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1. Full Text Thioredoxin-interacting protein A critical link between glucose toxicity and β-cell apoptosis
by Chen, Junqin and Saxena, Geetu and Mungrue, Imran N and Lusis, Aldons J and Shalev, Anath
Diabetes, ISSN 0012-1797, 04/2008, Volume 57, Issue 4, pp. 938 - 944
OBJECTIVE - In diabetes, glucose toxicity affects different organ systems, including pancreatic islets where it leads to beta-cell apoptosis, but the... 
OXIDATIVE STRESS | RECOMBINANT CONGENIC STRAINS | PANCREATIC-ISLETS | GLUCOTOXICITY | GENETIC COMPOSITION | ENDOCRINOLOGY & METABOLISM | TXNIP | IDENTIFICATION | EXPRESSION | UP-REGULATED PROTEIN-1 | DIABETES SUSCEPTIBILITY | Islets of Langerhans - drug effects | Insulin - analysis | Apoptosis - drug effects | Islets of Langerhans - chemistry | Male | Carrier Proteins - drug effects | Thioredoxins - genetics | Islets of Langerhans - metabolism | Thioredoxins - drug effects | Glucose - toxicity | Insulin-Secreting Cells - cytology | Thioredoxins - physiology | Cell Culture Techniques | Insulin Secretion | Carrier Proteins - physiology | Insulin-Secreting Cells - physiology | DNA Nucleotidylexotransferase - metabolism | Glucose - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Carrier Proteins - genetics | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Mice | Apoptosis - physiology | Index Medicus | Abridged Index Medicus
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2. Full Text Toxicity and biodegradability of imidazolium ionic liquids
by Romero, A and Santos, A and Tojo, J and Rodríguez, A
Journal of Hazardous Materials, ISSN 0304-3894, 2008, Volume 151, Issue 1, pp. 268 - 273
Several bioassays have been carried out to analyze the toxicity and biodegradability of several imidazolium ionic liquids (ILs) in aqueous phase. The... 
Imidazolium | Microtox | Biodegradability | Ionic liquid | PYRIDINIUM | biodegradability | microtox | PHYSICAL-PROPERTIES | CATALYSIS | SALTS | ionic liquid | MIXTURES | ENVIRONMENTAL SCIENCES | ENGINEERING, CIVIL | SOLVENTS | ENGINEERING, ENVIRONMENTAL | ANTIMICROBIAL ACTIVITIES | imidazolium | CHLORIDES | ETHANOL | METHYL-SULFATE | Biodegradation, Environmental | Photobacterium - drug effects | Photobacterium - metabolism | Adenosine Triphosphate - analysis | Glucose - metabolism | Imidazoles - analysis | Imidazoles - toxicity | Glucose - analysis | Oxygen - metabolism | Imidazoles - metabolism | Index Medicus
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3. Full Text Human IAPP-induced Pancreatic β cell toxicity and its regulation by autophagy
by Shigihara, Nayumi and Fukunaka, Ayako and Hara, Akemi and Komiya, Koji and Honda, Akira and Uchida, Toyoyoshi and Abe, Hiroko and Toyofuku, Yukiko and Tamaki, Motoyuki and Ogihara, Takeshi and Miyatsuka, Takeshi and Hiddinga, Henry J and Sakagashira, Setsuya and Koike, Masato and Uchiyama, Yasuo and Yoshimori, Tamotsu and Eberhardt, Norman L and Fujitani, Yoshio and Watada, Hirotaka
Journal of Clinical Investigation, ISSN 0021-9738, 08/2014, Volume 124, Issue 8, pp. 3634 - 3644
Pancreatic islets in patients with type 2 diabetes mellitus (T2DM) are characterized by loss of beta cells and formation of amyloid deposits derived from islet... 
