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Annual Review of Immunology, ISSN 0732-0582, 2007, Volume 25, Issue 1, pp. 297 - 336
Conference Proceeding
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 5, pp. 1827 - 1832
CD1d-restricted natural killer T (NKT) cells include two major subgroups. The most widely studied are Vα14Jα18⁺ invariant NKT (iNKT) cells that recognize the... 
T lymphocytes | Antigens | Reactivity | Natural killer T cells | Hybridomas | Antigen presenting cells | Lipids | Infections | Phospholipids | T cell antigen receptors | CD1 antigen presentation | Innate immunity | Adaptive immunity | Type II natural killer T cell | SULFATIDE | REPERTOIRE | POPULATION | ACTIVATION | MULTIDISCIPLINARY SCIENCES | DISTINCT | type II natural killer T cell | RECEPTOR | adaptive immunity | CD1 | innate immunity | KILLER T-CELLS | INNATE-LIKE | GLYCOLIPIDS | Hybridomas - metabolism | Corynebacterium glutamicum - genetics | Antigens, CD1d - immunology | Transferases (Other Substituted Phosphate Groups) - genetics | Antigens, Bacterial - immunology | Mycobacterium tuberculosis - immunology | Transferases (Other Substituted Phosphate Groups) - immunology | Cardiolipins - immunology | Galactosylceramides - metabolism | Receptors, Antigen, T-Cell, alpha-beta - immunology | Corynebacterium glutamicum - metabolism | Transferases (Other Substituted Phosphate Groups) - metabolism | Natural Killer T-Cells - metabolism | Myeloid Differentiation Factor 88 - immunology | Antigens, CD1d - genetics | Bacterial Proteins - immunology | Phospholipids - metabolism | Toll-Like Receptors - metabolism | Mycobacterium tuberculosis - metabolism | Phosphatidylglycerols - metabolism | Macrophages - immunology | Macrophages - microbiology | Cell Line | Antigen-Presenting Cells - metabolism | Mice, Inbred C57BL | Phosphatidylglycerols - immunology | Phospholipids - immunology | Toll-Like Receptors - immunology | Bacterial Proteins - genetics | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Cardiolipins - metabolism | Antigen-Presenting Cells - immunology | Antigens, CD1d - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Galactosylceramides - immunology | Hybridomas - immunology | Bacterial Proteins - metabolism | Mice | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Corynebacterium glutamicum - immunology | Antigens, Bacterial - metabolism | Myeloid Differentiation Factor 88 - metabolism | Natural Killer T-Cells - immunology | Autoimmunity | Killer cells | Antigen receptors, T cell | Receptors | Research | Properties | T cells | Biological Sciences | glycolipids | KazuyoshiKawakami | distinct | Mikrobiologi inom det medicinska området | VictorNizet | population | MoriyaTsuji | cd1 | receptor | Gurdyal S.Besra | Microbiology in the medical area | killer t-cells | innate-like | sulfatide | repertoire | KNizet | VBesra | GSTsuji | activation | Kawakami
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 05/2012, Volume 109, Issue 19, pp. E1143 - E1152
Obesity triggers a low-grade systemic inflammation, which plays an important role in the development of obesity-associated metabolic diseases. In searching for... 
Cluster of differentiation 1d | Alpha-galactosylceramide | Glycolipid-reactive T cells | Obesity-induced inflammation | glycolipid-reactive T cells | NKT CELLS | INKT CELLS | GLUCOSE-INTOLERANCE | MULTIDISCIPLINARY SCIENCES | GLYCOLIPID ANTIGENS | INNATE IMMUNE-SYSTEM | FATTY-ACIDS | NONALCOHOLIC STEATOHEPATITIS | DEFICIENT MICE | DANGER SIGNALS | alpha-galactosylceramide | cluster of differentiation 1d | obesity-induced inflammation | ADIPOSE-TISSUE | Adipose Tissue, White - immunology | Antigens, CD1d - immunology | Obesity - immunology | Adipose Tissue, White - metabolism | Lipids - immunology | Male | Dietary Fats - immunology | Galactosylceramides - administration & dosage | Obesity - genetics | Lymphocyte Activation - immunology | Flow Cytometry | Natural Killer T-Cells - metabolism | Dietary Fats - administration & dosage | Inflammation Mediators - metabolism | Antigens, CD1d - genetics | CD8-Positive T-Lymphocytes - metabolism | Female | Macrophages - immunology | Cytokines - immunology | Fatty Liver - genetics | Inflammation Mediators - immunology | Adipose Tissue, White - pathology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Inflammation - immunology | Lipids - administration & dosage | Galactosylceramides - physiology | Antigens, CD1d - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Galactosylceramides - immunology | Fatty Liver - immunology | Insulin Resistance - genetics | Inflammation - genetics | Insulin Resistance - immunology | Mice | CD8-Positive T-Lymphocytes - immunology | Natural Killer T-Cells - immunology | Biological Sciences | PNAS Plus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53531
Successful vaccination against intracellular pathogens requires the generation of cellular immune responses. Trehalose-6,6-dibehenate (TDB), the synthetic... 
