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PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0121046 - e0121046
A unique feature of cancer cells is to convert glucose into lactate to produce cellular energy, even under the presence of oxygen. Called aerobic glycolysis... 
CANCER-CELLS | MAMMALIAN TARGET | RAPAMYCIN | STRAND BREAK REPAIR | METABOLISM | PATHWAY | MULTIDISCIPLINARY SCIENCES | GROWTH | STRESS | UP-REGULATION | TRANSLATIONAL CONTROL | Oxygen Consumption - radiation effects | Hexokinase - antagonists & inhibitors | TOR Serine-Threonine Kinases - metabolism | HCT116 Cells | Humans | Radiation | Mitochondria - metabolism | Breast Neoplasms - metabolism | Breast Neoplasms - radiotherapy | Oxidative Phosphorylation - radiation effects | MCF-7 Cells | Adenosine Triphosphate - metabolism | Glycolysis - radiation effects | Energy Metabolism - radiation effects | Cell Line, Tumor | Glucose - metabolism | Mitochondria - radiation effects | Female | Hexokinase - metabolism | Cell Proliferation - radiation effects | Lactates | Glucose metabolism | Ionizing radiation | Colorectal cancer | Mitochondrial DNA | Glucose | Health aspects | Dextrose | Tumors | Energy metabolism | Genotoxicity | Cancer therapies | Proteins | Mitochondria | Cell growth | Enzymatic activity | Bioenergetics | Penicillin | Inhibition | Deoxyribonucleic acid--DNA | Radiation effects | Rapamycin | Metabolism | Survival | Radiation dosage | Gamma rays | Protein synthesis | Irradiation | Hypoxia | Mutation | TOR protein | Cell proliferation | Energy consumption | Phosphorylation | Glioblastoma | Reversing | Clinical trials | Oncology | Kinases | Autophagy | Colon cancer | Energy | Colon | Relocation | Oxygen | Cell survival | Tumor cells | Oxygen consumption | Breast cancer | Radiation therapy | Tumor cell lines | Hexokinase | Medicine | Oxidative phosphorylation | Glycolysis | Lactic acid | Electron transport | Prostate | Cancer | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Transfusion, ISSN 0041-1132, 06/2004, Volume 44, Issue 6, pp. 877 - 885
Journal Article
Plant Physiology, ISSN 0032-0889, 2/2013, Volume 161, Issue 2, pp. 1034 - 1048
The regulation of carbon metabolism in the diatom Phaeodactylum tricornutum at the cell, metabolite, and gene expression levels in exponential fed-batch... 
Enzymes | Scotophase | Photophase | Genes | SYSTEMES AND SYNTHETIC BIOLOGY | Lipids | Gene expression regulation | Biosynthesis | Diatoms | Gene expression | Fatty acids | FATTY-ACID COMPOSITION | MARINE DIATOM | GLYCOLYTIC PATHWAY | MICROALGAE | MITOTIC CHROMOSOME | DARK CYCLE | CELL-CYCLE | NUTRIENT LIMITATION | CARBOHYDRATE-METABOLISM | PHYTOPLANKTON | PLANT SCIENCES | Oligonucleotide Array Sequence Analysis | Diatoms - genetics | Carbohydrate Metabolism - genetics | Gene Expression Profiling | Phylogeny | Pyruvate Dehydrogenase Complex - genetics | Mitosis - genetics | Acclimatization - radiation effects | Diatoms - metabolism | Glycolysis - genetics | Diatoms - radiation effects | Mitochondria - genetics | Membrane Transport Proteins - genetics | Mitochondria - radiation effects | Plastids - genetics | Lipid Metabolism - genetics | Plastids - metabolism | Carbon - metabolism | Mitosis - radiation effects | Carbon Cycle - genetics | Lipid Metabolism - radiation effects | Mitochondria - metabolism | Photoperiod | Pyruvate Dehydrogenase Complex - classification | Carbohydrate Metabolism - radiation effects | Gluconeogenesis - genetics | Gluconeogenesis - radiation effects | Membrane Transport Proteins - classification | Glycolysis - radiation effects | Gene Expression Regulation - radiation effects | Acclimatization - genetics | Plastids - radiation effects | Circadian rhythms | Physiological aspects | Acclimatization | Genetic aspects | Genetic regulation | Carbon | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2017, Volume 23, Issue 19, pp. 5881 - 5891
Purpose: MUC1, an oncogene overexpressed in multiple solid tumors, including pancreatic cancer, reduces overall survival and imparts resistance to radiation... 
