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PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 2, p. e54442
.... SGLT2 inhibitors block reabsorption of filtered glucose by inhibiting SGLT2, the primary glucose transporter in the proximal tubular cell (PTC... 
MATRIX | STIMULATION | MULTIDISCIPLINARY SCIENCES | DISEASE | HIGH GLUCOSE | GENE-EXPRESSION | PPAR-GAMMA AGONISTS | TRANSFORMING GROWTH FACTOR-BETA | Diabetes Mellitus - pathology | Epithelial Cells - metabolism | Gene Expression - drug effects | Diabetes Mellitus - genetics | Transcription Factor AP-1 - genetics | Epithelial Cells - drug effects | Humans | Diabetic Nephropathies - drug therapy | NF-kappa B - metabolism | Smad3 Protein - metabolism | Transcription Factor AP-1 - metabolism | Sodium-Glucose Transporter 1 - genetics | Smad3 Protein - genetics | Sodium-Glucose Transporter 1 - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | Transforming Growth Factor beta1 - pharmacology | Sodium-Glucose Transporter 2 - genetics | Collagen Type IV - metabolism | Sodium-Glucose Transporter 1 - antagonists & inhibitors | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Glucosides - pharmacology | Kidney Tubules, Proximal - pathology | Interleukin-6 - genetics | Sodium-Glucose Transporter 2 - metabolism | Diabetic Nephropathies - metabolism | Diabetes Mellitus - drug therapy | Diabetes Mellitus - metabolism | Toll-Like Receptor 4 - genetics | Diabetic Nephropathies - genetics | Epithelial Cells - pathology | Glucose - pharmacology | Toll-Like Receptor 4 - metabolism | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | NF-kappa B - genetics | Kidney Tubules, Proximal - metabolism | Collagen Type IV - genetics | Protein Binding | Benzhydryl Compounds - pharmacology | Kidney Tubules, Proximal - drug effects | Physiological aspects | Care and treatment | Genetic aspects | Research | Transforming growth factors | Diabetic nephropathies | Chromatin | Immunoprecipitation | Transforming growth factor-b | Interleukin | Clinical trials | Systematic review | Smad3 protein | Glucose | Kinases | Interleukin 6 | Proteins | Hyperglycemia | Rodents | Toll-like receptors | Collagen (type IV) | Hypoglycemic agents | Inhibition | Growth factors | Deoxyribonucleic acid--DNA | Immune system | Binding | Glucose transporter | Medical research | NF-κB protein | Diabetes mellitus | Markers | Activator protein 1 | Blocking | Reabsorption | Inflammation | Gene expression | Glucose transport | Medicine | High mobility group proteins | Hospitals | Inhibitors | Nephropathy | Sodium | Fibrosis | Kidney diseases | Diabetes | Transporter | Adenosine triphosphatase | Kidney transplantation | Deoxyribonucleic acid | DNA
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2010, Volume 285, Issue 45, pp. 35123 - 35132
.... In this report, we demonstrate that an orphan transporter hNPT4 (human sodium phosphate transporter 4; SLC17A3... 
HYPERURICEMIA | ORGANIC ANION TRANSPORT | GOUT | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | EXCRETION | APICAL MEMBRANE | URIC-ACID | HOMINOID EVOLUTION | FAMILY | EXCHANGER | Glucose Transport Proteins, Facilitative - metabolism | Organic Anion Transporters, Sodium-Independent - genetics | Organic Anion Transport Protein 1 - metabolism | Diuretics - pharmacokinetics | Sodium-Phosphate Cotransporter Proteins, Type I - genetics | Humans | Hyperuricemia - metabolism | Mutation, Missense | Organic Anion Transporters - metabolism | Urate Oxidase - metabolism | Organic Anion Transporters - genetics | Urate Oxidase - genetics | Furosemide - adverse effects | Organic Anion Transporters, Sodium-Independent - metabolism | Kidney Tubules, Proximal - secretion | Ion Transport - drug effects | Glucose Transport Proteins, Facilitative - genetics | Bumetanide - adverse effects | Genetic Predisposition to Disease | Uric Acid - metabolism | Organic Cation Transport Proteins - metabolism | Diuretics - pharmacology | Furosemide - pharmacokinetics | Liver - metabolism | Xenopus laevis | Organic Anion Transport Protein 1 - genetics | Bumetanide - pharmacokinetics | Bumetanide - pharmacology | Hyperuricemia - genetics | Gout - metabolism | Animals | Models, Biological | Gout - genetics | Furosemide - pharmacology | Organic Cation Transport Proteins - genetics | Sodium-Phosphate Cotransporter Proteins, Type I - metabolism | Diuretics - adverse effects | Hyperuricemia - chemically induced | Ion Transport - genetics | Index Medicus | Transport Drugs | Membrane Biology | Gout | Epithelial Cell | Uric Acid | Diuretics | Kidney
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 484, Issue 7394, pp. 333 - 338
The prevalence of obesity and type 2 diabetes is increasing worldwide and threatens to shorten lifespan... 
