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Neuron, ISSN 0896-6273, 2010, Volume 68, Issue 1, pp. 113 - 126
Endocannabinoids and their receptor CB1 play key roles in brain function. Astrocytes express CB1Rs that are activated by endocannabinoids released by neurons.... 
ENDOGENOUS CANNABINOIDS | ACTIVATION | CA1 PYRAMIDAL CELLS | EXCITATORY SYNAPSES | NEURONS | LONG-TERM POTENTIATION | GLIAL-CELLS | NEUROSCIENCES | MODULATION | HIPPOCAMPAL SYNAPSES | PLASTICITY | Cannabinoid Receptor Modulators - metabolism | Cannabinoid Receptor Modulators - pharmacology | Glycine - analogs & derivatives | Synaptic Transmission - physiology | Synaptic Transmission - genetics | Calcium - metabolism | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Resorcinols - pharmacology | Drug Interactions | Photolysis | Piperidines - pharmacology | Pyramidal Cells - physiology | Egtazic Acid - analogs & derivatives | Synaptic Transmission - drug effects | Pyramidal Cells - drug effects | Electric Stimulation - methods | Endocannabinoids | Excitatory Postsynaptic Potentials - genetics | Pyrazoles - pharmacology | Animals, Newborn | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Biophysics | Chelating Agents - pharmacology | Hippocampus - cytology | Mice, Knockout | Astrocytes - physiology | Animals | Egtazic Acid - pharmacology | Glycine - pharmacology | Cannabinoid Receptor Modulators - antagonists & inhibitors | Benzoates - pharmacology | Thapsigargin - pharmacology | Mice | Pyridines - pharmacology | Receptor, Cannabinoid, CB1 - deficiency | In Vitro Techniques | Glutamate | Neurons | Studies | Retina | Transmitters | Index Medicus | Potentiation | Brain | Neurotransmitter release | Astrocytes | Glutamic acid receptors | Cannabinoid CB1 receptors | synaptic depression | Calcium signalling | Pyramidal cells | Synaptic transmission | Endogenous stimuli | Receptor mechanisms | Glutamic acid receptors (metabotropic) | Hippocampus | Neurotransmission
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 10/2012, Volume 32, Issue 43, pp. 15148 - 15157
Several behavioral effects of nicotine are mediated by changes in serotonin (5-HT) release in brain areas that receive serotonergic afferents from the dorsal... 
Electric Stimulation | Rats, Wistar | Dihydro-beta-Erythroidine - pharmacology | Male | Nicotinic Antagonists - pharmacology | Raphe Nuclei - cytology | Excitatory Postsynaptic Potentials - drug effects | Nicotine - pharmacology | Excitatory Postsynaptic Potentials - physiology | Muscarinic Antagonists - pharmacology | Egtazic Acid - analogs & derivatives | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Indoles - pharmacology | Tetrodotoxin - pharmacology | Cadmium Chloride - pharmacology | Cholinergic Agonists - pharmacology | Ryanodine - pharmacology | Animals, Newborn | Serotonergic Neurons - physiology | Presynaptic Terminals - drug effects | Acetylcholine - pharmacology | Enzyme Inhibitors - pharmacology | Glutamic Acid - physiology | Rats | Atropine - pharmacology | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Presynaptic Terminals - physiology | Aconitine - analogs & derivatives | Bicuculline - pharmacology | Chelating Agents - pharmacology | Aconitine - pharmacology | GABA Antagonists - pharmacology | Physostigmine - pharmacology | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Receptors, Nicotinic - physiology | Nicotine - analogs & derivatives | Serotonin - metabolism | In Vitro Techniques | Serotonergic Neurons - cytology | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2012, Volume 15, Issue 3, pp. 477 - 486
Efforts to study the development and function of the human cerebral cortex in health and disease have been limited by the availability of model systems.... 