FIBRIL FORMATION | MEDICINE, RESEARCH & EXPERIMENTAL | ISLET-AMYLOID-POLYPEPTIDE | WILD-TYPE | INTRACELLULAR AMYLOIDOGENESIS | ALZHEIMERS-DISEASE | INSULIN-SECRETION | COS-1 CELLS | TYPE-2 DIABETES-MELLITUS | GLUCOSE-TOLERANCE | TRANSGENIC MICE | Insulin-Secreting Cells - physiology | Cell Line | Microtubule-Associated Proteins - genetics | Cell Survival | Humans | Mice, Transgenic | Autophagy - physiology | Islet Amyloid Polypeptide - genetics | Recombinant Proteins - genetics | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Diabetes Mellitus, Type 2 - physiopathology | Cell Cycle | Insulin Resistance - physiology | Recombinant Proteins - toxicity | Diabetes Mellitus, Type 2 - etiology | Mice | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Islet Amyloid Polypeptide - physiology | Microtubule-Associated Proteins - deficiency | Islet Amyloid Polypeptide - toxicity | Index Medicus | Abridged Index Medicus
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4. Full Text Safety assessment of [3S, 3′S]-astaxanthin – Subchronic toxicity study in rats
by Buesen, R and Schulte, S and Strauss, V and Treumann, S and Becker, M and Gröters, S and Carvalho, S and van Ravenzwaay, B
Food and Chemical Toxicology, ISSN 0278-6915, 07/2015, Volume 81, pp. 129 - 136
Astaxanthin, a naturally occurring xanthophyll, is commercially used as a coloring agent in salmon feed, but also marketed as a dietary supplement. The... 
Rat | Astaxanthin | Subchronic toxicity | MICROALGAE | FOOD SCIENCE & TECHNOLOGY | HUMAN HEALTH | CHEMISTRY | NUTRITION | TOXICOLOGY | Body Weight | Rats, Wistar | Cholesterol - blood | Rats | Male | Alanine Transaminase - blood | Dose-Response Relationship, Drug | Xanthophylls - toxicity | Bilirubin - blood | Organ Size - drug effects | Xanthophylls - blood | Animals | Aspartate Aminotransferases - blood | Triglycerides - blood | Creatinine - blood | Female | Blood Glucose - metabolism | Drug Evaluation, Preclinical | Dietary Supplements | Toxicity Tests, Subchronic - methods | Alkaline Phosphatase - blood | Serum Albumin - metabolism | Histochemistry | Coloring matter | Dietary supplements | Index Medicus
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5. Full Text Acute toxicity due to the confirmed consumption of synthetic cannabinoids: clinical and laboratory findings
by Hermanns‐Clausen, Maren and Kneisel, Stefan and Szabo, Bela and Auwärter, Volker
Addiction, ISSN 0965-2140, 03/2013, Volume 108, Issue 3, pp. 534 - 544
Aims Recently, several synthetic cannabinoids were identified in herbal mixtures consumed as recreational drugs alternative to cannabis products. The aim was... 
122 | 210 | aminoalkylindoles | spice | 081 | JWH | Acute toxicity | synthetic cannabinoids | Aminoalkylindoles | JWH-122 | JWH-210 | Synthetic cannabinoids | Spice | JWH-081 | DRUG | PSYCHIATRY | CANNABIMIMETIC INDOLES | SUBSTANCE ABUSE | IDENTIFICATION | INTOXICATION | CB1 | PSYCHOSIS | RECEPTOR-BINDING | BRAIN | Substance Abuse Detection - methods | Cannabinoids - blood | Humans | Male | Young Adult | Plant Preparations - poisoning | Adolescent | Cannabinoids - poisoning | Emergency Treatment - statistics & numerical data | Plant Preparations - blood | Adult | Female | Street Drugs - blood | Retrospective Studies | Street Drugs - poisoning | Hypertension | Glucose metabolism | Blood sugar | Emergency medicine | Phenols | Marijuana | Consumption data | Psychotropic drugs | Toxicity | Emergency medical care | Index Medicus
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6. Full Text Skeletal muscle TRIB3 mediates glucose toxicity in diabetes and high-fat diet-induced insulin resistance
by Zhang, Wei and Wu, Mengrui and Kim, Teayoun and Jariwala, Ravi H and Garvey, W. John and Luo, Nanlan and Kang, Minsung and Ma, Elizabeth and Tian, Ling and Steverson, Dennis and Yang, Qinglin and Fu, Yuchang and Garvey, W. Timothy
Diabetes, ISSN 0012-1797, 08/2016, Volume 65, Issue 8, pp. 2380 - 2391
In the current study, we used muscle-specific TRIB3 over-expressing (MOE) and knockout (MKO) mice to determine whether TRIB3 mediates glucose-induced insulin... 
