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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2015, Volume 35, Issue 9, pp. 1945 - 1953
Journal Article
Kidney International, ISSN 0085-2538, 08/2013, Volume 84, Issue 2, pp. 338 - 352
Venous neointimal hyperplasia (VNH) is responsible for hemodialysis vascular access malfunction. Here we tested whether VNH formation occurs, in part, due to... 
veins | chronic kidney disease | restenosis | murine model | arteriovenous fistula | Restenosis | Arteriovenous fistula | Chronic kidney disease | Veins | Murine model | NEOINTIMAL HYPERPLASIA | MMP-9 SECRETION | MATRIX METALLOPROTEINASE-2 | VEGF-A | INTIMAL HYPERPLASIA | ARTERIOVENOUS-FISTULA | MOUSE MODEL | UROLOGY & NEPHROLOGY | ATRIAL MYOFIBROBLAST PROLIFERATION | MUSCLE-CELL-PROLIFERATION | ENDOTHELIAL GROWTH-FACTOR | NIH 3T3 Cells | Apoptosis - drug effects | Constriction, Pathologic | Hyperplasia | Actins - metabolism | Caspase 3 - metabolism | Graft Occlusion, Vascular - prevention & control | Male | Vascular Endothelial Growth Factor A - metabolism | Jugular Veins - surgery | Simvastatin - pharmacology | Jugular Veins - metabolism | Jugular Veins - drug effects | Myofibroblasts - metabolism | Matrix Metalloproteinase 9 - metabolism | Neointima | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cell Transdifferentiation - drug effects | Graft Occlusion, Vascular - metabolism | Disease Models, Animal | Jugular Veins - pathology | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Myofibroblasts - drug effects | Graft Occlusion, Vascular - genetics | Cell Movement - drug effects | Animals | Graft Occlusion, Vascular - etiology | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Graft Occlusion, Vascular - pathology | Signal Transduction - drug effects | Arteriovenous Shunt, Surgical - adverse effects | Carotid Arteries - surgery | Cell Proliferation - drug effects | Mice | Renal Dialysis | Basic Research
Journal Article
Circulation, ISSN 0009-7322, 10/2017, Volume 136, Issue 18, pp. 1749 - 1764
Journal Article
Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 289 - 306
Venous neointimal hyperplasia (VNH) causes hemodialysis vascular access failure. Here we tested whether VNH formation occurs in part due to local vessel... 
diabetes | VEGF | vascular access | OXIDATIVE STRESS | INCREASED EXPRESSION | NEOINTIMAL HYPERPLASIA | MATRIX METALLOPROTEINASE-2 | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | HEMODIALYSIS GRAFTS | INTIMAL HYPERPLASIA | RESTENOSIS | UROLOGY & NEPHROLOGY | INHIBITOR | ENDOTHELIAL GROWTH-FACTOR | RNA, Small Interfering - genetics | Cell Proliferation | Constriction, Pathologic | Hyperplasia | Caspase 3 - metabolism | Graft Occlusion, Vascular - prevention & control | Male | Vascular Endothelial Growth Factor A - metabolism | Jugular Veins - surgery | Vascular Endothelial Growth Factor A - genetics | Jugular Veins - metabolism | Adventitia - metabolism | Renal Insufficiency, Chronic - metabolism | Matrix Metalloproteinase 9 - metabolism | Neointima | Nephrectomy | RNA Interference | Time Factors | Renal Insufficiency, Chronic - genetics | Lentivirus - genetics | Graft Occlusion, Vascular - metabolism | Adventitia - surgery | Disease Models, Animal | Fibroblasts - metabolism | Transduction, Genetic | Jugular Veins - pathology | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Fibroblasts - pathology | Graft Occlusion, Vascular - genetics | Renal Insufficiency, Chronic - pathology | Animals | Graft Occlusion, Vascular - pathology | Arteriovenous Shunt, Surgical - adverse effects | Carotid Arteries - surgery | Adventitia - pathology | Mice | Genetic Vectors | Apoptosis | Cell Movement | Genetic Therapy - methods | RNA, Small Interfering - metabolism | Basic Research
Journal Article
Kidney International, ISSN 0085-2538, 09/2015, Volume 88, Issue 3, pp. 490 - 502
A major factor contributing to failure of arteriovenous fistulas (AVFs) is migration of smooth muscle cells into the forming neointima. To identify the source... 
