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Annals of Oncology, ISSN 0923-7534, 03/2004, Volume 15, Issue 3, pp. 460 - 466
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 54, Issue 4, pp. 773 - 794
Numerous investigations have shown that mitochondrial dysfunction is a major mechanism of drug-induced liver injury, which involves the parent drug or a... 
Gastroenterology and Hepatology | Drugs | Obesity | Oxidative stress | Mitochondria | Cell death | Lipids | Hepatotoxicity | Steatosis | TRIGLYCERIDE TRANSFER PROTEIN | DNA-POLYMERASE-GAMMA | PREGNANE X-RECEPTOR | PROLIFERATOR-ACTIVATED RECEPTOR | HEPATIC CYTOCHROME-P450 2E1 | ELEMENT-BINDING PROTEINS | FATTY-ACID OXIDATION | CONSTITUTIVE ANDROSTANE RECEPTOR | MANGANESE SUPEROXIDE-DISMUTASE | STRESS-RELATED PARAMETERS | GASTROENTEROLOGY & HEPATOLOGY | Carbohydrate Metabolism - drug effects | Reactive Oxygen Species - metabolism | Mitochondria, Liver - metabolism | Humans | Leptin - metabolism | Oxidative Phosphorylation - drug effects | Adipose Tissue - metabolism | Adiponectin - metabolism | Hepatitis C - complications | Cell Death - drug effects | Fatty Acids - metabolism | Chemical and Drug Induced Liver Injury - etiology | Diabetes Mellitus, Type 2 - complications | Genetic Predisposition to Disease | Fatty Liver - metabolism | Oxidation-Reduction | Obesity - complications | Insulin Resistance | Mitochondrial Membrane Transport Proteins - drug effects | Chemical and Drug Induced Liver Injury - genetics | Mitochondria, Liver - drug effects | Animals | Chemical and Drug Induced Liver Injury - metabolism | Models, Biological | Lipid Metabolism - drug effects | Alcoholic Intoxication - complications | Genome, Mitochondrial | Adipose Tissue - drug effects | Energy Metabolism - drug effects | Fatty Liver - etiology | Divalproex | Liver diseases | Thiols | Liver | Cytochrome P-450 | Mitochondrial DNA | Triglycerides | Stavudine | Permeability | Valproic acid | Fatty acids | Cells | Carnitine | Metabolites | Glutathione transferase | Acetaminophen | Physiological aspects | DNA polymerases | Health aspects | Zidovudine | Index Medicus | Reactive Oxygen Species | Fatty Acids | Adiponectin | Mitochondria, Liver | Life Sciences | Mitochondrial Membrane Transport Proteins | Cell Death | Diabetes Mellitus, Type 2 | Adipose Tissue | Alcoholic Intoxication | Hépatology and Gastroenterology | Oxidative Phosphorylation | Carbohydrate Metabolism | Lipid Metabolism | Drug-Induced Liver Injury | Fatty Liver | Human health and pathology | Energy Metabolism | Leptin | Hepatitis C
Journal Article
Journal of Periodontology, ISSN 0022-3492, 08/2013, Volume 84, Issue 8, pp. 1145 - 1157
Background: Simvastatin is a cholesterol‐lowering drug whose pleiotropic effects may have a therapeutic impact on bone. This study evaluates the effect of... 
hydroxymethylglutaryl‐CoA reductase inhibitors | inflammation | Alveolar bone loss | periodontitis | Inflammation | Hydroxymethylglutaryl-CoA reductase inhibitors | Periodontitis | OVARIECTOMIZED RATS | STATINS | IN-VITRO | METABOLISM | DENTISTRY, ORAL SURGERY & MEDICINE | NITRIC-OXIDE | hydroxymethylglutaryl-CoA reductase inhibitors | MEVALONATE PATHWAY | KAPPA-B LIGAND | PROTEIN PRENYLATION | Interleukin-1beta - drug effects | Simvastatin - therapeutic use | Bone Morphogenetic Protein 2 - drug effects | Rats, Wistar | Matrix Metalloproteinase 8 - drug effects | Alveolar Bone Loss - prevention & control | Receptor Activator of Nuclear Factor-kappa B - drug effects | RANK Ligand - drug effects | Malondialdehyde - analysis | Alanine Transaminase - drug effects | Gingiva - drug effects | Glutathione - drug effects | Anti-Inflammatory Agents - therapeutic use | Female | Osteoprotegerin - drug effects | Interleukin-10 - analysis | Nitrates - analysis | Peroxidase - drug effects | Matrix Metalloproteinase 1 - drug effects | Nitric Oxide Synthase Type II - drug effects | Rats | Aspartate Aminotransferases - drug effects | Alkaline Phosphatase - drug effects | Periodontitis - prevention & control | Antioxidants - therapeutic use | Animals | Tumor Necrosis Factor-alpha - drug effects | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Nitrites - analysis | Oxidative Stress - drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0116747
Cellular mechanisms of multidrug resistance (MDR) are related to ABC transporters, apoptosis, antioxidation, drug metabolism, DNA repair and cell... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | STEM-CELLS | MDR1 | TRANSPORTERS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | MUTANT P53 | TUMOR-SUPPRESSOR PROTEIN | DRUG-RESISTANCE | OVARIAN-CANCER | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Drug Resistance, Multiple - drug effects | Genes, Neoplasm | Humans | Apoptosis - genetics | Epithelial-Mesenchymal Transition - drug effects | Gene Expression Profiling | DNA Repair - genetics | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | Dose-Response Relationship, Drug | MCF-7 Cells | Neoplastic Stem Cells - metabolism | Inhibitory Concentration 50 | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | DNA Repair - drug effects | Drug Resistance, Multiple - genetics | Tumor Suppressor Protein p53 - metabolism | Signal Transduction - genetics | Down-Regulation - drug effects | Cell Shape - drug effects | Up-Regulation - drug effects | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Signal Transduction - drug effects | Doxorubicin - pharmacology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Drug resistance in microorganisms | Anthracyclines | Tumor proteins | Intermediate filament proteins | Genes | Bcl-2 protein | AKT protein | Cytotoxicity | Drug resistance | Cancer therapies | Doxorubicin | Cell morphology | Proteins | CD44 antigen | Drug metabolism | Repair | Drug dosages | Deoxyribonucleic acid--DNA | Enzymes | Ploidy | BRCA1 protein | Gene expression | Metabolism | 1-Phosphatidylinositol 3-kinase | Chemotherapy | Pharmacy | Stem cells | Mutation | Codons | Surface markers | Cell proliferation | Transcription | Mesenchyme | Gene regulation | Cytology | Kinases | DNA repair | Cell surface | E-cadherin | MDR1 protein | Clonal deletion | Rodents | Cell cycle | Deletion | Pharmaceutical sciences | Glutathione | Multidrug resistance | Breast cancer | Medicine | Hypotheses | Gene amplification | Apoptosis | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Plant Physiology and Biochemistry, ISSN 0981-9428, 01/2017, Volume 110, pp. 70 - 81
The role of silicon (Si) in alleviating biotic as well as abiotic stresses is well known. However, the potential of silicon nanoparticle (SiNP) in regulating... 
