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PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49701
Paeoniflorin (PF), the principal component of Paeoniae Radix prescribed in traditional Chinese medicine, has been reported to exhibit many pharmacological... 
STROKE | ACTIVATION | PATHWAY | NEUROVASCULAR UNIT | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | INTRACEREBRAL HEMORRHAGE | NITRIC-OXIDE | MECHANISMS | EXPRESSION | NEUROPROTECTION | Microglia - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Brain - metabolism | Inflammation - metabolism | Bridged-Ring Compounds - pharmacology | Monoterpenes | Neurons - metabolism | Disease Models, Animal | NF-kappa B - antagonists & inhibitors | bcl-2-Associated X Protein - metabolism | Rats | Brain - drug effects | Signal Transduction - drug effects | Brain Ischemia - drug therapy | Benzoates - pharmacology | Brain - pathology | Bridged-Ring Compounds - administration & dosage | Astrocytes - metabolism | Nitric Oxide Synthase Type II - metabolism | Cerebral Infarction - drug therapy | Brain Ischemia - metabolism | Cytochromes c - genetics | Hippocampus - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Interleukin-1beta - blood | Proto-Oncogene Proteins c-bcl-2 - metabolism | Lipoxygenase - metabolism | Cerebral Infarction - metabolism | Inflammation - drug therapy | Neurons - drug effects | bcl-2-Associated X Protein - genetics | Cerebral Infarction - pathology | Astrocytes - drug effects | Glucosides - pharmacology | Microglia - drug effects | Cytochromes c - metabolism | Benzoates - administration & dosage | Gene Expression Regulation - drug effects | Animals | Glucosides - administration & dosage | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Cyclooxygenase 2 - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Mitogen-Activated Protein Kinases - metabolism | Occlusion | Neuroprotection | Brain | Inflammatory response | Activation | Kinases | Carotid arteries | Signal transduction | Chinese medicine | Ischemia | Neurodegeneration | Cerebral blood flow | Rodents | Tumor necrosis factor-TNF | Inhibition | NF-κB protein | Stroke | Cytokines | Astrocytes | Traditional Chinese medicine | Extracellular signal-regulated kinase | MAP kinase | JNK protein | Pharmacology | Inflammation | Tumor necrosis factor-α | IL-1β | Nitric-oxide synthase | Microglia | Signaling | Brain research | Liquid oxygen | Hypoxia | Brain damage | Cyclooxygenase-2 | Laboratory animals | Apoptosis | Veins & arteries
Journal Article
Journal Article
The Plant Journal, ISSN 0960-7412, 10/2011, Volume 68, Issue 2, pp. 273 - 286
Summary Cyanogenic glucosides are amino acid‐derived defence compounds found in a large number of vascular plants. Their hydrolysis by specific β‐glucosidases... 
Sorghum bicolor | cytochrome P450 | Lotus japonicus | Manihot esculenta | gene clustering | cyanogenic glucosides | CONVERGENT EVOLUTION | SECONDARY METABOLISM | DIVERSIFICATION | CYTOCHROMES P450 | SORGHUM-BICOLOR | PLANT SCIENCES | SUBSTRATE-SPECIFICITY | HYDROXYNITRILE GLUCOSIDES | PLANTS | ARABIDOPSIS | EXPRESSION | Sorghum - enzymology | Multigene Family | DNA, Complementary - genetics | Cytochrome P-450 Enzyme System - metabolism | Manihot - enzymology | Phylogeny | Glucosyltransferases - metabolism | Glucosides - genetics | Gene Expression Regulation, Plant | Glucosides - chemistry | Molecular Structure | Plant Proteins - metabolism | Sorghum - genetics | Plant Leaves - enzymology | Glycosides - chemistry | Glucosyltransferases - genetics | Manihot - metabolism | Nitriles - metabolism | Genome, Plant - genetics | Tobacco - metabolism | Loteae - metabolism | Manihot - genetics | Sorghum - metabolism | RNA, Plant - genetics | Glucosides - metabolism | Biological Evolution | Gene Expression Regulation, Enzymologic | Hydrogen Cyanide - metabolism | Plant Proteins - genetics | Plant Leaves - genetics | Plant Leaves - metabolism | Glycosides - metabolism | Tobacco - genetics | Nitriles - chemistry | Cytochrome P-450 Enzyme System - genetics | Loteae - enzymology | Glucosides - biosynthesis | Mutation | Loteae - genetics | Hydrolysis | Legumes | Enzymes | Beans | Genes | Genetic research | Physiological aspects | Nitriles | Mimosaceae | Plant biology | Biosynthesis | Genomics | Plant resistance
Journal Article
Neuropharmacology, ISSN 0028-3908, 04/2017, Volume 116, pp. 412 - 420
Paeoniflorin (PF) is a major bioactive ingredient in roots that has low toxicity and has been shown to have neuroprotective effects. Our in vitro experiments... 