RADIATION-RESISTANCE | CELLS | MUC1 | POSITRON-EMISSION-TOMOGRAPHY | ONCOLOGY | ADENOCARCINOMA | MOUSE MODEL | DNA-DAMAGE | INHIBITOR | TUMOR-GROWTH | EXPRESSION | Cell Proliferation - genetics | Pancreatic Neoplasms - metabolism | Humans | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - radiotherapy | Radiation Tolerance - genetics | Pancreatic Neoplasms - genetics | Gene Knockdown Techniques | Xenograft Model Antitumor Assays | Animals | Glycolysis - radiation effects | Cell Line, Tumor | Glucose - metabolism | Gene Expression Regulation, Neoplastic - radiation effects | Mice | Signal Transduction - radiation effects | DNA Damage - radiation effects | Cell Proliferation - radiation effects | Mucin-1 - genetics | Phosphates | Cell culture | Toxicity | DNA damage | Radiation | Cytotoxicity | Biosynthesis | Alterations | Pentose | Biocompatibility | Inhibition | Pretreatment | Deoxyribonucleic acid--DNA | Pentose phosphate | Radiation effects | Damage assessment | Radiation therapy | Tumor cell lines | Metabolism | Pools | Gene expression | Studies | Experimental design | Pancreatic cancer | Cell lines | Pentose phosphate pathway | Radiation tolerance | Glycolysis | Solid tumors | Radiation damage | Tumors | Cancer | Index Medicus | cancer metabolism | pancreatic cancer | mucin | radiation resistance | nucleotide metabolism | bromopyruvate
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2016, Volume 291, Issue 9, pp. 4698 - 4710
Journal Article
Oncogene, ISSN 0950-9232, 08/2018, Volume 37, Issue 32, pp. 4385 - 4397
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2015, Volume 89, pp. 263 - 273
We investigated whether altering Warburg metabolism (aerobic glycolysis) by treatment with the metabolic agent dichloroacetate (DCA) could increase the... 
Fractionated/split-dose | Dichloroacetate (DCA) | Double-strand DNA break repair | Non-small cell lung cancer (NSCLC) | Warburg effect | INDIVIDUAL CELLS | CYCLE REGULATION | COMET ASSAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | BODY RADIATION-THERAPY | MAMMALIAN CELLS | CHINESE-HAMSTER CELLS | CLINICAL-TRIALS | ENDOCRINOLOGY & METABOLISM | Double strand DNA break repair | RADIOTHERAPY | NF-KAPPA-B | Carcinoma, Non-Small-Cell Lung - radiotherapy | Lung Neoplasms - drug therapy | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Lung Neoplasms - metabolism | DNA Repair - radiation effects | Lung Neoplasms - radiotherapy | Lung Neoplasms - pathology | Glycolysis - drug effects | DNA Breaks, Double-Stranded - radiation effects | Glycolysis - physiology | DNA Breaks, Double-Stranded - drug effects | Tumor Cells, Cultured | Carcinoma, Non-Small-Cell Lung - pathology | DNA Repair - drug effects | Cell Cycle - radiation effects | Carcinoma, Non-Small-Cell Lung - metabolism | Comet Assay | Radiation Tolerance - drug effects | X-Rays | Dichloroacetic Acid - pharmacology | Cell Proliferation - drug effects | Carcinoma, Non-Small-Cell Lung - drug therapy | Cell Cycle - drug effects | Cell Proliferation - radiation effects | Lactates | Glucose metabolism | Growth | Cancer cells | Physiological aspects | Glucose | Lung cancer, Non-small cell | Statistics | Dextrose | Medical colleges | Radiation | Respiratory agents | Radiotherapy | DNA repair | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100738 - e100738
Neoadjuvant chemoradiation therapy (CRT) is increasingly the standard of care for locally advanced oesophageal cancer. A complete pathological response to CRT... 
GENOMIC INSTABILITY | OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | NEOADJUVANT CHEMORADIOTHERAPY | PREOPERATIVE CHEMORADIOTHERAPY | IONIZING-RADIATION | DNA-REPAIR | EXPRESSION | COLON-CANCER-CELLS | EXPOSURE | HUMAN-TUMOR XENOGRAFTS | Mitochondrial Size - drug effects | Adenocarcinoma - pathology | Humans | Middle Aged | Male | Chemoradiotherapy, Adjuvant | Mutagenesis - drug effects | Esophageal Neoplasms - pathology | Mitochondrial Size - radiation effects | Adenocarcinoma - metabolism | Mutagenesis - radiation effects | Esophageal Neoplasms - metabolism | Energy Metabolism - radiation effects | Mitochondria - radiation effects | Adult | Female | Esophageal Neoplasms - therapy | Treatment Outcome | Mitochondria - metabolism | Mitochondria - drug effects | Radiation Tolerance - drug effects | Adenocarcinoma - therapy | Cell Line, Tumor | Aged | Energy Metabolism - drug effects | Adenocarcinoma | Glucose metabolism | Genetic aspects | Gene expression | Analysis | Esophageal cancer | Oxidative stress | Energy metabolism | Phosphorylation | Oxidative metabolism | Radiation | Radioresistance | Genomes | Mitochondrial DNA | Kinases | Cancer therapies | Mitochondria | Alterations | Energy | Bioenergetics | Surgery | Cell cycle | Pretreatment | Deoxyribonucleic acid--DNA | Radiation therapy | Metabolism | Esophagus | Pathology | Gene amplification | Oxidative phosphorylation | Medical prognosis | Glycolysis | Mutation | Electron transport | Cancer | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Cell death & disease, ISSN 2041-4889, 2014, Volume 5, Issue 5, pp. e1255 - e1255
Journal Article