INDUCED DESENSITIZATION | METABOLIC SYNDROME | TRIBBLES HOMOLOG | TRANSPORT SYSTEM | BIOCHEMICAL PATHWAYS | GLUT4 TRANSLOCATION | ENDOCRINOLOGY & METABOLISM | TRB3 | HEXOSAMINE BIOSYNTHESIS | GLUTAMINE-FRUCTOSE-6-PHOSPHATE AMIDOTRANSFERASE | TRANSGENIC MICE | Humans | Diet, High-Fat - adverse effects | Male | Muscle, Skeletal - metabolism | Promoter Regions, Genetic - genetics | Proto-Oncogene Proteins c-akt - genetics | Diabetes Mellitus, Experimental - blood | Insulin Resistance - physiology | Body Composition - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Glucose - toxicity | Diabetes Mellitus, Experimental - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Body Composition - physiology | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Calorimetry, Indirect | Signal Transduction - genetics | Cholesterol - metabolism | Mice, Knockout | Insulin - metabolism | Animals | Insulin Resistance - genetics | Glucose - metabolism | Signal Transduction - physiology | Mice | Analysis | Muscles | Insulin resistance | Diabetes | Research | Health aspects | Ketogenic diet | Musculoskeletal system | Phosphorylation | Glucose | Toxicity | Index Medicus | Abridged Index Medicus | Pathophysiology
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7. Full Text Deciphering Tacrolimus‐Induced Toxicity in Pancreatic β Cells
by Triñanes, J and Rodriguez‐Rodriguez, A. E and Brito‐Casillas, Y and Wagner, A and De Vries, A. P. J and Cuesto, G and Acebes, A and Salido, E and Torres, A and Porrini, E
American Journal of Transplantation, ISSN 1600-6135, 11/2017, Volume 17, Issue 11, pp. 2829 - 2840
β Cell transcription factors such as forkhead box protein O1 (FoxO1), v‐maf musculoaponeurotic fibrosarcoma oncogene homolog A (MafA), pancreatic and duodenal... 
molecular biology | animal models | diabetes: new onset/posttransplant | drug toxicity | clinical research/practice | kidney transplantation/nephrology | cellular biology | immunosuppressant | calcineurin inhibitor (CNI) | islets of Langerhans | basic (laboratory) research/science | SURGERY | ORGANS | IMMUNOGENICITY | HAND TRANSPLANTATION | PHASE-TRANSITION | CRYOPRESERVATION | COMPOSITE TISSUE ALLOTRANSPLANTATION | ALLOGRAFTS | FACE | CHILLING SENSITIVITY | OVARIAN-FUNCTION | TRANSPLANTATION | Diabetes Mellitus, Experimental - drug therapy | Cyclosporine - pharmacology | NFATC Transcription Factors - metabolism | Insulin Resistance | Rats | Obesity - physiopathology | Diabetes Mellitus, Type 2 - metabolism | Nerve Tissue Proteins - metabolism | Rats, Zucker | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Thinness - physiopathology | Insulin-Secreting Cells - drug effects | Calcineurin - pharmacology | Tacrolimus - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Immunosuppressive Agents - pharmacology | Index Medicus
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8. Full Text The loss of glucose-regulated protein 78 (GRP78) during normal aging or from siRNA knockdown augments human alpha-synuclein (α-syn) toxicity to rat nigral neurons
by Salganik, Maxim and Sergeyev, Valeriy G and Shinde, Vishal and Meyers, Craig A and Gorbatyuk, Marina S and Lin, Jonathan H and Zolotukhin, Sergey and Gorbatyuk, Oleg S
Neurobiology of Aging, ISSN 0197-4580, 2015, Volume 36, Issue 6, pp. 2213 - 2223
Abstract Age-related structural changes and gradual loss of key enzymes significantly affect the ability of the endoplasmic reticulum (ER) to facilitate proper... 
