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European Journal of Pharmaceutical Sciences, ISSN 0928-0987, 07/2015, Volume 74, pp. 95 - 102
Journal Article
Nature, ISSN 0028-0836, 04/2013, Volume 496, Issue 7444, pp. 238 - 242
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4223 - 4238
A high intake of dietary salt (NaCl) has been implicated in the development of hypertension, chronic inflammation, and autoimmune diseases. We have recently... 
MEDICINE, RESEARCH & EXPERIMENTAL | M2 MACROPHAGES | NA+ STORAGE | TISSUE | GENE-EXPRESSION | URINARY SODIUM | TRANSCRIPTION FACTOR | POTASSIUM EXCRETION | ALTERNATIVE ACTIVATION | BLOOD-PRESSURE | T-CELLS | Sodium Chloride, Dietary - pharmacology | Sodium Chloride - pharmacology | Male | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Oxidative Phosphorylation - drug effects | Interleukin-4 - pharmacology | Histone Code - drug effects | Bone Marrow Cells - drug effects | TOR Serine-Threonine Kinases - physiology | Wound Healing - drug effects | Macrophages - immunology | Macrophages - classification | Immunity, Innate - drug effects | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Inflammation | Proto-Oncogene Proteins c-akt - physiology | Mitochondria - drug effects | Random Allocation | Interleukin-13 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Sodium Chloride, Dietary - toxicity | Chitin - toxicity | Macrophages - drug effects | Mice | Macrophage Activation - drug effects | Salt | Medical research | Immune response | Medicine, Experimental | Research | Macrophages | Properties | Observations | Biological control systems | Health aspects | Hypertension | Wound healing | Sodium | Cytokines | Kinases | Metabolism | Gene expression | Experiments | Acquisitions & mergers | Index Medicus | Abridged Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 2014, Volume 20, Issue 11, pp. 1327 - 1333
Journal Article
Cell Metabolism, ISSN 1550-4131, 11/2016, Volume 24, Issue 5, pp. 657 - 671
Journal Article
Nature, ISSN 0028-0836, 10/2014, Volume 514, Issue 7524, pp. 628 - 632
Pancreatic ductal adenocarcinoma (PDAC) is one of the deadliest cancers in western countries, with a median survival of 6 months and an extremely low... 
STEM-CELLS | INHIBITION | MELANOMA | METABOLISM | GLUCOSE | MULTIDISCIPLINARY SCIENCES | IMATINIB | LEUKEMIA | AUTOPHAGY | INDUCTION | TUMOR-GROWTH | Recurrence | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Autophagy | Carcinoma, Pancreatic Ductal - genetics | Oxidative Phosphorylation - drug effects | Pancreatic Neoplasms - drug therapy | Lysosomes - metabolism | Neoplastic Stem Cells - metabolism | Cell Respiration - drug effects | Neoplastic Stem Cells - pathology | Female | Disease Models, Animal | Proto-Oncogene Proteins p21(ras) - metabolism | Cell Survival - drug effects | Neoplasm Recurrence, Local - prevention & control | Signal Transduction | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Mitochondria - metabolism | Genes, p53 - genetics | Mitochondria - drug effects | Mutation - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Glycolysis | Mice | Protein research | Ablation (Surgery) | Care and treatment | Pancreatic cancer | Oncology, Experimental | Genetic aspects | Research | Cancer | Mitochondria | Stem cells | Cell cycle | Lipids | Metabolism | Tumors | Apoptosis | Index Medicus
Journal Article
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 17, pp. 14777 - 14795
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2010, Volume 30, Issue 8, pp. 2967 - 2978
Journal Article
Cell Metabolism, ISSN 1550-4131, 01/2014, Volume 19, Issue 1, pp. 37 - 48
Journal Article
Journal of Experimental Biology, ISSN 0022-0949, 08/2012, Volume 215, Issue 15, pp. 2567 - 2578
Previous studies in two rainbow trout lines divergently selected for lean (L) or fat (F) muscle suggested that they differ in their ability to metabolise... 
