binding proteins | Health aspects | Risk factors | Post-traumatic stress disorder in children | PTSD | Emotional Abuse | Child Sexual Abuse | Predisposition | Epigenetics | Genetics | Genetic Markers | Child Abuse | Neurophysiology
Journal Article
Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7388, pp. 222 - 226
Cognitive decline is a debilitating feature of most neurodegenerative diseases of the central nervous system, including Alzheimer's disease(1). The causes... 
REMOTE MEMORIES | LONG-TERM-MEMORY | CDK5 | CHROMATIN | PHOSPHORYLATION | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | CLINICAL-IMPLICATIONS | SYNAPTIC PLASTICITY | EXPRESSION | TRANSGENIC MICE | Memory Disorders - physiopathology | Alzheimer Disease - complications | Memory Disorders - genetics | Peptide Fragments - toxicity | Humans | Neuronal Plasticity - drug effects | Receptors, Glucocorticoid - metabolism | Hippocampus - drug effects | RNA Polymerase II - metabolism | Memory Disorders - complications | Promoter Regions, Genetic - drug effects | Promoter Regions, Genetic - genetics | Gene Knockdown Techniques | Brain - metabolism | Neuronal Plasticity - genetics | Epigenesis, Genetic - drug effects | Phosphorylation - drug effects | Disease Models, Animal | Hydrogen Peroxide - toxicity | Histone Deacetylase 2 - genetics | Alzheimer Disease - physiopathology | Amyloid beta-Peptides - toxicity | Brain - physiopathology | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - complications | Brain - drug effects | Gene Expression Regulation - drug effects | Hippocampus - metabolism | Acetylation - drug effects | Animals | Neurodegenerative Diseases - physiopathology | Histone Deacetylase 2 - deficiency | Mice | Histone Deacetylase 2 - metabolism | Histones - metabolism | Alzheimer Disease - genetics | Epigenetic inheritance | Complications and side effects | Causes of | Nervous system | Genetic aspects | Degeneration | Cognition disorders | Health aspects | Epigenetics | Genetics | RNA polymerase | Kinases | Neurodegeneration | Cyclin-dependent kinases
Journal Article