CRUCIAL ROLE | IFN-GAMMA PRODUCTION | CASPASE-1 | INFLAMMASOME ACTIVATION | MULTIDISCIPLINARY SCIENCES | CYTOKINE | IL-1-BETA | INDUCTION | ANTIBODY-RESPONSES | T-CELLS | MYD88 | Adjuvants, Immunologic - administration & dosage | Cytoskeletal Proteins - genetics | Receptors, Interleukin-1 - genetics | Antigens, Bacterial - immunology | Interleukin-17 - immunology | Lectins, C-Type - immunology | Lectins, C-Type - genetics | Th1 Cells - immunology | Cytoskeletal Proteins - deficiency | Membrane Proteins - deficiency | Molecular Mimicry | Adjuvants, Immunologic - chemistry | Myeloid Differentiation Factor 88 - immunology | Interferon-gamma - genetics | Lectins, C-Type - deficiency | Tuberculosis Vaccines - immunology | Adaptive Immunity | Immunization | Vaccines, Subunit | Antigens, Bacterial - chemistry | Signal Transduction | Membrane Proteins - genetics | Receptors, Interleukin-1 - immunology | Cord Factors - chemistry | Tuberculosis Vaccines - administration & dosage | Gene Expression Regulation | Myeloid Differentiation Factor 88 - genetics | Interleukin-17 - genetics | Membrane Proteins - immunology | Cord Factors - immunology | Mice, Knockout | Tuberculosis - immunology | Animals | Apoptosis Regulatory Proteins | Cytoskeletal Proteins - immunology | Tuberculosis Vaccines - chemistry | CARD Signaling Adaptor Proteins | Interferon-gamma - immunology | Tuberculosis - prevention & control | Th17 Cells - immunology | Mice | Lectins | Antigens | Membrane lipids | Tuberculosis vaccines | Plant lipids | Vaccination | Trehalose | Helper cells | Intracellular signalling | Activation | Lymphocytes T | Interleukin 1 receptors | Cord factor | Experiments | Signal transduction | Immunology | Lymphocytes | Interleukin 1 | Immunoglobulins | Immune response | Cytokines | Neutrophils | Adjuvanticity | Interleukin 17 | Interleukin 18 | Infectious diseases | Hospitals | Tuberculosis | Glycolipids | Antigen-presenting cells | γ-Interferon | MyD88 protein | Syk protein | Hygiene
Journal Article
Immunity, ISSN 1074-7613, 2007, Volume 27, Issue 4, pp. 597 - 609
Invariant natural killer T (iNKT) cells are a subset of innate lymphocytes that recognize lipid antigens in the context of CD1d and mediate potent immune... 
CELLIMMUNO | DEFENSE | INFECTIONS | RECOGNITION | BIOSYNTHESIS | GLYCOLIPID ANTIGENS | KILLER T-CELLS | MICE | ISOGLOBOTRIHEXOSYLCERAMIDE | IMMUNOLOGY | GALACTOSYLCERAMIDE | INNATE IMMUNITY | Membrane Glycoproteins - metabolism | Coculture Techniques | Dendritic Cells - immunology | RNA, Messenger - analysis | Interferon-gamma - metabolism | Interferon Type I - immunology | Toll-Like Receptor 9 - immunology | Lymphocyte Activation - immunology | T-Lymphocytes - metabolism | Antigens, CD1d | Neoplasms, Experimental - immunology | Toll-Like Receptor 7 - immunology | Interferon Type I - metabolism | Killer Cells, Natural - immunology | Female | Membrane Glycoproteins - immunology | Dendritic Cells - metabolism | Antigens, CD1 - immunology | Toll-Like Receptor 7 - metabolism | Immunotherapy, Adoptive | Acidic Glycosphingolipids - immunology | Mice, Inbred C57BL | Cells, Cultured | Acidic Glycosphingolipids - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Animals | Antigens, CD1 - metabolism | Interferon-gamma - immunology | T-Lymphocytes - immunology | Mice | Killer Cells, Natural - metabolism | Neoplasms, Experimental - metabolism | Toll-Like Receptor 9 - metabolism | Studies | Liver | Melanoma | Lipids | T cell receptors | Software | Autoimmune diseases | Immune system | Killer Cells, Natural | Dendritic Cells | T-Lymphocytes | Lymphocyte Activation | Antigens, CD1 | Toll-Like Receptor 9 | Toll-Like Receptor 7 | Interferon Type I | Life Sciences | Immunology | Interferon Type II | Neoplasms, Experimental | Acidic Glycosphingolipids | Membrane Glycoproteins | RNA, Messenger
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2011, Volume 129, Issue 1, pp. 216 - 227.e6
Journal Article
Infection and Immunity, ISSN 0019-9567, 01/2013, Volume 81, Issue 1, pp. 311 - 316
Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
INFECTIOUS DISEASES | IMMUNOLOGY | RECOGNITION | CUTTING EDGE | ANTIGEN | Tumor Necrosis Factor-alpha - metabolism | Mycobacterium bovis - immunol