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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 50, pp. 21248 - 21255
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 10/2009, Volume 180, Issue 7, pp. 640 - 648
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 12/2012, Volume 18, Issue 12, pp. 1786 - 1796
Journal Article
1995, ISBN 0521461588, xi, 329
The colony-stimulating factors (CSFs) are the major regulators in the body of the production and activity of two types of white blood cells - granulocytes and... 
Colony-stimulating factors (Physiology) | Receptors, Colony-Stimulating Factor | Hematopoiesis | Colony-Stimulating Factors
Book
Stroke, ISSN 0039-2499, 10/2013, Volume 44, Issue 10, pp. 2681 - 2687
Journal Article
Immunity, ISSN 1074-7613, 07/2015, Volume 43, Issue 1, pp. 187 - 199
Journal Article
Cytokine, ISSN 1043-4666, 08/2019, Volume 120, pp. 191 - 191
The aim of this study was to mention some methodological issues in a study which investigate the effect of Granulocyte colony-stimulating factor on developing... 
Adverse event | Aortitis | Epidemiologic methods | Sparse data bias | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNOLOGY | CELL BIOLOGY | Granulocyte colony-stimulating factor | Epidemiology | Research institutes
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 7, pp. 1362 - 1370.e7
Background & Aims Patients with decompensated cirrhosis have significantly reduced survival without liver transplantation. Granulocyte colony-stimulating... 
Gastroenterology and Hepatology | DPO | Liver Regeneration | Chronic Liver Disease | Clinical Trial | SURVIVAL | PROGENITORS | STEM-CELLS | INFECTIONS | MOBILIZATION | REGENERATION | BONE-MARROW | NEOINTIMAL HYPERPLASIA | G-CSF | CHRONIC LIVER-FAILURE | GASTROENTEROLOGY & HEPATOLOGY | Liver - pathology | Prospective Studies | Shock, Septic - etiology | Humans | Middle Aged | Liver Cirrhosis - mortality | Erythropoietin - adverse effects | Male | Shock, Septic - prevention & control | Liver - physiopathology | Erythropoietin - analogs & derivatives | Liver - drug effects | Time Factors | Injections, Subcutaneous | Adult | Female | Drug Therapy, Combination | Granulocyte Colony-Stimulating Factor - adverse effects | Severity of Illness Index | Erythropoietin - administration & dosage | Liver Cirrhosis - drug therapy | Liver Cirrhosis - etiology | Liver Cirrhosis - diagnosis | Double-Blind Method | Drug Administration Schedule | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Disease Progression | India | Biopsy | Liver Cirrhosis - physiopathology | Darbepoetin alfa | Paracentesis | Granulocyte Colony-Stimulating Factor - administration & dosage | Liver Regeneration - drug effects | Colony-stimulating factors (Physiology) | Glycoproteins | Liver cirrhosis | Patient outcomes | Liver | Index Medicus | Abridged Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 11, pp. 4297 - 4309
Journal Article
Cancer Cell, ISSN 1535-6108, 06/2012, Volume 21, Issue 6, pp. 822 - 835
Cancer-associated inflammation is thought to be a barrier to immune surveillance, particularly in pancreatic ductal adenocarcinoma (PDA). Gr-1 CD11b cells are... 
OVEREXPRESSION | MICROENVIRONMENT | ONCOLOGY | DUCTAL ADENOCARCINOMA | INTERLEUKIN-1-BETA | MICE | SUPPRESSOR-CELLS | INDUCTION | DIFFERENTIATION | MAMMARY CARCINOMAS | MOUSE MODELS | CELL BIOLOGY | Immunohistochemistry | Adenocarcinoma - pathology | CD8-Positive T-Lymphocytes - pathology | Pancreatic Neoplasms - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Inflammation - metabolism | Adenocarcinoma - metabolism | RNA Interference | T-Lymphocytes - metabolism | Myeloid Cells - immunology | CD8-Positive T-Lymphocytes - metabolism | T-Lymphocytes - pathology | Carcinoma, Pancreatic Ductal - immunology | CD11b Antigen - immunology | Adenocarcinoma - immunology | Pancreatic Neoplasms - pathology | Receptors, Chemokine - metabolism | Mice, Transgenic | Inflammation - immunology | Receptors, Chemokine - immunology | Carcinoma, Pancreatic Ductal - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Mice, Knockout | Tumor Microenvironment - immunology | Models, Immunological | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pancreatic Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | CD11b Antigen - metabolism | Myeloid Cells - pathology | CD8-Positive T-Lymphocytes - immunology | Medical colleges | Analysis | Pancreatic cancer | Oncology, Experimental | Genetically modified organisms | Inflammation | Macrophage colony stimulating factor | Research | T cells | Tumors | Cancer | Index Medicus | granulocyte-macrophage colony stimulating factor | myeloid cells | T lymphocyte | carcinoma | pancreas
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 07/2008, Volume 26, Issue 21, pp. 3607 - 3613
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 08/2015, Volume 12, Issue 1, pp. 139 - 139
Background: Microglia are dependent upon colony-stimulating factor 1 receptor (CSF1R) signaling for their survival in the adult brain, with administration of... 
Neuroinflammation | Cognition | Alzheimer's disease | Therapeutics | CLEARANCE | ALZHEIMERS-DISEASE | MACROPHAGES | MOUSE | TREM2 | MODEL | IMMUNOLOGY | NEUROSCIENCES | TRAUMATIC BRAIN-INJURY | A-BETA | INFLAMMATORY RESPONSE | TRANSGENIC MICE | Medicine, Experimental | Medical research | Advertising executives | Analysis | Index Medicus
Journal Article