SUBTYPE | HUMAN ES | EVOLUTION | NEOCORTICAL NEURONOGENESIS | NEURONS | NEUROGENESIS | SUBVENTRICULAR ZONE | SPECIFICATION | DIFFERENTIATION | MODEL | NEUROSCIENCES | Disks Large Homolog 4 Protein | Embryonic Stem Cells - cytology | Humans | Synaptic Potentials - drug effects | Ki-67 Antigen - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Cerebral Cortex - cytology | Piperidines - pharmacology | Time Factors | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Fetus | Membrane Proteins - metabolism | Tetrodotoxin - pharmacology | Cell Differentiation - physiology | Potassium Channel Blockers - pharmacology | Cell Line | Synapses - drug effects | Synaptic Potentials - physiology | Pluripotent Stem Cells - cytology | Glutamic Acid - pharmacology | Synapses - physiology | Pluripotent Stem Cells - physiology | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Sodium Channel Blockers - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Embryonic Stem Cells - physiology | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Patch-Clamp Techniques | Cerebral Cortex - embryology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Neurogenesis - physiology | Pluripotent Stem Cells - drug effects | RNA, Messenger | Retinoids - pharmacology | Physiological aspects | Cerebral cortex | Research | Synapses | Stem cells | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2011, Volume 118, Issue 5, pp. 826 - 840
Inflammation contributes to neurodegeneration in post-ischemic brain, diabetes, and Alzheimer's disease. Participants in this inflammatory response include... 
gap junctions | Alzheimer’s disease | amyloid β‐peptide | cytokines | diabetes mellitus | pannexin | stroke | connexin | amyloid β-peptide | Alzheimer's disease | BRAIN-INJURY | GAP JUNCTIONAL COMMUNICATION | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMYLOID PLAQUES | ASTROCYTES | NEUROTOXICITY | NEUROSCIENCES | BETA | IN-VIVO | CHANNELS | RECEPTORS | amyloid beta-peptide | Tumor Necrosis Factor-alpha - metabolism | Oxygen - pharmacology | Cell Hypoxia - physiology | Amyloid beta-Peptides - pharmacology | Microtubule-Associated Proteins - pharmacology | Culture Media, Conditioned - pharmacology | Lanthanum - pharmacology | Peptide Fragments - pharmacology | Cerebral Cortex - cytology | Adenosine Triphosphate - pharmacology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Neuropeptides - pharmacology | Nerve Tissue Proteins - pharmacology | Neurons - physiology | Astrocytes - chemistry | Female | Cell Death - drug effects | Connexins - pharmacology | Neurons - drug effects | Animals, Newborn | Astrocytes - drug effects | Cell Hypoxia - drug effects | Connexin 43 - metabolism | Glutamic Acid - pharmacology | Cells, Cultured | Fluoresceins | Coculture Techniques - methods | Connexin 43 - deficiency | Connexins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Biotinylation - methods | Glutamic Acid - metabolism | Mice | Astrocytes - metabolism | Methyl aspartate | Neurosciences | Peptides | Neurons | Diabetes | Glucose | Glutamate | Dextrose | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2006, Volume 26, Issue 11, pp. 2852 - 2861
Glutamate receptor (GluR) ion channels mediate fast synaptic transmission in the mammalian CNS. Numerous crystallographic studies, the majority on the... 
Glutamate receptor | Kainate | Antagonist | Structure | Crystallography | Gating | ACTIVATION | NMDA RECEPTORS | MECHANISM | MODEL | kainate | antagonist | NEUROSCIENCES | structure | GLUTAMATE-RECEPTOR | crystallography | AMPA RECEPTORS | glutamate receptor | gating | DYNAMICS | PARTIAL AGONIST ACTION | ION CHANNELS | SUBUNIT | Excitatory Amino Acid Antagonists - metabolism | Receptors, Kainic Acid - chemistry | Protein Conformation - drug effects | Thymine - analogs & derivatives | Receptors, AMPA - chemistry | Alanine - chemistry | Crystallography, X-Ray | Receptors, Kainic Acid - antagonists & inhibitors | Oocytes | Quinoxalines - pharmacology | Alanine - analogs & derivatives | Recombinant Fusion Proteins - antagonists & inhibitors | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Excitatory Amino Acid Antagonists - chemistry | Thymine - pharmacology | Binding Sites | Alanine - pharmacology | Thymine - metabolism | Thymine - chemistry | Glutamic Acid - pharmacology | Xenopus laevis | Models, Molecular | Rats | Alanine - metabolism | Excitatory Amino Acid Antagonists - pharmacology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - metabolism | Recombinant Fusion Proteins - chemistry | Patch-Clamp Techniques | Animals | Hydrogen Bonding | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - chemistry | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Protein Binding | Glutamic Acid - metabolism | Glutamic Acid - chemistry | Receptors, AMPA - antagonists & inhibitors | Index Medicus | BIOLOGY | GENES | CRYSTAL STRUCTURE | MATERIALS SCIENCE | CHAINS | DIMERS | CONFORMATIONAL CHANGES
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2010, Volume 30, Issue 2, pp. 545 - 555
Marijuana is a widely used drug that impairs memory through interaction between its psychoactive constituent, Delta-9-tetrahydrocannabinol (Delta(9)-THC), and... 
