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Cell, ISSN 0092-8674, 10/2016, Volume 167, Issue 2, pp. 457 - 470.e13
Activated macrophages undergo metabolic reprogramming, which drives their pro-inflammatory phenotype, but the mechanistic basis for this remains obscure. Here,... 
succinate | reverse electron transport | toll-like receptors | innate immunity | immunometabolism | macrophage | succinate dehydrogenase | RESPIRATORY-CHAIN | DENDRITIC CELL | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | ALPHA | IL-1-BETA | INTERLEUKIN-10 IL-10 | DIMETHYL FUMARATE | HYPOXIA | ELECTRON-TRANSPORT | CELL-ACTIVATION | CELL BIOLOGY | Mitochondria - enzymology | Reactive Oxygen Species - metabolism | Transcriptome | Malonates - pharmacology | Lipopolysaccharides - immunology | Oxidative Phosphorylation - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Membrane Potential, Mitochondrial | Oxidation-Reduction - drug effects | Interleukin-10 - metabolism | Plant Proteins - metabolism | Macrophages - immunology | Oxidoreductases - metabolism | Mice, Inbred C57BL | Inflammation - immunology | Mitochondria - drug effects | Citric Acid Cycle | Macrophage Activation | Macrophages - metabolism | Succinate Dehydrogenase - genetics | Animals | Sequence Analysis, RNA | Succinic Acid - metabolism | Glycolysis | Inflammation - genetics | Succinate Dehydrogenase - metabolism | Mice | Oxidases | RNA sequencing | Glucose metabolism | Medical colleges | RNA | Gastrointestinal diseases | Genes | Research institutes | Macrophages | Gene expression | Mitogens
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