Neurology | Internal Medicine | Parkinson Disease | Recombinant Adeno-associated virus | Tyrosine hydroxylase | Dopamine | Substantia nigra pars compacta | Glucose regulated protein 78 | BIP | NEURODEGENERATION | ENDOPLASMIC-RETICULUM STRESS | DOPAMINERGIC-NEURONS | MODEL | NEUROSCIENCES | GERIATRICS & GERONTOLOGY | CHAPERONES | PURIFICATION | SEROTYPE-1 | EXPRESSION | RNA, Small Interfering - genetics | Molecular Chaperones | alpha-Synuclein - toxicity | Down-Regulation | Humans | Rats, Inbred F344 | Endoplasmic Reticulum - physiology | Homeostasis | Male | Parkinson Disease - genetics | Substantia Nigra - cytology | Protein Folding | Aging - pathology | Gene Knockdown Techniques | Heat-Shock Proteins - genetics | Animals | Aging - genetics | Dopaminergic Neurons - drug effects | Female | Heat-Shock Proteins - physiology | Aging - metabolism | Disease Models, Animal | Enzymes | Molecular genetics | Neurons | Glucose | Gene therapy | Health aspects | Dextrose | Index Medicus
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9. Full Text Fructose‐1,6‐bisphosphate aldolase (class II) is the primary site of nickel toxicity in Escherichia coli
by Macomber, Lee and Elsey, Scott P and Hausinger, Robert P
Molecular Microbiology, ISSN 0950-382X, 12/2011, Volume 82, Issue 5, pp. 1291 - 1300
Nickel is toxic to all forms of life, but the mechanisms of cell damage are unknown. Indeed, environmentally relevant nickel levels (8 µM) inhibit wild‐type... 
TRANSPORT-SYSTEM | HOMEOSTASIS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALCALIGENES-EUTROPHUS | PURIFICATION | FRUCTOSE-1,6-DIPHOSPHATE ALDOLASE | MICROBIOLOGY | SUPEROXIDE DISMUTASES | CROOKES STRAIN | IDENTIFICATION | COBALT | Fructose-Bisphosphate Aldolase - antagonists & inhibitors | Escherichia coli - enzymology | Enzyme Inhibitors - metabolism | Escherichia coli - drug effects | Carbohydrate Metabolism | Mutant Proteins - metabolism | Nickel - toxicity | Fructose-Bisphosphate Aldolase - isolation & purification | Enzyme Inhibitors - toxicity | Escherichia coli - metabolism | Fructose-Bisphosphate Aldolase - metabolism | Kinetics | Culture Media - chemistry | Nickel - metabolism | Mutant Proteins - antagonists & inhibitors | Glycerin | Lactates | Molecular genetics | Escherichia coli | Glycerol | Nickel | Glucose | Fructose | Dextrose | Mutation | E coli | Microbiology | Toxicity | Index Medicus | metalloprotein | metal substitution | allosteric regulation | glutathione | glycolysis
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10. Full Text The regulation of cellular apoptosis by the ROS-triggered PERK/EIF2α/chop pathway plays a vital role in bisphenol A-induced male reproductive toxicity
by Yin, Li and Dai, Yanlin and Cui, Zhihong and Jiang, Xiao and Liu, Wenbin and Liu, Jinyi and Han, Fei and Lin, Ao and Cao, Jia
Toxicology and Applied Pharmacology, ISSN 0041-008X, 01/2017, Volume 314, pp. 98 - 108
Bisphenol A (2,2-bis(4-hydroxyphenyl)propane, BPA) is ubiquitous in the environment, wildlife, and humans. Evidence from past studies suggests that BPA is... 
