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Cell Metabolism, ISSN 1550-4131, 06/2012, Volume 15, Issue 6, pp. 827 - 837
Journal Article
Gastroenterology, ISSN 0016-5085, 04/2018, Volume 154, Issue 5, pp. 1465 - 1479.e13
Cirrhosis results from accumulation of myofibroblasts derived from quiescent hepatic stellate cells (Q-HSCs); it regresses when myofibroblastic HSCs are... 
Metabolic Reprogramming | Liver Diseases | Hippo Pathway | Fibrogenesis | FIBROSIS | TRANSCRIPTIONAL ACTIVITY | PULMONARY-HYPERTENSION | FATTY LIVER-DISEASE | MECHANISMS | CANCER | METABOLISM | GLUCOSE | GROWTH | Fibro-genesis | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Liver - pathology | Cell Proliferation | Mitochondria, Liver - metabolism | Humans | Glutamine - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Smoothened Receptor - metabolism | Phosphoproteins - metabolism | Smoothened Receptor - genetics | Case-Control Studies | Cellular Reprogramming | Myofibroblasts - metabolism | Transfection | Hedgehog Proteins - genetics | RNA Interference | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis, Experimental - pathology | Cell Transdifferentiation | Ketoglutaric Acids - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | Mitochondria, Liver - pathology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Rats | Glutaminase - metabolism | Liver Cirrhosis, Experimental - genetics | Phosphoproteins - genetics | Mice, Knockout | Phenotype | Animals | Energy Metabolism | Adaptor Proteins, Signal Transducing - genetics | Liver Cirrhosis - pathology | Adaptor Proteins, Signal Transducing - metabolism | Liver Cirrhosis, Experimental - metabolism | Glucose metabolism | Metabolites | RNA | Analysis | Liver | Physiological aspects | Genetic research | Verteporfin | Development and progression | Mice | Liver cirrhosis | Glutamine | Index Medicus | Abridged Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 01/2012, Volume 15, Issue 1, pp. 110 - 121
Journal Article
Oncogene, ISSN 0950-9232, 01/2014, Volume 33, Issue 5, pp. 556 - 566
Retinoblastoma (Rb) protein is a tumor suppressor that is dysregulated in a majority of human cancers. Rb functions to inhibit cell cycle progression in part... 
Metabolism | Cancer | Glutamine | Retinoblastoma Protein | Tumor Suppressor | retinoblastoma protein | APOPTOSIS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | G CONTROL | tumor suppressor | CELL-PROLIFERATION | FACTOR-I | glutamine | CELL BIOLOGY | GLYCOLYSIS | INHIBITION | ONCOLOGY | GENETICS & HEREDITY | metabolism | cancer | GROWTH-FACTOR | ENERGY-METABOLISM | Cell Proliferation | Reactive Oxygen Species - metabolism | Glutamate-Cysteine Ligase - biosynthesis | Minor Histocompatibility Antigens | Glutamine - metabolism | Retinoblastoma-Like Protein p107 - genetics | Adenosine Triphosphate - biosynthesis | Oxygen - metabolism | Glutathione - biosynthesis | RNA Interference | Retinoblastoma-Like Protein p130 - metabolism | Fibroblasts | Biological Transport - genetics | Amino Acid Transport System ASC - genetics | Ketoglutaric Acids - metabolism | Glutaminase - biosynthesis | Promoter Regions, Genetic | Retinoblastoma-Like Protein p107 - metabolism | Retinoblastoma Protein - metabolism | Cells, Cultured | Amino Acid Transport System ASC - metabolism | Glutaminase - metabolism | Mice, Knockout | Animals | E2F3 Transcription Factor - biosynthesis | Cell Cycle | Retinoblastoma Protein - genetics | E2F3 Transcription Factor - metabolism | Mice | RNA, Small Interfering | Retinoblastoma-Like Protein p130 - genetics | Amino Acid Transport System ASC - biosynthesis | Glutaminase - genetics | Oncology, Experimental | Physiological aspects | Genetic research | Tumor suppressor genes | Genetic aspects | Research | Studies | Oncology | Tumors | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 12/2011, Volume 60, Issue 12, pp. 3121 - 3131
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1591 - 1602
The mechanistic target of rapamycin (mTOR) is hyperactivated in many types of cancer, rendering it a compelling drug-target; however, the impact of mTOR... 