Carbohydrates | Fish nutrition | Genetic selection | Gene expression | Metabolism | carbohydrates | ACTIVATED PROTEIN-KINASE | genetic selection | ONCORHYNCHUS-MYKISS | NUTRITIONAL REGULATION | TIME RT-PCR | LIPOGENIC ENZYME-ACTIVITIES | DIFFERENT FAMILIES | HEPATIC GLUCOSE-METABOLISM | BIOLOGY | GENE-EXPRESSION | fish nutrition | metabolism | SALMON SALMO-SALAR | gene expression | ATLANTIC SALMON | Gene Expression Regulation, Enzymologic - drug effects | Liver - enzymology | Ribosomal Protein S6 Kinases - metabolism | Gluconeogenesis - drug effects | RNA, Messenger - metabolism | Glycolysis - drug effects | Proto-Oncogene Proteins c-akt - genetics | Glycolysis - genetics | Adipose Tissue - metabolism | Lipogenesis - genetics | Liver - drug effects | Adenylate Kinase - metabolism | Oxidation-Reduction - drug effects | Postprandial Period - drug effects | Phosphorylation - drug effects | Muscles - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Fatty Acids - metabolism | Oncorhynchus mykiss - metabolism | Dietary Carbohydrates - pharmacology | RNA, Messenger - genetics | Oncorhynchus mykiss - genetics | Transcription Factors - genetics | Oncorhynchus mykiss - blood | Blotting, Western | Oncorhynchus mykiss - growth & development | Feeding Behavior - drug effects | Transcription Factors - metabolism | Animals | Gluconeogenesis - genetics | Lipogenesis - drug effects | Muscles - drug effects | Adenylate Kinase - genetics | Adipose Tissue - drug effects | Ribosomal Protein S6 Kinases - genetics | Adipose tissues | Hyperglycemia | Research | Diagnosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0135083 - e0135083
The chemotherapeutic use of cisplatin is limited by its severe side effects. In this study, by conducting different omics data analyses, we demonstrated that... 
APOPTOSIS | OXIDATIVE STRESS | P53 ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | TUBULAR EPITHELIAL-CELLS | DOWN-REGULATION | AUDITORY CELLS | DEATH | CANCER | NEPHROTOXICITY | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Membrane Potential, Mitochondrial - drug effects | Glycolysis - drug effects | Reactive Oxygen Species - agonists | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Cisplatin - toxicity | Tumor Suppressor Protein p53 - agonists | Epithelial Cells - cytology | Kidney Tubules - metabolism | Cell Line | Kidney Tubules - drug effects | Citric Acid Cycle - drug effects | Gene Expression Regulation | Injections, Intraperitoneal | Tumor Suppressor Protein p53 - metabolism | Rats | Rats, Sprague-Dawley | Kidney Tubules - cytology | Animals | L-Lactate Dehydrogenase - genetics | Acetylcysteine - pharmacology | Cell Proliferation - drug effects | Physiological aspects | Reactive oxygen species | Genetic aspects | Research | Cisplatin | Bioengineering | Tricarboxylic acid cycle | Oxidative stress | Correlation | Toxicity | p53 Protein | Genes | Science | Cytotoxicity | Kinases | Ovarian cancer | Mitochondria | Cell growth | Pathways | Metabolites | Rodents | Cell cycle | Inhibition | Adenosine triphosphate | Urine | Abnormalities | Mortality | Data processing | Interdisciplinary aspects | Metabolism | Gene expression | Side effects | Cell death | Glycolysis | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2015, Volume 112, Issue 8, pp. 2473 - 2478
The malignant progression of pancreatic ductal adenocarcinoma (PDAC) is accompanied by a profound desmoplasia, which forces proliferating tumor cells to... 
PROTEIN | MULTIDISCIPLINARY SCIENCES | GLYCOLYSIS | pancreatic cancer | PATHWAY | PROSTATE-CANCER | LIPID RAFTS | cholesterol | S-PHASE | metabolism | gemcitabine | CELL-CYCLE | KRAS | ACCUMULATION | SIMVASTATIN | LDLR | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Gene Silencing - drug effects | Pancreatic Neoplasms - metabolism | Prognosis | Epithelial Cells - drug effects | Humans | Deoxycytidine - pharmacology | Extracellular Signal-Regulated MAP Kinases - metabolism | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Adenocarcinoma - metabolism | Lipoproteins - metabolism | Clone Cells | Gene Expression Regulation, Neoplastic - drug effects | Receptors, LDL - genetics | Pancreatic Neoplasms - pathology | Adenocarcinoma - enzymology | Pancreatic Neoplasms - enzymology | Receptors, LDL - metabolism | Epithelial Cells - pathology | Up-Regulation - genetics | Cholesterol - metabolism | Adenocarcinoma - drug therapy | Up-Regulation - drug effects | Metabolic Networks and Pathways - genetics | Phenotype | Animals | MAP Kinase Signaling System - drug effects | Cell Compartmentation - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Metabolic Networks and Pathways - drug effects | Deoxycytidine - analogs & derivatives | Pancreatic cancer | Carcinoma, Ductal | Development and progression | Genetic aspects | Genetic transcription | Cholesterol metabolism | Health aspects | Lipids | Chemotherapy | Pancreas | Cells | Cholesterol | Tumors | Index Medicus | Biological Sciences
Journal Article