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PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 2, p. e54442
.... SGLT2 inhibitors block reabsorption of filtered glucose by inhibiting SGLT2, the primary glucose transporter in the proximal tubular cell (PTC... 
MATRIX | STIMULATION | MULTIDISCIPLINARY SCIENCES | DISEASE | HIGH GLUCOSE | GENE-EXPRESSION | PPAR-GAMMA AGONISTS | TRANSFORMING GROWTH FACTOR-BETA | Diabetes Mellitus - pathology | Epithelial Cells - metabolism | Gene Expression - drug effects | Diabetes Mellitus - genetics | Transcription Factor AP-1 - genetics | Epithelial Cells - drug effects | Humans | Diabetic Nephropathies - drug therapy | NF-kappa B - metabolism | Smad3 Protein - metabolism | Transcription Factor AP-1 - metabolism | Sodium-Glucose Transporter 1 - genetics | Smad3 Protein - genetics | Sodium-Glucose Transporter 1 - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | Transforming Growth Factor beta1 - pharmacology | Sodium-Glucose Transporter 2 - genetics | Collagen Type IV - metabolism | Sodium-Glucose Transporter 1 - antagonists & inhibitors | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Glucosides - pharmacology | Kidney Tubules, Proximal - pathology | Interleukin-6 - genetics | Sodium-Glucose Transporter 2 - metabolism | Diabetic Nephropathies - metabolism | Diabetes Mellitus - drug therapy | Diabetes Mellitus - metabolism | Toll-Like Receptor 4 - genetics | Diabetic Nephropathies - genetics | Epithelial Cells - pathology | Glucose - pharmacology | Toll-Like Receptor 4 - metabolism | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | NF-kappa B - genetics | Kidney Tubules, Proximal - metabolism | Collagen Type IV - genetics | Protein Binding | Benzhydryl Compounds - pharmacology | Kidney Tubules, Proximal - drug effects | Physiological aspects | Care and treatment | Genetic aspects | Research | Transforming growth factors | Diabetic nephropathies | Chromatin | Immunoprecipitation | Transforming growth factor-b | Interleukin | Clinical trials | Systematic review | Smad3 protein | Glucose | Kinases | Interleukin 6 | Proteins | Hyperglycemia | Rodents | Toll-like receptors | Collagen (type IV) | Hypoglycemic agents | Inhibition | Growth factors | Deoxyribonucleic acid--DNA | Immune system | Binding | Glucose transporter | Medical research | NF-κB protein | Diabetes mellitus | Markers | Activator protein 1 | Blocking | Reabsorption | Inflammation | Gene expression | Glucose transport | Medicine | High mobility group proteins | Hospitals | Inhibitors | Nephropathy | Sodium | Fibrosis | Kidney diseases | Diabetes | Transporter | Adenosine triphosphatase | Kidney transplantation | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2014, Volume 508, Issue 7495, pp. 258 - 262
In obesity and type 2 diabetes, Glut4 glucose transporter expression is decreased selectively in adipocytes(1... 
METABOLISM | MULTIDISCIPLINARY SCIENCES | MOUSE | LIVER | RESISTANCE | MICE | CDNA CLONING | EXPRESSION | SIRT1 | CANCER | ADIPOSE-TISSUE | Sirtuin 1 - metabolism | Acetyltransferases - metabolism | Adipocytes - secretion | Ornithine Decarboxylase - metabolism | Liver - enzymology | Glucose Transporter Type 4 - metabolism | Adipose Tissue, White - metabolism | Male | Diabetes Mellitus, Type 2 - metabolism | Nicotinamide N-Methyltransferase - metabolism | Glucose Intolerance | Glucose Transporter Type 4 - deficiency | Obesity - genetics | Thinness - metabolism | Gene Knockdown Techniques | Adipose Tissue - metabolism | Nicotinamide N-Methyltransferase - deficiency | Obesity - etiology | NAD - metabolism | Spermine - analogs & derivatives | Nicotinamide N-Methyltransferase - genetics | Thinness - enzymology | Glucose Transporter Type 4 - genetics | Niacinamide - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - enzymology | Insulin Resistance | Oxidoreductases Acting on CH-NH Group Donors - metabolism | Fatty Liver | Animals | Diet | Energy Metabolism | Adipocytes - metabolism | Obesity - prevention & control | Spermine - metabolism | Adipose Tissue, White - enzymology | Adipose Tissue - enzymology | Mice | Obesity - enzymology | S-Adenosylmethionine - metabolism | Adipose tissues | Type 2 diabetes | Obesity | Care and treatment | Analysis | Transferases | Physiological aspects | Genetic aspects | Research | Gene expression | Enzymes | Body fat | Adipocytes | Polyamines | Variance analysis | Proteins | Weight control | Metabolites | Rodents | Insulin resistance | Diabetes | Mass spectrometry | Food
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2006, Volume 354, Issue 24, pp. 2552 - 2563
In this study, serum levels of retinol-binding protein 4, a molecule secreted by adipocytes, correlated with the magnitude of insulin resistance in subjects with obesity, impaired glucose tolerance... 
