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Nature, ISSN 0028-0836, 2014, Volume 514, Issue 7521, pp. 181 - 186
Journal Article
Nature, ISSN 0028-0836, 01/2012, Volume 481, Issue 7382, pp. 511 - 515
Exercise has beneficial effects on human health, including protection against metabolic disorders such as diabetes(1). However, the cellular mechanisms... 
DISRUPTION | BECLIN-1 | GENE | DIET | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | CAUSES INSULIN-RESISTANCE | MOUSE | SENSITIVITY | MICE | TUMORIGENESIS | Physical Exertion - genetics | Protein Binding - genetics | Physical Exertion - physiology | Male | Muscle, Skeletal - metabolism | Autophagy - physiology | Muscle, Skeletal - cytology | Physical Endurance - physiology | Physical Endurance - genetics | Autophagy - drug effects | Running - physiology | Glucose Intolerance - prevention & control | Phosphorylation - genetics | Leptin - blood | Muscle, Skeletal - drug effects | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Glucose Intolerance - chemically induced | Autophagy - genetics | Homeostasis - drug effects | Beclin-1 | Proto-Oncogene Proteins - metabolism | Dietary Fats - adverse effects | Glucose Tolerance Test | Cells, Cultured | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Physical Conditioning, Animal - physiology | Apoptosis Regulatory Proteins - metabolism | Gene Knock-In Techniques | Myocardium - cytology | Adiponectin - blood | Food Deprivation - physiology | Animals | Glucose - metabolism | Mice | Mutation | Proto-Oncogene Proteins c-bcl-2 | Prevention | Autophagy (Cytology) | Metabolic diseases | Influence | Exercise | Health aspects | Muscular system | Glucose | Kinases | Rodents
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2016, Volume 126, Issue 11, pp. 4372 - 4386
Obese, insulin-resistant states are characterized by a paradoxical pathogenic condition in which the liver appears to be selectively insulin resistant.... 
RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | TRANSCRIPTION FACTOR FOXO1 | FATTY LIVER-DISEASE | OB/OB MICE | ELEMENT-BINDING PROTEIN | HEPATIC STEATOSIS | ADIPOSE-TISSUE | PLASMA TRIGLYCERIDES | Glucose Intolerance - metabolism | Glucose-6-Phosphatase - genetics | Fatty Liver - pathology | Humans | Male | Glucose - biosynthesis | Glucose Intolerance - pathology | Fatty Liver - chemically induced | Glycolysis - drug effects | Glycolysis - genetics | Lipogenesis - genetics | Female | Glucose Intolerance - chemically induced | Insulin - genetics | Nuclear Proteins - genetics | Fatty Liver - genetics | Forkhead Box Protein O1 - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Fructose - toxicity | Insulin Resistance | Glucose - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Glucose-6-Phosphatase - metabolism | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Forkhead Box Protein O1 - genetics | Obesity | Analysis | Insulin resistance | Genetic aspects | Genetic transcription | Research | Risk factors | Protein binding | Glucose | Metabolites | Rodents | Liver
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e106159
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2007, Volume 282, Issue 50, pp. 36275 - 36282
Compelling evidence indicates that excess consumption of sugar-sweetened beverages plays an important role in the epidemic of obesity, a major risk factor for... 
DIABETES-MELLITUS | HIGH-FAT | OBESITY | PRESENILINS | PROTEIN | DIET | LEARNING-DEFICITS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | MICE | US CHILDREN | Beverages - adverse effects | Glucose Intolerance - metabolism | Amyloid Precursor Protein Secretases - genetics | Diabetes Mellitus, Type 2 - genetics | Humans | Sweetening Agents - toxicity | Glucose Intolerance - pathology | Diabetes Mellitus, Type 2 - metabolism | Apolipoproteins E - metabolism | Obesity - genetics | Amyloidosis - chemically induced | Alzheimer Disease - pathology | Brain - metabolism | Hypercholesterolemia - pathology | Sucrose - toxicity | Amyloid beta-Peptides - genetics | Amyloidosis - genetics | Amyloid beta-Peptides - metabolism | Water - administration & dosage | Glucose Intolerance - chemically induced | Alzheimer Disease - chemically induced | Disease Models, Animal | Glucose Intolerance - genetics | Amyloidosis - pathology | Risk Factors | Insulin Resistance | Mice, Transgenic | Obesity - metabolism | Obesity - pathology | Amyloid Precursor Protein Secretases - metabolism | Animals | Apolipoproteins E - genetics | Obesity - epidemiology | Alzheimer Disease - metabolism | Brain - pathology | Mice | Diabetes Mellitus, Type 2 - chemically induced | Diabetes Mellitus, Type 2 - pathology | Hypercholesterolemia - chemically induced | Hypercholesterolemia - genetics | Alzheimer Disease - genetics | Hypercholesterolemia - metabolism | Amyloidosis - metabolism
Journal Article
Diabetes Care, ISSN 0149-5992, 2011, Volume 34, Issue 2, pp. 412 - 417
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2013, Volume 273, Issue 3, pp. 551 - 560
Air pollutants have been associated with increased diabetes in humans. We hypothesized that ozone would impair glucose homeostasis by altering insulin... 
Epinephrine | Aging | Air pollution | Ozone | Serum biomarkers | Metabolic syndrome | LEPTIN | LONG-TERM EXPOSURE | ENDOPLASMIC-RETICULUM STRESS | PARAMETERS | INFLAMMATORY MARKERS | AIR-POLLUTION | OBESITY | INSULIN-RESISTANCE | FISCHER-344 | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PARTICULATE MATTER | Diabetes Mellitus - pathology | Osteopontin - blood | Metabolic Diseases - pathology | Phosphorylation | Age Factors | Lipoproteins, HDL - blood | Male | Glucose Intolerance - pathology | Insulin Receptor Substrate Proteins - metabolism | Insulin - blood | Adipose Tissue - metabolism | Rats, Inbred BN | Leptin - blood | Liver - drug effects | Glucose Intolerance - chemically induced | Insulin Receptor Substrate Proteins - genetics | Biomarkers - metabolism | Glucose Tolerance Test | Endoplasmic Reticulum Stress - drug effects | Liver - metabolism | Risk Factors | Insulin Resistance | Rats | Metabolic Diseases - chemically induced | Ozone - toxicity | Reverse Transcriptase Polymerase Chain Reaction | Lipoproteins, IDL - blood | Adiponectin - blood | alpha-Macroglobulins - metabolism | Animals | Triglycerides - blood | Diabetes Mellitus - chemically induced | Adipose Tissue - drug effects | Glucose metabolism | Glucose intolerance | Leptin | Diabetes | Glucose | Dextrose | Index Medicus | RATS | 60 APPLIED LIFE SCIENCES | ADRENALINE | AIR POLLUTION | STRESSES | INSULIN | HYPERGLYCEMIA | BIOLOGICAL MARKERS | GLUCOSE | LIVER | OZONE | ADIPOSE TISSUE
Journal Article
Journal Article