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Cancer Cell, ISSN 1535-6108, 06/2012, Volume 21, Issue 6, pp. 822 - 835
Cancer-associated inflammation is thought to be a barrier to immune surveillance, particularly in pancreatic ductal adenocarcinoma (PDA). Gr-1 CD11b cells are... 
OVEREXPRESSION | MICROENVIRONMENT | ONCOLOGY | DUCTAL ADENOCARCINOMA | INTERLEUKIN-1-BETA | MICE | SUPPRESSOR-CELLS | INDUCTION | DIFFERENTIATION | MAMMARY CARCINOMAS | MOUSE MODELS | CELL BIOLOGY | Immunohistochemistry | Adenocarcinoma - pathology | CD8-Positive T-Lymphocytes - pathology | Pancreatic Neoplasms - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Inflammation - metabolism | Adenocarcinoma - metabolism | RNA Interference | T-Lymphocytes - metabolism | Myeloid Cells - immunology | CD8-Positive T-Lymphocytes - metabolism | T-Lymphocytes - pathology | Carcinoma, Pancreatic Ductal - immunology | CD11b Antigen - immunology | Adenocarcinoma - immunology | Pancreatic Neoplasms - pathology | Receptors, Chemokine - metabolism | Mice, Transgenic | Inflammation - immunology | Receptors, Chemokine - immunology | Carcinoma, Pancreatic Ductal - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Mice, Knockout | Tumor Microenvironment - immunology | Models, Immunological | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pancreatic Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | CD11b Antigen - metabolism | Myeloid Cells - pathology | CD8-Positive T-Lymphocytes - immunology | Medical colleges | Analysis | Pancreatic cancer | Oncology, Experimental | Genetically modified organisms | Inflammation | Macrophage colony stimulating factor | Research | T cells | Tumors | Cancer | Index Medicus | granulocyte-macrophage colony stimulating factor | myeloid cells | T lymphocyte | carcinoma | pancreas
Journal Article
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 04/2019, Volume 10, Issue 1, pp. 1935 - 15
Despite their location at the cell surface, several receptor tyrosine kinases (RTK) are also found in the nucleus, as either intracellular domains or full... 
TARGET | LOCALIZATION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | KINASE | ENDOCYTOSIS | CSF-1 RECEPTOR | EXPRESSION | MET | Chromatin - metabolism | RNA, Small Interfering - genetics | Humans | Leukemia, Myelomonocytic, Chronic - genetics | ets-Domain Protein Elk-1 - genetics | YY1 Transcription Factor - metabolism | Leukemia, Myelomonocytic, Chronic - metabolism | Cell Membrane - chemistry | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - genetics | ets-Domain Protein Elk-1 - metabolism | Early Growth Response Protein 1 - genetics | HEK293 Cells | Macrophage Colony-Stimulating Factor - pharmacology | Cell Membrane - metabolism | Leukemia, Myelomonocytic, Chronic - pathology | Chromatin - drug effects | Cell Membrane - drug effects | Chromatin - chemistry | Fluorescent Dyes - chemistry | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Signal Transduction | Gene Expression Regulation | THP-1 Cells | Macrophage Colony-Stimulating Factor - metabolism | Macrophages - cytology | YY1 Transcription Factor - genetics | Gene Editing | Macrophages - metabolism | Histones - genetics | CRISPR-Cas Systems | Protein Binding | Macrophages - drug effects | Histones - metabolism | Primary Cell Culture | Maleimides - chemistry | Early Growth Response Protein 1 - metabolism | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | RNA, Small Interfering - metabolism | Tyrosine | Myelomonocytic leukemia | Chromatin | Transcription factors | Immunoprecipitation | EGR-1 protein | Colonies | Leukemia | Gene regulation | Macrophage colony-stimulating factor | YY1 protein | Kinases | Gene expression | Macrophages | Cell surface | Recruitment | Proteins | Domains | Monocytes | Chronic myelomonocytic leukemia | Colony-stimulating factor | Index Medicus
Journal Article
The Journal of Infectious Diseases, ISSN 0022-1899, 7/2011, Volume 204, Issue 1, pp. 164 - 173
Journal Article
Human Gene Therapy, ISSN 1043-0342, 07/2013, Volume 24, Issue 7, pp. 644 - 654
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 12/2009, Volume 27, Issue 35, pp. 5911 - 5918
Journal Article
Human Gene Therapy, ISSN 1043-0342, 08/2017, Volume 28, Issue 8, pp. 667 - 680
In advanced and metastatic stages of colorectal cancer (CRC), reduced sensitivity to conventional strategies is still a major obstacle to successful... 