Paeoniflorin | Neuroprotective effect | Parkinson's disease | Phosphatidylinositol 3-kinase (PI3K)/Akt | OXIDATIVE STRESS | TYROSINE-HYDROXYLASE | SIGNAL PATHWAY | C57BL/6 MICE | KINASE | DEATH | DOPAMINERGIC NEUROTOXICITY | NEUROSCIENCES | ADENOSINE A RECEPTOR | PHARMACOLOGY & PHARMACY | GROWTH-FACTOR | PC12 CELLS | Dopamine Plasma Membrane Transport Proteins - metabolism | Motor Activity - physiology | Tyrosine 3-Monooxygenase - metabolism | Caspase 9 - metabolism | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Caspase 3 - metabolism | Motor Activity - drug effects | Male | Phosphatidylinositol 3-Kinases - metabolism | Substantia Nigra - metabolism | Corpus Striatum - metabolism | Monoterpenes - pharmacology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Neuroprotective Agents - pharmacology | Dopaminergic Neurons - metabolism | Dopaminergic Neurons - drug effects | MPTP Poisoning - pathology | bcl-Associated Death Protein - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Corpus Striatum - pathology | Cell Survival - physiology | Dopamine - metabolism | Cell Survival - drug effects | Monoamine Oxidase Inhibitors - pharmacology | Glucosides - pharmacology | Mice, Inbred C57BL | MPTP Poisoning - drug therapy | Substantia Nigra - drug effects | Animals | MPTP Poisoning - metabolism | Corpus Striatum - drug effects | Monoamine Oxidase - metabolism | Tyrosine | Analysis | Monoamine oxidase
Journal Article
The Plant Cell, ISSN 1040-4651, 4/2011, Volume 23, Issue 4, pp. 1536 - 1555
Journal Article
Nutrition, Metabolism and Cardiovascular Diseases, ISSN 0939-4753, 2013, Volume 23, Issue 11, pp. 1086 - 1092
Abstract Background and aims Resveratrol, the most investigated dietary compound in studies aimed at linking wine consumption to human health, is an extremely... 
