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Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/2013, Volume 210, Issue 11, pp. 2205 - 2221
Journal Article
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2013, Volume 369, Issue 1, pp. 20 - 31
Journal Article
American Journal of Transplantation, ISSN 1600-6135, 03/2013, Volume 13, Issue 3, pp. 663 - 675
Atypical hemolytic and uremic syndrome (aHUS) is a severe disease strongly associated with genetic abnormalities in the complement alternative pathway. In... 
Acute rejection | endothelial cell damage | mutations and polymorphisms in complement alternative pathway | thrombotic microangiopathy | atypical hemolytic and uremic syndrome | renal transplantation | SURGERY | FACTOR-B | HUS | FACTOR-I | FACTOR-H-AUTOANTIBODIES | TRANSPLANTATION | KIDNEY-TRANSPLANTATION | MUTATIONS | CD46 | PREDISPOSE | MEMBRANE COFACTOR PROTEIN | Recurrence | Genetic Testing | Prognosis | Humans | Middle Aged | Male | Young Adult | Hemolytic-Uremic Syndrome - genetics | Complement Factor B - genetics | Adult | Complement System Proteins - genetics | Female | Hemolytic-Uremic Syndrome - therapy | Retrospective Studies | Complement C3 - genetics | Biomarkers - metabolism | Risk Factors | Biomarkers - analysis | Kidney Transplantation | Mutation - genetics | Fibrinogen - genetics | Graft Survival - genetics | Adolescent | Membrane Cofactor Protein - genetics | Aged | Complement Factor H - genetics | Graft Rejection - genetics | Atypical Hemolytic Uremic Syndrome | Medical research | Kidneys | Genes | Organ transplant recipients | Medicine, Experimental | Genetic research | Transplantation | Clinical trials | Mutation | Transplants & implants | Drug therapy | Biological Markers | Complement C3 | Life Sciences | Immunology | Fibrinogen | Hemolytic-Uremic Syndrome | Antigens, CD46 | Graft Survival | Complement Factor H | Complement System Proteins | Complement Factor B | Graft Rejection
Journal Article
American Journal of Transplantation, ISSN 1600-6135, 07/2016, Volume 16, Issue 7, pp. 1982 - 1998
Interstitial fibrosis and tubular atrophy ( IFTA ) is found in approximately 25% of 1‐year biopsies posttransplant. It is known that IFTA correlates with... 
translational research/science | TCMR | organ transplantation in general | kidney transplantation/nephrology | Interstitial fibrosis and tubular atrophy | graft survival | genomics | kidney (allograft) function/dysfunction | immunobiology | rejection: T cell mediated | basic (laboratory) research/science | rejection: T cell mediated (TCMR) | SURGERY | basic (laboratory) research | PROTOCOL BIOPSIES | CELL-MEDIATED REJECTION | kidney transplantation | INJURY | GRAFT FAILURE | TUBULAR ATROPHY | dysfunction | TRANSPLANTATION | nephrology | B-CELLS | KIDNEY-TRANSPLANT RECIPIENTS | DECEASED DONORS | translational research | RENAL-ALLOGRAFT REJECTION | science | kidney (allograft) function | SUBCLINICAL REJECTION | Fibrosis - genetics | Glomerular Filtration Rate | Kidney Failure, Chronic - surgery | Prognosis | Humans | Nephritis, Interstitial - mortality | Risk Factors | Graft Survival | Survival Rate | Gene Expression Profiling | Fibrosis - mortality | Kidney Transplantation - methods | Kidney Function Tests | Kidney Failure, Chronic - genetics | Nephritis, Interstitial - genetics | Graft Rejection - mortality | Atrophy - genetics | Atrophy - mortality | Kidney Tubules - pathology | Graft Rejection - genetics | Kidney Tubules - metabolism | Analysis | Immunotherapy | Genes | Genomics | Transplantation of organs, tissues, etc | Genetic research | T cells | Gene expression | Atrophy | Immunosuppression | Phenotypes | Biopsy | Etiology | Fibrosis | Inflammation | Survival | Graft rejection
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 07/2015, Volume 26, Issue 7, pp. 1721 - 1731
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4094 - 4104
TNF, an inflammatory cytokine that is enriched in the tumor microenvironment, promotes tumor growth and subverts innate immune responses to cancer cells. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | INHIBITION | INFLAMMATION | MACROPHAGES | DEATH | EXPRESSION | CANCER | ANTITUMOR | T-CELLS | FACTOR-ALPHA | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Tumor Escape | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor-alpha - genetics | Caspase 8 - metabolism | Graft Rejection - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Caspase 8 - genetics | Signal Transduction - immunology | Neoplasms - genetics | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | T-Lymphocytes - pathology | Graft Rejection - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - biosynthesis | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Caspase 8 - immunology | Animals | Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | CASP8 and FADD-Like Apoptosis Regulating Protein - immunology | Graft Rejection - immunology | Myeloid Cells - pathology | Graft Rejection - genetics | Neoplasms - pathology | Tumor necrosis factor | Genetic aspects | Cellular signal transduction | Research | Properties | Gene expression | Apoptosis
Journal Article
Journal Article