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Cellular Microbiology, ISSN 1462-5814, 06/2006, Volume 8, Issue 6, pp. 961 - 971
Journal Article
FASEB Journal, ISSN 0892-6638, 08/2012, Volume 26, Issue 8, pp. 3321 - 3335
Journal Article
Acta Physiologica, ISSN 1748-1708, 05/2009, Volume 196, Issue 1, pp. 81 - 98
As the liver is central in the maintenance of glucose homeostasis and energy storage, knowledge of the physiology as well as physiopathology of hepatic energy... 
hepatic glucose production | fatty liver | type 2 diabetes | AMP-activated protein kinase | liver | energy metabolism | AMP‐activated protein kinase | Type 2 diabetes | Energy metabolism | Fatty liver | Hepatic glucose production | Liver | PHYSIOLOGY | LIPID-METABOLISM | CREB COACTIVATOR TORC2 | ACETYL-COA CARBOXYLASE | MICE LACKING ADIPONECTIN | LIVER-DISEASE | ISOLATED RAT HEPATOCYTES | SKELETAL-MUSCLE | FATTY-ACID OXIDATION | INSULIN-RESISTANCE | ADIPOSE-TISSUE | AMP-Activated Protein Kinases - metabolism | Liver - enzymology | Hypoglycemic Agents - metabolism | Humans | Homeostasis | Protein Subunits - metabolism | Liver Cirrhosis - metabolism | Aminoimidazole Carboxamide - metabolism | Energy Metabolism - physiology | Protein Subunits - genetics | Liver Cirrhosis - drug therapy | Dyslipidemias - drug therapy | Ribonucleotides - metabolism | Fatty Liver - metabolism | Fatty Liver - physiopathology | Lipid Metabolism | Mitochondria - metabolism | AMP-Activated Protein Kinases - chemistry | Fatty Liver - drug therapy | Gluconeogenesis - physiology | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Dyslipidemias - metabolism | Glucose - metabolism | Protein Conformation | Liver - cytology | Protein Subunits - chemistry | Liver Cirrhosis - physiopathology | AMP-Activated Protein Kinases - genetics | Dyslipidemias - physiopathology | Glucose metabolism | Enzymes | Liver diseases | Physiological aspects | Protein biosynthesis | Insulin resistance | Glucose | Protein kinases | Health aspects | Fatty acids | Dextrose | cytology | Protein Subunits | Dyslipidemias | AMP-Activated Protein Kinases | therapeutic | Hypoglycemic Agents | Aminoimidazole Carboxamide | Mitochondria | genetics | Gluconeogenesis | chemistry | drug therapy | physiology | Liver Cirrhosis | enzymology | Fatty Liver | physiopathology | Ribonucleotides | Energy Metabolism | analogs & derivatives | MP-activated protein kinase | metabolism
Journal Article
Trends in Endocrinology & Metabolism, ISSN 1043-2760, 10/2018, Volume 29, Issue 10, pp. 723 - 735
Salt-inducible kinases (SIKs) represent a subfamily of AMP-activated protein kinase (AMPK) family kinases. Initially named because SIK1 (the founding member of... 
kinase | second messengers | osteoporosis | inflammation | glucose metabolism | skin pigmentation | HEPATIC GLUCONEOGENESIS | GLUCOSE-HOMEOSTASIS | INFLAMMATORY-BOWEL-DISEASE | HISTONE DEACETYLASES | DENDRITIC CELLS | ENDOCRINOLOGY & METABOLISM | IL-10 PRODUCTION | DOWN-REGULATION | PARATHYROID-HORMONE | CREB COACTIVATOR TORC2 | POSTMENOPAUSAL WOMEN
Journal Article
Journal Article
Gut, ISSN 0017-5749, 12/2018, Volume 67, Issue 12, pp. 2192 - 2203
ObjectiveThe AAA+ ATPase Reptin is overexpressed in hepatocellular carcinoma and preclinical studies indicate that it could be a relevant therapeutic target.... 
diabetes mellitus | fatty liver | liver | glucose metabolism | lipid metabolism | HEPATOCELLULAR-CARCINOMA | CELL SIZE | ATPASE ACTIVITY | REPTIN | INSULIN-RESISTANCE | LIPOGENESIS | DOWNSTREAM | GROWTH | AKT | COMPLEX INTEGRITY | GASTROENTEROLOGY & HEPATOLOGY | Diet, High-Fat - adverse effects | Hepatocytes - metabolism | Glucose Intolerance - prevention & control | Insulin Resistance - physiology | ATPases Associated with Diverse Cellular Activities - physiology | Energy Metabolism - physiology | DNA Helicases - genetics | Lipogenesis - physiology | Glucose Intolerance - physiopathology | Disease Models, Animal | Fatty Liver - metabolism | Liver - metabolism | Fatty Liver - prevention & control | DNA Helicases - deficiency | Mechanistic Target of Rapamycin Complex 1 - metabolism | Mice, Knockout | Mechanistic Target of Rapamycin Complex 2 - metabolism | Animals | Glucose - metabolism | Signal Transduction - physiology | Body Weight - physiology | Adenosine Triphosphatases - physiology | Lipid Metabolism - physiology | DNA Helicases - physiology | Fatty Liver - etiology | Transcription | Body fat | Homeostasis | Lipids | Hepatocellular carcinoma | Glucose | Metabolic syndrome | Kinases | High fat diet | Proteins | Liver cancer | Glucose metabolism | Hyperglycemia | Fatty liver | Clonal deletion | Rodents | Lipid metabolism | Lipogenesis | Gluconeogenesis | Phenotypes | Liver diseases | Hyperlipidemia | Gene expression |