neointima formation | chronic kidney disease | fibroblast specific protein 1 | vascular smooth muscle cell | arteriovenous fistula | Notch | CARDIAC NEURAL CREST | HEMODIALYSIS VASCULAR ACCESS | BONE-MARROW-CELLS | VEIN GRAFTS | MESENCHYMAL TRANSITION | HYPERPLASIA | IN-VITRO | UNILATERAL URETERAL OBSTRUCTION | REGULATED KINASE 1 | UROLOGY & NEPHROLOGY | CIRCULATING PROGENITOR CELLS | Muscle, Smooth, Vascular - metabolism | S100 Calcium-Binding Protein A4 | Humans | Middle Aged | Carotid Artery, Common - surgery | Myocytes, Smooth Muscle - pathology | Male | Muscle, Smooth, Vascular - surgery | Jugular Veins - surgery | Muscle, Smooth, Vascular - physiopathology | Renal Insufficiency, Chronic - therapy | S100 Proteins - genetics | Neointima | Transfection | Blood Vessel Prosthesis Implantation - adverse effects | RNA Interference | Models, Animal | Graft Occlusion, Vascular - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Myocytes, Smooth Muscle - metabolism | Calcium-Binding Proteins - metabolism | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Signal Transduction | Carotid Artery, Common - metabolism | Blood Vessel Prosthesis Implantation - instrumentation | Cells, Cultured | Mice, Transgenic | S100 Proteins - metabolism | Receptor, Notch1 - metabolism | Muscle, Smooth, Vascular - pathology | Animals | Graft Occlusion, Vascular - etiology | Graft Occlusion, Vascular - pathology | Arteriovenous Shunt, Surgical - adverse effects | Carotid Artery, Common - pathology | Aged | Receptor, Notch1 - genetics | Graft Occlusion, Vascular - physiopathology | Calcium-Binding Proteins - genetics | Cell Movement | Renal Dialysis | Vascular Patency
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2011, Volume 31, Issue 1, pp. 95 - 101
OBJECTIVE—Annexin A5 (AnxA5) has antithrombotic, antiapoptotic, and antiinflammatory properties; we investigated its effectiveness against vascular... 
atherosclerosis | restenosis | annexin A5 | vein grafting | inflammation | CELLS | ATHEROTHROMBOSIS | ACCELERATED ATHEROSCLEROSIS | PREVENTION | MECHANISMS | VEIN GRAFTS | INHIBITION | PLACEMENT | DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | TRANSGENIC MICE | Inflammation - pathology | Apolipoproteins E - deficiency | Annexin A5 - pharmacology | Atherosclerosis - genetics | Graft Occlusion, Vascular - prevention & control | Veins - transplantation | Annexin A5 - administration & dosage | Hypercholesterolemia - drug therapy | Endothelium, Vascular - chemistry | Inflammation - metabolism | Chemotaxis, Leukocyte - drug effects | Time Factors | Mice, Mutant Strains | Anti-Inflammatory Agents - administration & dosage | Graft Occlusion, Vascular - metabolism | Cytokines - blood | Disease Models, Animal | Atherosclerosis - pathology | Atherosclerosis - drug therapy | Anti-Inflammatory Agents - pharmacology | Atherosclerosis - physiopathology | Cells, Cultured | Endothelium, Vascular - physiopathology | Inflammation Mediators - blood | Injections, Intraperitoneal | Cell Adhesion - drug effects | Vascular Patency - drug effects | Atherosclerosis - metabolism | Graft Occlusion, Vascular - genetics | Mice, Knockout | Animals | Graft Occlusion, Vascular - pathology | Endothelium, Vascular - metabolism | Apolipoproteins E - genetics | Inflammation - genetics | Endothelium, Vascular - pathology | Hypercholesterolemia - physiopathology | Inflammation - prevention & control | Mice | Hypercholesterolemia - genetics | Mutation | Vasodilation - drug effects | Nitric Oxide - metabolism | Hypercholesterolemia - metabolism | Graft Occlusion, Vascular - physiopathology | Inflammation - physiopathology
Journal Article
Journal Article