METABOLIC DEFECTS | MEDICINE, GENERAL & INTERNAL | PLASMA-CONCENTRATIONS | GLUCOSE | RETINOL-BINDING-PROTEIN | HYPERINSULINEMIA | ADIPOSITY | DYSLIPIDEMIA | CORONARY-HEART-DISEASE | FAT DISTRIBUTION | NIDDM | Cross-Sectional Studies | Glucose Transporter Type 4 - metabolism | Retinol-Binding Proteins, Plasma | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Male | Diabetes Mellitus, Type 2 - metabolism | Obesity - blood | Thinness - metabolism | Obesity - metabolism | Metabolic Syndrome - blood | Diabetes Mellitus, Type 2 - blood | Retinol-Binding Proteins - metabolism | Insulin Resistance - physiology | Adipocytes - metabolism | Glucose Metabolism Disorders - metabolism | Thinness - blood | Glucose Metabolism Disorders - blood | Adult | Female | Exercise - physiology | Blood Glucose - metabolism | Insulin resistance | Diabetes | Glucose | Insulin | Rodents | Obesity/blood/metabolism | Blood Glucose/metabolism | Adipocytes/metabolism | Thinness/blood/metabolism | Exercise/physiology | MEDICIN OCH HÄLSOVETENSKAP | Diabetes Mellitus | Glucose Transporter Type 4/metabolism | Type 2/blood/genetics/metabolism | Glucose Metabolism Disorders/blood/metabolism | Insulin Resistance/physiology | Metabolic Syndrome X/blood | Retinol-Binding Proteins/metabolism | MEDICAL AND HEALTH SCIENCES
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 10/2017, Volume 313, Issue 4, pp. C421 - C429
...) occurs through a facilitated glucose transport, through glucose transporter 1 (GLUT1), although other isoforms have been described at the BBB... 
Blood-brain barrier | Stem cells | Glucose | blood-brain barrier | stem cells | LOCALIZATION | glucose | PHYSIOLOGY | GLUT1 DEFICIENCY | ALZHEIMERS-DISEASE | IN-VITRO MODELS | CELL BIOLOGY | METABOLISM | NEURONS | RAT-BRAIN | BARRIER-SPECIFIC PROPERTIES | DEFICIENCY SYNDROME | HYPOTHALAMUS | Glucose Transport Proteins, Facilitative - metabolism | Blood-Brain Barrier - cytology | Glucose Transporter Type 4 - metabolism | Humans | Microvessels - metabolism | Glucose Transporter Type 1 - metabolism | Glucose Transporter Type 3 - metabolism | Sodium-Glucose Transporter 1 - genetics | Glucose Transporter Type 3 - genetics | Sodium-Glucose Transporter 1 - metabolism | Glucose Transport Proteins, Facilitative - genetics | Diffusion | Induced Pluripotent Stem Cells - metabolism | Microvessels - cytology | Cell Line | Glucose Transporter Type 4 - genetics | Induced Pluripotent Stem Cells - drug effects | Endothelial Cells - metabolism | Microvessels - drug effects | Electric Impedance | Blood-Brain Barrier - drug effects | Symporters - metabolism | Blood-Brain Barrier - metabolism | Phenotype | Glucose Transporter Type 1 - genetics | Symporters - genetics | Glucose - metabolism | Glucose Transport Proteins, Facilitative - antagonists & inhibitors | Endothelial Cells - drug effects | Physiological aspects | Glucose metabolism | Endothelium
Journal Article
EMBO reports, ISSN 1469-221X, 12/2005, Volume 6, Issue 12, pp. 1137 - 1142
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2015, Volume 10, Issue 12, p. e0146033
Journal Article