CA1 Region, Hippocampal - cytology | Receptor, Adenosine A1 - physiology | Neurons - cytology | Xanthines - pharmacology | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Receptor, Cannabinoid, CB1 - physiology | Excitatory Postsynaptic Potentials - physiology | Quinoxalines - pharmacology | Neural Inhibition - physiology | Receptor, Cannabinoid, CB1 - agonists | Piperidines - pharmacology | Caffeine - pharmacology | Dronabinol - pharmacology | Electric Stimulation - methods | Pyrazoles - pharmacology | Presynaptic Terminals - drug effects | Mice, Inbred C57BL | Morpholines - pharmacology | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Biophysics | Calcium Channel Blockers - pharmacology | GABA Antagonists - pharmacology | Mice, Knockout | Naphthalenes - pharmacology | Methoxyhydroxyphenylglycol - analogs & derivatives | Methoxyhydroxyphenylglycol - pharmacology | Animals | Analysis of Variance | Phosphinic Acids - pharmacology | Benzoxazines - pharmacology | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - pharmacology | Adenosine - metabolism | Picrotoxin - pharmacology | Presynaptic Terminals - metabolism | Glutamic Acid - metabolism | Mice | Receptor, Cannabinoid, CB1 - deficiency | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | In Vitro Techniques | Propanolamines - pharmacology | Neural Inhibition - drug effects | Index Medicus | brain slice | caffeine | memory | hippocampus | depolarization-induced suppression of excitation (DSE) | marijuana | learning
Journal Article
Neuron, ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 81 - 93
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2003, Volume 23, Issue 9, pp. 3588 - 3596
Journal Article
Neuron, ISSN 0896-6273, 2008, Volume 57, Issue 6, pp. 883 - 893
Cannabinoid receptors play key roles in brain function, and cannabinoid effects in brain physiology and drug-related behavior are thought to be mediated by... 
MOLNEURO | PURKINJE-CELLS | ENDOGENOUS CANNABINOIDS | CA2 | INTRACELLULAR CALCIUM | GLUTAMATE | SYNAPTIC-TRANSMISSION | CANNABINOID CB1 RECEPTOR | GLIAL-CELLS | NEUROSCIENCES | IN-SITU | HIPPOCAMPAL SYNAPSES | Cannabinoid Receptor Modulators - pharmacology | Calcium - metabolism | Cell Communication - physiology | Glial Fibrillary Acidic Protein - metabolism | Type C Phospholipases - pharmacology | Astrocytes - radiation effects | Cannabinoid Receptor Modulators - physiology | Photolysis | Piperidines - pharmacology | Egtazic Acid - analogs & derivatives | Neurons - physiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Neurons - drug effects | Electric Stimulation - methods | Endocannabinoids | Pyrazoles - pharmacology | Animals, Newborn | Astrocytes - drug effects | Mice, Inbred C57BL | Valine - analogs & derivatives | Excitatory Amino Acid Antagonists - pharmacology | Hippocampus - cytology | Mice, Knockout | Receptor, Cannabinoid, CB1 - metabolism | Astrocytes - physiology | Patch-Clamp Techniques | Animals | Egtazic Acid - pharmacology | Receptor, Cannabinoid, CB1 - genetics | Glutamic Acid - metabolism | Mice | Receptor, Cannabinoid, CB1 - deficiency | Valine - pharmacology | Neurons - radiation effects | Glutamate | Methyl aspartate | Neurons | Proteins | Brain | Transmitters | Rodents | Experiments | Communication | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2004, Volume 24, Issue 36, pp. 7821 - 7828
It is widely believed that long-term depression (LTD) and its counterpart, long-term potentiation (LTP), involve mechanisms that are crucial for learning and... 
