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1. Full Text Behavioral Phenotypes of Disc1 Missense Mutations in Mice
by Clapcote, Steven J and Lipina, Tatiana V and Millar, J. Kirsty and Mackie, Shaun and Christie, Sheila and Ogawa, Fumiaki and Lerch, Jason P and Trimble, Keith and Uchiyama, Masashi and Sakuraba, Yoshiyuki and Kaneda, Hideki and Shiroishi, Toshihiko and Houslay, Miles D and Henkelman, R. Mark and Sled, John G and Gondo, Yoichi and Porteous, David J and Roder, John C
Neuron, ISSN 0896-6273, 2007, Volume 54, Issue 3, pp. 387 - 402
To support the role of in human psychiatric disorders, we identified and analyzed two independently derived ENU-induced mutations in Exon 2 of mouse . Mice... 
SYSNEURO | HUMDISEASE | MOLNEURO | GATING DEFICITS | SCHIZOPHRENIC-PATIENTS | NEURITE OUTGROWTH | PREPULSE INHIBITION | PHOSPHODIESTERASE ENZYME | STRAIN DIFFERENCES | CHRONIC MILD STRESS | LATENT INHIBITION | BIPOLAR DISORDER | NEUROSCIENCES | CEREBRAL-CORTEX | Protein Binding - genetics | Brain - anatomy & histology | Cyclic Nucleotide Phosphodiesterases, Type 4 | Humans | Male | DNA Mutational Analysis - methods | Mice, Mutant Strains - physiology | Mutation, Missense - genetics | Alanine - genetics | Threonine - genetics | Behavior, Animal - drug effects | Female | Leucine - genetics | Neural Inhibition - genetics | 3',5'-Cyclic-AMP Phosphodiesterases - metabolism | Mice, Inbred C57BL | Behavior, Animal - physiology | Nerve Tissue Proteins - genetics | Subcellular Fractions - metabolism | Phenotype | Animals | Mice, Mutant Strains - anatomy & histology | Glutamine - genetics | Mice | Reflex, Acoustic - genetics | Genetic aspects | Proteins | Studies | Medical research | Signal transduction | Brain research | Mental disorders | Mutagenesis | Rodents | Memory | Mutation | Behavior
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2. Full Text Dental Caries and Enamelin Haplotype
by Chaussain, C and Bouazza, N and Gasse, B and Laffont, A.G and Opsahl Vital, S and Davit-Béal, T and Moulis, E and Chabadel, O and Hennequin, M and Courson, F and Droz, D and Vaysse, F and Laboux, O and Tassery, H and Carel, J.C and Alcais, A and Treluyer, J.M and Beldjord, C and Sire, J.Y
Journal of Dental Research, ISSN 0022-0345, 4/2014, Volume 93, Issue 4, pp. 360 - 365
In the literature, the enamelin gene ENAM has been repeatedly designated as a possible candidate for caries susceptibility. Here, we checked whether ENAM... 
polymorphism(s) | candidate gene analysis | enamel | mineralization | genetics | caries susceptibility | SUSCEPTIBILITY | PREVENTION | TWINS REARED APART | DENTISTRY, ORAL SURGERY & MEDICINE | GENES | DISEASE | EXPERIENCE | TRAITS | DMF Index | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Introns - genetics | Humans | Dental Caries Susceptibility - genetics | Linkage Disequilibrium - genetics | Exons - genetics | Male | Isoleucine - genetics | Mutation, Missense - genetics | Dental Caries - genetics | Proteins - genetics | Young Adult | Haplotypes - genetics | Extracellular Matrix Proteins | Arginine - genetics | Threonine - genetics | Glutamine - genetics | Polymorphism, Single Nucleotide - genetics | Amino Acid Substitution - genetics | Female | Genetic Variation - genetics | Child | Life Sciences | Biodiversity | Populations and Evolution
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3. Full Text MYC, metabolism, cell growth, and tumorigenesis
by Dang, Chi V
Cold Spring Harbor Perspectives in Medicine, ISSN 1943-0264, 08/2013, Volume 3, Issue 8, pp. a014217 - a014217
The MYC proto-oncogene is frequently activated in human cancers through a variety of mechanisms. Its deregulated expression, unconstrained by inactivation of... 
