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The Journal of clinical investigation, ISSN 1558-8238, 2015, Volume 125, Issue 11, pp. 4223 - 4238
.... We have recently shown that salt has a proinflammatory effect and boosts the activation of Th17 cells and the activation of classical, LPS-induced macrophages (M1... 
MEDICINE, RESEARCH & EXPERIMENTAL | M2 MACROPHAGES | NA+ STORAGE | TISSUE | GENE-EXPRESSION | URINARY SODIUM | TRANSCRIPTION FACTOR | POTASSIUM EXCRETION | ALTERNATIVE ACTIVATION | BLOOD-PRESSURE | T-CELLS | Sodium Chloride, Dietary - pharmacology | Sodium Chloride - pharmacology | Male | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Oxidative Phosphorylation - drug effects | Interleukin-4 - pharmacology | Histone Code - drug effects | Bone Marrow Cells - drug effects | TOR Serine-Threonine Kinases - physiology | Wound Healing - drug effects | Macrophages - immunology | Macrophages - classification | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Inflammation | Proto-Oncogene Proteins c-akt - physiology | Mitochondria - drug effects | Random Allocation | Interleukin-13 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Sodium Chloride, Dietary - toxicity | Chitin - toxicity | Macrophages - drug effects | Mice | Macrophage Activation - drug effects | Salt | Medical research | Immune response | Medicine, Experimental | Research | Macrophages | Properties | Observations | Biological control systems | Health aspects | Hypertension | Wound healing | Sodium | Cytokines | Kinases | Metabolism | Gene expression | Experiments | Acquisitions & mergers
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 8, pp. 2473 - 2478
The malignant progression of pancreatic ductal adenocarcinoma (PDAC) is accompanied by a profound desmoplasia, which forces proliferating tumor cells to... 
PROTEIN | MULTIDISCIPLINARY SCIENCES | GLYCOLYSIS | pancreatic cancer | PATHWAY | PROSTATE-CANCER | LIPID RAFTS | cholesterol | S-PHASE | metabolism | gemcitabine | CELL-CYCLE | KRAS | ACCUMULATION | SIMVASTATIN | LDLR | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Gene Silencing - drug effects | Pancreatic Neoplasms - metabolism | Prognosis | Epithelial Cells - drug effects | Humans | Deoxycytidine - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Adenocarcinoma - metabolism | Lipoproteins - metabolism | Clone Cells | Gene Expression Regulation, Neoplastic - drug effects | Receptors, LDL - genetics | Pancreatic Neoplasms - pathology | Adenocarcinoma - enzymology | Pancreatic Neoplasms - enzymology | Receptors, LDL - metabolism | Epithelial Cells - pathology | Up-Regulation - genetics | Cholesterol - metabolism | Adenocarcinoma - drug therapy | Up-Regulation - drug effects | Metabolic Networks and Pathways - genetics | Phenotype | Animals | MAP Kinase Signaling System - drug effects | Cell Compartmentation - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Metabolic Networks and Pathways - drug effects | Deoxycytidine - analogs & derivatives | Pancreatic cancer | Carcinoma, Ductal | Development and progression | Genetic aspects | Genetic transcription | Cholesterol metabolism | Health aspects | Biological Sciences
Journal Article
Molecular cancer, ISSN 1476-4598, 2016, Volume 15, Issue 1, p. 69
Journal Article
Stem cells (Dayton, Ohio), ISSN 1066-5099, 2016, Volume 34, Issue 5, pp. 1163 - 1176
..., including an enhanced Warburg effect, a greater carbon and energy source flexibility driven... 
Mitochondrial metabolism | Glutaminolysis | Cancer stem cells | Epithelial‐mesenchymal transition | Metabolic flux analysis | Warburg effect | Epithelial-mesenchymal transition | GLYCINE | NETWORK | FIBROBLASTS | GLUTAMINE-METABOLISM | CELL & TISSUE ENGINEERING | CELL BIOLOGY | HETEROGENEITY | SERINE | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH | HEMATOLOGY | EXPRESSION | PLASTICITY | FOLATE | Metabolomics | Transcription, Genetic - drug effects | Epithelial Cells - metabolism | Neoplastic Stem Cells - drug effects | Epithelial Cells - drug effects | Genes, Neoplasm | Humans | Spheroids, Cellular - pathology | Epithelial-Mesenchymal Transition - drug effects | Gene Expression Profiling | Epithelial-Mesenchymal Transition - genetics | Glycolysis - drug effects | Glycolysis - genetics | Amino Acids - metabolism | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Spheroids, Cellular - drug effects | NADP - metabolism | Pyruvate Dehydrogenase Complex - metabolism | Cell Proliferation - genetics | Spheroids, Cellular - metabolism | Citric Acid Cycle - drug effects | Epithelial Cells - pathology | Mitochondria - metabolism | Mitochondria - drug effects | Disease Progression | Citric Acid Cycle - genetics | Cell Line, Tumor | Glucose - metabolism | Cell Proliferation - drug effects | Oxidative Stress - drug effects | Mesoderm - pathology | Fatty Acids - biosynthesis | Hydrogen-Ion Concentration | Stem cells | Development and progression | Metastasis | Prostate cancer | Fatty acids | Cancer | Glutamine | Metabolism | epithelial-mesenchymal transition | metabolic flux analysis | mitochondrial metabolism | glutaminolysis
Journal Article
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 02/2010, Volume 30, Issue 8, pp. 2967 - 2978
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 8, p. e0135083
The chemotherapeutic use of cisplatin is limited by its severe side effects. In this study, by conducting different omics data analyses, we demonstrated... 
APOPTOSIS | OXIDATIVE STRESS | P53 ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | TUBULAR EPITHELIAL-CELLS | DOWN-REGULATION | AUDITORY CELLS | DEATH | CANCER | NEPHROTOXICITY | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Membrane Potential, Mitochondrial - drug effects | Glycolysis - drug effects | Reactive Oxygen Species - agonists | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Cisplatin - toxicity | Tumor Suppressor Protein p53 - agonists | Epithelial Cells - cytology | Kidney Tubules - metabolism | Cell Line | Kidney Tubules - drug effects | Citric Acid Cycle - drug effects | Gene Expression Regulation | Injections, Intraperitoneal | Tumor Suppressor Protein p53 - metabolism | Rats | Rats, Sprague-Dawley | Kidney Tubules - cytology | Animals | L-Lactate Dehydrogenase - genetics | Acetylcysteine - pharmacology | Cell Proliferation - drug effects | Physiological aspects | Reactive oxygen species | Genetic aspects | Research | Cisplatin | Bioengineering | Tricarboxylic acid cycle | Oxidative stress | Correlation | Toxicity | p53 Protein | Genes | Science | Cytotoxicity | Kinases | Ovarian cancer | Mitochondria | Cell growth | Pathways | Metabolites | Rodents | Cell cycle | Inhibition | Adenosine triphosphate | Urine | Oxygen | Abnormalities | Mortality | Data processing | Interdisciplinary aspects | Metabolism | Gene expression | Side effects | Cell death | Glycolysis | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 6, pp. E725 - E734
Journal Article