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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2019, Volume 116, Issue 5, pp. 1686 - 1691
While neuroinflammation is an evolving concept and the cells involved and their functions are being defined, microglia are understood to be a key cellular... 
Positron-emission tomography | C]CPPC | CSF1R | DAM | Neuroinflammation | positron-emission tomography | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | PROGENITOR-CELL | LIGANDS | neuroinflammation | INFLAMMATION | MOUSE MODEL | INHIBITOR | GLIA | CSF-1 RECEPTOR | EXPRESSION | [C-11]CPPC | Microglia - metabolism | Mice, Inbred C57BL | Male | Macrophage Colony-Stimulating Factor - metabolism | Positron-Emission Tomography - methods | Brain - metabolism | Radiopharmaceuticals - metabolism | Inflammation - metabolism | Animals | Alzheimer Disease - metabolism | Primates | Plaque, Amyloid - metabolism | Mice | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Disease Models, Animal | Physiological aspects | Cell receptors | PET imaging | Macrophages | Methods | Neuroimaging | Brain | Animal models | Dosimeters | Traumatic brain injury | Encephalomyelitis | Mental disorders | Radiation | Lipopolysaccharides | Head injuries | Demyelination | Autopsy | Radiochemistry | Tomography | Colony-stimulating factor | Dosimetry | Alzheimer's disease | Radioactivity | Medical imaging | Demyelinating diseases | Colonies | Positron emission | Macrophage colony-stimulating factor | Inflammation | Radiation dosimetry | Experimental allergic encephalomyelitis | Microglia | Positron emission tomography | Index Medicus | Biological Sciences | Physical Sciences | [11C]CPPC
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2018, Volume 143, Issue 6, pp. 1483 - 1493
Tumor‐associated macrophages can promote growth of cancers. In neuroblastoma, tumor‐associated macrophages have greater frequency in metastatic versus... 
neuroblastoma | tumor‐associated macrophage | MCS110 | BLZ945 | 5A1 | tumor-associated macrophage | SURVIVAL | NATURAL-KILLER-CELLS | M-CSF | CANCER | ONCOLOGY | GENE-EXPRESSION | RESISTANCE | TUMOR-ASSOCIATED MACROPHAGES | HIGH-RISK NEUROBLASTOMA | PROGRESSION | Cell Proliferation | Humans | Drug Resistance, Neoplasm | Monocytes - immunology | T-Lymphocytes, Cytotoxic - pathology | T-Lymphocytes, Cytotoxic - drug effects | Monocytes - pathology | Biomarkers, Tumor - metabolism | Antineoplastic Agents - pharmacology | Macrophages - immunology | Neuroblastoma - pathology | Disease Models, Animal | T-Lymphocytes, Cytotoxic - immunology | Picolinic Acids - pharmacology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - antagonists & inhibitors | Macrophages - pathology | Benzothiazoles - pharmacology | Cells, Cultured | Mice, SCID | Monocytes - drug effects | Xenograft Model Antitumor Assays | Animals | Neuroblastoma - drug therapy | Macrophages - drug effects | Mice, Inbred NOD | Mice | Neuroblastoma - metabolism | Apoptosis | Chemotherapy | Neuroblastoma | T cells | Macrophages | Analysis | Cancer | Animal models | Cytotoxicity | Lymphocytes T | Cerebrospinal fluid | CD14 antigen | Neuroblastoma cells | Metastases | Lymphocytes | Colony-stimulating factor | Cell survival | Immune response | Immunodeficiency | Topotecan | Gene expression | Patients | Survival | CD163 antigen | Monocytes | Cyclophosphamide | Depletion | Major histocompatibility complex | Medical prognosis | Tumors | Index Medicus
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 01/2008, Volume 28, Issue 1, pp. 29 - 43
Granulocyte-macrophage colony-stimulating factor (GM-CSF) is a hematopoietic cytokine responsible for the proliferation, differentiation, and maturation of... 
Cerebral ischemia | Hematopoietic factor | Programmed cell death | Gene expression | GM-CSF | Growth factor | FACTOR-RECEPTOR | APOPTOSIS | ERYTHROPOIETIN | RAT | 3-KINASE | FOCAL CEREBRAL-ISCHEMIA | INJURY | hematopoietic factor | NEUROSCIENCES | growth factor | STROKE | cerebral ischemia | DISEASE | ENDOCRINOLOGY & METABOLISM | BLOOD-BRAIN | programmed cell death | HEMATOLOGY | gene expression | Neurons - pathology | Rats, Wistar | Apoptosis - drug effects | Humans | Rats, Long-Evans | Cerebral Cortex - pathology | Brain Infarction - metabolism | Brain Ischemia - metabolism | Brain Infarction - pathology | Male | Phosphatidylinositol 3-Kinases - metabolism | Neurodegenerative Diseases - drug therapy | Cerebral Cortex - metabolism | Dose-Response Relationship, Drug | Neuroprotective Agents - pharmacology | Time Factors | Neurons - metabolism | bcl-X Protein - biosynthesis | Drug Evaluation, Preclinical | Proto-Oncogene Proteins c-akt - metabolism | Brain Infarction - drug therapy | Disease Models, Animal | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Neurodegenerative Diseases - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Rats | Neurodegenerative Diseases - metabolism | Blood-Brain Barrier - metabolism | Up-Regulation - drug effects | Blood-Brain Barrier - pathology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Myeloid Cells - metabolism | Cell Proliferation - drug effects | Myeloid Cells - pathology | Index Medicus
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 06/2010, Volume 181, Issue 12, pp. 1345 - 1354
Journal Article
Journal Article
Journal Article