Neoplasms - metabolism | Humans | Carcinogenesis - genetics | Gene Expression Regulation, Neoplastic | Glutamine - metabolism | Glucose - genetics | Signal Transduction - genetics | Genes, p53 - genetics | Transcription Factors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Mitochondria - genetics | Neoplasms - genetics | Ribosomes - genetics | Cell Transformation, Neoplastic - genetics | Genes, myc - genetics | Lipid Metabolism - genetics | Proto-Oncogene Proteins c-myc - genetics | B-CELLS | RNA-POLYMERASE-I | TARGET GENES | TUMOR PROGRESSION | MITOCHONDRIAL BIOGENESIS | C-MYC | CANCER METABOLISM | RIBOSOME BIOGENESIS | ONCOGENIC TRANSCRIPTION FACTOR | GLUTAMINE-METABOLISM | CELL BIOLOGY | 083
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4. Full Text Integration of Lupinus angustifolius L. (narrow-leafed lupin) genome maps and comparative mapping within legumes
by Wyrwa, Katarzyna and Książkiewicz, Michał and Szczepaniak, Anna and Susek, Karolina and Podkowiński, Jan and Naganowska, Barbara
Chromosome Research, ISSN 0967-3849, 9/2016, Volume 24, Issue 3, pp. 355 - 378
Narrow-leafed lupin (Lupinus angustifolius L.) has recently been considered a reference genome for the Lupinus genus. In the present work, genetic and... 
Life Sciences | Human Genetics | rDNA | Lupinus angustifolius | centromeres | chromosome markers | integrated map | Cell Biology | Animal Genetics and Genomics | Plant Genetics & Genomics | synteny | BRACHYPODIUM-DISTACHYON | ARABIDOPSIS-THALIANA | BACTERIAL ARTIFICIAL CHROMOSOMES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT GENOME | IN-SITU HYBRIDIZATION | RIBOSOMAL-RNA GENES | LOTUS-JAPONICUS | MEDICAGO-TRUNCATULA | GENETICS & HEREDITY | MOLECULAR CYTOGENETIC MAP | PHASEOLUS-VULGARIS | Aspartate Aminotransferases - genetics | Synteny - genetics | Genome, Plant - genetics | Membrane Proteins - genetics | Aspartate-Ammonia Ligase - genetics | In Situ Hybridization, Fluorescence | Phosphoenolpyruvate Carboxylase - genetics | RNA, Ribosomal - genetics | RNA, Ribosomal, 5S - genetics | Chromosomes, Artificial, Bacterial - genetics | Lupinus - genetics | Plant Proteins - genetics | Nitrogen Fixation - genetics | Centromere - genetics | Chromosome Mapping - methods | Genetic Markers - genetics | Glutamate-Ammonia Ligase - genetics | Tandem Repeat Sequences - genetics | Karyotype | DNA, Ribosomal - genetics | Genetic Linkage | Rhizobiaceae - genetics | Phosphates | Legumes | Genes | Genomics | Plant genetics | Fluorescence | Nitrogen | Mimosaceae | Soil microbiology | Beans | Fixation | Ligases | Genetic research | Asparagine | Comparative analysis | Glutamine | Flowers & plants | Chromosomes | Original
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5. Full Text Heat Shock Factor 1 Epigenetically Stimulates Glutaminase-1-Dependent mTOR Activation to Promote Colorectal Carcinogenesis
by Li, Jiaqiu and Song, Ping and Jiang, Tingting and Dai, Dongjun and Wang, Xian and Wang, Hanying and Sun, Jie and Zhu, Liyuan and Xu, Wenxia and Feng, Lifeng and Shin, Vivian Y and Morrison, Helen and Jin, Hongchuan
Molecular Therapy, ISSN 1525-0016, 07/2018, Volume 26, Issue 7, pp. 1828 - 1839
Heat shock factor 1 (HSF1) generally exhibits its properties under stress conditions. In tumors, HSF1 has a pleiotropic feature in regulating growth, survival,... 
