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Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 1, pp. 15 - 18
Ischemic postconditioning (IPoC) reduces infarct size following ischemia/reperfusion. Whether or not phosphorylation of RISK (reperfusion injury salvage... 
PERMEABILITY TRANSITION PORE | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | REPERFUSION | reperfusion injury | NECROSIS-FACTOR-ALPHA | ADENOSINE RECEPTORS | infarct size | INHIBITION | MYOCARDIAL INFARCT SIZE | cardioprotection | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | MOUSE HEARTS | SIGNAL TRANSDUCER | Glycogen Synthase Kinase 3 - physiology | MAP Kinase Kinase 2 - physiology | Nitriles - pharmacology | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Glycogen Synthase Kinase 3 beta | Ribosomal Protein S6 Kinases, 70-kDa - physiology | MAP Kinase Kinase 1 - physiology | Myocardial Reperfusion Injury - enzymology | Ribosomal Protein S6 Kinases, 70-kDa - biosynthesis | Protein Processing, Post-Translational - drug effects | Swine | Mitogen-Activated Protein Kinase 3 - biosynthesis | Myocardial Infarction - pathology | Swine, Miniature | Phosphorylation - drug effects | Myocardial Infarction - enzymology | MAP Kinase Kinase 1 - antagonists & inhibitors | Butadienes - pharmacology | Glycogen Synthase Kinase 3 - biosynthesis | Mitogen-Activated Protein Kinase 1 - physiology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Coronary Occlusion - enzymology | Enzyme Induction | Proto-Oncogene Proteins c-akt - physiology | Enzyme Activation - drug effects | Mitogen-Activated Protein Kinase 3 - physiology | Mitogen-Activated Protein Kinase 1 - biosynthesis | Proto-Oncogene Proteins c-akt - biosynthesis | Animals | Androstadienes - pharmacology | Myocardial Reperfusion | MAP Kinase Kinase 2 - antagonists & inhibitors | Coronary Occlusion - pathology | Phosphatidylinositol 3-Kinases - physiology | Protein Kinase Inhibitors - pharmacology | Myocardial Reperfusion Injury - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2014, Volume 192, Issue 3, pp. 1120 - 1129
Journal Article
American Journal of Reproductive Immunology, ISSN 1046-7408, 05/2015, Volume 73, Issue 5, pp. 460 - 478
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 05/2008, Volume 104, Issue 5, pp. 1452 - 1461
Journal Article
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 04/2013, Volume 18, Issue 4, pp. 451 - 460
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0136155 - e0136155
Tyrosine kinase inhibitors (TKIs) against EGFR and c-Met are initially effective when administered individually or in combination to non-small cell lung cancer... 
GROWTH-FACTOR-RECEPTOR | PATHWAY | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | T790M MUTATIONS | ACQUIRED-RESISTANCE | GENE-EXPRESSION | DOWN-REGULATION | BETA-CATENIN | WNT | Carcinoma, Non-Small-Cell Lung - pathology | Receptor, Epidermal Growth Factor - genetics | Glycogen Synthase Kinase 3 - biosynthesis | Carcinoma, Non-Small-Cell Lung - genetics | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Erlotinib Hydrochloride - administration & dosage | Glycogen Synthase Kinase 3 beta | GATA6 Transcription Factor - biosynthesis | Proto-Oncogene Proteins c-met - genetics | Drug Synergism | Protein Kinase Inhibitors - administration & dosage | Drug Resistance, Neoplasm - genetics | TOR Serine-Threonine Kinases - genetics | Wnt Signaling Pathway - drug effects | Glycogen Synthase Kinase 3 - genetics | Heterocyclic Compounds, 3-Ring - administration & dosage | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | GATA6 Transcription Factor - genetics | TOR Serine-Threonine Kinases - biosynthesis | Genetic aspects | Research | Lung cancer, Non-small cell | Drug therapy | Drug resistance | TOR protein | Cell proliferation | Wnt protein | Transcription | Laboratories | Lung cancer | Kinases | Cancer therapies | Modulators | c-Met protein | Proteins | β-catenin | Cell growth | Tumorigenesis | Inhibition | Protein-tyrosine kinase | Tyrosine | Immunoglobulins | Epidermal growth factor receptors | Cloning | Non-small cell lung carcinoma | Gene expression | Patients | Medicine | Gene amplification | Inhibitors | Medical prognosis | Cell lines | Mutation | Viability | Cancer | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 310, Issue 2, pp. H174 - H187
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2011, Volume 286, Issue 13, pp. 11009 - 11020
Lenalidomide plays an important role in our chemotherapeutic armamentarium against multiple myeloma, in part by exerting direct anti-proliferative and... 
ADHESION | REFRACTORY MULTIPLE-MYELOMA | PLUS DEXAMETHASONE | MECHANISM | PATHWAY | MEDIATED DRUG-RESISTANCE | COMBINATION THERAPY | BIOCHEMISTRY & MOLECULAR BIOLOGY | THALIDOMIDE | WNT | SYNTHASE KINASE 3 | Casein Kinase Ialpha - biosynthesis | Cyclin D1 - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Transcription, Genetic - drug effects | Multiple Myeloma - mortality | Humans | Casein Kinase Ialpha - genetics | Glycogen Synthase Kinase 3 beta | Thalidomide - pharmacology | Wnt Proteins - metabolism | Plasma Cells - pathology | Thalidomide - analogs & derivatives | Multiple Myeloma - drug therapy | Wnt Proteins - genetics | Plasma Cells - metabolism | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Glycogen Synthase Kinase 3 - biosynthesis | Proto-Oncogene Proteins c-myc - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Drug Resistance, Neoplasm - genetics | Multiple Myeloma - pathology | Cyclin D1 - genetics | Glycogen Synthase Kinase 3 - genetics | Signal Transduction - drug effects | Gene Expression Regulation, Enzymologic - genetics | Cell Line, Tumor | Wnt3 Protein | Proto-Oncogene Proteins c-myc - genetics | Transcription, Genetic - genetics | Wnt3A Protein | Multiple Myeloma - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus | Signal Transduction | Lenalidomide | Beta-catenin | Drug Action | Immunomodulatory Agent | Wnt Pathway | Drug Resistance | Multiple Myeloma
Journal Article
Journal Article