Male reproductive toxicity | Bisphenol A | PERK/EIF2α/chop pathway | Endoplasmic reticulum stress | CELLS | ER STRESS | MITOCHONDRIA | RATS | N-ACETYLCYSTEINE | ENDOPLASMIC-RETICULUM STRESS | UNFOLDED PROTEIN RESPONSE | CA2 | PHARMACOLOGY & PHARMACY | TOXICOLOGY | HEALTH | PERK/EIF2 alpha/chop pathway | EXPOSURE | Reactive Oxygen Species - metabolism | Endoplasmic Reticulum Stress - drug effects | Testis - cytology | Apoptosis - drug effects | Benzhydryl Compounds - toxicity | Phenols - toxicity | eIF-2 Kinase - metabolism | Male | Mitochondria - drug effects | Testis - drug effects | Animals | Eukaryotic Initiation Factor-2 - metabolism | Cell Proliferation - drug effects | Mice | Infertility, Male - chemically induced | Transcription Factor CHOP - metabolism | Index Medicus | TRANSCRIPTION FACTORS | APOPTOSIS | SPERMATOZOA | CELL PROLIFERATION | DEOXYURIDINE | TOXICITY | PROPANE | SPERMATOCYTES | 60 APPLIED LIFE SCIENCES | INOSITOL | WILD ANIMALS | ENDOPLASMIC RETICULUM | CYTOCHROMES | GLUCOSE | PHOSPHOTRANSFERASES | MICE
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11. Full Text Evaluation of toxicity and biodegradability of choline chloride based deep eutectic solvents
by Radošević, Kristina and Cvjetko Bubalo, Marina and Gaurina Srček, Višnje and Grgas, Dijana and Landeka Dragičević, Tibela and Radojčić Redovniković, Ivana
Ecotoxicology and Environmental Safety, ISSN 0147-6513, 02/2015, Volume 112, pp. 46 - 53
Deep eutectic solvents (DESs) have been dramatically expanding in popularity as a new generation of environmentally friendly solvents with possible... 
Choline chloride | In vitro toxicity | Phytotoxicity | Deep eutectic solvents | Biodegradability | IMIDAZOLIUM IONIC LIQUIDS | SEEDLINGS | CHLOROPLASTS | LENGTHS | MIXTURES | CHAIN | ENVIRONMENTAL SCIENCES | FISH | ASSAYS | TOXICOLOGY | CYTOTOXICITY | CELL-LINE | Oxalic Acid - metabolism | Animals | Glycerol - metabolism | Hydrogen Bonding | Glucose - metabolism | Choline - toxicity | Waste Water - microbiology | Solvents - toxicity | Triticum - drug effects | Glycerin | Glucose metabolism | Oxalic acid | Choline | Analysis | Green technology | Glycerol | Ecological footprint | Oxalates | Glucose | Dextrose | Index Medicus | Solvents | Biotechnology | Toxicity | Chlorides | Fish | In vitro testing
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12. Full Text Subchronic toxicity of rebaudioside A
by Curry, Leslie L and Roberts, Ashley
Food and Chemical Toxicology, ISSN 0278-6915, 2008, Volume 46, Issue 7, pp. S11 - S20
The safety of the stevia-derived sweetener, rebaudioside A (CAS No. 58543-16-1), was evaluated in two oral toxicity studies. In a 4-week study, Wistar rats... 
Subchronic | Rebaudioside A | Oral | Rebiana | Toxicity | toxicity | SPRAGUE-DAWLEY RATS | ZERO DOSE CONTROL | FOOD SCIENCE & TECHNOLOGY | STEVIA-REBAUDIANA | rebiana | INTESTINAL GLUCOSE-ABSORPTION | BODY-WEIGHT CHANGES | CRUDE EXTRACT | rebaudioside A | subchronic | ORAL STEVIOSIDE | NATURAL SWEETENER | HYPERTENSIVE RATS | oral | TOXICOLOGY | FOOD-CONSUMPTION | Bile Acids and Salts - blood | Rats, Wistar | Cholesterol - blood | Liver - metabolism | Diterpenes, Kaurane - toxicity | Rats | Sweetening Agents - toxicity | Bile Acids and Salts - metabolism | Male | Sex Characteristics | Energy Intake | Dose-Response Relationship, Drug | Eating - drug effects | Animals | Diet | Time Factors | Female | Weight Gain - drug effects | Diterpenes, Kaurane - administration & dosage | Sweetening Agents - administration & dosage | Index Medicus
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13.