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Journal of Pathology, ISSN 0022-3417, 2005, Volume 206, Issue 3, pp. 291 - 304
Journal Article
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 08/2010, Volume 19, Issue 15, pp. 3011 - 3020
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2006, Volume 126, Issue 1, pp. 107 - 120
The p53 tumor-suppressor protein prevents cancer development through various mechanisms, including the induction of cell-cycle arrest, apoptosis, and the... 
CANCER-CELLS | GLUCOSE-6-PHOSPHATE-DEHYDROGENASE | ARREST | OXIDATIVE STRESS | ACTIVATION | PENTOSE-PHOSPHATE PATHWAY | GENE | GLUCOSE-METABOLISM | FRUCTOSE-2,6-BISPHOSPHATASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | P53 | CELL BIOLOGY | Reactive Oxygen Species - metabolism | Humans | DNA Repair - physiology | Cell Survival - genetics | Genomic Instability - physiology | Molecular Sequence Data | Apoptosis - genetics | Fructose-Bisphosphatase - isolation & purification | Proteins - isolation & purification | Tumor Suppressor Protein p53 - genetics | Phosphoric Monoester Hydrolases - isolation & purification | Fructosediphosphates - metabolism | Glycolysis - genetics | DNA Damage - physiology | Cell Transformation, Neoplastic - genetics | Base Sequence | Energy Metabolism - genetics | Chromosomes, Human, Pair 12 - genetics | Amino Acid Sequence | Phosphoric Monoester Hydrolases - genetics | Oxidative Stress - genetics | Tumor Suppressor Protein p53 - metabolism | Gene Expression Regulation - physiology | Intracellular Signaling Peptides and Proteins | Cell Transformation, Neoplastic - metabolism | Down-Regulation - physiology | Proteins - genetics | Fructose-Bisphosphatase - metabolism | Proteins - metabolism | Cell Line, Tumor | Signal Transduction - physiology | Fructose-Bisphosphatase - genetics | Phosphoric Monoester Hydrolases - metabolism | Glycolysis | Genetic research | Tumor suppressor genes | Research | Apoptosis | Prevention | Oncology, Experimental | Tumor proteins | Fructose | Cancer | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2016, Volume 126, Issue 11, pp. 4372 - 4386
Obese, insulin-resistant states are characterized by a paradoxical pathogenic condition in which the liver appears to be selectively insulin resistant.... 
RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | TRANSCRIPTION FACTOR FOXO1 | FATTY LIVER-DISEASE | OB/OB MICE | ELEMENT-BINDING PROTEIN | HEPATIC STEATOSIS | ADIPOSE-TISSUE | PLASMA TRIGLYCERIDES | Glucose Intolerance - metabolism | Glucose-6-Phosphatase - genetics | Fatty Liver - pathology | Humans | Male | Glucose - biosynthesis | Glucose Intolerance - pathology | Fatty Liver - chemically induced | Glycolysis - drug effects | Glycolysis - genetics | Lipogenesis - genetics | Female | Glucose Intolerance - chemically induced | Insulin - genetics | Nuclear Proteins - genetics | Fatty Liver - genetics | Forkhead Box Protein O1 - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Fructose - toxicity | Insulin Resistance | Glucose - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Glucose-6-Phosphatase - metabolism | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Forkhead Box Protein O1 - genetics | Obesity | Analysis | Insulin resistance | Genetic aspects | Genetic transcription | Research | Risk factors | Protein binding | Glucose | Metabolites | Rodents | Liver | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2007, Volume 282, Issue 42, pp. 31038 - 31045
Methylglyoxal is a highly reactive dicarbonyl degradation product formed from triose phosphates during glycolysis. Methylglyoxal forms stable adducts primarily... 