glutamine metabolism | MIR137 | GLS1 | colorectal cancer | HSF1 | MIR137HG | MEDICINE, RESEARCH & EXPERIMENTAL | MULTIFACETED ROLES | PROTEIN | DNA METHYLATION | TRANSCRIPTION FACTOR-1 | KINASE | BREAST-CANCER | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | CARCINOMA | PROGRESSION | Cell Proliferation - genetics | Colorectal Neoplasms - genetics | Humans | Mice, Inbred C57BL | Carcinogenesis - genetics | Heat Shock Transcription Factors - genetics | Apoptosis - genetics | Male | Signal Transduction - genetics | Promoter Regions, Genetic - genetics | Heat-Shock Response - genetics | DNA (Cytosine-5-)-Methyltransferases - genetics | Animals | TOR Serine-Threonine Kinases - genetics | Epigenesis, Genetic - genetics | Cell Line, Tumor | Mice | MicroRNAs - genetics | Gene Expression Regulation, Neoplastic - genetics | Glutaminase - genetics | TOR protein | Transcription factors | Colorectal carcinoma | Genomes | mRNA | Kinases | Carcinogenesis | Experiments | Heat shock factors | Proteins | Cell growth | Azoxymethane | Metabolites | DNA methylation | Dextran sulfate | Tumorigenesis | HSF1 protein | Deoxyribonucleic acid--DNA | Enzymes | Cell survival | MiRNA | Metabolism | Gene expression | Glutaminase | Dextran | Sodium | Medical prognosis | Protein expression | DNA methyltransferase | Binding sites | Cancer | Apoptosis | Glutamine | Tumors | Original
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6. Full Text Development of Genetic Circuitry Exhibiting Toggle Switch or Oscillatory Behavior in Escherichia coli
by Atkinson, Mariette R and Savageau, Michael A and Myers, Jesse T and Ninfa, Alexander J
Cell, ISSN 0092-8674, 2003, Volume 113, Issue 5, pp. 597 - 607
Analysis of the system design principles of signaling systems requires model systems where all components and regulatory interactions are known. Components of... 
NITROGEN STARVATION | PROTEIN | RHYTHMS | CYANOBACTERIA | PRODUCT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | GLUTAMINE-SYNTHETASE | OPERON | EXPRESSION | GLNA PROMOTER | CELL BIOLOGY | Genes, Bacterial | Reproducibility of Results | Repressor Proteins - genetics | Lac Operon - genetics | Mutation - genetics | Promoter Regions, Genetic - genetics | Bacterial Proteins | Carrier Proteins - genetics | Gene Expression Regulation, Bacterial - genetics | Membrane Transport Proteins - biosynthesis | Nitrogen Fixation - genetics | Carrier Proteins - metabolism | Escherichia coli Proteins | Escherichia coli - genetics | Gene Expression Regulation, Enzymologic - genetics | Membrane Transport Proteins - genetics | Biological Clocks - genetics | Glutamate-Ammonia Ligase - genetics | Monosaccharide Transport Proteins | Symporters | Escherichia coli - metabolism | Glutamate-Ammonia Ligase - biosynthesis | Repressor Proteins - metabolism | Molecular electronics | Analysis | Genetic aspects | System development | Research | Gene expression | Genetic regulation
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7. Full Text Cleft Lip with Cleft Palate, Ankyloglossia, and Hypodontia are Associated with TBX22 Mutations
by Kantaputra, P.N and Paramee, M and Kaewkhampa, A and Hoshino, A and Lees, M and McEntagart, M and Masrour, N and Moore, G.E and Pauws, E and Stanier, P
Journal of Dental Research, ISSN 0022-0345, 4/2011, Volume 90, Issue 4, pp. 450 - 455
X-linked cleft palate and ankyloglossia (CPX) are caused by mutations in the TBX22 transcription factor. To investigate whether patients with ankyloglossia... 