NR2B | Depotentiation | LTP | NMDARs | LTD | NR2A | HIPPOCAMPAL SYNAPTIC PLASTICITY | RAT | D-ASPARTATE RECEPTORS | PERIRHINAL CORTEX | depotentiation | SUBUNIT COMPOSITION | NEUROSCIENCES | FUNCTIONAL-PROPERTIES | IN-VITRO | AMPA RECEPTORS | GLUTAMATE TRANSPORTERS | EXPRESSION | Receptors, Metabotropic Glutamate - drug effects | Protein Subunits | 2-Amino-5-phosphonovalerate - pharmacology | Neurotransmitter Uptake Inhibitors - pharmacology | Dicarboxylic Acids - pharmacology | Long-Term Synaptic Depression - drug effects | Excitatory Postsynaptic Potentials - drug effects | N-Methylaspartate - pharmacology | Pyrrolidines - pharmacology | Quinoxalines - pharmacology | Neurons - ultrastructure | Long-Term Potentiation - drug effects | Piperidines - pharmacology | Neurons - physiology | Long-Term Potentiation - physiology | Cerebral Cortex - drug effects | Neurons - drug effects | Phenols - pharmacology | Long-Term Synaptic Depression - physiology | Receptors, N-Methyl-D-Aspartate - drug effects | Glutamic Acid - pharmacology | Rats | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Receptors, N-Methyl-D-Aspartate - physiology | Animals | Picrotoxin - pharmacology | Cerebral Cortex - physiology | Dizocilpine Maleate - pharmacology | Aspartic Acid - pharmacology | Index Medicus
Journal Article
Glia, ISSN 0894-1491, 11/2011, Volume 59, Issue 11, pp. 1600 - 1611
We have previously demonstrated that human astrocytes are GABAergic cells. Throughout the adult human brain, they express the GABA synthesizing enzyme GAD 67,... 
serine | calcium chelation | glycine | GABA transporters | GABA receptors | glutamate | glutamate receptors | Glutamate receptors | D-serine | Glutamate | Glycine | Calcium chelation | CA2+ INFLUX | CELLS | TRANSPORTER | RAT | SYNAPTIC-TRANSMISSION | RECEPTOR | IDENTIFICATION | NEUROSCIENCES | INDICATORS | BRAIN | Receptors, Glutamate - metabolism | Calcium - metabolism | gamma-Aminobutyric Acid - metabolism | Humans | Calcium - analysis | RNA, Messenger - biosynthesis | Vigabatrin - pharmacology | Culture Media | Egtazic Acid - analogs & derivatives | Astrocytes - chemistry | Real-Time Polymerase Chain Reaction | Cell Survival - drug effects | Glutamic Acid - pharmacology | RNA, Messenger - genetics | Cells, Cultured | Excitatory Amino Acid Agonists - pharmacology | Chelating Agents - pharmacology | DNA Primers | Vesicular Glutamate Transport Proteins - metabolism | Vesicular Glutamate Transport Proteins - genetics | Blotting, Western | Vesicular Glutamate Transport Proteins - antagonists & inhibitors | Egtazic Acid - pharmacology | Cyclohexanecarboxylic Acids - pharmacology | Astrocytes - metabolism | Cell culture | Enzymes | Glutamic acid | Calcium (intracellular) | 4-Aminobutyrate transaminase | Calcium | Astrocytes | Glutamic acid receptors (ionotropic) | Data processing | N-Methyl-D-aspartic acid receptors | Glutamate decarboxylase | Glutamic acid receptors | mRNA | gamma -Aminobutyric acid transporter | gamma -Aminobutyric acid B receptors | Neurotransmitters | gamma -Aminobutyric acid A receptors | Chelating agents | Index Medicus
Journal Article