Antioxidants | Oxidative stress | Silicon | Silicon nanoparticle | UV-B radiation | Nitric oxide | L. SEEDLINGS | ACID | CHLOROPHYLL FLUORESCENCE | ASCORBATE-GLUTATHIONE CYCLE | RADIATION | ANTIOXIDANT PARAMETERS | PLANT SCIENCES | HIGHER-PLANTS | GROWTH | NITRIC-OXIDE | Lipid Peroxidation - radiation effects | Triticum - radiation effects | Antioxidants - metabolism | Radiation-Protective Agents - administration & dosage | Plant Development - drug effects | Triticum - metabolism | Ascorbate Peroxidases - metabolism | Plant Development - radiation effects | Silicon - pharmacology | Ultraviolet Rays | X-Ray Diffraction | Lipid Peroxidation - drug effects | Superoxides - metabolism | Radiation-Protective Agents - pharmacology | Oxidative Stress - radiation effects | Plant Leaves - drug effects | Plant Proteins - metabolism | Superoxide Dismutase - metabolism | Photosynthesis - radiation effects | Metal Nanoparticles - chemistry | Radiation-Protective Agents - chemistry | Plant Leaves - radiation effects | Seedlings - drug effects | Hydrogen Peroxide - metabolism | Particle Size | Metal Nanoparticles - administration & dosage | Plant Leaves - metabolism | Hydroponics - methods | Photosynthesis - drug effects | Oxidative Stress - drug effects | Seedlings - radiation effects | Silicon - chemistry | Seedlings - metabolism | Triticum - drug effects | Enzymes | Phytochemistry | Radiation | Physiological aspects | Superoxide | Photosynthesis | Wheat
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, p. 6761
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e62087
Silica nanoparticles have become promising carriers for drug delivery or gene therapy. Endothelial cells could be directly exposed to silica nanoparticles by... 
PATHWAYS | MAINTENANCE | AIR-POLLUTION | OXIDATIVE STRESS | CHK1 | PARTICLES | MULTIDISCIPLINARY SCIENCES | DISEASE | MITOCHONDRIA | SIZE-DEPENDENT CYTOTOXICITY | CANCER | Protein Kinases - metabolism | Reactive Oxygen Species - metabolism | Nanoparticles - chemistry | Apoptosis - drug effects | Humans | Membrane Potential, Mitochondrial - drug effects | Necrosis | Flow Cytometry | G2 Phase Cell Cycle Checkpoints - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Intracellular Space - drug effects | Subcellular Fractions - drug effects | Endocytosis - drug effects | Nanoparticles - ultrastructure | Hydrodynamics | Static Electricity | Comet Assay | Nanoparticles - toxicity | Subcellular Fractions - metabolism | Particle Size | Human Umbilical Vein Endothelial Cells - enzymology | Silicon Dioxide - toxicity | Intracellular Space - metabolism | Checkpoint Kinase 1 | Human Umbilical Vein Endothelial Cells - pathology | DNA Damage | Oxidative Stress - drug effects | Drugs | Nanoparticles | Drug delivery systems | DNA | DNA damage | Superoxide | Radiation, Background | DNA repair | Silica | Vehicles | Endothelium | Cdc2 protein | Reactive oxygen species | Intravenous administration | Toxicity | Cytology | Cytotoxicity | Superoxide dismutase | Drug delivery | Kinases | Cyclin B1 | Silicon dioxide | Signal transduction | Mitochondria | Peroxidase | Membrane potential | Damage | Heart diseases | Deoxyribonucleic acid--DNA | Glutathione | Glutathione peroxidase | Outdoor air quality | CHK1 protein | Lactate dehydrogenase | Exposure | Hazards | Malondialdehyde | L-Lactate dehydrogenase | Endothelial cells | Signaling | Lactic acid | Mutation | Gene therapy | Cardiovascular diseases | Viability | Apoptosis | Deoxyribonucleic acid
Journal Article
Schizophrenia Bulletin, ISSN 0586-7614, 09/2012, Volume 38, Issue 5, pp. 958 - 966
Journal Article