Cardiovascular | Phytoestrogen | Red wine | Resveratrol metabolism | Resveratrol | ESTROGEN-RECEPTORS | CARDIAC & CARDIOVASCULAR SYSTEMS | ASSAY | ALPHA | BETA | BREAST-CANCER CELLS | NUTRITION & DIETETICS | IN-VIVO | ENDOCRINOLOGY & METABOLISM | HEALTH | Nuclear Receptor Coactivator 2 - agonists | Humans | Neoplasm Proteins - antagonists & inhibitors | Stilbenes - chemistry | Stilbenes - pharmacology | Estrogen Receptor beta - metabolism | Adenocarcinoma - metabolism | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - genetics | Glucuronides - metabolism | Glucuronides - pharmacology | MCF-7 Cells | Sulfates - chemistry | Estrogen Antagonists - chemistry | Glucosides - chemistry | Antineoplastic Agents, Phytogenic - metabolism | Clone Cells | Neoplasm Proteins - genetics | Binding Sites | Estrogen Receptor beta - antagonists & inhibitors | Peptide Fragments - genetics | Phytoestrogens - pharmacology | Glucuronides - chemistry | Recombinant Proteins - chemistry | Estrogen Receptor alpha - antagonists & inhibitors | Sulfates - pharmacology | Adenocarcinoma - drug therapy | Breast Neoplasms - drug therapy | Glucosides - metabolism | Estrogen Receptor alpha - genetics | Peptide Fragments - agonists | Peptide Fragments - antagonists & inhibitors | Cell Line, Tumor | Phytoestrogens - metabolism | Stereoisomerism | Phytoestrogens - chemistry | Estrogen Antagonists - pharmacology | Stilbenes - metabolism | Nuclear Receptor Coactivator 2 - metabolism | Neoplasm Proteins - metabolism | Breast Neoplasms - metabolism | Estrogen Receptor beta - agonists | Estrogen Receptor alpha - metabolism | Female | Recombinant Proteins - metabolism | Glucosides - pharmacology | Peptide Fragments - metabolism | Sulfates - metabolism | Antineoplastic Agents, Phytogenic - chemistry | Estrogen Antagonists - metabolism | Nuclear Receptor Coactivator 2 - antagonists & inhibitors | Neoplasm Proteins - agonists | Antineoplastic Agents, Phytogenic - pharmacology | Nuclear Receptor Coactivator 2 - genetics | Metabolites | Beverages
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2012, Volume 52, Issue 2, pp. 314 - 327
Enhanced oxidative stress due to high glucose contributes to pathological changes in diabetes-related liver complications. Reducing oxidative stress may... 
Anthocyanin | CREB | Free radicals | GSH | PKA | OXIDATIVE STRESS | METABOLIC SYNDROME | RISK-FACTORS | FATTY LIVER-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN ENDOTHELIAL-CELLS | RESPONSE ELEMENT | INSULIN-RESISTANCE | CELLULAR GLUTATHIONE | ENDOCRINOLOGY & METABOLISM | GLUTAMATE-CYSTEINE LIGASE | SUBUNIT GENE-EXPRESSION | Up-Regulation | Liver - pathology | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Male | Hepatocytes - metabolism | fas Receptor - metabolism | Hyperglycemia - drug therapy | Glutathione - biosynthesis | MAP Kinase Kinase 4 - metabolism | fas Receptor - genetics | Anthocyanins - therapeutic use | Glucosides - therapeutic use | Glutamate-Cysteine Ligase - metabolism | Cyclic AMP - metabolism | Hepatocytes - drug effects | Cytoprotection | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Survival - drug effects | Anthocyanins - pharmacology | Gene Expression | Glucosides - pharmacology | Signal Transduction | Liver - metabolism | Fatty Liver - prevention & control | Thiobarbituric Acid Reactive Substances - metabolism | Antioxidants - pharmacology | Enzyme Activation - drug effects | Hep G2 Cells | Hyperglycemia - metabolism | Antioxidants - therapeutic use | Animals | Cyclic AMP Response Element-Binding Protein - metabolism | Protein Binding | Glutamate-Cysteine Ligase - genetics | Mice, Obese | Mice | Apoptosis | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 05/2016, Volume 65, Issue 5, pp. 1190 - 1196
Pharmacologically induced glycosuria elicits adaptive responses in glucose homeostasis and hormone release. In type 2 diabetes (T2D), along with decrements in... 