RAPAMYCIN | MEDICINE, RESEARCH & EXPERIMENTAL | PATHWAY | MUTATION | KINASE INHIBITORS | PROLIFERATION | CELL-GROWTH | PTEN-DEFICIENT | CANCER | CONTRIBUTES | Gas Chromatography-Mass Spectrometry | Humans | Glutamine - metabolism | Male | Neoplasm Proteins - antagonists & inhibitors | Purines - administration & dosage | Indoles - administration & dosage | Benzophenanthridines - therapeutic use | Glioblastoma - metabolism | Purines - therapeutic use | Neoplasm Proteins - genetics | Glutaminase - biosynthesis | Magnetic Resonance Spectroscopy | Benzophenanthridines - administration & dosage | Brain Neoplasms - drug therapy | Benzophenanthridines - pharmacology | Drug Synergism | Energy Metabolism | Signal Transduction - drug effects | Mice, Nude | Glioblastoma - pathology | Cell Line, Tumor | Indoles - therapeutic use | Mice | Mice, Inbred BALB C | Glioblastoma - drug therapy | Glutaminase - physiology | Neoplasm Proteins - physiology | Brain Neoplasms - pathology | Molecular Targeted Therapy | Brain Neoplasms - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Indoles - pharmacology | Ketoglutaric Acids - metabolism | Neoplasm Proteins - biosynthesis | Purines - pharmacology | RNA, Small Interfering - pharmacology | Temporal Lobe - metabolism | Glutaminase - antagonists & inhibitors | Citric Acid Cycle | Xenograft Model Antitumor Assays | Rotarod Performance Test | Animals | Metabolome - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Glycolysis | Signal Transduction - physiology | Aged | Glutamic Acid - metabolism | Protein Kinase Inhibitors - pharmacology | Drug Resistance, Neoplasm - physiology | Glutaminase - genetics | Pharmacology, Experimental | Oncology, Experimental | Glutamine metabolism | Research | Drug therapy | Drug resistance | Health aspects | Glioblastoma multiforme | Cancer | Medical research | Enzymes | Brain cancer | Neurosurgery | Glucose | Kinases | Metabolism | Carbon | Cancer therapies | Medicine | Cell growth | Brain research | Laboratory animals | Tumors | University graduates | Index Medicus | Abridged Index Medicus | Oncology
Journal Article
Science Signaling, ISSN 1945-0877, 12/2017, Volume 10, Issue 508, p. eaan4667
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 10, pp. 3535 - 3545
Altered glycolytic flux in cancer cells (the "Warburg effect") causes their proliferation to rely upon elevated glutamine metabolism ("glutamine addiction").... 
BREAST-CANCER | TARGETED THERAPY | ACTIVATION | KIDNEY-TYPE GLUTAMINASE | MECHANISM | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | RENAL MITOCHONDRIAL GLUTAMINASE | PROLIFERATION | ANTITUMOR-ACTIVITY | Drug Resistance, Multiple | Humans | Molecular Conformation | Sulfides - chemistry | Glutamine - metabolism | Crystallography, X-Ray | Drug Resistance, Neoplasm | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Triple Negative Breast Neoplasms - drug therapy | Antineoplastic Agents - metabolism | Thiadiazoles - chemistry | Enzyme Inhibitors - chemistry | Triple Negative Breast Neoplasms - pathology | Antineoplastic Agents - pharmacology | Benzeneacetamides - chemistry | Glutamine - chemistry | Sulfides - metabolism | Thiadiazoles - metabolism | Neoplasm Proteins - genetics | Benzeneacetamides - pharmacology | Sulfides - pharmacology | Thiadiazoles - pharmacology | Binding, Competitive | Recombinant Proteins - metabolism | Enzyme Inhibitors - metabolism | Allosteric Site - drug effects | Enzyme Inhibitors - pharmacology | Models, Molecular | Recombinant Proteins - chemistry | Neoplasm Proteins - chemistry | Glutaminase - metabolism | Antineoplastic Agents - chemistry | Glutaminase - antagonists & inhibitors | Glutaminase - chemistry | Glutamine - antagonists & inhibitors | Hydrogen Bonding | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Benzeneacetamides - metabolism | Cell Proliferation - drug effects | Mutation | Glutaminase - genetics | Amino Acid Substitution | Index Medicus | anticancer drug | crystallography | BPTES | glutaminase | cancer | Metabolism | CB-839
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2010, Volume 185, Issue 2, pp. 1037 - 1044
Journal Article