DIABETES-MELLITUS | FATTY-ACID SYNTHESIS | GENE | INSULIN-RESISTANCE | LIPOGENESIS | MULTIDISCIPLINARY SCIENCES | CARBOHYDRATE-RESPONSE ELEMENT | LIVER | BINDING-PROTEIN CHREBP | UCSC GENOME BROWSER | TRANSGENIC MICE | Transcription Factors - chemistry | Diabetes Mellitus - genetics | Humans | Male | RNA, Messenger - metabolism | Adipose Tissue - metabolism | Protein Isoforms - chemistry | Nuclear Proteins - deficiency | Lipogenesis | Body Mass Index | Glucose Transporter Type 4 - genetics | Adipose Tissue - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - chemistry | Genotype | Glucose - pharmacology | Nuclear Proteins - chemistry | Mice, Knockout | Insulin - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Insulin Resistance - genetics | Glucose - metabolism | Mice | Blood Glucose - metabolism | Homeostasis - genetics | Cohort Studies | Diabetes Mellitus - blood | Body Weight | Glucose Transporter Type 4 - metabolism | Transcription Factors - deficiency | Adipose Tissue - cytology | Molecular Sequence Data | Obesity - genetics | Promoter Regions, Genetic - genetics | Protein Isoforms - metabolism | Adiposity | Female | Nuclear Proteins - genetics | Insulin - pharmacology | Glucose Intolerance - genetics | Cross-Sectional Studies | Gene Expression Regulation - genetics | RNA, Messenger - genetics | Cells, Cultured | Diabetes Mellitus - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Obesity - metabolism | Transcription Factors - metabolism | Animals | Glucose Transporter Type 4 - biosynthesis | Adipocytes - metabolism | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Protein Isoforms - genetics
Journal Article
Journal Article
Journal Article
Journal of Psychiatric Research, ISSN 0022-3956, 2016, Volume 76, pp. 66 - 73
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 12/2016, Volume 60, Issue 12, pp. 7407 - 7414
The glucose transporter PfHT is essential to the survival of the malaria parasite Plasmodium falciparum and has been shown to be a druggable target with high potential for pharmacological intervention... 
LIFE-CYCLE | TARGET | HEXOSE TRANSPORTER | PARASITES | MECHANISM | PLASMODIUM-FALCIPARUM | VALIDATION | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | ANTIMALARIAL | CANCER | EXPRESSION | Glucose Transporter Type 2 - genetics | Small Molecule Libraries - pharmacology | Glucose Transporter Type 4 - metabolism | Species Specificity | Protozoan Proteins - antagonists & inhibitors | Humans | Glucose Transporter Type 1 - metabolism | Tritium | Structure-Activity Relationship | Glucose Transporter Type 3 - metabolism | Plasmodium falciparum - drug effects | Protozoan Proteins - genetics | Glucose Transporter Type 2 - metabolism | Glucose Transporter Type 3 - genetics | Protozoan Proteins - metabolism | HEK293 Cells | Plasmodium falciparum - metabolism | Monosaccharide Transport Proteins - metabolism | Monosaccharide Transport Proteins - antagonists & inhibitors | Monosaccharide Transport Proteins - genetics | Gene Expression | Glucose Transporter Type 4 - genetics | Cells, Cultured | Glucose - antagonists & inhibitors | Antimalarials - pharmacology | Erythrocytes - drug effects | Small Molecule Libraries - chemistry | High-Throughput Screening Assays | Glucose Transporter Type 1 - genetics | Antimalarials - chemistry | Erythrocytes - metabolism | Glucose - metabolism | Fluorescence Resonance Energy Transfer - methods | Plasmodium falciparum - growth & development | Erythrocytes - parasitology
Journal Article
Blood, ISSN 1528-0020, 2012, Volume 119, Issue 20, pp. 4686 - 4697
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 10/2017, Volume 313, Issue 4, pp. C421 - C429
...) occurs through a facilitated glucose transport, through glucose transporter 1 (GLUT1), although other isoforms have been described at the BBB... 
Blood-brain barrier | Stem cells | Glucose | blood-brain barrier | stem cells | LOCALIZATION | glucose | PHYSIOLOGY | GLUT1 DEFICIENCY | ALZHEIMERS-DISEASE | IN-VITRO MODELS | CELL BIOLOGY | METABOLISM | NEURONS | RAT-BRAIN | BARRIER-SPECIFIC PROPERTIES | DEFICIENCY SYNDROME | HYPOTHALAMUS | Glucose Transport Proteins, Facilitative - metabolism | Blood-Brain Barrier - cytology | Glucose Transporter Type 4 - metabolism | Humans | Microvessels - metabolism | Glucose Transporter Type 1 - metabolism | Glucose Transporter Type 3 - metabolism | Sodium-Glucose Transporter 1 - genetics | Glucose Transporter Type 3 - genetics | Sodium-Glucose Transporter 1 - metabolism | Glucose Transport Proteins, Facilitative - genetics | Diffusion | Induced Pluripotent Stem Cells - metabolism | Microvessels - cytology | Cell Line | Glucose Transporter Type 4 - genetics | Induced Pluripotent Stem Cells - drug effects | Endothelial Cells - metabolism | Microvessels - drug effects | Electric Impedance | Blood-Brain Barrier - drug effects | Symporters - metabolism | Blood-Brain Barrier - metabolism | Phenotype | Glucose Transporter Type 1 - genetics | Symporters - genetics | Glucose - metabolism | Glucose Transport Proteins, Facilitative - antagonists & inhibitors | Endothelial Cells - drug effects | Physiological aspects | Glucose metabolism | Endothelium
Journal Article
Journal Article