INSULIN | KETOACIDOSIS | SGLT2 | ENDOCRINOLOGY & METABOLISM | Glucose Intolerance - metabolism | Glycosuria - chemically induced | Carbohydrate Metabolism - drug effects | Lipolysis - drug effects | Benzhydryl Compounds - administration & dosage | Humans | Glucagon-Like Peptide 1 - blood | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Glucagon - blood | Glucose Intolerance - blood | Glucose Intolerance - drug therapy | Benzhydryl Compounds - adverse effects | Hypoglycemic Agents - administration & dosage | Time Factors | Glucosides - therapeutic use | C-Reactive Protein - analysis | Benzhydryl Compounds - therapeutic use | Insulin Secretion | Glucosides - adverse effects | Hypoglycemic Agents - therapeutic use | 3-Hydroxybutyric Acid - blood | Sodium-Glucose Transporter 2 - metabolism | Glucose Intolerance - urine | 3-Hydroxybutyric Acid - agonists | Renal Elimination - drug effects | Sodium-Glucose Transporter 2 Inhibitors | Membrane Transport Modulators - administration & dosage | Diabetes Mellitus, Type 2 - urine | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Algorithms | Glucosides - administration & dosage | 3-Hydroxybutyric Acid - metabolism | Lipid Metabolism - drug effects | Membrane Transport Modulators - therapeutic use | Glucagon - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Membrane Transport Modulators - adverse effects | Hypoglycemic Agents - adverse effects | Energy Metabolism - drug effects | Type 2 diabetes | Care and treatment | Glucagon | Dosage and administration | Triglycerides | Research | Insulin
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 09/2012, Volume 342, Issue 3, pp. 654 - 664
Nodakenin, a coumarin isolated from the roots of Angelicae gigas, has been reported to possess neuroprotective, antiaggregatory, antibacterial, and... 
ACTIVATION | CYTOKINES | CYCLOOXYGENASE-2 | RAW264.7 CELLS | NITRIC-OXIDE SYNTHASE | KINASE | SEPTIC SHOCK | PHARMACOLOGY & PHARMACY | MEDIATORS | SEPSIS | ANGELICA-GIGAS | Transcription, Genetic - drug effects | Interleukin-6 - antagonists & inhibitors | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | TNF Receptor-Associated Factor 6 - antagonists & inhibitors | Interleukin-1beta - genetics | Promoter Regions, Genetic - drug effects | Mitogen-Activated Protein Kinase Kinases - metabolism | Inflammation - metabolism | Phosphorylation - genetics | Drug Interactions | Ubiquitination - drug effects | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Phosphorylation - drug effects | TNF Receptor-Associated Factor 6 - genetics | Interleukin-6 - metabolism | Interleukin-1beta - antagonists & inhibitors | NF-KappaB Inhibitor alpha | NF-kappa B - antagonists & inhibitors | Interleukin-6 - genetics | Anti-Inflammatory Agents - pharmacology | Dinoprostone - genetics | MAP Kinase Kinase Kinases - genetics | MAP Kinase Kinase Kinases - metabolism | Signal Transduction - genetics | Down-Regulation - genetics | Coumarins - pharmacology | Dinoprostone - metabolism | Macrophages - metabolism | Signal Transduction - drug effects | Nitric Oxide - genetics | Lipopolysaccharides - pharmacology | Mice | Transcription, Genetic - genetics | Dinoprostone - antagonists & inhibitors | Nitric Oxide Synthase Type II - metabolism | Tumor Necrosis Factor-alpha - metabolism | Mitogen-Activated Protein Kinase Kinases - genetics | Shock, Septic - prevention & control | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Shock, Septic - metabolism | I-kappa B Kinase - metabolism | I-kappa B Kinase - antagonists & inhibitors | Inflammation - drug therapy | Nitric Oxide Synthase Type II - antagonists & inhibitors | I-kappa B Proteins - antagonists & inhibitors | Shock, Septic - drug therapy | Glucosides - pharmacology | Nitric Oxide - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | I-kappa B Kinase - genetics | Down-Regulation - drug effects | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Cyclooxygenase 2 - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Inflammation - genetics | Macrophages - drug effects | Nitric Oxide - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Ubiquitination - genetics
Journal Article
The Plant Journal, ISSN 0960-7412, 11/2012, Volume 72, Issue 3, pp. 411 - 422
Summary Glucosinolates (GSLs) are nitrogen‐ and sulfur‐containing metabolites that contribute to human health and plant defense. The biological activities of... 