Research Articles | oncolytic adenovirus | colorectal cancer | immune | decorin | GM-CSF | MEDICINE, RESEARCH & EXPERIMENTAL | SYSTEMIC DELIVERY | CELLS | FACTOR-BETA | TGF-BETA | CANCER | IN-VITRO | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | MEDIATED INHIBITION | PHASE-I | EXPRESSION | T-Lymphocyte Subsets - immunology | Genetic Therapy | Genetic Vectors - administration & dosage | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Colorectal Neoplasms - genetics | Humans | Cytopathogenic Effect, Viral | Carcinogenesis - metabolism | Colorectal Neoplasms - therapy | Epithelial-Mesenchymal Transition | Adenoviridae - genetics | Transgenes | Gene Order | Colorectal Neoplasms - metabolism | Decorin - genetics | Disease Models, Animal | Gene Expression | Transduction, Genetic | Cytokines - metabolism | Signal Transduction | Immunomodulation | Carcinogenesis - genetics | Oncolytic Viruses - genetics | Genetic Vectors - genetics | Oncolytic Virotherapy | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Xenograft Model Antitumor Assays | Animals | Colorectal Neoplasms - immunology | Virus Replication | T-Lymphocyte Subsets - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Decorin - metabolism | Decorin | Mesenchyme | Toxicity | CD8 antigen | Transforming growth factor-b | Colorectal carcinoma | Lung | Colorectal cancer | Clinical trials | Cytotoxicity | Lymphocytes T | Infections | Metastasis | Tissues | Macrophages | Blood | Metastases | Angiogenesis | Granzyme B | Granulocytes | Lymphocytes | Coding | Xenografts | Oncolysis | Colony-stimulating factor | Inhibition | Perforin | Vascular endothelial growth factor | Growth factors | Activation analysis | Spleen | Medical research | Immune response | Granulocyte-macrophage colony-stimulating factor | Colonies | Gene expression | Patients | Adenoviruses | Antitumor activity | Leukocytes (granulocytic) | Tumors | Apoptosis | Cancer | Index Medicus
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2017, Volume 23, Issue 19, pp. 5703 - 5710
Purpose: Binding of colony-stimulating factor 1 (CSF1) ligand to the CSF1 receptor (CSF1R) regulates survival of tumor-associated macrophages, which generally... 
METASTASIS | INHIBITION | ONCOLOGY | IMMUNOTHERAPY | MACROPHAGES | BLOCKADE | CSF-1 | ADOPTIVE CELL TRANSFER | CANCER | EXPRESSION | PROGRESSION | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Interleukin-1 - blood | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - blood | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Treatment Outcome | Drug-Related Side Effects and Adverse Reactions - pathology | Neoplasms - drug therapy | Antibodies, Monoclonal - blood | Dose-Response Relationship, Drug | Maximum Tolerated Dose | Macrophage Colony-Stimulating Factor - blood | Neoplasms - genetics | Antibodies, Monoclonal - administration & dosage | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Aged, 80 and over | Female | Aged | Macrophage Colony-Stimulating Factor - genetics | Neoplasms - pathology | Alkaline phosphatase | Intravenous administration | Toxicity | Macrophages | Anticancer properties | Quality | Safety engineering | Colony-stimulating factor | Binding | Edema | Deafness | Pharmacodynamics | Cell survival | Colonies | Macrophage colony-stimulating factor | Fatigue | Nausea | Pharmacology | Disease control | Patients | Immunosuppression | Experimental design | Antitumor activity | Ligands | Aspartate aminotransferase | Receptor mechanisms | Solid tumors | Pharmacokinetics | Tumors | Cancer | Index Medicus
Journal Article
Journal Article