microdontia | hypodontia | cleft lip and palate | ankyloglossia | TBX22 | dental anomaly | TRANSCRIPTIONAL REPRESSION | CPX | FAMILY | GENE | FREQUENT CAUSE | DENTISTRY, ORAL SURGERY & MEDICINE | MALFORMATION | MICE | EXPRESSION | Conserved Sequence - genetics | Tongue Diseases - genetics | Humans | Child, Preschool | Male | Cleft Palate - genetics | Promoter Regions, Genetic - genetics | Mutation, Missense - genetics | Anodontia - genetics | Proline - genetics | Arginine - genetics | Female | Child | Leucine - genetics | Tryptophan - genetics | Serine - genetics | Exons - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | Labial Frenum - abnormalities | Cleft Lip - genetics | Phenotype | Adolescent | Glutamine - genetics | Polymorphism, Genetic - genetics | Genetic Variation - genetics | Transcription, Genetic - genetics | Cohort Studies | Tyrosine - genetics
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8. Full Text Genetic networks of liver metabolism revealed by integration of metabolic and transcriptional profiling
by Ferrara, Christine T and Wang, Ping and Neto, Elias Chaibub and Stevens, Robert D and Bain, James R and Wenner, Brett R and Ilkayeva, Olga R and Keller, Mark P and Blasiole, Daniel A and Kendziorski, Christina and Yandell, Brian S and Newgard, Christopher B and Attie, Alan D
PLoS Genetics, ISSN 1553-7390, 03/2008, Volume 4, Issue 3, p. e1000034
Although numerous quantitative trait loci (QTL) influencing disease-related phenotypes have been detected through gene mapping and positional cloning,... 
QUANTITATIVE TRAIT LOCI | AMINO-ACID TRANSPORTER | STIMULATED INSULIN-SECRETION | GLUTAMINE | DISEASE | GENETICS & HEREDITY | MICE | HEPATIC-ENCEPHALOPATHY | COMPLEX TRAITS | EXPRESSION | MOLECULAR-MECHANISMS | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Male | Gene Expression Profiling | Leptin - genetics | Hepatocytes - metabolism | RNA, Messenger - metabolism | Phenotype | Animals | Metabolic Networks and Pathways | Mice, Mutant Strains | Female | Mice | Models, Genetic | Quantitative Trait Loci | Crosses, Genetic | Studies | Gene loci | Metabolites | Metabolism | Kinases | Genomics
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9. Full Text Background-dependent effects of polyglutamine variation in the Arabidopsis thaliana gene ELF3
by Soledad Francisca Undurraga and Maximilian Oliver Press and Matthieu Legendre and Nora Bujdoso and Jacob Bale and Hui Wang and Seth J. Davis and Kevin J. Verstrepen and Christine Queitsch
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2012, Volume 109, Issue 47, pp. 19363 - 19367
Tandem repeats (TRs) have extremely high mutation rates and are often considered to be neutrally evolving DNA. However, in coding regions, TR copy number... 
Phenotypes | Hypocotyls | Genetic variation | Alleles | Medical genetics | Huntington disease | Evolutionary genetics | Flowering | Plants | Hybridity | Circadian clock | Microsatellite | Hybrid incompatibility | REPEAT EXPANSION | PROTEIN | MULTIDISCIPLINARY SCIENCES | CLINICAL-FEATURES | circadian clock | HYPOCOTYL GROWTH | NATURAL VARIATION | microsatellite | FLOWERING TIME | HUNTINGTONS-DISEASE | ONSET | hybrid incompatibility | AGE | Haploinsufficiency - genetics | Arabidopsis Proteins - genetics | Peptides - genetics | Circadian Clocks - genetics | Transcription Factors - genetics | Mutation - genetics | Arabidopsis - genetics | Phenotype | Genes, Plant - genetics | Hybridization, Genetic | Tandem Repeat Sequences - genetics | Crosses, Genetic | Arabidopsis thaliana | Plant genetics | Physiological aspects | Genetic aspects | Research | Health aspects | Glutamine | Biological Sciences
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10. Full Text Allelic variants of XRCC1 and XRCC3 repair genes and susceptibility of oral cancer in Brazilian patients
by dos Reis, Mariana Bisarro and Losi‐Guembarovski, Roberta and de Souza Fonseca Ribeiro, Enilze Maria and Cavalli, Iglenir João and Morita, Maria Celeste and Ramos, Gyl Henrique Albrecht and de Oliveira, Benedito Valdecir and Mizuno, Lauro Toyoshi and Rogatto, Silvia Regina and de Syllos Cólus, Ilce Mara
Journal of Oral Pathology & Medicine, ISSN 0904-2512, 02/2013, Volume 42, Issue 2, pp. 180 - 185
J Oral Pathol Med (2013)42: 180–185 Background:  The capacity for DNA repair is essential in maintaining cellular functions and homeostasis; however, this... 