glucosinolate | benzoate | sinapoylation | Arabidopsis | benzoylation | serine carboxypeptidase‐like acyltransferases | serine carboxypeptidase-like acyltransferases | SECONDARY METABOLISM | CARBOXYPEPTIDASE-LIKE PROTEIN | BENZOIC-ACID BIOSYNTHESIS | CELL-CULTURES | PLANT SCIENCES | SALICYLIC-ACID | GENE | THALIANA | SERINE CARBOXYPEPTIDASE | PETUNIA FLOWERS | ATTED-II | Arabidopsis - enzymology | Coenzyme A Ligases - genetics | Coumaric Acids - chemistry | Benzoates - chemistry | Benzoates - metabolism | Substrate Specificity | Glucosinolates - metabolism | Benzoylcholine - metabolism | Acyltransferases - metabolism | Glucosinolates - chemistry | Acyltransferases - genetics | Genetic Complementation Test | Propanols - metabolism | Coenzyme A Ligases - metabolism | Arabidopsis Proteins - metabolism | Propanols - chemistry | Cinnamates - metabolism | Glucosides - chemistry | Cinnamates - chemistry | Acyl Coenzyme A - metabolism | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Glucosinolates - analysis | Seeds - metabolism | Seeds - genetics | Coumaric Acids - metabolism | Biosynthetic Pathways | Glucosides - metabolism | Carboxypeptidases | Esterification | Arabidopsis - metabolism | Arabidopsis - genetics | Phenotype | Benzoates - pharmacology | Kinetics | Mutation | Benzoylcholine - chemistry | Arabidopsis thaliana | Amino acids | Anopheles | Metabolites | Developmental biology | Transferases | Plant biology
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3178 - 3189
The purpose of the present study was to investigate the effect of salidroside (Sal) on myocardial injury in lipopolysaccharide (LPS)‐induced endotoxemic in... 
H9C2 | salidroside | myocardial injury | PI3K/Akt/mTOR | LPS | ROS | MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | PHOSPHATIDYLINOSITOL 3-KINASE | RATS | ACUTE LUNG INJURY | ISCHEMIA REPERFUSION INJURY | NLRP3 INFLAMMASOME | CELL BIOLOGY | MITOCHONDRIAL ROS | SIGNALING PATHWAY | TANSHINONE IIA SULFONATE | KAPPA-B PATHWAY | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | TOR Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - genetics | Endotoxemia - chemically induced | Endotoxemia - pathology | Interleukin-1beta - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Lipopolysaccharides | Myocardial Reperfusion Injury - pathology | Dexamethasone - pharmacology | TOR Serine-Threonine Kinases - genetics | Interleukin-1beta - metabolism | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-6 - metabolism | Myocardial Reperfusion Injury - genetics | Phenols - pharmacology | Glutathione Peroxidase - metabolism | Interleukin-6 - genetics | Signal Transduction | Catalase - genetics | Endotoxemia - drug therapy | Rats | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Glutathione Peroxidase - genetics | Myocytes, Cardiac - pathology | Reactive Oxygen Species - antagonists & inhibitors | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Hemodynamics - drug effects | Nitric Oxide Synthase Type II - metabolism | Tumor Necrosis Factor-alpha - metabolism | Endotoxemia - genetics | Glutathione - metabolism | Myocardial Reperfusion Injury - chemically induced | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | TOR Serine-Threonine Kinases - antagonists & inhibitors | Myocardial Reperfusion Injury - drug therapy | Superoxide Dismutase - metabolism | Cell Line | Glucosides - pharmacology | Gene Expression Regulation | Myocardium - pathology | Catalase - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | Nitric Oxide Synthase Type II - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Mitogens | Cysteine | PI3K | mTOR | Akt | Original
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 07/2015, Volume 172, Issue 13, pp. 3284 - 3301
Journal Article