DIABETIC-RETINOPATHY | ACTIVATION | ADVANCED GLYCATION | SP1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | O-GLYCOSYLATION | MICE | 3 MAJOR PATHWAYS | EXPRESSION | HYPERGLYCEMIC DAMAGE | GLYOXALASE-I | Tumor Necrosis Factor-alpha - metabolism | Transcription, Genetic - drug effects | Kidney - pathology | Sweetening Agents - metabolism | Angiopoietin-2 - genetics | Protein Processing, Post-Translational - genetics | Diabetes Mellitus, Experimental - genetics | N-Acetylglucosaminyltransferases - genetics | Glycolysis - drug effects | Acetylglucosamine - metabolism | Glycolysis - genetics | Pyruvaldehyde - metabolism | Kidney - metabolism | Diabetic Angiopathies - pathology | Protein Processing, Post-Translational - drug effects | Arginine - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Response Elements - genetics | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Repressor Proteins - metabolism | Sp3 Transcription Factor - metabolism | Diabetic Angiopathies - metabolism | Sweetening Agents - pharmacology | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Sp3 Transcription Factor - genetics | Angiopoietin-2 - biosynthesis | Repressor Proteins - genetics | Glucose - pharmacology | Acetylglucosamine - genetics | N-Acetylglucosaminyltransferases - metabolism | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Intercellular Adhesion Molecule-1 - genetics | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Endothelial Cells - pathology | Arginine - metabolism | Cell Line, Transformed | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2003, Volume 100, Issue 14, pp. 8466 - 8471
Type 2 diabetes mellitus (DM) is characterized by insulin resistance and pancreatic β cell dysfunction. In high-risk subjects, the earliest detectable... 
Body mass index | Biological Sciences | Fasting | Diabetes complications | Genes | Insulin resistance | Type 2 diabetes mellitus | Diabetes | Lipid metabolism | Family history | Insulin | OBESE PATIENTS | PIMA-INDIANS | MITOCHONDRIAL BIOGENESIS | LIPID-CONTENT | MULTIDISCIPLINARY SCIENCES | EXPRESSION PROFILE | FATTY-ACIDS | MELLITUS | HUMAN SKELETAL-MUSCLE | GAMMA COACTIVATOR-1 | MICROARRAY DATA | Prediabetic State - metabolism | Prediabetic State - genetics | Oligonucleotide Array Sequence Analysis | Diabetes Mellitus - genetics | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Transcription Factors - deficiency | Male | Muscle, Skeletal - metabolism | Gene Expression Profiling | Nuclear Respiratory Factors | Trans-Activators - physiology | Glycolysis - genetics | Adult | Female | Transcription, Genetic | Nuclear Respiratory Factor 1 | Obesity | DNA-Binding Proteins - physiology | Genetic Predisposition to Disease | Transcription Factors - physiology | Gene Expression Regulation - genetics | Oxidative Phosphorylation | Diabetes Mellitus - metabolism | Receptors, Cytoplasmic and Nuclear - physiology | Transcription Factors - genetics | Citric Acid Cycle - genetics | Mexican Americans - genetics | Biopsy | Insulin Resistance - genetics | NF-E2-Related Factor 1 | Lipid Peroxidation - genetics | Muscle, Skeletal - pathology | Type 2 diabetes | Genetic aspects | Genetics | Metabolism | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2012, Volume 15, Issue 5, pp. 725 - 738
Mammalian target of rapamycin complex 2 (mTORC2) phosphorylates and activates AGC kinase family members, including Akt, SGK1, and PKC, in response to... 
TRANSCRIPTION FACTORS | LIPID-METABOLISM | INSULIN-RESISTANCE | ELEMENT-BINDING PROTEIN-1C | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | MOTIF PHOSPHORYLATION | DIABETIC-NEPHROPATHY | GLUCOSE-UTILIZATION | CELL-GROWTH | PHOSPHOINOSITIDE 3-KINASE | CELL BIOLOGY | Glucose Intolerance - metabolism | Phosphorylation | Liver - enzymology | TOR Serine-Threonine Kinases - metabolism | Homeostasis | Glycogen Synthase Kinase 3 beta | Male | Hepatocytes - metabolism | Mechanistic Target of Rapamycin Complex 2 | Hyperglycemia - genetics | Proto-Oncogene Proteins c-akt - genetics | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Forkhead Transcription Factors - metabolism | Trans-Activators - genetics | Hyperinsulinism - genetics | Insulin - genetics | Lipogenesis | Proto-Oncogene Proteins c-akt - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Gluconeogenesis | Hyperinsulinism - metabolism | Glucose Intolerance - genetics | Signal Transduction | Liver - metabolism | Glucokinase - genetics | Glucose - genetics | Lipid Metabolism | Forkhead Transcription Factors - genetics | Glucokinase - metabolism | Glycogen Synthase Kinase 3 - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Proteins - genetics | Insulin - metabolism | Animals | Proteins - metabolism | Trans-Activators - deficiency | Glycogen Synthase Kinase 3 - genetics | Sterol Regulatory Element Binding Protein 1 - genetics | Glucose - metabolism | Glycolysis | Trans-Activators - metabolism | Forkhead Box Protein O1 | Mice | Transcription Factors | Glucose metabolism | Hyperglycemia | Glucose | Isoenzymes | Dextrose | Index Medicus
Journal Article