XRCC1 | histopathological parameters | polymorphisms | oral cancer | XRCC3 | Oral cancer | Polymorphisms | Histopathological parameters | HEAD | STRAND BREAK REPAIR | RISK | PATHOLOGY | XPD | NECK-CANCER | ACID SUBSTITUTION VARIANTS | DENTISTRY, ORAL SURGERY & MEDICINE | SQUAMOUS-CELL CARCINOMA | DNA-REPAIR | ASSOCIATION | SINGLE-NUCLEOTIDE POLYMORPHISMS | Gene Frequency - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | DNA Repair - genetics | Case-Control Studies | Young Adult | Arginine - genetics | Threonine - genetics | Aged, 80 and over | Adult | Female | Codon - genetics | Mouth Neoplasms - genetics | Genetic Predisposition to Disease - genetics | Risk Factors | Tryptophan - genetics | Genotype | DNA-Binding Proteins - genetics | Alleles | Glutamine - genetics | Lymphatic Metastasis - genetics | Mouth Neoplasms - pathology | Polymorphism, Single Nucleotide - genetics | Polymorphism, Genetic - genetics | Aged | Genetic Variation - genetics | X-ray Repair Cross Complementing Protein 1 | Methionine - genetics | Squamous cell carcinoma | Oncology, Experimental | Genes | Disease susceptibility | Research | Cancer patients | Anopheles | Codon | Analysis | DNA | Genetic research | Genetic aspects | Mouth cancer | Cancer | Codons
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11. Full Text Sequence features governing aggregation or degradation of prion-like proteins
by Cascarina, Sean M and Paul, Kacy R and Machihara, Satoshi and Ross, Eric D
PLoS Genetics, ISSN 1553-7390, 07/2018, Volume 14, Issue 7, p. e1007517
Enhanced protein aggregation and/or impaired clearance of aggregates can lead to neurodegenerative disorders such as Alzheimer's Disease, Huntington's Disease,... 
EXPOSED HYDROPHOBICITY | SUBSTRATE-SPECIFICITY | GENETICS & HEREDITY | QUALITY-CONTROL DEGRADATION | ALS | AMYOTROPHIC-LATERAL-SCLEROSIS | MUTATIONS | FRONTOTEMPORAL LOBAR DEGENERATION | CHAPERONE | SACCHAROMYCES-CEREVISIAE | YEAST PRION | Prions - metabolism | Neurodegenerative Diseases - pathology | Prions - genetics | Peptide Termination Factors - genetics | Humans | Glutamine - metabolism | Heterogeneous-Nuclear Ribonucleoprotein Group A-B - metabolism | Neurodegenerative Diseases - genetics | Amino Acid Sequence - genetics | Saccharomyces cerevisiae Proteins - genetics | Peptide Termination Factors - metabolism | Protein Domains - genetics | Protein Aggregation, Pathological - pathology | Proteolysis | Asparagine - metabolism | Glutamine - genetics | Saccharomyces cerevisiae Proteins - metabolism | Heterogeneous-Nuclear Ribonucleoprotein Group A-B - genetics | Protein Aggregation, Pathological - genetics | Asparagine - genetics | Protein research | Nucleotide sequence | Protein folding | Prions | Genetic research | Genetic aspects | Research | Nervous system diseases | Analysis | Quality control | Control systems | Glycine | Genetic screening | Quality management | Biodegradation | Yeast | Disease | Neurodegenerative diseases | Amino acids | Biochemistry | Mammals | Proteins | Algorithms | Asparagine | Mutation | Molecular biology | Prion protein | Protein interaction | Glutamine
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